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1 s characterized by an eosinophil-predominant inflammatory infiltrate.
2  and complement deposition and a conspicuous inflammatory infiltrate.
3 L-1alpha and attraction of a pro-tumorigenic inflammatory infiltrate.
4  dysregulated angiogenesis and a substantial inflammatory infiltrate.
5 muscle fibers, myophagocytosis, and a sparse inflammatory infiltrate.
6 ized by a spindle-cell proliferation with an inflammatory infiltrate.
7  divide and that they do so by triggering an inflammatory infiltrate.
8  the increased expression of VEGF-C, the SCC inflammatory infiltrate.
9 serum creatine kinase levels and a prominent inflammatory infiltrate.
10 s accompanied by a disproportionately sparse inflammatory infiltrate.
11 mmatory gene synthesis and resolution of the inflammatory infiltrate.
12 mesenchymal cells and was independent of the inflammatory infiltrate.
13  of tissues and are key orchestrators of the inflammatory infiltrate.
14  represented 10% or more of the interstitial inflammatory infiltrate.
15 liferation of basal keratinocytes and a skin inflammatory infiltrate.
16  expression of PPAR-gamma in the spinal cord inflammatory infiltrate.
17 our incidence correlates with wound size and inflammatory infiltrate.
18 rel had a significant effect on reducing the inflammatory infiltrate.
19 ells with characteristic morphology among an inflammatory infiltrate.
20 lls, consistent with Th9 lymphocytes, in the inflammatory infiltrate.
21 athological CD207(+) dendritic cells with an inflammatory infiltrate.
22 is in which T and NK cells dominate a severe inflammatory infiltrate.
23  to periodontitis displayed higher levels of inflammatory infiltrate.
24 acrophages or fibrin as well as intervillous inflammatory infiltrates.
25 ntestinal epithelial cells (IEC) and colonic inflammatory infiltrates.
26  gland function is preserved despite intense inflammatory infiltrates.
27 treatment was indicated through detection of inflammatory infiltrates.
28 nohistochemical analysis for the presence of inflammatory infiltrates.
29 e weakness and the presence of intramuscular inflammatory infiltrates.
30 ApoE((-/-)) versus ApoE((-/-)) mice, as were inflammatory infiltrates.
31 d histopathologic analysis showed no greater inflammatory infiltrates.
32 ological changes consisting of peribronchial inflammatory infiltrates.
33 3 is highly expressed in postmortem MS brain inflammatory infiltrates.
34 g of cerebral tissue was performed to detect inflammatory infiltrates.
35 hich are characterized by early perivascular inflammatory infiltrates.
36 ere mucosal erosion, villous congestion, and inflammatory infiltrates.
37 vascular changes, epidermal alterations, and inflammatory infiltrates.
38 rved within the islet endocrine cells and in inflammatory infiltrates.
39  nonspecific chronic hepatitis (14.3%), mild inflammatory infiltrates (10.7%), and steatosis (3.5%).
40    Surviving grafts were essentially free of inflammatory infiltrates 15 weeks after transplantation.
41 hoid follicles (32%), a plasma cell-enriched inflammatory infiltrate (63%), and central perivenulitis
42  in PsA by reducing, but not abolishing, the inflammatory infiltrate, adhesion molecule expression, a
43 suppress airway hyperreactivity, bronchiolar inflammatory infiltrate and allergen-specific IgE produc
44 rogenitors, are a prominent component of the inflammatory infiltrate and are believed to directly con
45 rous connective tissue response with minimal inflammatory infiltrate and bone formation.
46 val immune processes, including an increased inflammatory infiltrate and edema seen with IVCM.
47    Histology and immunostaining demonstrated inflammatory infiltrate and focal C3 deposition in early
48 e complex cellular composition of the portal inflammatory infiltrate and HPC/DR niche in NAFLD will s
49 iated skin and was associated with decreased inflammatory infiltrate and increased angiogenesis.
50  stages of infection, with a more pronounced inflammatory infiltrate and increased specific productio
51 e investigated the composition of the portal inflammatory infiltrate and its relationship to the duct
52 CLs and highlights the interplay between the inflammatory infiltrate and malignant B cells in these t
53 determine the effects of methotrexate on the inflammatory infiltrate and on cytokine and metalloprote
54 stained sections were used to study cellular inflammatory infiltrate and quantitative histomorphometr
55 followed by the characterization of cellular inflammatory infiltrate and quantitative histomorphometr
56 28 significantly reduces the formation of an inflammatory infiltrate and reduces bone loss, measured
57 placental factors, such as the nature of the inflammatory infiltrate and slightly higher expression o
58                    The interplay between the inflammatory infiltrate and tissue resident cell populat
59 aque segments, demonstrating the presence of inflammatory infiltrate and various compositional elemen
60 athological features include T-cell mediated inflammatory infiltrates and aberrant accumulations of p
61 fer of myelin-specific T cells, resulting in inflammatory infiltrates and demyelination in the centra
62 ed neurological functional recovery, reduced inflammatory infiltrates and demyelination, and increase
63 ssion of HIF, which initiated an increase in inflammatory infiltrates and edema in the colon.
64                                              Inflammatory infiltrates and enlarged blood vessels were
65 L-1 beta, and IL-8 and increased severity of inflammatory infiltrates and fibrosis were observed in i
66 reased atherosclerosis severity and arterial inflammatory infiltrates and increased neoangiogenesis w
67 was mediated, in part, by the suppression of inflammatory infiltrates and inflammatory cytokines/medi
68 , and histopathologists should be aware that inflammatory infiltrates and late presentation are poten
69  greater cellularity and significantly fewer inflammatory infiltrates and lipid pools in the ISR grou
70 -/-) mice had a lower number of perivascular inflammatory infiltrates and mast cells than those found
71                                              Inflammatory infiltrates and neuronal damage were presen
72 nonuclear cells at the junction of the focal inflammatory infiltrates and normal acinar tissue in bot
73 characterized by a gradual loss of myocytes, inflammatory infiltrates and replacement by fatty and fi
74                         Interactions between inflammatory infiltrates and resident tubular epithelial
75 active EAE of C57Bl/6 mice, decreased spinal inflammatory infiltrates and spinal infiltration of Th1
76                 Moreover, the total cellular inflammatory infiltrates and the CD3(+), CD4(+), HLA-DR(
77 rized by the proliferation of spindle cells, inflammatory infiltrate, and aberrant angiogenesis cause
78  antagonist IRS954 reduced pancreatic edema, inflammatory infiltrate, and apoptosis.
79  of a thin choroid, a perivascular choroidal inflammatory infiltrate, and atrophic changes within the
80 rhage, overall inflammation, intraepithelial inflammatory infiltrate, and edema.
81 parietal cell loss, induction of a prominent inflammatory infiltrate, and emergence of SPEM.
82 ss, temperature, virus titres, the pulmonary inflammatory infiltrate, and interferon [IFN]-gamma leve
83 dual viable tumor, necrosis, fibrous tissue, inflammatory infiltrate, and Ki-67 proliferative index w
84 dontal disease, bleeding index, intensity of inflammatory infiltrate, and loss of connective tissue i
85 ac allograft vasculopathy, the nature of the inflammatory infiltrate, and origin of endothelial cells
86 embrane attack complex, the character of the inflammatory infiltrate, and the primacy of capillary ab
87 o acute worsening of EAE symptoms, increased inflammatory infiltrate, and was associated with the dea
88 ged with H. pylori, and the bacterial loads, inflammatory infiltrates, and cytokine responses were ev
89 educing acanthosis (thickening of the skin), inflammatory infiltrates, and expression of Th17 cytokin
90 ice exhibited more extensive tubular injury, inflammatory infiltrates, and fibrosis than wild-type mi
91                The severity of the fibrosis, inflammatory infiltrates, and glomerular and tubular cha
92 ells, interstitial endothelium, interstitial inflammatory infiltrates, and glomerular cells.Primary V
93 ed ductules, and have well-developed stroma, inflammatory infiltrates, and pushing margins.
94 c asthma include airway hyperresponsiveness, inflammatory infiltrates, and structural changes in the
95 okine production (cytokine storm), excessive inflammatory infiltrates, and virus-induced tissue destr
96 nd histopathologic presentation and reducing inflammatory infiltrates, antibody treatment during alle
97 s that both products of the bacteria and the inflammatory infiltrate are capable of stimulating gastr
98                                              Inflammatory infiltrates are common but are small and of
99                                              Inflammatory infiltrates are frequently observed in pros
100        Axonal injury was correlated with the inflammatory infiltrate as well as clinical characterist
101 in diseased rats, which correlated well with inflammatory infiltrates as determined by histology.
102 y fewer T lymphocytes and macrophages in the inflammatory infiltrate, as well as less edema and cardi
103 ons of the epididymides showed a focal acute inflammatory infiltrate at 2 weeks p.i.
104        DC and T cells form aggregates in the inflammatory infiltrates at active disease sites in huma
105                                  Analysis of inflammatory infiltrates by fluorescence-activated cell
106 -field microscopy, (2) histology to quantify inflammatory infiltrates by immunostaining for macrophag
107 ive correlations could be observed among the inflammatory infiltrate, c-kit, and tryptase in individu
108 gic examination, a layer of collagen tissue, inflammatory infiltrate cells, perivascular infiltrate,
109 f clinical manifestations, the nature of the inflammatory infiltrating cells, the cytokine response p
110 hronic pancreatitis characterized by a mixed inflammatory infiltrate centered around pancreatic ducts
111 per stromal remodeling and perdurance of the inflammatory infiltrate characteristic of pancreatic tum
112 t and tryptase mast cells and the density of inflammatory infiltrate, cluster of differentiation 4 (C
113                     Despite these equivalent inflammatory infiltrates, CNS tissues from CXCR3(-/-) mi
114 logy of C3(-/-) mice revealed a reduction in inflammatory infiltrate compared with C3(+/+) mice.
115  of regenerative fibers, with a reduction in inflammatory infiltrates, compared with those in vehicle
116 c pathologists semiquantitatively graded the inflammatory infiltrate components and compared the resu
117 QTS/CPVT patients revealed mild but distinct inflammatory infiltrates composed of T lymphocytes and m
118 prominent spleen, liver, and skeletal muscle inflammatory infiltrates comprised of NK, dendritic, B a
119 marked edema of the papillary dermis with an inflammatory infiltrate consisting mainly of mature neut
120                                     An acute inflammatory infiltrate consisting of polymorphonuclear
121 perproliferation, elongated rete ridges, and inflammatory infiltrates consisting of T cells, macropha
122  skin transplanted onto NSG mice develops an inflammatory infiltrate, consisting predominately of hos
123 tis), i.e. replacememt of bone marrow fat by inflammatory infiltrates containing macrophages, T lymph
124 es in dermal fibroblasts that, like the skin inflammatory infiltrate, contribute to tumour formation.
125                                              Inflammatory infiltrate, DCs, chemokines, classification
126 gy scores, vascular densities, and levels of inflammatory infiltrates decreased significantly compare
127 own efficacy in the neoadjuvant setting with inflammatory infiltrates demonstrated in tumors.
128 nted into C57Bl/6 recipients exhibited mixed inflammatory infiltrates, destruction of the mucosa, and
129      At 3 days postinfection, well-organized inflammatory infiltrates developed in the spleen and liv
130   Later in the course of this illness, focal inflammatory infiltrates disappeared from the spinal cor
131   These results highlight differences in the inflammatory infiltrates during Lyme arthritis and cardi
132 idermis caused epidermal hyperplasia, dermal inflammatory infiltrate, dysmorphic hair follicles, and
133                 AD shows a Th2 cell-dominant inflammatory infiltrate, elevated serum IgE levels, a pe
134 alomyelitis (EAE), characterized by lesions, inflammatory infiltrates, elevated STAT activation, and
135 , there were reductions in the extent of the inflammatory infiltrate, endovasculitis, myocardial edem
136                          A rapid pericentral inflammatory infiltrate enriched in F4/80(+) monocytes c
137 prostaglandins and is associated with edema, inflammatory infiltrate, fever, and pain.
138 y of the muscle showed a mainly perivascular inflammatory infiltrate, fibrotic degeneration, and neov
139 d with the induction of an intense cutaneous inflammatory infiltrate following topical application of
140                           We discovered that inflammatory infiltrates from the affected tissue are do
141                       On day 3, however, the inflammatory infiltrate had subsided in the control grou
142                Synovial tissues with diffuse inflammatory infiltrates had high levels of TSP2, wherea
143 ntinued axon loss for months after acute CNS inflammatory infiltrates had subsided, and massive astro
144                                  We detected inflammatory infiltrates, hepatocellular necrosis, and a
145                  T cells are the predominant inflammatory infiltrate in affected tissue and are thoug
146                                          The inflammatory infiltrate in atherosclerotic plaque is com
147                            Data on placental inflammatory infiltrate in biopsies from 68 cases and 65
148 AC expression is elevated selectively in the inflammatory infiltrate in human and murine colitis.
149 hilic infiltrate compared with a mononuclear inflammatory infiltrate in normal mice.
150     Macrophages account for 5% to 30% of the inflammatory infiltrate in periodontitis and are activat
151 ameters, periodontal bacterial profiles, and inflammatory infiltrate in placental chorionic villi are
152 egulating the pattern and persistence of the inflammatory infiltrate in RA and suggest a new and impo
153              A characteristic feature of the inflammatory infiltrate in rheumatoid arthritis is the s
154                      Characterization of the inflammatory infiltrate in selected cases used a novel s
155                  This resulted in a profound inflammatory infiltrate in the adventitia and necrotic c
156  abscesses within the epidermis, and a mixed inflammatory infiltrate in the dermis.
157 , we analyzed the monocytic component of the inflammatory infiltrate in the draining popliteal lymph
158                                          The inflammatory infiltrate in the heart includes neutrophil
159 e congestion, alveolar hemorrhage, and acute inflammatory infiltrate in the interstitial compartment
160    Neutrophils were the predominant cellular inflammatory infiltrate in the lungs of superinfected mi
161 in serum IL-1beta and IL-18, reduced myeloid inflammatory infiltrate in the skin and spleen, and subs
162                                          The inflammatory infiltrates in active lesions were characte
163 cells and plasma cells are components of the inflammatory infiltrates in both active and chronic MS l
164 lls of normal colonic lamina propria, and in inflammatory infiltrates in colon tissues from Crohn's d
165                 Although islets demonstrated inflammatory infiltrates in CVB3-protected mice, insulin
166  that IFNgamma influences the composition of inflammatory infiltrates in Lyme carditis by promoting t
167             Macrophages constitute the major inflammatory infiltrates in musculoskeletal tissues duri
168 n was localized mainly in cardiomyocytes and inflammatory infiltrates in peri-infarct areas, but not
169  or growth restriction were characterized by inflammatory infiltrates in placentas, functional defici
170 s, and in neutrophil survival, which affects inflammatory infiltrates in RA.
171 trol study, in which we immunophenotyped the inflammatory infiltrates in renal biopsy specimens with
172                                          The inflammatory infiltrates in some models of chronic autoi
173 rity and incidence of EAE, the appearance of inflammatory infiltrates in spinal cord, and the subsequ
174 an skin scores and significantly fewer focal inflammatory infiltrates in submandibular salivary gland
175 nical course of EAE and showed markedly less inflammatory infiltrates in the CNS than wild-type mice.
176 ls were co-localized with CD4(+) Th cells in inflammatory infiltrates in the lungs of mycoplasma-infe
177 onchoalveolar lavage fluid, allergen-induced inflammatory infiltrates in the lungs, and Bla g 1-speci
178 nation in the cerebral cortex was related to inflammatory infiltrates in the meninges, which was pron
179 ase of unknown etiology, is characterized by inflammatory infiltrates in the pancreas with conspicuou
180 s, total serum Ig isotypes, lymphadenopathy, inflammatory infiltrates in the salivary gland and kidne
181  acute clinical disease and showed only mild inflammatory infiltrates in the spinal cord white and gr
182 glycolysis associated with paralysis-causing inflammatory infiltrates in the spinal cord; and (iii) t
183 vo clinical examination at 24 hours revealed inflammatory infiltrates in the vitreous and retina, whi
184 treated mice have reduced fibrosis and fewer inflammatory infiltrates in their adipose tissues.
185                                          The inflammatory infiltrates in various organs predominantly
186  the development of chronic small-intestinal inflammatory infiltrates in vivo.
187  oxidase and myeloperoxidase activities, and inflammatory infiltrate) in chronically infected hearts.
188 udies of patients have shown the presence of inflammatory infiltrates, in some cases with features of
189 ccompanied by a significant reduction of the inflammatory infiltrate including macrophages, dendritic
190 elitis and decreased the accumulation of CNS inflammatory infiltrates, including dendritic cells, B c
191 NBS-treated mice contained acute and chronic inflammatory infiltrates, increased collagen, fibrogenic
192                              Analysis of the inflammatory infiltrate indicated a reduction of cytotox
193 s have similarities in their desmoplasia and inflammatory infiltrates, indicating overlapping inflamm
194 inophils, all cell types present in the lung inflammatory infiltrate induced by RSV infection.
195 echanisms underlying the recruitment of this inflammatory infiltrate into the involved lymph nodes ar
196 omicrobial peritonitis, leading to increased inflammatory infiltrate into the peritoneum and target o
197 is abundant EBV replication but the expected inflammatory infiltrate is absent.
198                                         This inflammatory infiltrate is predominately composed of T l
199 r-treated NAFLD livers had notably fewer pro-inflammatory infiltrating macrophages and a significantl
200                                   The portal inflammatory infiltrate may contribute directly to fibro
201 riodontal disease, these cells, found in the inflammatory infiltrate, may drive the progressive perio
202 ing cholangitis is characterized by a portal inflammatory infiltrate mostly composed of memory T cell
203 atures such as eosinophilic and neutrophilic inflammatory infiltrates, necrosis, vascular hyalinizati
204                                          The inflammatory infiltrate, NOS-2-positive macrophages, myo
205                                              Inflammatory infiltrates occurred in the majority of HIV
206 cells were found in increased numbers in the inflammatory infiltrate of CD lesions and induced expres
207 hocytes and plasma cells) predominate in the inflammatory infiltrate of human chronic periodontitis.
208 rophils are the predominant cell in the lung inflammatory infiltrate of infants with respiratory sync
209         The ear bud area develops an intense inflammatory infiltrate of OT-I cells.
210  response to kidney injury includes a robust inflammatory infiltrate of PMNs and macrophages.
211 ant, revealed staining in the perifollicular inflammatory infiltrate of rosacea for both proteins.
212                                          The inflammatory infiltrate of SeTA was similar to SeA but c
213 IL-1beta, accompanied by significantly fewer inflammatory infiltrates of macrophages and neutrophils
214 sed expression of CXCL13 was detected in the inflammatory infiltrates of the control NZB/NZW mice.
215        The increase in CD4(+) T cells in the inflammatory infiltrates of the lacrimal gland, and the
216                                              Inflammatory infiltrates, often with granulomatous arran
217   No ratios were correlated with presence of inflammatory infiltrate or foamy macrophages.
218  mice; in contrast, there was no evidence of inflammatory infiltrates or responses in any other tissu
219  increased the intensity and duration of the inflammatory infiltrate (P < 0.05).
220 -derived LCs was associated with nonspecific inflammatory infiltrates (P=0.009).
221 cells cause enhanced disease, with increased inflammatory infiltrate, particularly rich in neutrophil
222      Likewise, at 2 weeks, acute and chronic inflammatory infiltrates persisted in the bladders and k
223 d; P < .001), presence of neutrophils in the inflammatory infiltrate (predominantly mononuclear vs mi
224 gs; fibrous hyperplasia; a low-grade chronic inflammatory infiltrate, predominantly consisting of lym
225                 Adenoviral infection induced inflammatory infiltrates primarily made of T and B cells
226                            The prevalence of inflammatory infiltrate, proinflammatory cytokines, and
227 ator synthesis and mediate resolution of the inflammatory infiltrate, promoting formation of a scar.
228 d tumours, colorectal cancer exhibits immune/inflammatory infiltrates, referred to as 'tumour-elicite
229 maller, less aggressive lesions with reduced inflammatory infiltrate relative to tumors generated by
230 a-Cell replication stimulated by the initial inflammatory infiltrate results in increased production
231                              Analyses of the inflammatory infiltrate revealed a classic acute inflamm
232 ent bacterial colonization and heterogeneous inflammatory infiltrates rich in CD15+ neutrophils, as h
233                              Despite similar inflammatory infiltrates, RRV-infected C3(-/-) mice exhi
234 gmentation and loss, Wallerian degeneration, inflammatory infiltrates, Schwann cell proliferation, an
235 ght account in part for the heterogeneity of inflammatory infiltrates seen in lungs of asthmatics.
236                                The resulting inflammatory infiltrate serves to aid bacterial clearanc
237                              Analysis of CNS inflammatory infiltrates showed that female but not male
238    Histologic examination revealed increased inflammatory infiltrate, significantly increased immunos
239                                       In the inflammatory infiltrate subjacent to the pocket epitheli
240 ariable numbers of malignant cells within an inflammatory infiltrate, suggesting that these tumors es
241                  The presence of a CD8+ rich inflammatory infiltrate suggests an ongoing immune respo
242 ivascular interstitial edema associated with inflammatory infiltrates surrounding larger pulmonary ve
243 ys of GTC-treated mice had less fibrosis and inflammatory infiltrate than kidneys of vehicle-treated
244 eed-Sternberg (RS) cells within an extensive inflammatory infiltrate that includes abundant T helper
245      The remaining tissue is comprised of an inflammatory infiltrate that includes myeloid cells.
246 cosal diseases are often characterized by an inflammatory infiltrate that includes polymorphonuclear
247 ancer is frequently characterized by a large inflammatory infiltrate that includes T cells.
248 y to be one of several cytokines produced by inflammatory infiltrates that contribute to this and sub
249 monia (peak days, 4-5) and chronic pulmonary inflammatory infiltrates that lasted up to 154 days afte
250 , associated dysfunction, and development of inflammatory infiltrates that result in abnormal tears.
251    Although the IL-1R(-/-) mice developed an inflammatory infiltrate, the number of infiltrating neut
252 ions with respect to demyelinating activity, inflammatory infiltrates, the presence of meningeal infl
253 Western blot were used to evaluate levels of inflammatory infiltrate, TLR2, TLR4, and RAGE expression
254    Fibrosis was present in most with similar inflammatory infiltrate to viremic cases.
255  vivo EP also recruited large mixed cellular inflammatory infiltrates to the site of inoculation.
256  examined for the presence of CNS tissue and inflammatory infiltrates using direct microscopy, enhanc
257             In this study we used MRI to map inflammatory infiltrates using high-field MRI and fluore
258                                        Liver inflammatory infiltrate was characterized by immunohisto
259 bserved in the urinary bladder, although the inflammatory infiltrate was delayed by approximately a w
260                                              Inflammatory infiltrate was evaluated and quantified in
261         However, when the composition of the inflammatory infiltrate was examined by flow cytometric
262                            In hairless mice, inflammatory infiltrate was found around the rudimentary
263 was observed in IL-6(-/-) mice, in which the inflammatory infiltrate was markedly reduced and synovia
264                          The formation of an inflammatory infiltrate was measured in hematoxylin-and-
265 ogical examination revealed that the hepatic inflammatory infiltrate was mixed but dominated by eosin
266               On day 1, a moderate to severe inflammatory infiltrate was noted in both the diabetic a
267 gnificant histopathological reduction of the inflammatory infiltrate was observed after clobetasol tr
268 ttern dictated by the human K14 promoter, an inflammatory infiltrate was observed in skin and esophag
269   At 3 weeks p.i., a mixed acute and chronic inflammatory infiltrate was observed in the urethra that
270 ull were not different from those in WT, the inflammatory infiltrate was reduced in SP/ON-null PDL.
271 tive responses increased, although the total inflammatory infiltrate was reduced.
272                            After 3 days, the inflammatory infiltrate was significantly higher in the
273                         Retention of chronic inflammatory infiltrates was demonstrated by histologica
274                 The predominant cells in the inflammatory infiltrate were T cells and plasma cells.
275 lar channels, and adventitial and periaortic inflammatory infiltrates were assessed in an equal numbe
276               Axonal swelling and perineural inflammatory infiltrates were detectable at every time p
277                                              Inflammatory infiltrates were not associated with neurog
278 ubstantial amounts of perivascular edema and inflammatory infiltrates were noted in the lungs of Tg m
279                           Renal interstitium inflammatory infiltrates were observed in 50% of cases.
280              However, histologically similar inflammatory infiltrates were present in livers of both
281                 Lymphocytic and neutrophilic inflammatory infiltrates were present in the subglottic
282                    Villus blunting and heavy inflammatory infiltrates were seen in the 5-FU-challenge
283                                   Persistent inflammatory infiltrates were seen in the lungs and brai
284 gical features, such as inclusion bodies and inflammatory infiltrates, were more severe and prominent
285 en, proinflammatory cytokine production, and inflammatory infiltrate while simultaneously preventing
286  mucosal biopsy showed a slight perivascular inflammatory infiltrate while skin biopsy was normal.
287 , gammaHV68 induced a neutrophilic meningeal inflammatory infiltrate, while gammaHV68-M3.stop induced
288 ical disease can occur in the presence of an inflammatory infiltrate with eosinophilic, neutrophilic,
289 n muscle biopsy--namely, rimmed vacuoles, an inflammatory infiltrate with invasion of non-necrotic mu
290 acterized by epidermal thickening and dermal inflammatory infiltrates with a dominant Th2 profile dur
291 tic islets from these transgenic mice showed inflammatory infiltrates with corresponding destruction
292                   CRTH-positive Th2-dominant inflammatory infiltrate, with increased IL-4 expression,
293 on, accompanied by a significantly increased inflammatory infiltrate within the central nervous syste
294 ear layers of the retina, and a perivascular inflammatory infiltrate within the choroid.
295 ons are required for the generation of early inflammatory infiltrate within the injured brain, and wh
296                      Characterization of the inflammatory infiltrate within the skeletal muscle tissu
297 ls were present in perifollicular and dermal inflammatory infiltrates within acne lesions, suggesting
298  mice had similar numbers and composition of inflammatory infiltrates within hind limb skeletal muscl
299 o concanavalin A stimulation, and absence of inflammatory infiltrates within the brain.
300 stologic analysis showed absent to low-grade inflammatory infiltrates without cardiomyocyte necrosis.

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