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1 arrest preempting organogenesis (flowers and inflorescence).
2 g the lengths of the siliques along the main inflorescence.
3 ach stage of development of the pea compound inflorescence.
4 tral domain and premature termination of the inflorescence.
5 ls during and after vernalization and in the inflorescence.
6 ssed, resulting in ectopic expression in the inflorescence.
7 to the 3' region of a gene expressed in the inflorescence.
8 ed MIR genes are expressed in shoot, root or inflorescence.
9 sis that BRs promote masculinity of the male inflorescence.
10 in establishing these boundaries within the inflorescence.
11 in functioning meristems of both rosette and inflorescence.
12 function in promoting cell elongation in the inflorescence.
13 ility of the lateral spikelets on the barley inflorescence.
14 rized by reduced and uneven branching of the inflorescence.
15 be key to future evo-devo work on the grass inflorescence.
16 the way in which flowers are arranged on an inflorescence.
17 , organized in structurally similar compound inflorescences.
18 primordium founder cell fate in Arabidopsis inflorescences.
19 ads to a strong decrease of FT expression in inflorescences.
20 ey role in determining simple versus complex inflorescences.
21 AD in three different extracts of G. globosa inflorescences.
22 ons in these genes delayed degreening of the inflorescences.
23 re highly influenced by the architectures of inflorescences.
24 resulted in shortened pedicels and clustered inflorescences.
25 and in leaves directly underneath developing inflorescences.
26 did to their own alarm pheromone on natural inflorescences.
27 development and inhibited degreening of the inflorescences.
28 AMOSA2 and RAMOSA3, generate highly branched inflorescences.
29 of mature miR160 was greatly reduced in foc inflorescences.
30 etative shoots replace seminiferous (sexual) inflorescences.
31 id concentrations were reduced in developing inflorescences.
32 ecture in a natural system displaying closed inflorescences.
33 n drastically increased PA levels within the inflorescences.
34 at CML36 and ACA8 are co-expressed mainly in inflorescences.
35 trescine, spermidine, and spermine in mutant inflorescences.
36 t this depends on pollinator behavior within inflorescences.
37 c function during the evolution of head-like inflorescences.
39 , gene expression changes in arrested apical inflorescences after fruit removal resembled changes obs
40 ILAMENTOUS FLOWER (FIL) is also required for inflorescence and floral meristem establishment and flow
41 hoot development, including leaf patterning, inflorescence and floral meristem function, and seed set
42 by directly activating key genes involved in inflorescence and floral meristem identity, including FR
43 ic gene regulation, maintain the identity of inflorescence and floral meristems after floral inductio
47 sting prior to initiating organogenesis, and inflorescence and flower meristems exhibiting a phenotyp
50 The distribution of the metabolites in the inflorescence and root parts were mainly affected by var
53 itutively expressed in Arabidopsis stems and inflorescences and shows highly coordinated herbivore-in
54 hgrass aboveground organs (leaves, stems and inflorescences) and underground organs (crowns and roots
55 ed for the dynamic attainment of vegetative, inflorescence, and floral meristem (VM, IM, FM) cell fat
56 changes in meristem fate from vegetative to inflorescence, and to floral, leading to flower formatio
60 ighly expressed in vegetative shoot buds and inflorescence apices, specifically at boundaries between
61 To identify genes that may contribute to inflorescence architecture and thus have the potential t
62 nt development, the flowering transition and inflorescence architecture are modulated by two homologo
63 MKK4/MKK5 and downstream of ER in regulating inflorescence architecture based on both gain- and loss-
64 lateral organ, the pulvinus, and influences inflorescence architecture by impacting the angle of lat
67 ssion patterns of CorTFL1 and CorAP1 and the inflorescence architecture in a natural system displayin
69 efine a fundamental mechanism that regulates inflorescence architecture in one of the most widely gro
70 ider implications for future manipulation of inflorescence architecture in related legume crop specie
72 aturation and that evolutionary diversity in inflorescence architecture is modulated by heterochronic
73 , Ishii and colleagues show that a change in inflorescence architecture is sufficient to increase see
75 ng plants, has been attributed to the unique inflorescence architecture of the family, which superfic
77 retention and modification of vegetative and inflorescence architecture that ultimately contributed t
78 e genes in maize modifies flowering time and inflorescence architecture through maintenance of the in
80 ng of flowering can have profound effects on inflorescence architecture, flower production and yield.
90 plants, raising the question of how diverse inflorescence architectures arise from seemingly common
91 dial branches produces a remarkable array of inflorescence architectures, but little is known about t
97 s, through which grains were retained on the inflorescence at maturity, enabling effective harvesting
98 oms include the formation of multiple female inflorescences at subapical nodes of the stalk because o
100 dened floral bracts and modifications to the inflorescence axis of grasses have been hypothesized to
101 5g14090) most similar to LAZY1 increased the inflorescence branch angle to 81 degrees from the wild t
103 elated nightshades (Solanaceae), new lateral inflorescence branches develop on the flanks of older br
106 genetic screen for the enhancement of maize inflorescence branching and the discovery of a new regul
108 netics approach reveals that the program for inflorescence branching is initiated surprisingly early
109 enetic studies in grass models, that is that inflorescence branching is regulated by novel localized
112 by reducing the number of flowers probed per inflorescence, but supporting evidence has been equivoca
113 ir up-regulation was suppressed in dark-held inflorescences by glucose treatment, which promoted macr
115 tion, and intermolecular interactions of the inflorescence cell wall using solid-state nuclear magnet
116 Thus, evolutionary diversity in Solanaceae inflorescence complexity is determined by subtle modific
117 e family members, we achieved a continuum of inflorescence complexity that allowed breeding of higher
120 than the B73 allele in hybrid B73-Mo17 F(1) inflorescences, consistent with the complete absence of
121 gain of function as the basis of bp and pny inflorescence defects and reveal how antagonism between
123 maize fuzzy tassel (fzt) mutant has striking inflorescence defects with indeterminate meristems, fasc
124 ing reporter lines to the abscission mutants inflorescence deficient in abscission (ida) and blade-on
126 tative ligand required for organ separation, INFLORESCENCE DEFICIENT IN ABSCISSION (IDA), suggesting
127 eparation event is controlled by the peptide INFLORESCENCE DEFICIENT IN ABSCISSION (IDA), which signa
128 by cell-wall remodeling, which involves the INFLORESCENCE DEFICIENT IN ABSCISSION (IDA)-derived pept
129 s reported for plants ectopically expressing INFLORESCENCE DEFICIENT IN ABSCISSION, a predicted signa
130 In tomato plants, formation of multiflowered inflorescences depends on a precisely timed process of m
131 operiod- and Ppd-H1-dependent differences in inflorescence development and flower fertility were asso
133 is critical for regulation of flowering and inflorescence development and identifying it as a homolo
136 As knowledge of the gene networks regulating inflorescence development in Arabidopsis thaliana improv
138 anding of the mechanisms underlying compound inflorescence development in pea and may have wider impl
141 nder long and short days, whereas successful inflorescence development occurred only under long days.
143 porter, exhibit a dosage-dependent defect in inflorescence development under B-limited conditions, in
144 synthesis and signaling pathways suppressing inflorescence development under long-day conditions.
145 reasing literature on genes regulating grass inflorescence development, an effective model of inflore
146 (tls1) mutant has defects in vegetative and inflorescence development, comparable to the effects of
147 into the genetic regulation of Brachypodium inflorescence development, we generated fast neutron mut
148 argue that the existing framework for grass inflorescence development, which invokes homeotic shifts
156 ELLIC ACID INSENSITIVE (SpGAI)) and observed inflorescence expression in females two-fold higher than
161 RF5 resulted in dwarfism, delayed growth and inflorescence formation, and up-regulation of Oskn2.
163 tioned by loss of expression of the COMPOUND INFLORESCENCE gene, is driven by delaying the maturation
166 r-mediated selection on floral display area, inflorescence height and corolla length of R. purpurea b
171 n of AhAI was observed in A. hypochondriacus inflorescence; however, it was not detected in the seed.
172 seeds on Arabidopsis (Arabidopsis thaliana) inflorescences, i.e. global proliferative arrest (GPA) d
174 is phenomenon reverts after emergence of the inflorescence in the cold or upon shift to 20 degrees C.
179 transcriptome-profiling of leaves and young inflorescences in wild and domesticated tetraploid wheat
180 d in developmental stages of L. x intermedia inflorescence indicating that the production of 1,8 cine
181 ercentage of RG-II dimers is reduced in tls1 inflorescences, indicating that the defects may result f
183 flowers early and converts the multiflowered inflorescence into a solitary flower as a result of prec
184 and this expression transforms multiflowered inflorescences into normal solitary flowers resembling t
185 opic defects, most notably simplification of inflorescences into single flowers, resembling tmf mutan
186 dence suggest that the reduced growth of the inflorescence is a result of carbohydrate starvation.
187 Brachypodium (Brachypodium distachyon), the inflorescence is an unbranched spike with a terminal spi
188 t flowering family, have a unique compressed inflorescence known as a capitulum, which resembles a so
190 T, including the differentiation of pin-like inflorescence, loss of apical dominance, leaf fusion, an
192 emic acquired resistance induction in female inflorescences mainly involves accumulation of salicylic
193 xpressed in meristematic tissues such as the inflorescence meristem (IM), floral meristem (FM), and c
195 d number of axillary meristems produced from inflorescence meristem compared with the wild type.
196 meristem, uniform expression of GhLFY in the inflorescence meristem defines the capitulum as a determ
201 We present a hypothesis that variation in inflorescence meristem size affects kernel row number (K
203 the shoot apical meristem is converted to an inflorescence meristem that forms flowers, and this tran
204 or, MYB and zinc-finger protein expressed in inflorescence meristem transcription factors were increa
205 ich repeat protein, was induced in the early inflorescence meristem, and flor1 mutations delayed flow
206 delayed conversion of vegetative meristem to inflorescence meristem, and repetitive initiation and ou
209 xpression in the shoot apical versus lateral inflorescence meristems is controlled through distinct c
210 CHEL (WUS) promotes stem cell maintenance in inflorescence meristems of Arabidopsis thaliana WUS, whi
215 ed whether the evolutionary modifications of inflorescence morphology result from shifts in their exp
216 us species that display four types of closed inflorescence morphology using quantitative real-time po
220 d that developmental arrest in the lox3 lox4 inflorescence occurs with the production of an abnormal
226 ession were studied in root, male and female inflorescences of maize under local and systemic fungal
231 ining the arrangement of flowers on a barley inflorescence, opening new doors for increasing grain yi
233 variation in the density of grains along the inflorescence, or spike, of modern cultivated barley (Ho
242 The data provide evidence of the G. globosa inflorescences potential as a source of anti-inflammator
243 the influence of photoperiod was studied on inflorescence primordia differentiation and floral pathw
244 tomato and related nightshades (Solanaceae), inflorescences range from solitary flowers to highly bra
246 ter early allocation to increased numbers of inflorescences, reduction in rosette leaf photosynthesis
248 ranscriptome profiling at 24 h revealed that inflorescence response at 24 h had a large carbon-depriv
256 onsequently, pin1 mutants give rise to naked inflorescence stalks with few or no flowers, indicating
258 reduction in total lignin amount in the main inflorescence stem and a compositional shift of the rema
259 otein, however, was localized throughout the inflorescence stem and at the base of lateral meristems,
260 double mutants no longer exhibited the short inflorescence stem and lignification phenotypes but stil
261 e control of cuticular wax deposition during inflorescence stem development in Arabidopsis (Arabidops
262 5 knockout mutant, the expansion rate of the inflorescence stem is halved compared with the wild type
263 dopsis (Arabidopsis thaliana) flowering, the inflorescence stem undergoes rapid growth, with elongati
264 aracterized by difficulties in developing an inflorescence stem was visible when plants were grown fo
265 ity, outgrowth of laminar-like tissue on the inflorescence stem, and early arrest of floral meristems
266 fectively restored the elongation of primary inflorescence stem, application to 7-week-old plants ena
267 ced visible phenotype is the extremely short inflorescence stem, but how deficient DGDG biosynthesis
268 ered only in the epidermal cells of the any1 inflorescence stem, whereas they were transverse to the
270 ressed in the vascular tissue of embryos and inflorescence stems and overexpression of QUA2 resulted
272 asurements of atlazy1 hypocotyls and primary inflorescence stems showed a significantly reduced bendi
273 te system as well as in Arabidopsis thaliana inflorescence stems that PIN-mediated auxin transport is
275 nd decreased stature with shorter leaves and inflorescence stems, thus supporting DAO1 IAA oxidase fu
276 ogenous promoter are small and have multiple inflorescence stems, twisted leaves, deformed leaf epide
281 entally typical flower buds are the terminal inflorescence structures observed in both the laboratory
283 zygous plants developed defective flowers on inflorescences that were eventually terminated by the fo
284 istems during the early development of maize inflorescences, the tassel and the ear, and has been imp
286 rmal seed development, while activity in the inflorescences themselves is required for proper loading
287 ead to ectopic expression of the gene in the inflorescences, thus conferring vegetative traits to rep
288 nt or produce secondary shoots terminated by inflorescences, thus increasing the number of infloresce
289 nome-wide screen for DELLA-bound loci in the inflorescence tip, revealed that DELLAs limit meristem s
290 Its overall activity is integrated across an inflorescence to determine final organ size, which is la
292 inance is gradually transferred from growing inflorescences to maturing seeds, allowing offspring con
294 d by the elongation of internodes to make an inflorescence upon which lateral branches and flowers ar
296 lia which included leaves, stems, roots, and inflorescences were collected from two Brazilian states
298 provement was an increase in seed number per inflorescence, which enhanced yield and simplified harve
299 ns of the Arabidopsis (Arabidopsis thaliana) inflorescence, while changes in the 3,320/2,944 cm(-1) i
300 in plants with increased branching, shorter inflorescences with fewer flowers, and dramatic changes
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