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1 Decreasing TIA1 also inhibited the accumulation of tau oligomers at the expense of

2 Triggering of ILT3 inhibited the activation of Akt kinase upon B-cell receptor (

3 The presence of Rev1 inhibited the activity of Polzeta and greatly increased the r

4 in neurons both in vivo and in vitro Increased Trib3 levels inhibited the activity of the kinase Akt by interacting with

5 al and biochemical analyses revealed that UbVs specifically inhibited the activity of UBE4B or phosphorylated CBL by bloc

6 In vitro, crocin inhibited the assembly of pure tubulin as well as the assembl

7 Vinblastine inhibited the binding of crocin to tubulin while podophylloto

8 -molecular-weight Delta123 was poly specific, given that it inhibited the binding of human bNAbs directed to three major

9 anscription factor, specificity protein 1 (Sp1), which then inhibited the binding of Sp1 to the VEGF promoter to reduce V

10 Interestingly, macrophage depletion with clodronate inhibited the development of colitis, while the absence of IL

11 Akt2 deficiency profoundly inhibited the development of liver lesions in mice, whereas A

12 Ectopic expression of miR-K6-5p specifically inhibited the expression of endogenous CD82 and strongly prom

13 cumulation and tolerogenic activity of MDSCs in tumors, and inhibited the expression of immunosuppressive factors arginas

14 The cFLIP long isoform (cFLIPL) inhibited the expression of IRF7-controlled natural or synthe

15 Silencing PV+ interneurons of the NAc selectively inhibited the expression of locomotor sensitization following

16 Fgf8, a downstream secreted factor of Tbx1, inhibited the expression of Sema3c in cNCCs via activation of

17 Mechanistically, we found that increased HOTAIR expression inhibited the expression of the TRAIL receptor death receptor

18 PDGFRalpha inhibited the formation of adipocytes from these precursors w

19 In htel-25, the G/APs inhibited the formation of parallel strands and these adopted

20 ost cell death in the context of L. monocytogenes infection inhibited the generation of protective immunity and specifica

21 17f inhibited the growth of acute and chronic myeloid leukemia ce

22 When administered orally, mastic oil inhibited the growth of colon carcinoma tumors in mice.

23 6OTD inhibited the growth of GSCs more potently than it did the gr

24 5.0 and 7.5 muM) for 24 and 48 h significantly (P < 0.001) inhibited the growth of melanoma cells but not normal melanoc

25 Intraperitoneal administration of S17 significantly inhibited the growth of MGC803 cells in vivo in a xenograft m

26 l inhibition of CK1alpha and the proteasome synergistically inhibited the growth of multiple RAS-mutant human cancer cell

27 In contrast, overexpression of COY1 inhibited the growth of mutant strains deficient in fusion ac

28 , the most potent antimicrobial of this suite of compounds, inhibited the growth of Mycobacterium smegmatis with an MIC80

29 Consistently, KDM3A depletion inhibited the growth of subcutaneously implanted cisplatin-re

30 a chronically stimulated Th1 phenotype, lack of IL-4Ralpha inhibited the induction of IL-10.

31 tment of HNF-1beta mutant mIMCD3 cells with hypertonic NaCl inhibited the induction of osmoregulated genes, including Nr1

32 on by NO2-OA in TNBC cells were multifaceted, as NO2-OA (a) inhibited the inhibitor of NF-kappaB subunit kinase beta phos

33 In contrast to 1B6, 1F11 inhibited the interaction of CXCL10 with GAGs, did not recogn

34 oting beta-catenin phosphorylation and degradation, it also inhibited the phosphorylation of p38 mitogen-activated protei

35 show in murine HSCs that the telomere binding protein POT1a inhibited the production of reactive oxygen species, and reju

36 We also inhibited the production of the proinflammatory secretome of

37 ed in the loss of antifungal activity by tissue Mvarphi and inhibited the production of tumor necrosis factor alpha (TNF-

38 ibitor 4mu8C could suppress the production of beta-catenin, inhibited the proliferation of colon cancer cells, repressed

39 -signaling, enhanced insulin-stimulated glucose uptake, and inhibited the proliferation of DLD-1 colorectal adenocarcinom

40 n the adaptive immune system, as serum from stroke patients inhibited the proliferation of healthy donor-derived lymphocy

41 of one allele of Dkk1 in Osx-expressing cells in adult mice inhibited the recovery of BM stem and progenitor cells and of

42 SIRT1 loss altered the production of many cytokines, inhibited the recruitment of macrophages, neutrophils, and ma

43 SIRT1 deficiency in the epidermis inhibited the regeneration of both the epidermis and the derm

44 Finally, siRNA knockdown of LRRC8C+LRRC8D strongly inhibited the release of all osmolytes.

45 In addition, the suppression of TLR1 inhibited the release of IL-33.

46 Fremanezumab pretreatment selectively inhibited the responsiveness of Adelta neurons, but not C-fib

47 Moreover, MSA effectively inhibited the sprouts of mouse aortic rings and neoangiogenes

48 E/K465E) knockin mice, we found that decreased Akt activity inhibited the survival of T cells during the effector-to-memo

49 Additionally, we showed that LJ001 inhibited the transmission of IHNV from infected fish to heal

50 When these neurons are optogenetically inhibited, the activity of these neuroendocrine axes are supp