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1 loss, and distinctive myelin outfolding and infolding.
2 large macroendosomes (MEs) via bulk membrane infolding.
3 nduction velocity and myelin outfoldings and infoldings.
4 s, the RPE had dilated and fewer basolateral infoldings.
5 a of intracellular cisterns and plasmalemmal infoldings.
6 y decreased both apical microvilli and basal infoldings.
7 oelectron microscopy at microvilli and basal infoldings.
8 hitectural distortion of the plasma membrane infoldings.
9 and the appearance of large intramembraneous infoldings.
11 results from this study suggest that myelin infolding and paranodal damage may represent pathogenic
12 plement of sensillae before hatching; 2) the infolding and rotations that form the deep groove are co
14 FM1-43 can be rinsed) delivers vesicles via infoldings and cisternae selectively to a reserve pool w
15 own to lead to loss of postsynaptic membrane infoldings and disorganization of the NMJ microenvironme
16 y distributed cisternae and surface membrane infoldings and enter vesicle clusters spaced at regular
18 9P) knockin mice exhibited loss of the basal infoldings and vacuolization, with accumulation of amorp
19 tes were of normal size, contained glandular infoldings, and maintained high secretory epithelium, an
20 luding basal laminar deposits, loss of basal infoldings, and vacuoles in the retinal pigment epitheli
23 mination, whereas the potential for carboxyl infolding by hydrogen bonding seems to favor glucuronida
24 olation, loss or disruption of the RPE basal infoldings, choroidal atrophy, and focal thickening of a
27 e and stubby apical microvilli, and no basal infoldings, induced maturation of microvilli and the for
33 unoreactivity for Na/K-ATPase revealed basal infolding of lamellate cells in type-I, abluminal inters
38 n abnormalities, such as tomacula and myelin infoldings/outfoldings, centered around the paranodes an
39 nificant loss of apical microvilli and basal infoldings, reduced retinal adhesion, and epithelial-mes
40 helial) cells in type-II, and labyrinth-like infolding structures opening towards the lumen in type-I
42 by decreased epithelial height and glandular infolding through 24 weeks of age, differentiation marke
43 ong apical microvilli and of elaborate basal infoldings typical of these cells, and characterize the
45 ion of microvilli and the formation of basal infoldings without changing moesin expression levels.
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