ライフサイエンスコーパス: inhalation injury

1 ore TBSA burn similar in age, TBSA burn, and inhalation injury.

2 episodes correlate with injury severity and inhalation injury.

3 cal studies of patients with burns and smoke inhalation injury.

4 - and anti-inflammation early after burn and inhalation injury.

5 sfunction in sheep subjected to severe smoke inhalation injury.

6 ion in an established, large animal model of inhalation injury.

7 injury secondary to combined burn and smoke inhalation injury.

8 acious therapy for burn patients with severe inhalation injury.

9 ry, acute respiratory distress syndrome, and inhalation injury.

10 lmonary pathology in burn victims with smoke inhalation injury.

11 tion of burn patients with concomitant smoke inhalation injury.

12 lmonary pathology in burn victims with smoke inhalation injury.

13 ding to TBSA burn and presence or absence of inhalation injury.

14 nstriction following combined burn and smoke inhalation injury.

15 ARDS resulting from combined burn and smoke inhalation injury.

16 in sheep with severe combined burn and smoke inhalation injury.

17 in previous studies to reduce the degree of inhalation injury.

18 eaths administered (24, 36, 48) for a graded inhalation injury.

19 hat neutrophils play a pivotal role in smoke inhalation injury.

20 40 percent of body-surface area burned, and inhalation injury.

21 at are used commonly are age, burn size, and inhalation injury.

22 excision, wound healing, scar formation, and inhalation injury.

23 = nonsignificant), and clinical diagnosis of inhalation injury (39% vs. 35%, p = nonsignificant).

24 Following combined burn and smoke inhalation injury (40% of total body surface area, third

25 urn size, incidence in sepsis (20% vs. 26%), inhalation injury (46% vs. 27%), or mortality (8% vs. 7%

26 - 11, P < 0.0001), and a higher incidence of inhalation injury (8% vs. 13%, P = 0.024).

27 rface area third-degree flame burn and smoke inhalation injury after tracheostomy.

28 Patients with the most severe inhalation injuries also had a greater requirement for t

29 would mitigate acute lung injury after smoke inhalation injury and burn.

30 ctive and lumenal obstructive response after inhalation injury and identifies low-dose nebulization o

31 s been placed on the early identification of inhalation injury and its impact on fluid resuscitation,

32 ry response is enhanced with worse grades of inhalation injury and that those who die of injuries hav

33 his association is independent of burn size, inhalation injury, and age.

34 concentrate on the pathophysiology of burns, inhalation injury, and edema formation.

35 s, nutritional support, incidence of sepsis, inhalation injury, and mortality were noted.

36 s' demographics, extent of burn, presence of inhalation injury, and mortality.

37 BSA) burned, age > or =40 years, presence of inhalation injury, and ventilator days were found to be

38 fering from a 60% TBSA burn with concomitant inhalation injury are more likely to develop sepsis if t

39 underlying lung inflammation in toxic smoke inhalation injury are unknown.

40 were then compared to the graded severity of inhalation injury as determined by Abbreviated Injury Sc

41 in sheep with severe combined burn and smoke inhalation injury by preventing the formation of airway

42 Inhalation injury (coefficient -0.622) played a variable

43 controlling for age and % TBSA, presence of inhalation injury continues to be significant.

44 Inhalation injury continues to be the most significant c

45 Inhalation injury contributes to the morbidity and morta

46 l, regardless of age, burn size, presence of inhalation injury, delay in resuscitation, or laboratory

47 Lower age, larger burn size, presence of inhalation injury, delayed intravenous access, lower adm

48 e aimed to determine whether the severity of inhalation injury evokes an immune response measurable a

49 ep in the sepsis group received cotton smoke inhalation injury followed by instillation of Pseudomona

50 Combined burn and smoke inhalation injury frequently results in acute lung injur

51 Mortality was associated with TBS, FTBS, inhalation injury, FWI, and age.

52 sma IL-1RA also correlated with % TBSA burn, inhalation injury grade, fluid resuscitation, Baux score

53 ing for the effects of age, % TBSA burn, and inhalation injury grade, plasma IL-1RA remained signific

54 When compared in terms of low inhalation injury (grades 1-2) vs. high inhalation injur

55 low inhalation injury (grades 1-2) vs. high inhalation injury (grades 3-4), we found significant dif

56 Those who presented with worse grades of inhalation injury had higher plasma levels of carboxyhem

57 Total body surface area, age, and inhalation injury had significant effects on the subdist

58 These data reveal that the degree of inhalation injury has basic and profound effects on burn

59 ventilation in the management of early smoke inhalation injury has not been studied.

60 The severity of smoke inhalation injury has systemically reaching effects, whi

61 found that patients with >60% TBSA burn with inhalation injury have an 8% risk of developing sepsis i

62 hstanding significant % TBSA and presence of inhalation injury, have significantly declined compared

63 gender, total body surface area burned, and inhalation injury (hazard ratio, 1.73; 95% CI, 1.18-2.54

64 57.8 +/- 18.2 for children) and presence of inhalation injury in 38% of the adults and 54.8% of the

65 ng effects, which argue in favor of treating inhalation injury in a graded manner.

66 the neonatal and pediatric population, treat inhalation injury in pediatric and adult patients, and a

67 Smoke inhalation injury is a serious medical problem that incr

68 ecreased fluid flux in a combined burn/smoke inhalation injury model.

69 e patient demographics, incidence of sepsis, inhalation injury, mortality, serum constitutive protein

70 o patient age, total body surface area burn, inhalation injury, number of units of blood transfused o

71 One patient died of an inhalation injury on day 7.

72 tient age, total body surface area burn, and inhalation injury on the probability of discharge and de

73 teristics of age, burn size, and presence of inhalation injury, outcome was correctly predicted in on

74 We recorded age, burn size, inhalation injury, resuscitation, packed-cell volume at

75 ere at their highest in those with the worst inhalation injury scores (grades 3 and 4), the greatest

76 a immune mediators were increased with worse inhalation injury severity, even after adjusting for age

77 e (FTBS, percentage body surface area), age, inhalation injury, sex, and fungal-status category.

78 Patients with severe burn and/or smoke inhalation injury suffer both systemic and pulmonary vas

79 Twenty-four hours after smoke inhalation injury, the animals were reanesthetized and s

80 nderstanding of the pathophysiology of smoke inhalation injury, the best evidence-based treatments, a

81 ose with limited donor sites, those who have inhalation injury, those with delays in resuscitation, a

82 ingly, future investigations should consider inhalation injury to be a graded phenomenon.

83 than 55 years with severe burn injuries and inhalation injury to survive these devastating condition

84 ace area involvement of 54% +/- 4%, 63% with inhalation injury) underwent tracheostomy a mean of 3.9

85 catheters and underwent an LD50 cotton smoke inhalation injury via a tracheostomy under halothane ane

86 Of these, inhalation injury was graded into 1 of 5 categories (0,

87 Inhalation injury was induced by 48 breaths of cotton sm

88 Age, total body surface area burned, and inhalation injury were also significantly associated wit