ライフサイエンスコーパス: inhaled anesthetic

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1 lly relevant targets for clinical actions of inhaled anesthetics.

2 ted by clinically relevant concentrations of inhaled anesthetics.

3 els, which helps explain how closely related inhaled anesthetics achieve specific actions and suggest

4 ons may represent a fundamental mechanism of inhaled anesthetic action.

5 ecular volume is an important determinant of inhaled anesthetic activity at the GABA(A) receptor.

6 ments, T1 measurements obtained with several inhaled anesthetic agents and propofol dissolved in sali

7 All exposed children received inhaled anesthetic agents, and anesthesia duration range

8 Ligand-gated ion channels are a target for inhaled anesthetics and alcohols in the central nervous

9 specific or nonspecific interactions between inhaled anesthetics and proteins are more likely to unde

10 ther the weak molecular interactions between inhaled anesthetics and proteins, we studied the charact

11 inergic neurons, immobilizing actions of the inhaled anesthetics and sedative effects of halothane we

12 The inhaled anesthetics are low affinity volatile compounds

13 Physiologic sites for inhaled anesthetics are presumed to be cavities within t

14 Inhaled anesthetics are thought to alter the conformatio

15 chanism(s) and site(s) of action of volatile inhaled anesthetics are unknown in spite of the clinical

16 tance of aromatic residues as one feature of inhaled anesthetic binding sites.

17 Halothane, an inhaled anesthetic, destabilizes the folded structure of

18 e molecular substrates for select actions of inhaled anesthetics; for immobilization, which is spinal

19 ein the location of the binding site for the inhaled anesthetic halothane at the amino acid residue l

20 The inhaled anesthetics halothane and isoflurane potentiate

21 nstrictors during concomitant exposure to an inhaled anesthetic (halothane); and b) to determine if e

22 y was used to define the kinetics with which inhaled anesthetics interact with (Aalpha(2)-L1M/L38M)(2

23 Similarly, the inhaled anesthetic isoflurane triggered a persistent inc

24 linically relevant concentrations of another inhaled anesthetic, isoflurane.

25 Halogenated inhaled anesthetics modulate voltage-gated ion channels

26 l membrane proteins are potential targets of inhaled anesthetics, most currently favored membrane pro

27 Molecular targets of inhaled anesthetics must be represented in the group tha

28 ) M(-1), higher than any previously reported inhaled anesthetic-protein interaction.

29 led novel activation of K(v) channels by the inhaled anesthetic sevoflurane.

30 Previous work suggested that n-alcohols and inhaled anesthetics stabilize the closed state of the Sh

31 Alkanols and halogenated inhaled anesthetics such as halothane and isoflurane inh

32 onstrate specific and discrete binding of an inhaled anesthetic to a mammalian-soluble protein, and f

33 is thought to involve the direct binding of inhaled anesthetics to ligand-gated ion channels.

34 Saturable binding of various inhaled anesthetics to serum albumin has been shown with

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