ライフサイエンスコーパス: inhalation

1 entry into nasal cavity lymphatics following inhalation.

2 sa in >90% of animals from 5 to 60 min after inhalation.

3 lood and sputum mDCs and pDCs after allergen inhalation.

4 thmatic patients before and after salbutamol inhalation.

5 ent in >90% of animals 5 to 60 min following inhalation.

6 /- mice were subjected to lipopolysaccharide inhalation.

7 (-1)), floor dust ingestion and personal air inhalation.

8 ter the lumen of lymphatic vessels following inhalation.

9 tions of 75% accuracy in 70-90 ms after odor inhalation.

10 the treatment of respiratory indications via inhalation.

11 ailed role of CXCR7 in a murine model of LPS inhalation.

12 nchiolar epithelium at the early steps after inhalation.

13 athway that is comparable to or greater than inhalation.

14 responsiveness decreases 3 h after allergen inhalation.

15 timated dermal uptakes are small compared to inhalation.

16 pathogen that initiates infection following inhalation.

17 ucial in determining the outcome of allergen inhalation.

18 sols and those produced for drug delivery by inhalation.

19 sibility was determined after bronchodilator inhalation.

20 roduction and mucus secretion after allergen inhalation.

21 e prevalence, where exposure is typically by inhalation.

22 o greater than 90% with once-daily dosing by inhalation.

23 ponsible for respiratory disorders following inhalation.

24 Carbon dioxide (CO2) inhalation, a biological challenge and pathologic marker

25 Initiation of CO inhalation after SAH rescued the absence of microglial H

26 MA and MDPV vapor inhalation also altered activity on a running wheel in a

27 HDM inhalation also increased serum IL-4 and IL-17 levels an

28 nd approved treatment for poisoning by ricin inhalation, although there have been major improvements

29 and perfluorooctyl sulfonate (PFOS) through inhalation and biotransformation of PFAS precursors in a

30 e we construct a mathematical model of spore inhalation and clearance by concerted actions of macroph

31 tion of food, drinking water, dust/soil, air inhalation and dermal exposure the daily intake of PAEs

32 We assessed congener-specific inhalation and dietary exposure for 78 adolescent childr

33 several emerging flame retardants (EFRs) via inhalation and dust ingestion.

34 jected to chronic intermittent ethanol (CIE) inhalation and examined changes in local inhibitory cont

35 the changes in humidity caused by cycles of inhalation and exhalation to electrical signals.

36 association was found between diesel exhaust inhalation and flow-mediated dilation.

37 0A8 was administered to BALB/c mice by nasal inhalation and genes induced over a time-course assessed

38 early detection of prions in blood following inhalation and has implications for horizontal prion tra

39 on about the putative toxicity of AgNWs upon inhalation and incomplete understanding of the propertie

40 dwater is also a contributing factor to both inhalation and ingestion routes.

41 Carbogen inhalation and oral corticosteroids were also given.

42 led formaldehyde ([(13)CD2]-formaldehyde) by inhalation and performed ultrasensitive mass spectrometr

43 By inhalation and subsequent pulmonary infection, it may di

44 encountered by respiratory viruses following inhalation and the primary site of respiratory viral rep

45 MWCNTs (which could contact lung cells upon inhalation) and approximately 40 ppm as free-standing MW

46 late residential intake from dust ingestion, inhalation, and dermal uptake from air, and then identif

47 netic disorder typically triggered by potent inhalation anesthetics and/or the depolarizing muscle re

48 effects following a drug ingestion/injection/inhalation are mydriasis, miosis, and nystagmus.

49 luded the following: dry powder mannitol for inhalation as a bronchial provocation test is FDA approv

50 te of (99m)Tc-sestamibi from the lungs after inhalation as an aerosol has been hypothesized to be reg

51 are increasingly being used to manage smoke inhalation-associated acute lung injury.

52 o 800, because the air curtain effect during inhalation becomes strongest in this range.

53 The role of inhalation behaviors as predictors of nicotine uptake wa

54 ytoxin congeners exert toxic effects through inhalation both in natural environments and in the surro

55 When exposed to peanut flour by inhalation, both BALB/c and C57BL/6 mice developed peanu

56 d in particular to the effects of ozone (O3) inhalation, but the underlying mechanisms remain unclear

57 suppressed acute lung injury provoked by LPS inhalation by suppressing mast-cell activation and induc

58 It is considered a biological weapon, and inhalation can trigger lung injury and sometimes respira

59 investigations of the health impacts of PCB inhalation carefully consider certain aspects of study d

60 In addition, we found that CO2 inhalation caused BNST acidosis and that acidosis was su

61 Diacetyl inhalation caused dose-dependent increases in bronchial

62 e of Feno levels following positive specific inhalation challenge (SIC) was restricted to phenotypes

63 s with OA ascertained by a positive specific inhalation challenge (SIC) with NRL gloves and in 25 sym

64 diagnosing OA defined as a positive specific inhalation challenge (SIC).

65 the most accurate investigation is specific inhalation challenge in an experienced centre, this is a

66 Pharmacological blockade of CysLT2R prior to inhalation challenge of Ptges(-/-) mice with aspirin blo

67 sought (i) to examine the effect of allergen inhalation challenge on expression levels of receptors f

68 r exposure to LMW and HMW agents by specific inhalation challenge test (SIC).

69 on testing, methacholine challenge, specific inhalation challenge to argan powder, skin prick tests,

70 oline challenges at baseline of the specific inhalation challenge, at the workplace, and outside work

71 ween 1983 and 2011 and having had a specific inhalation challenge.

72 ith mild atopic asthma underwent rising dose inhalation challenges with allergen or methacholine to d

73 lergic asthma underwent diluent and allergen inhalation challenges.

74 mmation in response to repetitive Alternaria inhalation challenges.

75 lfur dioxide (SO2; 5.6 ppm), in a whole-body inhalation chamber.

76 gic airway inflammation following dry powder inhalation combined with a moderate systemic GR-effect,

77 previously demonstrated that carbon dioxide inhalation could induce panic anxiety in a group of rare

78 Thus, microplastic exposure via diet or inhalation could occur, the human health effects of whic

79 itis (EP) in the presence of cigarette smoke inhalation (CSI).

80 nd identified crystalline forms suitable for inhalation devices.

81 d neutrophilia, we demonstrated that Sul-121 inhalation dose-dependently prevented LPS-induced airway

82 Moreover, mean daily inhalation doses were decreased from 5.0 to 4.3 in BUD/F

83 efficiency for the carrier based dry powder inhalation (DPI).

84 mitral/tufted cell spiking in the absence of inhalation-driven sensory input and further increased sp

85 investigate the effects of short-term ozone inhalation during implantation on fetal growth outcomes

86 sis that cigarette smoking-relevant nicotine inhalation during pregnancy impairs cardiovascular funct

87 organophosphate flame retardants (PFRs) via inhalation, dust ingestion, and dermal absorption using

88 ive assessment of human exposure to PFRs via inhalation, dust ingestion, and dermal absorption was co

89 of DPI is an important factor for increasing inhalation efficiency.

90 s year were randomly assigned to receive, by inhalation, either the LABA indacaterol (110 mug) plus t

91 In addition to neutrophil influx, O3 inhalation elicited the appearance of eosinophils and IL

92 Direct mucosal exposure to HBCD during HDM inhalation enhanced IL-4 or IL-17A production, depending

93 However, the inhalation exposure assessment points to a minimum risk

94 air sample could generate a larger bias for inhalation exposure assessment.

95 However, PCB inhalation exposure assessments have been lacking for No

96 ll, and CYP2A13/2F1-humanized mice following inhalation exposure at an occupationally relevant dose (

97 perceived as critical for consumers because inhalation exposure can occur to potentially toxic nanop

98 oses (0.5-100 mug/mL) corresponding to human inhalation exposure durations at the consumer level of 8

99 mechanisms of adverse health effects through inhalation exposure is critical to mitigating its potent

100 Dermal and inhalation exposure levels were assessed by collecting h

101 ic analysis to show that some children's PCB inhalation exposure may be equal to or higher than their

102 In this study, we performed inhalation exposure of mice fed normal chow or a high-fa

103 Our studies demonstrated that inhalation exposure of mice fed normal chow to concentra

104 performed to estimate infectious risks after inhalation exposure of pathogens aerosolized during cent

105 als gained notoriety in the early 2000s when inhalation exposure of the flavoring chemical diacetyl w

106 Hepatotoxicity was observed in oral and inhalation exposure studies in several studies in animal

107 tudy show that after rats experienced a 6-hr inhalation exposure to 20- and 110-nm AgNP at a single p

108 sed genes and microRNAs following subchronic inhalation exposure to A. fumigatus.

109 Inhalation exposure to airborne respirable crystalline s

110 f gastrointestinal infection associated with inhalation exposure to airborne zoonotic pathogens emitt

111 racterizing in vitro pathway-based risks via inhalation exposure to ambient PAHs.

112 responses was evaluated following subchronic inhalation exposure to Aspergillus fumigatus conidia.

113 Inhalation exposure to indoor air pollutants and cigaret

114 In certain contexts, inhalation exposure to PCBs may contribute more to total

115 Further investigation revealed that inhalation exposure to PM2.5 induced hepatic autophagy i

116 igated the complex effects and mechanisms of inhalation exposure to PM2.5 on hepatic steatosis, a pre

117 The health risk of inhalation exposure to polychlorinated biphenyls (PCB) c

118 Inhalation exposure to the microbial and allergenic cont

119 indoor air and estimate the contribution of inhalation exposure to total PCB exposure for select age

120 rinated congeners (e.g., PCBs 40+41+71, 52), inhalation exposure was as high as one-third of the tota

121 Congener-specific PCB inhalation exposure was modeled using 293 measurements o

122 occurs from handling camping tents and that inhalation exposure will likely occur while inside a ten

123 er and lung) in male and female B6C3F1 mice (inhalation exposure) and at one site (liver) in male B6C

124 In relation to inhalation exposure, dust ingestion appears to be the ma

125 exhaled air concentrations resulting from an inhalation exposure.

126 larly involves respiratory disease following inhalation exposure.

127 treated textiles and measures of dermal and inhalation exposure.

128 , and account for the majority of children's inhalation exposure.

129 s as high as one-third of the total (dietary+inhalation) exposure.

130 This study evaluates infant and adult inhalation exposures and respiratory tract deposited dos

131 rticularly important in the workplace, where inhalation exposures can affect lung function and cause

132 Inhalation exposures were performed with rats to evaluat

133 We conducted contemporaneous PCB inhalation exposures with whole-body and nose-only expos

134 matching placebo three times a day via oral inhalation for 2 weeks, followed by a 2 week washout, an

135 ere switched to FP/FM-pMDI and slow and deep inhalation for 2-3 seconds was carried out thoroughly.

136 10, or 100 ppm; 75 mice/exposure group) via inhalation for 28 days (6 hr/day for 5 days/week).

137 when combined with postoperative 50% oxygen inhalation for 3 days, a number comparable to that requi

138 Local administration of therapeutics by inhalation for treatment of lung diseases has the abilit

139 , naive mice were exposed to peanut flour by inhalation for up to 4 weeks.

140 summation operatorPCB inhalation (geometric mean (SE)) was greater for urban m

141 rs of palytoxin, a highly potent toxin whose inhalation hazard is however unknown.

142 roperties and thus may pose an asbestos-like inhalation hazard.

143 Upon inhalation, house dust mite (HDM) allergens are deposite

144 e of lung cancer after cigarette smoking via inhalation; however, exposure through untreated groundwa

145 ndings challenge the safety of pure nicotine inhalation, i.e., E-cigarettes.

146 cally high levels in the United States, with inhalation (ie, smoking and vaping) being one of the mos

147 nt for occupational exposure to chemicals by inhalation in LCA.

148 isation of the lung following low dose spore inhalation in non-neutropenic hosts.

149 n vitro and in a model of lipopolysaccharide inhalation in vivo.

150 osure, plus airway Lipopolysaccharides (LPS) inhalation, in building our COPD mouse model.

151 Allergen inhalation increased basophil numbers in the airways and

152 Using an acid inhalation-induced lung injury model, we explored the me

153 n B. pseudomallei survival in vivo following inhalation infection and the antibacterial signaling net

154 he risks posed by Cd and the contribution of inhalation, ingestion, and dermal contact pathways to th

155 hing of BPA can result in human exposure via inhalation, ingestion, and dermal routes.

156 Inhalation injury (coefficient -0.622) played a variable

157 gender, total body surface area burned, and inhalation injury (hazard ratio, 1.73; 95% CI, 1.18-2.54

158 Total body surface area, age, and inhalation injury had significant effects on the subdist

159 57.8 +/- 18.2 for children) and presence of inhalation injury in 38% of the adults and 54.8% of the

160 Smoke inhalation injury is a serious medical problem that incr

161 tient age, total body surface area burn, and inhalation injury on the probability of discharge and de

162 Age, total body surface area burned, and inhalation injury were also significantly associated wit

163 nderstanding of the pathophysiology of smoke inhalation injury, the best evidence-based treatments, a

164 ore TBSA burn similar in age, TBSA burn, and inhalation injury.

165 cal studies of patients with burns and smoke inhalation injury.

166 s 4.0 mug/(mug/m(3) in air) compared with an inhalation intake of 3.8 mug/(mug/m(3) in air).

167 s 3.1 mug/(mug/m(3) in air) compared with an inhalation intake of 3.9 mug/(mug/m(3) in air).

168 surviving in the external environment before inhalation into a new potential host-steps influenced by

169 In atopic asthmatics, allergen inhalation is associated with an influx of inflammatory

170 Maternal smoking with obligatory nicotine inhalation is associated with preterm delivery, low birt

171 the mass flux of water to the droplet during inhalation is crucial.

172 location, and health-related impacts of AgNP inhalation is limited.

173 fficacy and safety of liposomal amikacin for inhalation (LAI) in treatment-refractory pulmonary nontu

174 Silica inhalation leads to the development of the chronic lung

175 uorescence with only a slight enhancement of inhalation-linked responses to odorant.

176 and standard surgical defect creation under inhalation/local anesthesia.

177 Carbon dioxide (CO2) inhalation lowers brain pH and induces anxiety, fear, an

178 onic cigarette-type technology to facilitate inhalation, male Wistar rats were exposed to vaporized m

179 HDM inhalation markedly increased airway goblet cell numbers

180 se materials that are traditionally used for inhalation may contain biologic activities to block TRPC

181 ome indoor settings and for some age groups, inhalation may contribute more to total PCB exposure tha

182 igmaPFRs was the highest with stationary air inhalation (median =34 ng.kg bw(-1).day(-1), IQR = 38 ng

183 imple control measures to reduce particulate inhalation might reduce the incidence of invasive bacter

184 following pulmonary infection in the murine inhalation model of cryptococcosis but still causes brai

185 Here we describe a mouse inhalation model of P. aeruginosa chronic infection that

186 This clinically relevant nicotine aerosol inhalation (NAI) induced transient reduction and irregul

187 Respiration-gated images were acquired after inhalation of (68)Ga-carbon nanoparticles and administra

188 omized to receive pretreatment with a single inhalation of 100 mug salmeterol 30 min before bronchial

189 C57bl/6N mice were exposed for 7 days by inhalation of 25 ppm neat amine or equivalant concentrat

190 ts with TRD were randomly assigned to 1-hour inhalation of 50% nitrous oxide/50% oxygen or 50% nitrog

191 amine and steroid administration, as well as inhalation of a beta 2 stimulant.

192 sets of baseline images were acquired before inhalation of a bronchodilating agent (salbutamol 400 mu

193 were mild in severity and resolved soon with inhalation of a bronchodilator.

194 ath-hold (19)F gas wash-in MR imaging during inhalation of a normoxic fluorinated gas mixture (perflu

195 ich the subject's head was scanned following inhalation of a single puff of smoke from a cigarette co

196 onic tularemia in humans typically occurs by inhalation of aerosolized bacteria, the rat model has re

197 Inhalation of aerosolized C20 resulted in rapid transloc

198 We show that inhalation of aerosolized H5N1 influenza virus in cynomo

199 These data establish inhalation of aerosolized virus as a critical source of

200 In allergic asthma, inhalation of airborne allergens such as the house dust

201 gevity of asthma-like responses triggered by inhalation of allergen together with environmentally rel

202 ease in the magnitude of responses linked to inhalation of ambient air, as well as modest increases i

203 Moreover, inhalation of an anticholinergic bronchodilator reduced

204 llergic immune responses were induced by the inhalation of antigen during IL-1alpha release and iBALT

205 inhaled antigen alone but were augmented by inhalation of antigen together with HDE.

206 Inhalational anthrax is caused by inhalation of Bacillus anthracis spores.

207 However, whether the inhalation of bacteria triggers ASL secretion and whethe

208 Whether the inhalation of bacteria triggers ASL secretion, and the r

209 In patients with CF, the inhalation of bacteria would fail to trigger ASL secreti

210 Inhalation of benzene at levels below the current exposu

211 h2 cells, termed BT-II, followed by repeated inhalation of Blo t 5 expands these cells in the lung >1

212 In contrast, inhalation of C110 resulted in a gradual but progressive

213 Inhalation of capsaicin, the extract of hot chili pepper

214 Chronic inhalation of cigarette smoke is the major cause of ster

215 increased per 10 min of exposure during the inhalation of coarse CAP when compared with changes duri

216 Inhalation of coarse PM from a rural location is associa

217 Disease typically occurs following inhalation of contaminated aerosols, resulting in an ini

218 eptor-mediated ASL secretory response to the inhalation of cystic fibrosis relevant bacteria.

219 unotoxicological outcomes following repeated inhalation of dry Aspergillus fumigatus spores aerosoliz

220 Inhalation of elevated levels of particulate air polluti

221 Epidemiological studies associate inhalation of fine-sized particulate matter (PM2.5) duri

222 Slow and deep inhalation of FP/FM-pMDI is effective in many asthmatic

223 cases and the effectiveness of slow and deep inhalation of FP/FM-pMDI.

224 Repeated inhalation of fungal aerosols resulted in significant pu

225 he health outcomes that result from repeated inhalation of fungal bioaerosols in contaminated environ

226 a health risk to susceptible users from the inhalation of garden hose aerosols.

227 entially hazardous to the airways because of inhalation of house dust.

228 Finally, inhalation of hydrogen gas in adolescence significantly

229 The results showed that repeated inhalation of hydrogen-oxygen mixed gas [67%:33% (V/V)]

230 ELISA analyses showed that inhalation of hydrogen-oxygen mixed gas blocked CMS-indu

231 ienced anaphylactic reactions just after the inhalation of Inavir (Laninamivir Octanoate Hydrate) to

232 Inhalation of infected brain homogenate results in trans

233 nd can transmit cryptococcosis to humans via inhalation of inoculated bird excreta.

234 se of toxic lung injury which occurred after inhalation of legal highs, the so-called "artificial has

235 whereas lung inflammation was assessed after inhalation of lipopolysaccharide.

236 Whether inhalation of metabolites of environmental bacteria cont

237 TB) is a significant human disease caused by inhalation of Mycobacterium tuberculosis.

238 t and further increased spiking responses to inhalation of odorless air and to odorants.

239 (*)OH production in lungs following inhalation of particulate matter (PM) can result from re

240 We show that inhalation of self-assembling virus-like nanoparticles f

241 neal injection of propofol and maintained by inhalation of sevoflurane for 1 hr.

242 Chronic inhalation of silica particles causes lung fibrosis and

243 ts experienced some adverse reactions during inhalation of SK-1211, all of which were mild in severit

244 ction of mice abrogated tolerance induced by inhalation of soluble ovalbumin, suppressing the normal

245 In mice inhalation of SplD without adjuvant induced lung inflamm

246 Inhalation of the bacterium Yersinia pestis results in p

247 g dynamic (19)F gas washout MR imaging after inhalation of the gas mixture was finished for a total o

248 Seven-day treatment with inhalation of the nonsteroidal glucocorticoid receptor a

249 The effect of vapor inhalation of the stimulants was found comparable to the

250 Inhalation of these contaminants significantly suppresse

251 utbreaks of respiratory tularemia, caused by inhalation of this bacterium, are poorly understood.

252 tion to a civilian or military population by inhalation of toxic bioaerosol.

253 Inhalation of traffic-associated atmospheric particulate

254 Inhalation of traffic-related particulate matter (e.g.,

255 re volatile, these observations suggest that inhalation of vapor-phase PCBs is an important route of

256 IE) exposure (postnatal days 28-42) by vapor inhalation on different aspects of executive functioning

257 his study was to assess the effect of silica inhalation on telomere length and the regulation of RTEL

258 ty (RH) of the aerosol prior to, and during, inhalation on the deposition pattern is explored, and fo

259 ith each study drug administered by means of inhalation once daily for 28 days.

260 can use information occurring <100 ms after inhalation onset to identify odors.

261 e dose of vaccine by means of either aerosol inhalation or a subcutaneous injection.

262 Dust inhalation or exposure to high temperatures promoted pro

263 Inhalation or ingestion of ricin may even lead to death.

264 Flow Volume after 240s of hypertonic saline inhalation; p = 4.81*10(-4)) and CD14 rs2569190 was asso

265 After a 2-week run-in period receiving one inhalation per day via single-dose dry-powder inhaler of

266 of an albuterol rescue inhaler (2.8 and 3.0 inhalations per week, respectively; P=0.69), unscheduled

267 hotopic tumors via i.v. injection or aerosol inhalation, respectively.

268 man health effects, but data specific to the inhalation route are scarce.

269 Inhalation seems to be the major exposure pathway for Si

270 Rapid inhalation should also be encouraged.

271 assess safety and efficacy of aztreonam for inhalation solution (AZLI) in patients with non-cystic f

272 In this study, we showed in mice that after inhalation, Staphylococcus aureus enterotoxin A rapidly

273 Inhalation studies in mice showed that this formulation

274 products" (SFP) that are representative for inhalation studies.

275 high confidence due to the lack of rigorous inhalation studies.

276 varium, thus was preferred for long-term PCB inhalation studies.

277 In a macaque model of inhalation TB, suppression of IDO activity reduced bacte

278 Inhalation technique was assessed in a random sample of

279 sthma, adherence to treatment, and a correct inhalation technique were qualified as having severe ref

280 osure, nose-only CS exposure, and airway LPS inhalation testing.

281 nical model of opioid self-administration by inhalation that does not require surgery and reliably pr

282 n the third day following an acute exposure (inhalation) that would lead to an effective dose of 0.1

283 At any point in time during inhalation the depositional flux of the aerosol is a fun

284 The results show that, during inhalation, the anatomical structure of the oropharynx c

285 ations are incompletely prevented by maximal inhalation therapy in patients with severe chronic obstr

286 k and 19-month-old male rats were exposed by inhalation to 10 mg/m(3) of TiO2 nano-aerosol (6 hrs/day

287 ive B6C3F1/N mice were exposed via nose-only inhalation to A. fumigatus viable conidia, heat-inactiva

288 Thus, we show oxygen inhalation to be a simple and promising approach to succ

289 Coupling CO2 inhalation to retrieval increased activation of amygdala

290 VOC in vitro can be a valuable complement to inhalation toxicological evaluations.

291 e burn and 48 breaths of cooled cotton smoke inhalation under deep anesthesia and analgesia.

292 me compounds, the modeled ratio of dermal-to-inhalation uptake is large.

293 have the largest modeled ratios of dermal-to-inhalation uptake; for such compounds, the estimates rep

294 The calculated exposure due to air inhalation was substantially lower when the stationary a

295 s for whom direct and indirect exposures via inhalation were characterized using multiple measurement

296 N-gamma, as well as the effect of ozone (O3) inhalation, were studied on recirculation of pulmonary d

297 Diesel exhaust inhalation, which is the model traffic-related air pollu

298 volume resuscitation, and salbutamol sulfate inhalation, which resulted in an improvement of the symp

299 taxel offers a potential delivery system for inhalation with improved anti-cancer efficacy, prolonged

300 iratory Flow after 240s of hypertonic saline inhalation with respect to the age- and ancestry-matched