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1 nfective dose of 10(4)B. anthracis cells for inhalation anthrax.
2 ely devoid of virulence, in a mouse model of inhalation anthrax.
3 ternative clinical treatment regimen against inhalation anthrax.
4 the incubation period distribution for human inhalation anthrax.
5 that accurately predict the effects of human inhalation anthrax.
6  effective emergency therapy of postexposure inhalation anthrax.
7 racis may be critical in the early stages of inhalation anthrax.
8 AD is highly attenuated in a mouse model for inhalation anthrax.
9                                              Inhalation anthrax, an often fatal infection, is initiat
10 lete solution for population protection from inhalation anthrax and has been associated with concerns
11  the onset of symptoms in all 6 survivors of inhalation anthrax and in 7 of 11 survivors of cutaneous
12 ients with bioterrorism-related cutaneous or inhalation anthrax and in a patient with laboratory-acqu
13                  Since the initial events in inhalation anthrax are believed to be uptake of B. anthr
14  the protective activity of gammaDPGA Abs in inhalation anthrax are known.
15                    During advanced stages of inhalation anthrax, Bacillus anthracis accumulates at hi
16 F detection as a tool for early diagnosis of inhalation anthrax before the onset of fulminant systemi
17  cells were detectable in all 6 survivors of inhalation anthrax but in only 2 of 7 patients with cuta
18 hylococcus aureus hematogenous pneumonia and inhalation anthrax but no activity against Streptococcus
19              We show that protection against inhalation anthrax by an irradiated spore vaccine depend
20 toxin may play a role in the pathogenesis of inhalation anthrax by facilitating the dissemination of
21  Systemic sepsis, most often associated with inhalation anthrax, can cause massive ascites, electroly
22 f-life confer significant protection against inhalation anthrax despite their lack of Fc regions.
23 otic that is used in combination therapy for inhalation anthrax disease.
24  LF, PA, and PGA levels during the course of inhalation anthrax in five rhesus macaques.
25  To better understand the pathogenesis of an inhalation anthrax infection, we propose a two-compartme
26 ategy for prophylaxis or treatment following inhalation anthrax infection.
27                                              Inhalation anthrax is a deadly disease for which there i
28        Successful postexposure treatment for inhalation anthrax is thought to include neutralization
29 xin genes and is capable of causing a severe inhalation anthrax-like illness.
30  conferred complete protection in the rabbit inhalation anthrax model.
31 us in the two most accepted animal models of inhalation anthrax, nonhuman primates and rabbits, but r
32                                Prevention of inhalation anthrax requires early and extended antibioti
33                                              Inhalation anthrax results in high-grade bacteremia and
34  the site of entry for Bacillus anthracis in inhalation anthrax, the deadliest form of the disease.
35  spore-laced letters resulted in 22 cases of inhalation anthrax, with five fatalities.
36 emination and lethality in a murine model of inhalation anthrax, yet the roles for the toxins during

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