ライフサイエンスコーパス: influx

1 tion), but reduced SULTR1;1 expression (root influx).

2 expression, inhibitor binding, and substrate influx.

3 en mRNA levels of these genes and neutrophil influx.

4 ter release by inhibiting presynaptic Ca(2+) influx.

5 ne prevents TRPM7-dependent cytosolic Zn(2+) influx.

6 r (NCX) as predominant mechanisms of calcium influx.

7 romoter activity and TRPC6-dependent calcium influx.

8 hannels that control excitability and Ca(2+) influx.

9 talization of NMDA receptor-mediated calcium influx.

10 control axon outgrowth by regulating Ca(2+) influx.

11 cillation frequency and SOCE-mediated Ca(2+) influx.

12 hich was accompanied by a decrease in Ca(2+) influx.

13 n potential broadening and increased calcium influx.

14 istically enhanced degranulation and calcium influx.

15 rmis in a day against the interstitial fluid influx.

16 rmeable AMPARs, contributing to toxic Ca(2+) influx.

17 eurotransmitter release downstream of Ca(2+) influx.

18 (SOCE) as the major pathway for this Ca(2+) influx.

19 neurotransmitter release by limiting calcium influx.

20 roteins, and was highly dependent on calcium influx.

21 membrane and reduce voltage-dependent Ca(2+) influx.

22 effector functions require sustained calcium influx.

23 no effect on the mechanically induced Ca(2+) influx.

24 that open cooperatively, facilitating Ca(2+) influx.

25 d Orai couple to each other, allowing Ca(2+) influx.

26 gments mechanosensitive intracellular Ca(2+) influx.

27 r decreasing action potential-evoked calcium influx.

28 urrents and triggered NMDAR-dependent Ca(2+) influx.

29 s were initiated by an extracellular calcium influx across the cell membrane nearest to the jetting f

30 ssed in cardiomyocytes and decreases calcium influx across the L-type Ca(2+) channel.

31 esulting in membrane depolarization, calcium influx, aldosterone production, and cell proliferation.

32 Ca(2+) influx also regulates insulin synthesis and insulin gran

33 The reduced Ca(2+) influx alters the kinetics and amplitude of the global C

34 imicked the effect of leptin, causing Ca(2+) influx, AMPK activation, and increased trafficking of KA

35 cluding reduced depolarization-evoked Ca(2+)-influx and beta-cell exocytosis.

36 tivity in coupled neurons depends on calcium influx and calcium-initiated signalling pathways.

37 ritical for feedback control of both calcium influx and cell excitability.

38 stigated: cell death (apoptosis), neutrophil influx and cytokine/chemokine expression.

39 n endometrial apoptosis preceding neutrophil influx and cytokine/chemokine induction during active me

40 eceptor (NMDA-R) agonist, leading to calcium influx and downstream cell signaling events and neurotox

41 l and polarity; and (3) multiple patterns of influx and efflux carriers for maintaining an auxin patt

42 ns of interlinked levels and localisation of influx and efflux carriers.

43 tern formation requires coordination between influx and efflux carriers.

44 ansporters involved in mitochondrial calcium influx and efflux have recently been identified.

45 mphasis on nonequilibrium factors, including influx and efflux of lipid molecules.

46 Compared to the influx and efflux rates of most amino acids, similar one

47 orphogenesis, restrictive barrier formation, influx and efflux transporters with relevance to underst

48 cellular lactate, inhibiting monocarboxylate influx and efflux.

49 containing channels resulted in large Ca(2+) influx and enhanced migration, while in normal cerebella

50 quently, this impairs activity-driven Ca(2+) influx and exocytosis at nerve terminals.

51 ind that the spatial coupling between Ca(2+) influx and exocytosis changes from nanodomain in low-fre

52 Moreover, Gipc3 disruption enhanced Ca(2+) influx and exocytosis in IHCs, reversed the spatial grad

53 ted view of current models concerning Ca(2+) influx and extrusion mechanisms, where most of the recen

54 gy minimization, closely resembling synaptic influx and extrusion of Ca(2+), respectively.

55 optive transfer of ILC2s restored eosinophil influx and IL-4, IL-5 and IL-13 production in lung tissu

56 st activation kinetics in response to Ca(2+) influx and inactivation gating that could be modified by

57 e on TRPV1 channels was dependent on calcium influx and linked to calcium/calmodulin-dependent protei

58 his treatment was associated with macrophage influx and M1-like polarization, along with increased T

59 annels constitute a major pathway for Ca(2+) influx and mediate many essential signalling functions i

60 nule precursors, only small levels of Ca(2+) influx and no enhanced migration were observed.

61 residual T cells were able to induce Ca(2+) influx and nuclear factor (NF) kappaB signaling, whereas

62 necroptosis, MLKL-dependent calcium (Ca(2+)) influx and phosphatidylserine (PS) exposure on the outer

63 sed in brain and kidney and mediates calcium influx and promotes cell migration.

64 rough SGLT1 cotransporters can induce Ca(2+) influx and release of GLP-1 as a result of electrical ac

65 arize them to voltages necessary for calcium influx and synaptic vesicle fusion.

66 aquifer behaviors despite continuous oxygen influx and the annual hydrologically induced oxidation e

67 t functional coupling between CaV1.3 calcium influx and the intermediate-conductance KCa3.1 potassium

68 metry was used to determine LPS-induced cell influx and TNF-alpha production in peritoneal cells.

69 nnata of sulfate transporters SULTR1;2 (root influx) and SULTR2;1 (translocation), but reduced SULTR1

70 nel beta2 subunits modulate AP-evoked Ca(2+)-influx, and (3) beta2 subunits maintain successful AP pr

71 ndy expression, enhanced cytoplasmic citrate influx, and augmented hepatic lipogenesis in vivo.

72 ut requires membrane depolarization, calcium influx, and calcineurin signaling.

73 d increased chemokine expression, neutrophil influx, and mucosal damage.

74 uced urine abnormalities, renal myeloid cell influx, and NCGN.

75 e effect of HMBA was downstream of a calcium influx, and required the pattern recognition receptor an

76 bations in firing through changes in calcium influx, and translates this into compensatory changes in

77 a result of excessive intracellular calcium influx, and we have previously shown that angiotensin II

78 ss, result in multiple mechanisms of calcium influx around epithelial wounds.

79 A zinc-influx assay demonstrated that TRPM6 has the potential t

80 stimate that CaM senses NMDA receptor Ca(2+) influx at approximately 9 nm from the channel pore.

81 itability, thereby limiting excessive Ca(2+) influx at subthreshold potentials or during Ca(2+)-depen

82 to the lowered outward current (gluconate(-) influx) at membrane potential of 100 mV.

83 ignificantly affect the shear induced Ca(2+) influx, but at 10 mM it produced significant inhibition.

84 nsmission are converted to excitatory sodium influx by acid-sensing ion channels (ASICs).

85 cells directly or indirectly regulate their influx by altering the chemotactic milieu in the islets.

86 es and chemokines that regulate inflammatory influx by leukocytes in response to infection.

87 receptors in hCMEC/D3 cells induces a Ca(2+) influx by opening CNG channels in a cAMP-dependent manne

88 endent currents (e.g., NMDAR-mediated Ca(2+) influx) by synaptic input.

89 Here, we demonstrate that upon Ca(2+) influx, Ca(2+)/calmodulin (Ca(2+)/CaM) binding to the N-

90 iated reuptake rather than changes in Ca(2+) influx capacity.

91 sis is partly regulated by NPF3 acting as an influx carrier and that GA-ABA interaction may occur at

92 h ECH and ARF1 mediate the secretion of AUX1 influx carrier to the plasma membrane from the TGN durin

93 same auxin pattern, from different levels of influx carriers with the same nonpolar localisation, req

94 quires non-uniform and polar distribution of influx carriers; (2) the emergence of the same auxin pat

95 lity by binding to and inhibiting the Ca(2+) influx channel Orai1.

96 olecular components of store-operated Ca(2+) influx channels (SOCs) in proliferative and migratory va

97 findings stress the role of multiple Ca(2+) influx channels in VSMCs and are the first to show the r

98 se (nsEP) exposure generates reduced calcium influx compared to a unipolar (UP) nsEP.

99 n structure and how self-renewal and de novo influx contribute to the maintenance of memory compartme

100 ntly activated by calcium sparks and calcium influx during action potentials and control the one-to-o

101 echanisms and pathways mediating this Ca(2+) influx during agonist-induced ASMC contraction are not w

102 tput of central neurons by providing calcium influx during repetitive inputs.

103 luence AHPs in OT neurons by reducing Ca(2+) influx during spiking.

104 st and negatively correlated with the Ca(2+) influx EC50.

105 sensor (eUnaG) to detect transporter-coupled influx/efflux of organic compounds.

106 sensors/regulators, soluble chaperones, and influx/efflux transporters that control the Cu(+) levels

107 flux since last century, with the maximum Hg influx enrichment ratios of 5.4 and 3.5 in Lake Qinghai

108 balanced such that, in the steady state, Ca influx equals Ca efflux on every beat.

109 reover, we ask whether TRPV4-mediated Ca(++)-influx evokes mast cell degranulation.

110 ial propagation failure nor depressed Ca(2+) influx explained loss of evoked synaptic transmission.

111 channel (CNGC) family members mediate Ca(2+) influx from cellular stores in plants.

112 ide signalling, which is triggered by Ca(2+) influx from L-type voltage-gated Ca(2+) channels, not po

113 They elicited calcium influx, hyperalgesia and induced pro-nociceptive peptide

114 HQ elicited Ca(2+) influx in a subpopulation of mouse sensory neurons sensi

115 cation potentiated capsaicin-induced calcium influx in a subset of sensory neurons.

116 stos exposure increased mitochondrial Ca(2+) influx in alveolar macrophages from wild-type, but not M

117 Ca(2+) oscillations and SOCE-mediated Ca(2+) influx in ASMCs within mouse precision-cut lung slices.

118 onfirmed to be directly controlled by Ca(2+) influx in beta-cells.

119 mmol/L glucose, ANP readily elicited Ca(2+) influx in control beta-cells.

120 s regulate the membrane potential and Ca(2+) influx in human effector memory T (TEM) cells.

121 ersed the spatial gradient of maximal Ca(2+) influx in IHCs, and increased the maximal firing rate of

122 hway functions to promote endosomal membrane influx in infected macrophages, and is required to prese

123 n addition, we observed an increased calcium influx in KCl-depolarised cells expressing mutated hippo

124 brane calcium channel ORAI1 mediates calcium influx in LECs and activates calmodulin to facilitate a

125 ically, TCR stimulation induced rapid sodium influx in Napa(hyh/hyh) CD4 T cells, which reduced intra

126 To test the role of auxin influx in nodulation we used the auxin influx inhibitors

127 mulation increases the intracellular calcium influx in OPCs as well as the gene expression of L-type

128 brain and suggest that voltage-gated Ca(2+) influx in OPCs is critical for remyelination.

129 ) channel expression and ATP-induced calcium influx in podocytes of Podo-GC-A KO mice.

130 that Ang II-dependent stimulation of Ca(2+) influx in the podocytes is precluded by blocking either

131 hannel open time for outward currents (Cl(-) influx) increases linearly as the amplitude of single ch

132 Furthermore, excessive Ca(2+) influx induced by an optogenetic approach also inhibits

133 obetric integrator (CaMPARI) reports calcium influx induced by synaptic and neural activity.

134 auxin influx in nodulation we used the auxin influx inhibitors 1-naphthoxyacetic acid (1-NOA) and 2-N

135 Na(+)-influx inhibitors did not improve relaxation or prevent

136 er expression and tested the effect of Na(+)-influx inhibitors on relaxation in isolated myocardium f

137 The lusitropic effect of Na(+)-influx inhibitors ranolazine, furosemide, and amiloride

138 napses, while leaving NMDAR-mediated calcium influx intact.

139 hair cell stimulation leads to robust Ca(2+) influx into afferent terminals.

140 teasome-mediated degradation to limit Ca(2+) influx into mitochondria.

141 lculations suggest that increased root water influx into plant conducting-tissues overnight maintains

142 liposomal models and promote sodium chloride influx into the cytosol.

143 itional possibilities for regulating calcium influx into the cytosol.

144 C60-serPF velocity, and the time of compound influx into the intra- and extra-vascular space suggest

145 ltimeric ion channel which, by tuning Ca(2+) influx into the mitochondrial matrix, finely regulates m

146 tion (TA) spectroscopy to monitor the energy influx into the monolayer MoS2 in the process of ET from

147 y a series of C balance equations to track C influxes into and effluxes out of individual pools in ea

148 D-95 induced by a chronic increase in Ca(2+) influx is a critical molecular event in homeostatic down

149 s voltage and calcium indicated that calcium influx is induced by voltage depolarizations, similar to

150 horylation of phospholipase Cgamma or Ca(2+) influx, it was associated with significantly increased a

151 to have been a loss of diversity in calcium-influx mechanisms at the plasma membrane.

152 These results suggest that Ca(2+) influx mediated by L-VGCCs in oligodendroglial cells is

153 cytosolic Ca(2+) levels under a capacitative influx model and ER re-uptake of calcium, increasing the

154 F animals restored IgG deposition, leukocyte influx, NF-kappaB activation, and proinflammatory gene e

155 tallisation history of Toba quartz traces an influx of a low-delta(18)O component into the magma rese

156 uppression of regulatory T cells and greater influx of activated CD8(+) T cells.

157 stent with the historically attested earlier influx of Africans into Colombia.

158 infection of naive wild-type mice induced an influx of airway neutrophils and CD11b(+) exudative macr

159 ma pathogenesis by enabling the paracellular influx of allergens, toxins, and microbes to the submuco

160 Following stroke, a large influx of astrocytes and microglia releasing proinflamma

161 It is driven by the influx of Ca(2+) across the sarcolemma triggering Ca(2+)

162 obilization of intracellular Ca(2+) leads to influx of Ca(2+) and Na(+) through TRPV4 channels to evo

163 Intra-mitochondrial influx of Ca(2+) induces and potentiates CoMIC, and lead

164 tensioned at rest, resulting in a continuous influx of Ca(2+) into the cell.

165 STATEMENT During synaptic transmission, the influx of Ca(2+) into the presynaptic nerve terminal act

166 y trigger for Hebbian synaptic plasticity is influx of Ca2+ into postsynaptic dendritic spines.

167 hair cell near potentials that promoted the influx of calcium necessary for synaptic vesicle fusion.

168 rapid influx of calcium, we observe a second influx of calcium that spreads to neighboring cells beyo

169 Approximately 45 s after this rapid influx of calcium, we observe a second influx of calcium

170 uring intestinal injury, concomitant with an influx of CCDC88B(+)lymphoid and myeloid cells.

171 istently elevated in inflamed islets and the influx of CD11c(+) cells was partially dependent on thei

172 kdown of the blood-brain barrier and a large influx of CD8(+) effector T cells.

173 matrix proteins but also by controlling the influx of chemokines through the regulation and shedding

174 This response was driven by an influx of CRON DP-associated taxa.

175 by melanoma, which testified CTR1 specially influx of Cu(I).

176 riven assembly operates under the continuous influx of energy and results in superstructures that exi

177 ntraction and Ca(2+) oscillations require an influx of extracellular Ca(2+) , although the mechanisms

178 e energy transfer imaging, we found that the influx of extracellular Ca(2+) subsequent to LLO-mediate

179 tegrity of the plasma membrane, enabling the influx of extracellular nanoagents into cells to promote

180 ncrease in intracellular free Zn(2+) through influx of extracellular Zn(2+) ) were mostly independent

181 Increased influx of fatty acids and inflammatory molecules from ot

182 imary role is to mediate selective uptake or influx of HDL-derived cholesteryl esters into cells and

183 Following the initial influx of immune cells, T cell clusters develop, acceler

184 We also observed a remarkable influx of inflammatory cells and upregulation of cytokin

185 ocular surface response while the secondary influx of inflammatory cells leading to the recruitment

186 which were widely infected and caused marked influx of interstitial macrophages and neutrophils.

187 monary inflammation, which including reduced influx of leukocytes and down regulated tumor-promoting

188 disease characterized by massive transmural influx of leukocytes and lymphocytes, resulting in villo

189 mulation by bacterial lipopolysaccharide; an influx of macrophages and appearance of live bacteria we

190 ivo fluorescence microscopy revealed a large influx of macrophages in the pancreas colocalizing with

191 However, these methods required an influx of metal ions to increase their concentrations fo

192 Ischemia-reperfusion injury induced influx of monocytes, DCs, macrophages, and neutrophils i

193 vity of BALB/c mice induced a time-dependent influx of mononuclear cells that were primarily macropha

194 Thus, influx of Na(+) via voltage-gated Na(+) channels (NaV )

195 There has been an increasing influx of nanopesticides into agriculture in recent year

196 on in allergic mice with SD was marked by an influx of neutrophils (mainly) and eosinophils and secre

197 orptive transporters and may not require the influx of neutrophils that accompanies infection.

198 ell memory subsets by measuring the rates of influx of new cells and using detailed timecourses of DN

199 uch expression was dependent on a continuous influx of newly synthesized PTC-containing mRNAs, indica

200 tion, and hence the tricarboxylic acid cycle influx of pyruvate-derived acetyl-CoA relative to beta-o

201 ing glacier runoff, but the influence of the influx of riverine organic matter on the trophodynamics

202 active cellular processes that can create an influx of RNA into RNP granules, such as transcription,

203 aling events, including cytosolic efflux and influx of select ions, have been suggested.

204 nding to Toll-Like receptors (TLR) induce an influx of superoxide anion in the ensuing endosomes.

205 found that MCT8 was necessary for polarized influx of the active form of TH across the BBB.

206 high intracellular Ca(2+) , suggesting that influx of the divalent ion via more Ca(2+) -permeable no

207 owever, we also find signals consistent with influxes of non-local people, most likely from northern

208 Our results suggest that seasonal influxes of O2 and NO3(-) may cause only localized mobil

209 forecasting the response of rivers to large influxes of sediment triggered by earthquakes or storms.

210 e the contributions of STIM1-mediated Ca(2+) influx on electrical and mechanical properties of normal

211 ation induces cell death by sustained Ca(2+) influx or by enhancing cytokine production, aggravating

212 n of input fibres does not depend on calcium influx or dynamics.

213 Pharmacological blockade of NMDA-R, calcium influx, or calpain activity abolished SSC and glutamate

214 e data suggest that SOCE is the major Ca(2+) influx pathway for ASMCs with respect to sustaining agon

215 rated Ca(2+) entry (SOCE) is the main Ca(2+) influx pathway in lymphocytes and is essential for T cel

216 Orai in cardiac myocytes to produce a Ca(2+) influx pathway that can prolong the AP duration and load

217 ma membrane Orai channels to induce a Ca(2+) influx pathway.

218 Ionomycin-induced Ca(2+) influx promoted p-Akt, an effect blocked by PDK1, and/or

219 Parametric distribution volume (VT) and influx rate (K1) were compared with those obtained from

220 e trapping rate (k3) and delivery rate (K1), influx rate (Ki ) constants, and tissue-to-blood activit

221 ermine the insulin-mediated (18)F-FDG tissue influx rate (Ki) in the whole-body region by using the P

222 However, changes in net influx rate (Ki) may better reflect treatment effects th

223 le binding (BPND) for [(11)C]carfentanil and influx rate constant (Ki) values for [(18)F]fluorodopa w

224 me VTDuring ABCB1 inhibition, retinal VT and influx rate constant K1 were significantly, by 1.4 +/- 0

225 f tissue-specific insulin-mediated (18)F-FDG influx rates, tissue depots, and M value.

226 fluorine 18 ((18)F) fluorodeoxyglucose (FDG) influx rates, tissue depots, and whole-body insulin sens

227 cell death due to profoundly enhanced Ca(2+) influx, resulting from altered channel function.

228 Instead, we suggest that the sediment influx should result in an elevated sand flux, leading t

229 , Lake Qinghai and Nam Co, show increased Hg influx since last century, with the maximum Hg influx en

230 ction of NMDA-receptor-dependent LTP, Ca(2+) influx stimulates recruitment of synaptic AMPA (alpha-am

231 ay hyperresponsiveness and inflammatory cell influx suggesting the presence of a novel receptor that

232 algae and marine angiosperms requires Na(+) influx, suggesting that Na(+) /Pi symporters also functi

233 periplasmic AGP-Ca(2+) resulting in a Ca(2+) influx that activates exocytosis of wall precursors.

234 lt of glucose transporter 1-mediated glucose influx that drives glucose metabolism and ATP production

235 ing within the skin, preventing an epidermal influx that is associated with lesion development.

236 nucleotide-gated channels allowing a Ca(2+) influx that opens Ca(2+)-activated Cl(-) channels, gener

237 significantly reduced neointimal macrophage influx that was accompanied by increased GCLC messenger

238 oltage pulses in tumors enables high calcium influxes that trigger cell death.

239 dant system through up-regulation of glucose influx, the pentose phosphate pathway, and NAD salvaging

240 In response to gradual changes in oxygen influx, this model abruptly transitions between an oxic

241 nic neurons, acts to reduce excessive Ca(2+) influx through Cav channels and thus permits normal axon

242 ically, this requires local dendritic Ca(2+) influx through Dalpha7nAChRs or through LVA channels fol

243 traburst firing rates require axonal calcium influx through Dmca1D channels, likely to enhance sodium

244 in release from beta-cells depends on Ca(2+) influx through high voltage-gated Ca(2+) channels (HVCCs

245 , when we controlled the frequency of Ca(2+) influx through individual channels, we found that a kine

246 Ca(2+)-influx through L-type Ca(2+)-channels (LTCCs) is associa

247 Ca(2+) influx through Orai1 channels is crucial for several T c

248 +) concentration occur in response to Ca(2+) influx through plasma membrane channels and Ca(2+) relea

249 ase in the amplitude and frequency of Ca(2+) influx through T-type and L-type Ca(2+) channels.

250 v2.2-NOS1 complexes voltage-triggered Ca(2+) influx through the Cav channels reliably initiates enzym

251 Ca(2+) influx through the dendritic high-threshold voltage-gate

252 ratio of lactate production flux to pyruvate influx through the mitochondrial pyruvate carrier) was c

253 e bacterial flagellum is powered by a proton influx through the peptidoglycan (PG)-tethered stator ri

254 tant partner in regulating immediate calcium influx through the surface membrane readily accounts for

255 Calcium influx through the voltage-dependent L-type calcium chan

256 a signalling cascade that stimulates cation influx through transient receptor potential (TRP) melast

257 Furthermore, Ca(2+) influx through TRPM7 is essential for the maintenance of

258 We found that Ca(2+) influx through TRPV1 is necessary for capsaicin-induced

259 Our findings demonstrate how Ca(2+) influx through TRPV4 channels can activate SK channels i

260 This limits calcium influx through voltage-dependent calcium channels and da

261 SK channels were also activated by Ca(2+) influx through voltage-gated Ca(2+) channels and synapti

262 ular Ca(2+) concentration, by varying Ca(2+) influx through voltage-gated Ca(2+)-channels or Ca(2+) u

263 Ca(2+) influx through voltage-gated L-type Ca(2+) channels (LTC

264 t extends the contribution of CaV1.3 calcium influx to a time frame well beyond a brief input train.S

265 ht be targeted to promote greater antibiotic influx to increase cytoplasmic antibiotic concentration

266 stained Ca(2+) oscillations require a Ca(2+) influx to replenish Ca(2+) losses across the plasma memb

267 es and chemokines that regulate inflammatory influx to sites of infection and tissue damage.

268 itable cells harness voltage-coupled calcium influx to transmit intracellular signals, typically stud

269 ated calcium channels (VACCs) mediate Ca(2+) influx to trigger action potential-evoked neurotransmitt

270 1 (hCTR1) is the major high affinity copper influx transporter in mammalian cells, and specially res

271 s thaliana auxin efflux transporter pin1 and influx transporter lax2 mutants showed reduced vein numb

272 e involvement of the AUX/LAX family of auxin influx transporters in nodulation.

273 C30A10 and SLC39A14 are manganese efflux and influx transporters, respectively.

274 At the cellular level, calcium influx triggered by beta-alanine is also unchanged in cu

275 ulin 1 (INS1) cells for which localized Ca2+ influx triggers exocytosis with high probability and min

276 Here, we show that this constitutive Ca(2+) influx, usually considered as potentially deleterious fo

277 Thus, non-specific sodium influx via bonafide calcium channels disrupts unexpected

278 to a reduction in activity-dependent calcium influx via Cav channels.

279 Ageing maintained the integrated Ca(2+) influx via ICa-L but decreased peak ICa-L .

280 ide efflux, leading to compensatory chloride influx via NCC activation at the cost of increasing sodi

281 the sarcoplasmic reticulum, although Ca(2+) influx via plasma membrane channels is also necessary to

282 on collagen-induced aggregation or on Ca(2+) influx via TRPC6 or Orai1 channels and caused only a min

283 GCCs), we show that SOCE, rather than Ca(2+) influx via VGCCs, provides the major Ca(2+) entry pathwa

284 This massive endosomal influx was accompanied by dramatic recruitment of the in

285 d-brain barrier, although the rate of SAA2.1 influx was approximately 5 times faster than that of SAA

286 The influx was inhibited by MK-801, a specific pore blocker

287 experiments in vitro indicated that calcium influx was similar in fibroblasts and cancer cells.

288 tion of emphysema pathogenesis to macrophage influx, we evaluated the inflammatory changes and lung h

289 alyzing swelling as osmotically driven water influx, we find the H2O membrane permeability of the rod

290 voltage-gated Ca(2+) channels to this Ca(2+) influx, we generated a comprehensive mathematical model,

291 mbrane transporters probably mediating Cu(+) influx were among those largely repressed upon Cu(+) str

292 d an inward depolarizing current and calcium influx, whereas other analogs did not exhibit these effe

293 both intracellular Ca(2+) release and Ca(2+) influx, which activates low-level nitric oxide productio

294 oligomers could be observed to induce Ca(2+) influx, which could be inhibited by the addition of a na

295 ptic currents and, more relevant, to calcium influx, which could be involved in the modulation of pre

296 (CNG) channels and thereby induces a Ca(2+) influx, which leads to the increase in gap junction coup

297 e of the Ca transient (without decreasing Ca influx) will therefore increase diastolic [Ca(2+) ]i .

298 calcium channel so as to synchronize calcium influx with membrane fusion.

299 buffering with EGTA-AM or decreasing calcium influx with omega-agatoxin IVA decreased the amount of a

300 functions downstream of presynaptic calcium influx with separable activities to stabilize baseline t