ライフサイエンスコーパス: inflammatory reaction

1 d, to decipher how syndecan-1 impacts on the inflammatory reaction.

2 nse by microglia while triggering a moderate inflammatory reaction.

3 an important role in the coordination of an inflammatory reaction.

4 ng at 48 hours and delayed resolution of the inflammatory reaction.

5 and mesenteric lymph nodes, causing an acute inflammatory reaction.

6 cells that are recruited and involved in the inflammatory reaction.

7 of proper neuro-vascular associations and an inflammatory reaction.

8 y in pulmonary sensory neurons during airway inflammatory reaction.

9 ional knockout mouse brain invoked a massive inflammatory reaction.

10 betes within a week, characterized by strong inflammatory reaction.

11 ng to the pathogenesis of this novel type of inflammatory reaction.

12 cosal healing, such as the persistence of an inflammatory reaction.

13 l was occasionally associated with a limited inflammatory reaction.

14 , and dendritic cells, in the local allergic inflammatory reaction.

15 ase pattern that is associated with an acute inflammatory reaction.

16 layed oral mucosa healing in the presence of inflammatory reaction.

17 phase of sepsis is characterized by a strong inflammatory reaction.

18 lial clearance of misfolded proteins and the inflammatory reaction.

19 and stimulates an innate and adaptive immune inflammatory reaction.

20 interplay of gut microbiota and host immune/inflammatory reaction.

21 synthesis is known to optimize local tissue inflammatory reaction.

22 Lactococcus lactis G121 to prevent allergic inflammatory reactions.

23 amage inferred from attenuation of glial and inflammatory reactions.

24 T cells (Tregs) are thought to downmodulate inflammatory reactions.

25 by IL-33 probably play an important role in inflammatory reactions.

26 of mitochondria or in induction of untoward inflammatory reactions.

27 ons for bone disorders but may cause serious inflammatory reactions.

28 ocytic, phagocytic, proinflammatory, or anti-inflammatory reactions.

29 role of mast cells and IL-1 in NBP-mediated inflammatory reactions.

30 ns and for preventing excessive or prolonged inflammatory reactions.

31 to facilitate the resolution of pathological inflammatory reactions.

32 ling pathway to protect against exacerbating inflammatory reactions.

33 ide production is believed to participate to inflammatory reactions.

34 betaR) activation controls and downregulates inflammatory reactions.

35 opment and progression of local and systemic inflammatory reactions.

36 and prevents the development of exacerbated inflammatory reactions.

37 g homeostasis and preventing excessive acute inflammatory reactions.

38 dating the molecular mechanisms that lead to inflammatory reactions.

39 re prominent contributor to the pathology of inflammatory reactions.

40 genetically engineered mice induced similar inflammatory reactions.

41 conditions associated with retinal vascular inflammatory reactions.

42 onist could be useful to treat skin-mediated inflammatory reactions.

43 leukocyte recruitment in the development of inflammatory reactions.

44 eloped over the years to rectify deregulated inflammatory reactions.

45 leads to the rapid manifestation of allergic inflammatory reactions.

46 s play an orchestrating role in immune-based inflammatory reactions.

47 lasts in bone or into giant cells in chronic inflammatory reactions.

48 y out immune surveillance and participate in inflammatory reactions.

49 (IL-1) plays a prominent role in immune and inflammatory reactions.

50 ers in the absence of aneurysm formation and inflammatory reactions.

51 ly titanium dioxide) leading to peri-implant inflammatory reactions.

52 as an immunomodulatory agent in allergic and inflammatory reactions.

53 athogens are sometimes accompanied by strong inflammatory reactions.

54 tively recruited to the lung during allergic inflammatory reactions.

55 receptors to elicit and amplify a variety of inflammatory reactions.

56 ced gene 6 (TSG-6), which is up-regulated in inflammatory reactions.

57 municable diseases and are driven by chronic inflammatory reactions.

58 responders" to tissue stress and drivers of inflammatory reactions.

59 n and from reduced allergic, autoimmune, and inflammatory reactions.

60 regulation, orchestrating both pro- and anti-inflammatory reactions.

61 es to control pathologies with overexuberant inflammatory reactions.

62 lations in the steady-state and during acute inflammatory reactions.

63 function in pathogen- and autoimmune-induced inflammatory reactions.

64 d the types of T cells involved in different inflammatory reactions.

65 icant and influenced by diet, metabolic, and inflammatory reactions.

66 dictating the magnitude of subsequent acute inflammatory reactions.

67 itical role in myeloid cell-mediated in vivo inflammatory reactions.

68 om repeated injections, a potential toxic or inflammatory reaction after exposure to the biologic age

69 tin-like domain of TM resulted in a stronger inflammatory reaction after Stx2/LPS with more neutrophi

70 chistosomes results in a CD4 T cell-mediated inflammatory reaction against parasite eggs that varies

71 VCAM-1 in regulating the immune response and inflammatory reactions against viral infections.

72 the poorly understood instant blood-mediated inflammatory reaction and a thorough understanding of ea

73 factors and antigens that stimulate a local inflammatory reaction and activation of the innate immun

74 In pulmonary fibrosis, an inflammatory reaction and differentiation of myofibrobla

75 ating that TLR2 activation limited the toxic-inflammatory reaction and maintained assembly of the dru

76 fective means of controlling a virus-induced inflammatory reaction and may act mainly by the effects

77 is critically involved in the postinfarction inflammatory reaction and mediates adverse dilative remo

78 We examined neuronal death, inflammatory reaction and neurogenesis in hippocampus 72

79 f MRSA pneumonia by attenuating an excessive inflammatory reaction and protecting the lung from patho

80 portant for resolution of the postinfarction inflammatory reaction and regulates fibroblast function.

81 t foot is also characterised by a pronounced inflammatory reaction and the pathogenic significance of

82 formed via nitric oxide-dependent oxidative inflammatory reactions and are found at concentrations o

83 an AD patients is limited because of adverse inflammatory reactions and cerebral hemorrhaging associa

84 EX1, NOTCH3, and Col4A1 are known to provoke inflammatory reactions and damage the brain in a wide va

85 teine proteases, have been shown to increase inflammatory reactions and lymphocyte apoptosis.

86 of CD200 in follicular epithelium attenuates inflammatory reactions and may play a role in maintainin

87 Thus, Foxj1 likely modulates inflammatory reactions and prevents autoimmunity by anta

88 Thus, Foxd1 modulates inflammatory reactions and prevents autoimmunity by dire

89 ugh the CD200 receptor (CD200R) to attenuate inflammatory reactions and promote immune tolerance.

90 Inhalation of agricultural dusts causes inflammatory reactions and symptoms such as headache, fe

91 d determined the magnitude of neuroglial and inflammatory reactions and their cytokine expression pro

92 odies and immune complexes that induce local inflammatory reactions and tissue destruction.

93 ortant in bacterial killing, but also drives inflammatory reactions and tissue oxidation.

94 eoporosis but have also been found to induce inflammatory reactions and to delay the progression of b

95 shown to cause foreign-body sensation, local inflammatory reaction, and conjunctival hyperemia.

96 n the brain, a fulminant microglial-mediated inflammatory reaction, and neuronal death.

97 ing immunomodulatory effects in allergic and inflammatory reactions, and also plays a key regulatory

98 a central role in initiating and amplifying inflammatory reactions, and is implicated in the pathoge

99 ns, including IgE-dependent allergies, other inflammatory reactions, and mastocytosis.

100 f regulatory T cells, which help to suppress inflammatory reactions, and plays a significant role in

101 tors, FPR1 and FPR2, play critical roles for inflammatory reactions, and receptor-specific antagonist

102 tions, such as inadequate bone regeneration, inflammatory reactions, and wound infection.

103 Although inflammatory reactions are beneficial and necessary for

104 Both inflammatory and anti-inflammatory reactions are normal components of the same

105 complement system, known to produce a local inflammatory reaction, are associated with the plaques a

106 ar TA injection markedly decreased the acute inflammatory reaction associated with intravitreous Ad-L

107 Controlling the overwhelming inflammatory reaction associated with polymicrobial seps

108 o bone (entheses), the key territory for the inflammatory reaction associated with PsA, being subject

109 nvolved in the generation of the more severe inflammatory reaction associated with the late central n

110 ole for VCAM-1 in promoting successful local inflammatory reactions associated with efficient viral c

111 trate a robust but transient postreperfusion inflammatory reaction, associated with a rapid up-regula

112 This results in an inflammatory reaction at several sites, including the sy

113 MCs) from infected cows, despite a continued inflammatory reaction at sites of infection.

114 We also assessed oxidative stress and the inflammatory reaction at the injury site, neuronal and o

115 ipants (40%) experienced a localized sterile inflammatory reaction at the site of the injection, whic

116 ies show the presence of a lymphohistiocytic inflammatory reaction at the sites of delivery of alloge

117 involvement in immediate as well as chronic inflammatory reactions at both local and distal sites po

118 s suggest that vitamin D modulates cutaneous inflammatory reactions, at least in part, by increasing

119 transcription factors has a central role in inflammatory reactions, being stimulated by multiple cyt

120 sue-resident macrophages during infection or inflammatory reactions, besides self-replication of tiss

121 ltalpxP32::P(lpxL) lpxL) stimulated a strong inflammatory reaction but sickened mice before recovery

122 In contrast, CD39-null mice had inflammatory reactions but developed only minor pancreat

123 n is important because TNF not only furthers inflammatory reactions but might also be neuroprotective

124 -1 (ICAM-1) is critical in immunological and inflammatory reactions but, like other adhesive interact

125 onse to chemical or physical stimuli repress inflammatory reactions, but cells that undergo nonapopto

126 MKP-1 serves to limit the inflammatory reaction by inactivating JNK and p38, thus

127 ther propose that dynamic modulation of this inflammatory reaction by interrupting the vicious cycle

128 naling through cysLT(1)R might contribute to inflammatory reactions by cooperating with IL-4 in enhan

129 esion molecules is widely thought to promote inflammatory reactions by facilitating leukocyte recruit

130 This inflammatory reaction can spread throughout the systemic

131 Isolated intraocular inflammatory reactions can be an initial manifestation o

132 ces between PPC and CCC in corneal edema, AC inflammatory reaction, capsular fibrosis, ACO, and PCO.

133 occurs during isolation and from the severe inflammatory reactions caused by the transplantation pro

134 9 transgenic mice mounted an enhanced innate inflammatory reaction characterized by increased express

135 egimen-dependent induction of an acute local inflammatory reaction, characterized in part by rapid ne

136 Inflammatory reaction clears dead cells and matrix debri

137 hases of atherogenesis, including well-known inflammatory reactions consequent to intimal trapping an

138 oil' to the stem cells' seed), such as local inflammatory reactions, contribute to successful tissue

139 ith ridge height; thereafter, a more intense inflammatory reaction corresponded to reduction in alveo

140 m day 4 to day 14 (P < 0.001), with distinct inflammatory reaction demonstrated by lymphocyte and neu

141 Although frequently accompanied by local inflammatory reactions detectable in cerebrospinal fluid

142 An inflammatory reaction developed in all nine of the survi

143 A mild inflammatory reaction disappeared after 12 weeks.

144 Loss of T and B cell inflammatory reactions does not protect from impaired ly

145 syndrome is characterized by an overwhelming inflammatory reaction driven by excessive expansion of T

146 itations apply, most notably a chronic local inflammatory reaction due to permanent implantation of a

147 izing autoantibodies, who developed a severe inflammatory reaction during anti-tuberculosis treatment

148 zing bacteria, which is likely to impact the inflammatory reaction during mucosal diseases.

149 n regulates immune cell infiltration and the inflammatory reaction during sepsis, suggesting that act

150 t bear therapeutic potential by reducing the inflammatory reaction during the early phase of AILI.

151 y, focusing on muscle satellite cells (SCs), inflammatory reaction, fibrosis, and formation of new bl

152 gists when there is a need to review diverse inflammatory reactions; for identification of fungi, par

153 lected: VA, degree of anterior and posterior inflammatory reaction, foveal thickness measured by opti

154 hase plasma protein, is a major component of inflammatory reactions functioning as a mediator of inna

155 vs 45 years, P = .05), presence of vitreous inflammatory reactions >2+ (35.9% vs 6.2%, P = .04), vas

156 The remaining inflammatory reactions had a change in the ratio of CD4

157 Systemic inflammatory reactions have been postulated to exacerbat

158 re main effector cells of allergic and other inflammatory reactions; however, only a few anti-MC agen

159 poor survival of donor cells in response to inflammatory reactions, hypoxia, oxidative stress, or nu

160 lets can result in an instant blood-mediated inflammatory reaction (IBMIR) that contributes to early

161 iveness because of an instant blood-mediated inflammatory reaction (IBMIR) that occurs immediately af

162 t has been termed the instant blood-mediated inflammatory reaction (IBMIR).

163 nts without HIV infection caused intraocular inflammatory reaction in all cases and demonstrated more

164 autoimmune disease characterized by a local inflammatory reaction in and around islets followed by s

165 ficant increase of blood vessel presence and inflammatory reaction in control compared with treated e

166 ation of active B burgdorferi infection, the inflammatory reaction in mice with persistent joint infl

167 t cytosolic RLR-RNA-sensing pathway mediated inflammatory reaction in response to acute liver IR.

168 PE-PLD-deficient mice fail to mount a normal inflammatory reaction in response to carrageenan adminis

169 nterica serotype Typhimurium causes an acute inflammatory reaction in the ceca of streptomycin-pretre

170 nterica serotype Typhimurium causes an acute inflammatory reaction in the ceca of streptomycin-pretre

171 SV-1 infection has been shown to initiate an inflammatory reaction in the cornea that leads to tissue

172 h herpes simplex virus 1 (HSV-1) sets off an inflammatory reaction in the cornea which leads to both

173 This infection usually results in a blinding inflammatory reaction in the cornea, orchestrated mainly

174 ve colitis are caused by an excessive immune-inflammatory reaction in the intestinal wall.

175 eficit in short term memory (STM) and strong inflammatory reaction in the ipsilateral cortex and hipp

176 Poly(I:C) injection induced a sustained inflammatory reaction in the SN and in the dorsolateral

177 wild-type or CXCR3-/- mice, they elicited an inflammatory reaction in wild-type but not in CXCR3-/- m

178 epidermis of adult mice also led to stronger inflammatory reactions in a tape-stripping test and in a

179 rmine whether dendritic cells participate in inflammatory reactions in amyotrophic lateral sclerosis

180 Inflammatory reactions in anterior segments developed in

181 ection against disease onset, and attenuated inflammatory reactions in arthritic mice.

182 Interleukin 6 plays a key role in mediating inflammatory reactions in autoimmune diseases and cancer

183 Inflammatory reactions in both IRI and infections share

184 invading microorganisms but also potentiate inflammatory reactions in cases of excessive or misdirec

185 R knocked down cholangiocytes fail to induce inflammatory reactions in control large cholangiocytes.

186 lving histamine-induced and allergen-induced inflammatory reactions in forearm skin.

187 sual sphingolipids that are known to promote inflammatory reactions in gingival fibroblasts and Toll-

188 e receptors (TLR) activate multiple steps in inflammatory reactions in innate immune responses.

189 FI is promising for noninvasive detection of inflammatory reactions in IR myocardium because of its m

190 that establish chronic infections and elicit inflammatory reactions in mammals.

191 dampen, rather than provoke, T cell-mediated inflammatory reactions in many clinical conditions where

192 ifs" were recently shown to block pathologic inflammatory reactions in murine models of autoimmune di

193 and IL-1beta was associated with comparable inflammatory reactions in patients and controls.

194 tenance of immune homeostasis or breakout of inflammatory reactions in patients with chronic inflamma

195 3CL1 mRNA, providing fine-tuning of cellular inflammatory reactions in response to IFN-gamma stimulat

196 recurrent self-resolving attacks of systemic inflammatory reactions in the absence of infection or au

197 fied as important factors amplifying the pro-inflammatory reactions in these pathologies.

198 -1 in the negative control of MAPK-regulated inflammatory reactions in vivo.

199 vation and has been used to study immune and inflammatory reactions in which nitric oxide (NO) is a v

200 evalence of ophthalmic findings (intraocular inflammatory reactions [including transient, isolated re

201 gates" for transmigrating neutrophils in all inflammatory reactions investigated (elicited by leukotr

202 A well-orchestrated inflammatory reaction involves the induction of effector

203 Resolution of inflammatory reactions involves emigration of monocyte-d

204 s do not express COX-2), intensifies a broad inflammatory reaction involving numerous cytokines and o

205 ins unknown but a timely and well-controlled inflammatory reaction is essential for the integrity and

206 tants of B. cenocepacia, suggesting that the inflammatory reaction is likely induced by an as yet unc

207 The inflammatory reaction is orchestrated by gamma interfero

208 nd an inducible chemokine, whose role in the inflammatory reaction is poorly known.

209 ized to the vessel wall, the etiology of the inflammatory reaction is unknown.

210 A characteristic feature of chronic inflammatory reactions is their persistence and predilec

211 hibit a cell death-type modulatory effect on inflammatory reactions is unknown.

212 (FA) dietary supplementation would modulate inflammatory reactions leading to periodontal disease in

213 though beneficial at early stages, excessive inflammatory reaction leads to cell death and tissue dam

214 The current VPC regime induced local skin inflammatory reaction limited to subcutaneous injection

215 To promote the inflammatory reaction, lipopolysaccharide was administra

216 Failure to terminate such inflammatory reactions may lead to detrimental systemic

217 in vivo, suggesting that local and systemic inflammatory reactions may trigger hepatic regeneration

218 pools that may have important effects during inflammatory reactions mediated by monocytes, most notab

219 During granulomatous inflammatory reactions, myeloid cells can differentiate

220 No intraocular inflammatory reaction occurred after intravitreous injec

221 tachment appeared once more, however without inflammatory reaction of the anterior chamber.

222 ryptamine (5-HT)) is implicated in enhancing inflammatory reactions of skin, lung, and gastrointestin

223 Although signals that trigger acute inflammatory reactions often stimulate dendritic cell ma

224 (i.e. similar to untreated tissues), with no inflammatory reaction or fibrous encapsulation occurring

225 t delta 31-61, did not cause any cell death, inflammatory reaction or oxidative damage.

226 There were no intraocular hemorrhages, inflammatory reactions, or pigment dispersion and the IO

227 The inflammatory reaction over several days will drive robus

228 yed healing was associated with a persistent inflammatory reaction, over-production of osteopontin, e

229 That the human whole blood inflammatory reactions paralleled the host response show

230 c strategies incorporating methods to elicit inflammatory reactions, particularly NK cell-driven lysi

231 2 entities characterized by a granulomatous inflammatory reaction pattern but with unique genetic an

232 s play a crucial role during stroke, and the inflammatory reaction promotes neurodegeneration.

233 mostasis and minimizing the ensuing pain and inflammatory reactions provoked by these mediators.

234 (anterior chamber cells and flare, vitreous inflammatory reactions, retinal whitening), without clin

235 -evoked responses are elevated during airway inflammatory reaction, revealing the potential convergen

236 ted disease (IgG4-RD) is characterized by an inflammatory reaction rich in IgG4-positive plasma cells

237 sed impaired life span and intensive cardiac inflammatory reactions, showing that the cardiotoxic IFN

238 E. coli may contribute to the resolution of inflammatory reactions since E. coli can inhibit hydroxy

239 Although the granulocytes contribute to the inflammatory reaction, stem cells may promote tissue rep

240 yelination and persists despite the residual inflammatory reaction subsiding to levels seen in contro

241 occurs as part of the central nervous system inflammatory reaction suggests a potential mechanism of

242 The type of inflammatory reaction suggests cytotoxic CD8+ T lymphocy

243 r the cause, or a feature of severe systemic inflammatory reaction syndrome, whatever the cause.

244 pates in the molecular events underlying the inflammatory reaction that accompanies cerebral ischemia

245 iated with inflammation, but its role in the inflammatory reaction that accompanies cerebral ischemia

246 rative disorders are accompanied by an acute inflammatory reaction that can contribute to neuronal da

247 ayed immune response, followed by an intense inflammatory reaction that causes widespread tissue dama

248 Myocardial necrosis triggers an inflammatory reaction that clears the wound from dead ce

249 The injury causes a rapid inflammatory reaction that includes activation of the ti

250 Granulomas represent a localized inflammatory reaction that is characteristically observe

251 Myocardial infarction triggers an inflammatory reaction that is involved in cardiac remode

252 regeneration are closely associated with an inflammatory reaction that is usually characterized by s

253 ing that this is associated with the intense inflammatory reaction that occurs in this condition.

254 Surgery launches a systemic inflammatory reaction that reaches the brain and associa

255 Infarct healing is dependent on an inflammatory reaction that results in leukocyte infiltra

256 y preterm labor or preeclampsia, triggers an inflammatory reaction that results in spontaneous preter

257 rystals into the rat joint provoked a marked inflammatory reaction that was significantly inhibited (

258 innate immune response involves a variety of inflammatory reactions that can result in inflammatory d

259 TLRs activate multiple steps in the inflammatory reactions that help to eliminate the invadi

260 deposits but damaged blood vessels caused by inflammatory reactions that lead to ischemia, amyloid ac

261 Bone loss in periodontitis results from inflammatory reactions that stimulate osteoclastic bone

262 ipoprotein cholesterol, initiating a chronic inflammatory reaction, the result of which is a vulnerab

263 ponse to pathological stimuli by suppressing inflammatory reactions through AdipoR1/AMPK-dependent me

264 counteracts scar resolution by perpetuating inflammatory reactions through release of proinflammator

265 osapentaenoic acid (EPA, 20:5n-3)], modulate inflammatory reactions through various mechanisms, inclu

266 Macrophages play vital functions in host inflammatory reaction, tissue repair, homeostasis and im

267 n of CCR7 implies that during a TH2-mediated inflammatory reaction, TLSP-activated CD1c(+) DCs are re

268 The immune-mediated inflammatory reaction to allogeneic and xenogeneic stem

269 important role in the initiation of the host inflammatory reaction to clear the invading pathogen.

270 The inflammatory reaction to coated discs was assessed in th

271 letion of NK cells significantly altered the inflammatory reaction to corneal wounding, as evidenced

272 corneal healing by limiting the innate acute inflammatory reaction to corneal wounding.

273 Local inflammatory reaction to inoculation with E coli was att

274 may serve as a surrogate marker of unwanted inflammatory reaction to microbicide products topically

275 Periodontal disease is an inflammatory reaction to the bacteria in dental plaque.

276 Modulation of the inflammatory reaction to the wear products represents a

277 test subject to assess cytotoxicity and pro-inflammatory reactions to diatom-biosilica.

278 ppreciated mechanism that prevents prolonged inflammatory reactions to Gram-negative bacteria and LPS

279 f transplantation is associated with instant inflammatory reactions to the graft and subsequent islet

280 ate HLA to evade host immune responses, host inflammatory reactions upregulate HLA, and differences a

281 behavioral deficits, neuronal injury and the inflammatory reaction using the kainic acid (KA) seizure

282 nly poised to initiate potentially injurious inflammatory reactions via immune complex formation asso

283 This inflammatory reaction was dependent on the interaction o

284 f (3)H-TPP and (18)F-FDG in inflammation, an inflammatory reaction was induced by subcutaneous inject

285 In 1 case an inflammatory reaction was observed in the anterior chamb

286 events, though a local sterile conjunctival inflammatory reaction was observed.

287 arization were increased, while the extended inflammatory reaction was repressed in the relaxin-treat

288 To study a role of cysLTs in inflammatory reactions we have characterized the gene st

289 lymphatic system is a critical regulator of inflammatory reactions, we tested the hypothesis that ob

290 ith virulent pathogens often induce a strong inflammatory reaction, what drives the increased immune

291 culature and towards the core of the ensuing inflammatory reaction where they exert effector function

292 Th1 lymphocytes participate in cell-mediated inflammatory reactions, where the selectins play a role

293 resent the major effector cells driving this inflammatory reaction whereas Foxp3(+) regulatory T cell

294 role of Foxp3(+)CD4(+) Tregs during ongoing inflammatory reactions, which is the topic of this artic

295 In contrast, persistent inflammatory reactions, which may be driven by infection

296 of action of neuropeptides and on immune and inflammatory reactions will likely yield new treatment o

297 the central nervous system (CNS) trigger an inflammatory reaction with potentially devastating conse

298 , and the orchestration of acute and chronic inflammatory reactions within the arterial wall.

299 afts in four recipients with MC developed an inflammatory reaction without skin graft loss.

300 These results affirm that metabolic and inflammatory reactions yield electrophilic products that