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1  and against colonic pathology in a model of infectious colitis.
2 te to stressor-enhanced C. rodentium-induced infectious colitis.
3 ness and treat disease in an animal model of infectious colitis.
4 or evaluation and treatment of patients with infectious colitis.
5 culture should be performed in patients with infectious colitis.
6 in the Citrobacter rodentium model of murine infectious colitis.
7 ogy, pathobiology, and mechanisms underlying infectious colitis.
8 t advances in knowledge of pathogens causing infectious colitis.
9 novel strategies for preventing and treating infectious colitis.
10 for improving intestinal barrier function in infectious colitis.
11 nced susceptibility to C. rodentium-enhanced infectious colitis.
12 as in Crohn disease, ulcerative colitis, and infectious colitis.
13 hown to be associated with susceptibility to infectious colitis and liver abscess due to Entamoeba hi
14 r rodentium model mimics the pathogenesis of infectious colitis and requires sequential contributions
15 , Crohn's disease, irritable bowel syndrome, infectious colitis, and microscopic colitis, as well as
16 rotective cytokines and confer protection in infectious colitis, and their cell numbers in the colon
17 enders mice susceptible to a more aggressive infectious colitis caused by Citrobacter rodentium.
18                  No patient had identifiable infectious colitis, colonic stricture, bowel obstruction
19 estinal infections continues to increase and infectious colitis contributes to significant morbidity
20 e with cancer but in none of the ischemic or infectious colitis controls (P<0.03).
21           The role of innate immune cells in infectious colitis, however, is less well understood.
22  significantly enhanced C. rodentium-induced infectious colitis in resistant mice, as measured by inc
23                           Also patients with infectious colitis, in particular Salmonella and Campylo
24                                              Infectious colitis is diagnosed in someone with diarrhea
25  not reduce all aspects of stressor-enhanced infectious colitis, it did significantly reduce pathogen
26 therapy should be initiated for all forms of infectious colitis other than STEC.
27 dium difficile remains an important cause of infectious colitis, particularly in healthcare facilitie
28 t; one patient receiving sorafenib died from infectious colitis while on treatment and four patients

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