ライフサイエンスコーパス: infecting @2 with

1 h sets of antagomir effects were mimicked by infecting cells with a p220 cDNA-encoding adenoviral vec

2 MAPK pathway was constitutively activated by infecting cells with a v-mos retrovirus.

3 tidylinositol 3-kinase/protein kinase Akt or infecting endothelial cells with a dominant-negative Akt

4 terial can be generated using this system by infecting insect cells with a baculovirus expressing the

5 We investigated this hypothesis by infecting MDCK cells with a transfectant influenza virus

6 s an opportunistic human pathogen capable of infecting patients with a deficient immune system.

7 PI anti-PnPs antibody levels to the presumed infecting serotypes, with a geometric mean level of 0.83

8 d purified lipooligosaccharide of homologous infecting strains and with a series of mutants deficient

9 vivo depleted these cells from RMs prior to infecting the primates with a pathogenic strain of SIV.

10 complementing the cells with human CD81, or infecting them with a strain of HCV with less restricted

11 -phase induction in quiescent human cells by infecting them with Ad N-terminal E1A mutants with mutat

12 B-/- double knockout mouse lens epithelia by infecting primary cells with Ad12-SV40 hybrid virus.

13 Infecting cells with adenovirus encoding constitutively

14 In addition, stabilizing HIF1/2alpha by infecting mammalian cells with adenoviruses containing t

15 n bieneusi is the most common microsporidian infecting patients with AIDS.

16 als could be generated quickly and easily by infecting susceptible blastocysts with ALV-based retrovi

17 This hypothesis was tested by infecting cells with an M protein mutant virus defective

18 ocked the binding of AP-1 proteins to DNA by infecting keratinocytes with an adenovirus encoding a do

19 Upon infecting macrophages separately with antimony drug-sens

20 Infecting glial progenitors with Cre-recombinant retrovi

21 G10P[11] strains were also seen infecting older children with dehydrating gastroenteriti

22 study, we use a sequential infection model, infecting AG129 mice with DENV-1, followed by DENV-2 6-8

23 the control of a pathogen that is capable of infecting multiple hosts with different rates of transmi

24 e cells to the JNK inhibitor SP600125 and by infecting cells with dominant-negative JNK or AMPK adeno

25 en validated these analytical predictions by infecting mice with doses below or above the predicted t

26 These cells were produced by infecting fibroblasts with dox-inducible lentiviruses, r

27 y chemically inhibiting actin assembly or by infecting mammalian cells with EHEC mutants that translo

28 ally maintaining T cells was investigated by infecting mice with either a demyelinating, paralytic (V

29 her Tir modification could not be induced by infecting cells with EPEC, suggesting that Tir must be c

30 broad range, with laboratory-adapted viruses infecting all clones with equal efficiencies and primary

31 Specifically, infecting mice with H3N2 influenza before challenging wi

32 ta-lipoproteins may reduce the efficiency of infecting hepatocytes with HCV by competitively inhibiti

33 ng the brain via HBCE cells and subsequently infecting microglial cells with high efficiency, leading

34 d and primary T-cell targets was examined by infecting cells with HIV-1 reporter viruses containing k

35 00 in infected patients, regardless of their infecting strain, correlated with increased production o

36 g the plasmid into recipient cells, and then infecting those cells with influenza virus.

37 d when pre-integration complexes are made by infecting cells with integrase-minus virus, demonstratin

38 eld-isolate reovirus strains were capable of infecting cells transfected with JAM-A but not those tra

39 loviruses and nudiviruses, a group of insect-infecting viruses with key roles in biocontrol.

40 of HIF-1alpha or HIF-2alpha was abrogated by infecting astrocytes with lentiviral particles encoding

41 chronic infection were moderately better at infecting cells with low CCR5 concentrations.

42 -infection V1-V2 sequences showed a trend in infecting cells with low CD4 concentrations more efficie

43 ence in inflamed tissues was demonstrated by infecting transgenic mice with MCMV recombinant virus co

44 of these mutations was further evaluated by infecting naive mice with MHV-JHM variants isolated from

45 could be used to predict the genotype of the infecting virus with moderate success.

46 Infecting Ramos cells with MV did not result directly in

47 viral escape from autophagic degradation, as infecting DC with Nef- or Env-deficient HIV strains did

48 ur knowledge has never been described as the infecting organism with oropharyngeal candidiasis (OPC).

49 oglobulin G (IgG) concentrations against the infecting serotype compared with other vaccine serotypes

50 ed with lower IgG concentrations against the infecting serotype compared with other vaccine serotypes

51 f IOMT-overexpressing plants with CuCl(2) or infecting these plants with Phoma medicaginis leads to g

52 human cells with recombinant plasmids and by infecting insect cells with recombinant baculoviruses.

53 DS has primarily consisted of experimentally infecting macaques with related simian immunodeficiency

54 gastric cancer cell line was investigated by infecting these cells with retroviral construct (MFG) ex

55 ed or increased p53 activity were created by infecting VSMCs with retroviruses containing a dominant-

56 ing cells with a proteasomal inhibitor or by infecting cells with small interfering (si)RNA against S

57 Geminiviruses are plant-infecting viruses with small circular single-stranded DN

58 Infecting BFTE cells with sporulated oocysts provided a

59 le to directly correlate the genotype of the infecting antigen with the antibody response.

60 erminal-repeat circle junctions derived from infecting cells with the mutant viruses indicated that t

61 in the five major commercial turkey lines by infecting each with the parental B. avium strain and thr

62 By infecting mice with the attaching and effacing bacteria

63 Moreover, infecting mice with the LRV1-cured Leishmania guyanensis

64 In these studies, we demonstrate that infecting mice with the Th1-inducing parasite Toxoplasma

65 The infecting phages colocalized with the polar protein mark

66 ne response of these animals was explored by infecting them with the Th1-inducing parasite Toxoplasma

67 Mice are primarily responsible for infecting ticks with the Lyme disease agent.

68 nteractions compare with those of vertebrate-infecting viruses and with the Sigma rhabdovirus that in

69 ere we report a high-efficiency protocol for infecting yeast with the [PSI+] prion using amyloids com

70 directly the effects of a human transgene by infecting them with Theiler's murine encephalomyelitis v

71 After co-infecting C. elegans with these organisms, we observed t

72 Given the difficulty of infecting laboratory mice with these diarrhea-causing pa

73 By infecting nonpermissive cells with this library and scre

74 specifically destroy the malignant cells by infecting them with toxic cargo.

75 We determined this by infecting monocytes with two strains of ZIKV: South Amer

76 o induce DNA repair was also demonstrated by infecting NWTb3 cells with UV-irradiated adenovirus.

77 avital microscopy and ex vivo analyses after infecting mice with vaccinia virus (VV), a large DNA vir

78 By infecting animals with various doses of Listeria and ana

79 The latter possibility was studied by dually infecting rhesus macaques with X4 and R5 chimeric simian