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1 hylaxis that occurs 3-6 hours after red meat ingestion.
2 e vaccine, similar to those caused by gluten ingestion.
3 me retardants (EFRs) via inhalation and dust ingestion.
4 lected 2 times/d before and after RHM or PHM ingestion.
5 xercise brought about by warm and cool fluid ingestion.
6 difference in decline in pain 2 hours after ingestion.
7 ntake being more important than frequency of ingestion.
8 libitum pizza intake 150 min after beverage ingestion.
9 ere obtained every 10 min until 90 min after ingestion.
10 cytokine responses within 12 h of bacterial ingestion.
11 measure Cfree and the chemicals taken up via ingestion.
12 tory input and hunger state to modulate food ingestion.
13 r emergency patient evaluation after caustic ingestion.
14 he most devastating complications of caustic ingestion.
15 termine the stability of LHRHa upon possible ingestion.
16 als, that reduces muscle sensitivity to food ingestion.
17 llergy due to IgE mediated anaphylaxis after ingestion.
18 er of sea turtles globally at risk of debris ingestion.
19 engage segregated brain networks to motivate ingestion.
20 BOEP and triphenyl phosphate (TPHP) via dust ingestion.
21 Grade 3 to 5 anaphylactic reactions upon egg ingestion.
22 g gut PCs accumulate in patients upon gluten ingestion.
23 l striatum with fructose relative to glucose ingestion.
24 racheobronchial necrosis (TBN) after caustic ingestion.
25 ere not different from the response to water ingestion.
26 iplatelet effects compared with whole tablet ingestion.
27 ppressed by food-related sensory cues before ingestion.
28 efore and at 2, 4, 7, 24, and 48h after GSPE ingestion.
29 rease muscle protein synthesis after protein ingestion.
30 d tissues that were fungally degraded before ingestion.
31 re miRNAs were relatively stable after blood ingestion.
32 pid disintegration and dissolution upon oral ingestion.
33 veral health benefits, are metabolized after ingestion.
34 ed to siRNAs by EAB larvae within 72 h after ingestion.
35 tion of the networks subsequent to Ayahuasca ingestion.
36 ence physiological responses to carbohydrate ingestion.
37 a potentially toxic calcium channel blocker ingestion (1D); 2) as first-line therapies (prioritized
38 .5 +/- 0.4 degrees C; P = 0.27) or LRG fluid ingestion (7 degrees C: 35.3 +/- 0.6 degrees C; 22 degre
39 n all fluid temperatures following SML fluid ingestion (7 degrees C: 35.7 +/- 0.5 degrees C; 22 degre
40 otypic changes associated with microparticle ingestion, a consistently sparse population of resident
42 ise temporal dynamics by which water or food ingestion affect VPpp neuron activity, we directly recor
46 ed sucrose pellets both pre- and post reward ingestion and also reduced responding under simpler sche
47 fundamental roles that the stomach plays in ingestion and digestion notwithstanding, little morpholo
48 ntial for the incretin effect after nutrient ingestion and is critical for the actions of dipeptidyl
50 ed that HIV-infection progression due to Coc ingestion and therapeutic effects of highly specific dru
51 flame retardants (PFRs) via inhalation, dust ingestion, and dermal absorption using different samplin
52 human exposure to PFRs via inhalation, dust ingestion, and dermal absorption was conducted with indi
53 ed by Cd and the contribution of inhalation, ingestion, and dermal contact pathways to these risks.
55 ea turtles are at the highest risk of debris ingestion, and olive ridley turtles are the most at-risk
56 ucine lower blood glucose after oral glucose ingestion, and the intraduodenal infusion of leucine dec
57 (95% CI 176.2-342.4) mumol/L 3.5 hours after ingestion, and the median half-life was 73.0 (interquart
58 In relation to inhalation exposure, dust ingestion appears to be the major exposure pathway to FR
59 ho took 10 mg/d of biotin for 7 days, biotin ingestion-associated interference was found in 9 of the
60 and the sensation of coolness, whereas fluid ingestion at 22 and 7 degrees C increased shivering and
61 cold compared to 37 degrees C, whereas fluid ingestion at 22 and 7 degrees C led to equivalent increa
62 red to shivering and thermal sensations with ingestion at 37 degrees C (M: 215 +/- 47 W, EMG: 3.9 +/-
64 ferences in core and skin temperature, fluid ingestion at 52 degrees C rapidly decreased shivering an
66 cooler thermal sensations were observed with ingestion at 7 degrees C (M: 179 +/- 55 W, WBTS: 29 +/-
67 lues were different (all P < 0.05) following ingestion at 7 degrees C (M: 269 +/- 77 W, EMG: 5.5 +/-
68 ts mainly from the fear of accidental peanut ingestion but is also due to dietary and social restrict
69 istory stage is the best predictor of debris ingestion, but the best-fit model also incorporates enco
72 id radiation, suggesting that apoptotic body ingestion by CD8(+) DCs initiates tolerance induction.
75 fferentially phosphorylated following fungal ingestion by macrophages, thereby indicating global repr
79 We modelled the risk (probability of debris ingestion) by incorporating exposure to debris and conse
80 both chemical uptake from water or via food ingestion can be controlled, thus enabling the discrimin
87 contrast, exercise performed before fructose ingestion does not significantly alter fructose oxidatio
88 vity is modified by both warm and cold fluid ingestion during heat stress, independently of differenc
89 vity is modified by both warm and cold fluid ingestion during heat stress, independently of differenc
91 ires the identification of accompanying drug ingestions, especially in the setting of intentional poi
92 ve under extended periods of wakefulness and ingestion events, daily eating pattern offers a new pote
93 0-min exercise either 75 min before fructose ingestion [exercise, then fructose condition (ExFru)] or
94 evealed relative importance of soil and dust ingestion exposure pathways and associated Pb intake rat
95 s of the small intestine and released by fat ingestion, facilitates fatty acid translocation in rat i
96 dy the fate and behaviour of AgNPs upon oral ingestion following an in vitro model that included sali
98 ing plants, recorded at 24-hr intervals post-ingestion for three days, was >90% and 10%, respectively
99 condition (ExFru)] or 90 min after fructose ingestion [fructose, then exercise condition (FruEx)].
100 ased circulating insulin level after glucose ingestion; furthermore, the change in acyl-ghrelin and i
102 ficantly between the high and medium plastic ingestion group for sumPCBs, sumDDTs, and sumPBDEs.
103 study examines the association between 6-MP ingestion habits and 6-MP adherence, red cell thioguanin
104 ators, there was no association between 6-MP ingestion habits and relapse risk (6-MP with food: hazar
107 n vivo chemical complexity it manifests upon ingestion has impeded efforts to unambiguously identify
112 ions for safe upper limits on chronic iodine ingestion in general and on iodine supplementation in pa
113 ardiac repolarization in response to glucose ingestion in LQT2 patients with functional mutations in
114 can silence genes of matching sequence upon ingestion in many invertebrates and is therefore being d
123 xercise performed immediately after fructose ingestion increases fructose oxidation and decreases fru
126 Ms to calculate residential intake from dust ingestion, inhalation, and dermal uptake from air, and t
129 st habitats, which implies that microplastic ingestion is not likely to increase the exposure to and
131 f SGLT1 in the small intestine after glucose ingestion is promoted by glucose-dependent disinhibition
133 f glucose homeostasis in response to glucose ingestion is unclear.We sought to determine the metaboli
137 ations in DNA methylation 160 min after meal ingestion mainly reflect changes in the estimated leukoc
138 s biotin in blood due to supplemental biotin ingestion may affect biotin-streptavidin binding, leadin
140 kg bw(-1).day(-1)), followed by surface dust ingestion (median = 13 ng.kg bw(-1).day(-1), IQR = 28 ng
141 only practiced restrictions surrounding 6-MP ingestion might not influence outcome but may hinder adh
142 )CO2 production), fructose storage (fructose ingestion minus (13)C-fructose oxidation), fructose conv
143 carried into the human body by means of food ingestion, mostly via red meat, dairy products and fatty
148 samples were collected before and after the ingestion of 170 g pork (36 g protein and 3 g fat) to as
152 ial muscle protein synthesis rates after the ingestion of 25 g whey protein. kg(-1) . d(-1); n = 12)
154 muscle protein synthetic responses after the ingestion of a protein-dense food source across a range
156 ral contractions and oro-cecal transit after ingestion of a solid meal were investigated by MRI and (
163 evidence to address the hypothesis that the ingestion of baked hen's egg or cow's milk results in mo
164 ed severe neurologic disorder occurred after ingestion of BIA 10-2474 at the highest dose level used
168 Energy expenditure did not increase after ingestion of capsulated or uncapsulated mustard compared
170 ically susceptible subjects triggered by the ingestion of cereal gluten proteins for which the only t
172 etabolites over time was higher after the co-ingestion of cocoa flavanols and methylxanthines than af
174 t disease in low-income countries (following ingestion of contaminated food and/or water), S. sonnei
175 nfections occur via open wounds or following ingestion of contaminated seafood, most infamously oyste
179 e result of an exercise challenge test after ingestion of egg was negative, and no allergic symptoms
180 ic response to a greater extent than did the ingestion of egg whites (P= 0.04).We show that the inges
181 myofibrillar protein synthesis than did the ingestion of egg whites, despite being matched for prote
184 that C1q promotes macrophage survival during ingestion of excess cholesterol, as well as improves foa
187 els, either therapeutically or following the ingestion of food, could alter the excitability of neuro
188 em orchestrates how the body responds to the ingestion of foods, employing a diversity of hormones to
191 xtraintestinal symptoms, or both, related to ingestion of gluten-containing grains, with symptomatic
194 ommitted effective doses due to (210)Po from ingestion of honey for infants, children and adults acco
197 studies have indicated associations between ingestion of inorganic arsenic and ischemic heart diseas
198 rts have described anaphylaxis caused by the ingestion of jellyfish, but the allergens in jellyfish h
199 rlying nutrient-sensing mechanisms after the ingestion of lean pork between obese, overweight, and he
200 ne the uptake of inorganic resources and the ingestion of living prey, are ubiquitous in marine ecosy
201 xication on driving abilities, unintentional ingestion of marijuana products by children, the relatio
203 The isotope ratio results suggest direct ingestion of metallic Hg by C. pagurus but do not offer
208 ore, this study aimed to investigate whether ingestion of modified LDL in the presence of C1q alters
210 esearch knowledge on health effects posed by ingestion of mycotoxins-contaminated food and feed by hu
211 s disease has been proposed to develop after ingestion of neurotoxicants that affect the brain-gut ax
215 study explores two exposure routes of OHCs, ingestion of OHCs (i) via house dust and (ii) via cat fo
216 ing maintenance treatment, consisting of the ingestion of one chicken egg per day; no allergic sympto
218 fter an exercise challenge combined with the ingestion of only Citrus unshiu, an anaphylactic reactio
219 ate (beta-HB) in the apical medium following ingestion of OS by human fetal RPE and ARPE19 cells cult
224 ocedure (HECP), with and without concomitant ingestion of protein (n = 15) or an amount of leucine th
226 ofibrillar protein synthetic response to the ingestion of protein-dense food in overweight and obese
227 human exposure to xenobiotics occurs through ingestion of reclaimed wastewater-irrigated produce, pro
228 ment before, but not after, the delivery and ingestion of reward, indicating a selective effect on in
229 of glutamatergic activation of aNAcSh on the ingestion of rewarding stimuli as well as its effect in
237 ro and in the intracellular environment upon ingestion of the bacteria by macrophages; indicating tha
238 Correlation analysis of data obtained from ingestion of the caffeine solution revealed an associati
245 We observed a modest association between ingestion of water with higher THMs (>95th percentile vs
246 tercress should be avoided to ensure maximum ingestion of watercress-derived beneficial phytochemical
247 ial period was similar (P= 0.75) between the ingestion of whole eggs (68% +/- 1%) and egg whites (66%
248 ion of egg whites (P= 0.04).We show that the ingestion of whole eggs immediately after resistance exe
250 ons could be selected to cope with important ingestions of abrasive particles in the context of inten
251 s was studied before, the effects of ethanol ingestion on ependymal cilia function have not been inve
252 n (BTT), and as definitive therapy for toxic ingestion or idiopathic liver failure (DT) in a level 1
253 ine appearance rates increased after protein ingestion (P < 0.01) with no differences between treatme
254 iate analysis, late TBN (P = 0.017) and acid ingestion (P = 0.002) were predictors of mortality.
255 ecreased plasma glucose levels after glucose ingestion (P=0.02) with more symptoms of hypoglycemia (P
258 rice samples although it represents a vital ingestion portion for a real estimation of human health
261 VC bypasses bioavailability barriers of oral ingestion, provides pharmacological concentrations in ti
263 of several key feeding processes, including ingestion rate and assimilation efficiency, that affect
271 Results showed that Cu influx rates and ingestion rates decreased as Cu exposures of the algal m
273 tive was to estimate site-specific soil/dust ingestion rates through reevaluation of the lead absorpt
279 r intravenous amino acid infusion or protein ingestion, reduces insulin-stimulated glucose disposal.
280 as the amount of water retained at 2 h after ingestion relative to that observed after the ingestion
283 we estimated lifetime exposure to THMs from ingestion, showering/bathing, and hours of swimming pool
284 issue concentrations of POPs between plastic ingestion subgroups, fugacity calculations, and bioaccum
285 The close alignment of the circulatory and ingestion systems, as well as other morphological charac
287 Flies showed discrete, temporally precise ingestion that was regulated by hunger state and sucrose
289 t during the HECP without protein or leucine ingestion, the grand mean +/- SEM plasma 3-HIB concentra
290 ity within seconds, beginning prior to water ingestion, upon presentation of water-predicting cues.
292 thways and associated Pb intake rates; water ingestion was also a main pathway, especially for infant
293 sulin levels after both glucose and fructose ingestion was associated with increased hypothalamic, th
294 tration, is available to nectarivores during ingestion whereas post-ingestive information about resou
295 m baseline, peaking at 45 min after fructose ingestion, whereas breath hydrogen peaked later, at 75 m
300 e years of life, often after the first known ingestion with typical rapid onset IgE-mediated symptoms
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