ライフサイエンスコーパス: inhibitor of apoptosis protein

1 iapoptotic factors Bcl-xL and XIAP (X-linked Inhibitor of Apoptosis Protein).

2 lovirus IAP repeat) domain of XIAP (X-linked inhibitor of apoptosis protein).

3 We denote K7 as vIAP (viral inhibitor-of-apoptosis protein).

4 Dark adaptors and negative control via IAPs (inhibitor of apoptosis proteins).

5 nhibitors of apoptosis proteins and X-linked inhibitor of apoptosis protein.

6 ession, and induction of X chromosome-linked inhibitor of apoptosis protein.

7 or anti-apoptotic factors, such as x-linked inhibitor of apoptosis protein.

8 y, including the Bcl-2 homologue protein and inhibitor of apoptosis protein.

9 tivation by proteasome activity and X-linked inhibitor of apoptosis protein.

10 of PDA chemotherapy resistance, the X-linked inhibitor of apoptosis protein.

11 Smac mimetics and regulation of apoptosis by inhibitor of apoptosis proteins.

12 dependent apoptosis by binding and degrading inhibitor of apoptosis proteins.

13 dent apoptosis in the cytoplasm by degrading inhibitor of apoptosis proteins.

14 ase activity in pupal retina through hid and Inhibitor of Apoptosis Proteins.

15 tiapoptotic Bcl-2 family member proteins and inhibitors of apoptosis proteins.

16 ate the expression of bcl-2, bcl-xL, bax, or inhibitors of apoptosis proteins.

17 ates inhibitor of apoptosis protein cellular-inhibitor of apoptosis protein 1 (c-IAP-1).

18 ve detected an up-regulation of the cellular inhibitor of apoptosis protein 1 (c-IAP1).

19 with millimolar affinities to both cellular inhibitor of apoptosis protein 1 (cIAP1) and X-linked in

20 Cellular inhibitor of apoptosis protein 1 (cIAP1) is a cellular E

21 we show that LAR-RPTPs act through cellular inhibitor of apoptosis protein 1 (cIAP1) to modulate cas

22 ceptor FN14, the adaptor TRAF2, and cellular inhibitor of apoptosis protein 1 (cIAP1).

23 ered the proteasomal degradation of cellular inhibitor of apoptosis protein 1 and 2 (c-IAP1 and c-IAP

24 odel, does not rely on depletion of cellular inhibitor of apoptosis protein 1 and 2 (cIAP1/2).

25 lator of TNFalpha induction whereas cellular inhibitor of apoptosis protein 1 negatively regulates TN

26 a protein referred to as DIAP1 (D rosophila Inhibitor of Apoptosis Protein 1) that acts to supress a

27 d Akt B-cell lymphoma-xL, survivin, cellular inhibitor of apoptosis protein 1, and cellular inhibitor

28 , including those linked with cell survival (inhibitor of apoptosis protein 1, Bcl-2, Bcl-xL, survivi

29 ll survival (Bcl-2, Bcl-x(L), cellular FLIP, inhibitor of apoptosis protein 1, inhibitor of apoptosis

30 y correlated with downregulation of cellular inhibitor of apoptosis protein 1, suggesting suppressive

31 Cellular inhibitor of apoptosis proteins 1 and 2 (c-IAP1/2) have

32 ubiquitin-conjugating enzyme Ubc13, cellular inhibitor of apoptosis proteins 1 and 2 (c-IAP1/2), Ikap

33 ibitor-of-apoptosis protein (XIAP), cellular inhibitor-of-apoptosis protein 1 (cIAP-1), cIAP-2, and s

34 cIAPs (cellular inhibitor of apoptosis proteins) 1 and 2 are able to reg

35 We report herein a critical role of cellular inhibitor of apoptosis protein-1 (cIAP1) in controlling

36 ding the anti-apoptotic Bcl-2 homolog A1 and inhibitor of apoptosis protein-1 (IAP-1) by APC in umbil

37 clin D1 and c-Myc), antiapoptotic (survivin, inhibitor of apoptosis protein-1 (IAP1), IAP2, X-chromos

38 eases in the antiapoptotic proteins cellular inhibitor of apoptosis protein-1 and X-linked inhibitor

39 the expression of osteoprotegerin, BCL-2, or inhibitor of apoptosis protein-1 by RNA interference sho

40 acting protein), TRAF1, and cIAP-1 (cellular inhibitor of apoptosis protein-1), forming a survival si

41 included osteoprotegerin, BCL-2, and IAP-1 (inhibitor of apoptosis protein-1).

42 some-linked inhibitor of apoptosis, cellular inhibitor of apoptosis protein-1, and survivin were sign

43 s (cyclin D1, c-myc, Bcl-2, Bcl-xL, cellular inhibitor of apoptosis protein-1, cyclooxygenase-2, matr

44 atrix metalloproteinase-9 (MMP-9), survivin, inhibitor-of-apoptosis protein-1 (IAP1), IAP2, X chromos

45 spase inhibitory system mediated by cellular inhibitor of apoptosis protein 2 (c-IAP2).

46 The E3 ligase cellular inhibitor of apoptosis protein 2 (cIAP2) was upregulated

47 Here, we show that Drosophila melanogaster inhibitor of apoptosis protein 2 (DIAP2) controls the le

48 linked to impaired upregulation of cellular inhibitor of apoptosis protein 2 and an excessive produc

49 uloviral IAP repeat (BIR) domains of c-IAP2 (inhibitor of apoptosis protein 2) fused with portions of

50 of the inhibitor of apoptosis gene, cellular inhibitor of apoptosis protein 2, (c-IAP2), is significa

51 ular FLIP, inhibitor of apoptosis protein 1, inhibitor of apoptosis protein 2, and survivin), invasio

52 hibitor of apoptosis protein 1, and cellular inhibitor of apoptosis protein 2.

53 s in colon cancer cells and induces cellular inhibitor of apoptosis protein-2 (c-IAP2) via a cAMP-res

54 (BIM), which both lowered FLIP and cellular inhibitor of apoptosis protein-2 (cIAP-2) protein levels

55 Moreover, PR39 increased inhibitor of apoptosis protein-2 (IAP-2) gene and protei

56 Our recent study showed hypoxic induction of inhibitor of apoptosis protein-2 (IAP-2).

57 B activity as well as expression of cellular inhibitor of apoptosis protein-2 and FLICE inhibitory pr

58 with the down-regulation of bcl-2, cellular inhibitor of apoptosis protein-2, cyclin D1, cyclooxygen

59 cation mechanism involving interactions with inhibitor of apoptosis proteins, activation of caspase-3

60 by facilitating association between X-linked inhibitor of apoptosis protein, an inhibitor of caspase

61 of virus-mediated depletion of the cellular inhibitor-of-apoptosis protein, an event that initiates

62 e, including p53, Apaf-1, and members of the inhibitor of apoptosis protein and Bcl-2 gene families,

63 proteolytic activity of Omi/HtrA2 toward the inhibitor of apoptosis proteins and beta-casein and indu

64 e survival following toxin challenge include inhibitors of apoptosis proteins and X-linked inhibitor

65 ression of epidermal growth factor receptor, inhibitor of apoptosis protein, and survivin pathways, c

66 IAP-2, Bcl-2, Bcl-xL, survivin, and X-linked inhibitor of apoptosis protein, and up-regulated the pro

67 olypeptides, including Bcl-2 family members, inhibitor of apoptosis proteins, and several protein kin

68 spase function: serpins, p35 family members, inhibitor of apoptosis proteins, and viral FLICE-inhibit

69 mimetics to their target, the XIAP (X-linked inhibitor of apoptosis) protein, and their binding affin

70 B-PAC-1 in combination with an inhibitor of apoptosis protein antagonist (Smac066) syne

71 ase-dependent manner by interacting with the inhibitor of apoptosis protein as well as in a caspase-i

72 by interacting with the X-chromosome linked inhibitor of apoptosis protein, as well as in a caspase-

73 suppressor of caspase-8, but not by X-linked inhibitor of apoptosis protein-baculovirus IAP repeat-3

74 hondria-derived activator of caspases/direct inhibitor of apoptosis protein-binding protein with low

75 l-derived activator of caspase (SMAC)/direct inhibitor of apoptosis protein-binding protein with low

76 The inhibitor of apoptosis protein BIRC5 (survivin), a known

77 as been implicated in the degradation of the inhibitor-of-apoptosis protein BRUCE.

78 h cAMP-mediated, rapid induction of cellular inhibitor of apoptosis protein (c-IAP)-2 and delayed ind

79 le survival factor genes, including cellular inhibitor of apoptosis protein (c-IAP1), c-IAP2, TNF rec

80 dance of the apoptotic inhibitor, a cellular inhibitor of apoptosis proteins (c-IAP-1), was markedly

81 inhibits caspase activation and up-regulates inhibitor of apoptosis protein cellular-inhibitor of apo

82 osphorylation of Bad; and high expression of inhibitor of apoptosis proteins chicken IAP1, IAP3, and

83 cells in HGSCs expressing high levels of the inhibitor of apoptosis protein cIAP in the CA125-negativ

84 ave shown that Smac mimetics target cellular inhibitor of apoptosis protein (cIAP)-1/2 for degradatio

85 Here, we show that cellular inhibitor of apoptosis proteins (cIAPs) attenuate TNF si

86 We have shown that cellular inhibitor of apoptosis proteins (cIAPs) impair clearance

87 Cellular inhibitors of apoptosis proteins (cIAPs) are essential r

88 eletion of Smac) with expression of X-linked inhibitor of apoptosis protein decreases the sensitivity

89 f-101 before reperfusion attenuated X-linked inhibitor of apoptosis protein degradation and inhibited

90 optosis-inducing factor, as well as X-linked inhibitor of apoptosis protein degradation, but these de

91 chrome c- mediated apoptosis progresses from inhibitor of apoptosis protein-dependent to -independent

92 The Drosophila inhibitor of apoptosis protein DIAP1 ensures cell viabil

93 The inhibitor of apoptosis protein DIAP1 suppresses apoptosi

94 as blocked by overexpression of the cellular inhibitor-of-apoptosis proteins DIAP1 and SfIAP but not

95 reases expression of the endogenous X-linked inhibitor of apoptosis protein expression by translation

96 Conversely, negating X-linked inhibitor of apoptosis protein expression or function in

97 that IL-6 translationally regulates X-linked inhibitor of apoptosis protein expression reveal a novel

98 se localization at the membrane and X-linked inhibitor of apoptosis protein expression, respectively,

99 Survivin is a member of the inhibitor of apoptosis protein family and has an essenti

100 Survivin is a unique member of the inhibitor of apoptosis protein family, and its expressio

101 dy, we showed that survivin, a member of the inhibitor of apoptosis protein family, is expressed in t

102 the expression of survivin, a member of the inhibitor of apoptosis protein family, its expression wa

103 Survivin, a member of the inhibitor of apoptosis protein family, was found to be s

104 protein contains structural features of the inhibitor of apoptosis protein family.

105 Survivin is a member of the IAP (inhibitor of apoptosis) protein family, defined in part

106 XIAP and cIAP1 are two members of the inhibitors of apoptosis protein family whose expression

107 Survivin, a member of the inhibitors of apoptosis protein family, plays important

108 y receptor (DcR) 1 and DcR2, Flame-1, or the inhibitors of apoptosis proteins family of proteins.

109 is effect appears to be mediated by the IAP (inhibitor of apoptosis protein) family of anti-apoptotic

110 nhibitor of apoptosis protein-1 and X-linked inhibitor of apoptosis protein, Fas-induced apoptosis in

111 ity is initiated by a sublethal pulse of the inhibitor of apoptosis protein (IAP) antagonist reaper.

112 to DNA damage--converges on transcription of inhibitor of apoptosis protein (IAP) antagonists reaper,

113 chemoresistant role for RIP1 that maintains inhibitor of apoptosis protein (IAP) expression by relea

114 the expression of molecules in the Bcl-2 and inhibitor of apoptosis protein (IAP) families.

115 c-IAP1, a member of the inhibitor of apoptosis protein (IAP) family and a RING f

116 XIAP and cIAP1 are members of the inhibitor of apoptosis protein (IAP) family and are key

117 -ESO-1 (cancer/testis antigen) and survivin (inhibitor of apoptosis protein (IAP) family antigen).

118 Members of the Inhibitor of Apoptosis Protein (IAP) family are essentia

119 T-infected cells, we found that survivin, an Inhibitor of Apoptosis Protein (IAP) family member, was

120 d activation of caspases by sequestering the inhibitor of apoptosis protein (IAP) family of potent ca

121 The founding member of the inhibitor of apoptosis protein (IAP) family was original

122 We examined whether the inhibitor of apoptosis protein (IAP) family, in particul

123 urvivin, which is the smallest member of the inhibitor of apoptosis protein (IAP) family, is a chromo

124 rotein (XIAP), the most potent member of the inhibitor of apoptosis protein (IAP) family, plays a cru

125 Survivin, a member of the inhibitor of apoptosis protein (IAP) family, regulates m

126 naling is dependent on XIAP, a member of the inhibitor of apoptosis protein (IAP) family.

127 of the human cIAP1 protein, a member of the inhibitor of apoptosis protein (IAP) family.

128 Inhibitor of apoptosis protein (IAP) suppresses apoptosi

129 We have investigated the role of human inhibitor of apoptosis protein (IAP) surviving in regula

130 I kappa B, inhibitor of apoptosis protein (IAP)-1, and cellular Fas

131 Inhibitor of apoptosis protein (IAP)-like protein-1 (ILP

132 OX-2), cyclin D1, and c-myc], antiapoptosis [inhibitor of apoptosis protein (IAP)1, IAP2, X-chromosom

133 af1/CIP1)) and antiapoptotic proteins of the inhibitor of apoptosis proteins (IAP) and Bcl-2 families

134 The inhibitor of apoptosis proteins (IAP) are endogenous cas

135 Smac mimetic compounds targeting the inhibitor of apoptosis proteins (IAP) baculoviral IAP re

136 quitin-proteasome pathway is mediated by the inhibitor of apoptosis proteins (IAP) E3 ligases.

137 se-3, nuclear factor kappaB (NF-kappaB), and inhibitor of apoptosis proteins (IAP) were assayed by en

138 pases, which are negatively regulated by the inhibitor of apoptosis proteins (IAP); however, death ef

139 trix metalloproteinase-9, survivin, X-linked inhibitor-of-apoptosis protein (IAP), IAP1, Bcl-x(L), A1

140 poptotic protein that functions by relieving inhibitor-of-apoptosis protein (IAP)-mediated suppressio

141 Inhibitors of apoptosis proteins (IAP) are key regulator

142 Inhibitors of apoptosis proteins (IAP) exert critical co

143 Upregulated expression of inhibitors of apoptosis proteins (IAP) has been implicat

144 ing the caspase-inhibitory properties of the inhibitors of apoptosis proteins (IAP), particularly XIA

145 resistance is the expression and activity of inhibitors of apoptosis proteins (IAP).

146 Inhibitor of apoptosis proteins (IAPs) act as endogenous

147 iapoptotic factor structurally distinct from inhibitor of apoptosis proteins (IAPs) and Bcl-2 family

148 Degradation of certain inhibitor of apoptosis proteins (IAPs) appears to be cri

149 The inhibitor of apoptosis proteins (IAPs) are believed to b

150 Inhibitor of Apoptosis Proteins (IAPs) are frequently ov

151 Inhibitor of apoptosis proteins (IAPs) are important reg

152 Inhibitor of apoptosis proteins (IAPs) c-IAP1 and c-IAP2

153 Inhibitor of Apoptosis Proteins (IAPs) can bind to and i

154 Many inhibitor of apoptosis proteins (IAPs) function as E3 li

155 The inhibitor of apoptosis proteins (IAPs) have generated co

156 The inhibitor of apoptosis proteins (IAPs) have recently bee

157 We investigated the roles of inhibitor of apoptosis proteins (IAPs) in the regulation

158 Furthermore, SMAC and agents that mimic the inhibitor of apoptosis proteins (IAPs) inhibition functi

159 Inhibitor of apoptosis proteins (IAPs) interact with and

160 Inhibitor of apoptosis proteins (IAPs) play a major role

161 Inhibitor of apoptosis proteins (IAPs) prevent apoptosis

162 Inhibitor of apoptosis proteins (IAPs) regulate apoptosi

163 s to baculoviral IAP repeat (BIR) domains of inhibitor of apoptosis proteins (IAPs) releasing their i

164 The inhibitor of apoptosis proteins (IAPs) represent the onl

165 Inhibitor of apoptosis proteins (IAPs) such as XIAP, cIA

166 Although early studies of inhibitor of apoptosis proteins (IAPs) suggested that cI

167 RNAs together with a chemical antagonist of Inhibitor of apoptosis proteins (IAPs) that simulates th

168 Despite the potential of the inhibitor of apoptosis proteins (IAPs) to block cytochro

169 icated in promoting cell death by binding to inhibitor of apoptosis proteins (IAPs) via its amino-ter

170 Resistance to apoptosis is afforded by inhibitor of apoptosis proteins (IAPs) which bind to and

171 poptotic B cell lymphoma 2 (BCL-2) proteins, inhibitor of apoptosis proteins (IAPs), and murine doubl

172 Loss of inhibitor of apoptosis proteins (IAPs), particularly cIA

173 LCL161 antagonizes the function of inhibitor of apoptosis proteins (IAPs), thereby promotin

174 ators of cell death function by antagonizing Inhibitor of Apoptosis Proteins (IAPs), which themselves

175 downstream caspase activation was blocked by inhibitor of apoptosis proteins (IAPs).

176 in known to bind and inhibit the function of inhibitor of apoptosis proteins (IAPs).

177 ys demonstrated that Omi/HtrA2 could degrade inhibitor of apoptosis proteins (IAPs).

178 ces apoptosis through its action on cellular inhibitor of apoptosis proteins (IAPs).

179 sis to relieve inhibition of caspases by the inhibitor of apoptosis proteins (IAPs).

180 designed and prepared as antagonists of the inhibitor of apoptosis proteins (IAPs).

181 event their inappropriate activation through inhibitor of apoptosis proteins (IAPs).

182 h Effector Domains (DED); (5) BIR domains of Inhibitor of Apoptosis Proteins (IAPs); (6) Bcl-2 homolo

183 ecroptosis, the TLR adaptor protein TRIF and inhibitor of apoptosis proteins (IAPs: X-linked IAP, cel

184 The inhibitor-of-apoptosis proteins (IAPs) regulate programm

185 ctivity was because of the inhibition by the inhibitor-of-apoptosis proteins (IAPs), such as XIAP, an

186 Inhibitors of Apoptosis Protein (IAPs) are guardian ubiq

187 these events contribute to up-regulation of inhibitors of apoptosis proteins (IAPs) and MDR1 (multid

188 Inhibitors of apoptosis proteins (IAPs) are important re

189 Inhibitors of apoptosis proteins (IAPs) interact with ca

190 Inhibitors of apoptosis proteins (IAPs) were originally

191 parvum led to upregulation of genes encoding inhibitors of apoptosis proteins (IAPs), including c-IAP

192 kt axis on the expression of a member of the inhibitors of apoptosis proteins (IAPs), the neuronal ap

193 hich are opposed by a pro-survival family of inhibitors of apoptosis proteins (IAPs).

194 adenovirus was used to express the X-linked inhibitor of apoptosis protein in these cells.

195 breast cancer cells, we found here that the inhibitors of apoptosis proteins, including c-IAP1 and X

196 XIAP (X-chromosome-linked inhibitor of apoptosis protein) is an inhibitor of apopt

197 XIAP (X-linked inhibitor of apoptosis protein) is involved in the media

198 und that survivin, the mitosis regulator and inhibitor of apoptosis protein, is deregulated in these

199 GM-CSF prevented GC-induced loss of X-linked inhibitor of apoptosis protein, late activation of JNK,

200 FkappaB, which increased X chromosome-linked inhibitor of apoptosis protein levels.

201 demonstrate striking down-regulation of the inhibitor of apoptosis protein livin/ML-IAP.

202 metabolism MURR1 domain) and XIAP (X-linked inhibitor of apoptosis protein) may also be required for

203 igh-titer IgG antibodies recognized melanoma inhibitor of apoptosis protein (ML-IAP), a caspase antag

204 suppressive roles for this and likely other inhibitor of apoptosis proteins on the stability and/or

205 However, pharmacological inhibition of inhibitor of apoptosis proteins or proteasome function l

206 utation in the thread gene, which encodes an inhibitor of apoptosis protein, produces a branchless ar

207 rotein 1 and atrogin-1), caspase-3 activity, inhibitor of apoptosis proteins, proinflammatory cytokin

208 Inhibition of DIABLO/Smac, which blocks inhibitor of apoptosis proteins, protected cells from de

209 h increased expression of survivin/BIRC5, an inhibitor of apoptosis protein, suggesting a survival ad

210 c acid greatly reduced the expression of the inhibitor of apoptosis protein survivin and cdc2 activit

211 ion of the targets of miR-708 identified the inhibitor of apoptosis protein survivin as important.

212 ion increased the level of expression of the inhibitor of apoptosis protein survivin in breast cancer

213 The inhibitor of apoptosis protein survivin is expressed in

214 ssion of COX-2 leads to stabilization of the inhibitor of apoptosis protein survivin resulting in the

215 e encoding secretory chemokine CCL21 and the inhibitor of apoptosis protein survivin, overexpressed b

216 Avicin D also decreased expression of the inhibitor of apoptosis protein survivin, the anti-apopto

217 The inhibitor-of-apoptosis protein survivin is expressed in

218 ssor p53 in regulating the expression of the inhibitor-of-apoptosis protein survivin, which may play

219 The inhibitor of apoptosis protein, survivin, has been impli

220 The overexpression of the inhibitor of apoptosis protein, survivin, may provide tu

221 c, cyclooxygenase-2 (COX-2), Bcl-2, cellular inhibitor of apoptosis protein, survivin, vascular endot

222 Survivin is an inhibitor of apoptosis protein that also functions durin

223 Survivin is an inhibitor of apoptosis protein that also plays critical

224 uitination of NEMO is mediated by c-IAP1, an inhibitor of apoptosis protein that is a component of th

225 Survivin is an inhibitor of apoptosis protein that regulates cell cycle

226 vated caspases, which can be counteracted by inhibitor of apoptosis proteins, there is little underst

227 Smac mimetics block inhibitor of apoptosis proteins to trigger TNFalpha-depe

228 ochondrial protein that binds XIAP (X-linked inhibitor of apoptosis protein) upon entering the cytoso

229 ) family members (cIAP1, cIAP2, and X-linked inhibitor of apoptosis protein), was overexpressed by RM

230 Survivin, an inhibitor-of-apoptosis protein, was also downregulated i

231 ased by 40% above basal wild-type levels and inhibitors of apoptosis proteins were down-regulated, bu

232 s, including combination with antagonists of inhibitor of apoptosis proteins, were revealed for PAK1.

233 poptotic proteins bcl-2, mcl-1, and X-linked inhibitor of apoptosis protein which occurs as early as

234 d significantly increased levels of X-linked inhibitor of apoptosis protein, which also correlated wi

235 Survivin is an inhibitor of apoptosis protein, which is over-expressed

236 Inhibitors of apoptosis proteins X-chromosome-linked inh

237 the inhibitory interaction between X-linked inhibitor of apoptosis protein (XIAP) and caspase-9 rema

238 o bind to the antiapoptotic protein X-linked inhibitor of apoptosis protein (XIAP) and showed cellula

239 X-linked inhibitor of apoptosis protein (XIAP) antagonist N-[(5R)

240 imic the binding of Smac protein to X-linked inhibitor of apoptosis protein (XIAP) are being pursued

241 rexpression studies have identified X-linked inhibitor of apoptosis protein (XIAP) as a potent inhibi

242 We have identified endogenous X-linked inhibitor of apoptosis protein (XIAP) as the essential p

243 , interacts with and phosphorylates X-linked inhibitor of apoptosis protein (XIAP) at residue serine-

244 due to the high-level expression of X-linked inhibitor of apoptosis protein (XIAP) by CLL cells.

245 expressing the BIR1/BIR2 domains of X-linked inhibitor of apoptosis protein (XIAP) decreased all anti

246 d down-regulation of active AKT and X-linked inhibitor of apoptosis protein (XIAP) expression and wer

247 tic and therapeutic significance of X-linked inhibitor of apoptosis protein (XIAP) in melanoma throug

248 t on the prognostic significance of X-linked inhibitor of apoptosis protein (XIAP) in melanoma.

249 Our previous studies reported that X-linked inhibitor of apoptosis protein (XIAP) interacted with Rh

250 The X-linked inhibitor of apoptosis protein (XIAP) is a potent caspas

251 X-linked inhibitor of apoptosis protein (XIAP) is a potent caspas

252 The X-linked inhibitor of apoptosis protein (XIAP) is a potent cellul

253 activated CASP7 (p19/p12-CASP7) by X-linked inhibitor of apoptosis protein (XIAP) is a potential mec

254 The X-linked inhibitor of apoptosis protein (XIAP) is a well known po

255 X-chromosome-linked inhibitor of apoptosis protein (XIAP) is an endogenous c

256 We show here that X-linked inhibitor of apoptosis protein (xIAP) is essential for t

257 The X chromosome-linked inhibitor of apoptosis protein (XIAP) is the most potent

258 Apaf-1 and X-linked inhibitor of apoptosis protein (Xiap) mRNA were differen

259 as cells overexpressing full-length X-linked inhibitor of apoptosis protein (XIAP) or the BIR1/BIR2 d

260 y overexpressing either full-length X-linked inhibitor of apoptosis protein (XIAP) or the BIR1/BIR2 d

261 X-linked inhibitor of apoptosis protein (XIAP) overexpression has

262 -dependent apoptosis induction, and X-linked inhibitor of apoptosis protein (XIAP) plays a critical r

263 to apoptosis and determines if the X-linked inhibitor of apoptosis protein (XIAP) regulates bile sal

264 that the E3 ubiquitin ligase named x-linked inhibitor of apoptosis protein (XIAP) restricts bacteria

265 h negative regulation of NF-kappaB- X-linked inhibitor of apoptosis protein (XIAP) signaling pathway.

266 hich activate apoptosis and inhibit X-linked inhibitor of apoptosis protein (XIAP) suppression of exe

267 Unexpectedly, binding of X-linked inhibitor of apoptosis protein (XIAP) to the Reaper moti

268 Endogenous caspase inhibitor X-linked inhibitor of apoptosis protein (XIAP) underwent complete

269 The X-linked inhibitor of apoptosis protein (XIAP) uses its second ba

270 ondria and subsequent inhibition of X-linked inhibitor of apoptosis protein (XIAP) was required to in

271 Epithelial expression of X-linked inhibitor of apoptosis protein (XIAP), activation of NF-

272 chemical interactions occur between x-linked inhibitor of apoptosis protein (XIAP), an E3 ubiquitin l

273 eukemia/lymphoma-2 protein (Bcl-2), X-linked inhibitor of apoptosis protein (XIAP), and myeloid cell

274 ular SLAM-associated protein (SAP), X-linked inhibitor of apoptosis protein (XIAP), and perforin expr

275 osis proteins, including c-IAP1 and X-linked inhibitor of apoptosis protein (XIAP), are obligate Hsp7

276 The protein levels of X-linked inhibitor of apoptosis protein (XIAP), Bcl-xL, caspase-1

277 of antiapoptotic proteins including X-linked inhibitor of apoptosis protein (XIAP), c-IAP1, and Bcl-X

278 ppaB-dependent antiapoptotic genes: X-linked inhibitor of apoptosis protein (XIAP), cIAP1, cIAP2, Bcl

279 a strong decrease of expression of X-linked inhibitor of apoptosis protein (XIAP), compared with the

280 ression of the apoptotic regulator, X-linked inhibitor of apoptosis protein (XIAP), has been associat

281 X-linked inhibitor of apoptosis protein (XIAP), the most potent m

282 , we found that CSR1 interacts with X-linked Inhibitor of Apoptosis Protein (XIAP), using yeast two-h

283 or of cleaved caspase-9, X chromosome-linked inhibitor of apoptosis protein (XIAP), we observed that

284 optotic proteins, such as Mcl-1 and X-linked inhibitor of apoptosis protein (XIAP), were rapidly redu

285 2) is associated with deficiency in X-linked inhibitor of apoptosis protein (XIAP), with poorly under

286 esveratrol induced p53-independent, X-linked inhibitor of apoptosis protein (XIAP)-mediated transloca

287 o and HCT-116 Smac(-/-), similar to X-linked inhibitor of apoptosis protein (XIAP)-overexpressing HeL

288 of apoptosis inhibitor, X chromosome-linked inhibitor of apoptosis protein (XIAP).

289 d forms a protein assembly with the X-linked inhibitor of apoptosis protein (XIAP).

290 targeting the antiapoptotic protein X-linked inhibitor of apoptosis protein (XIAP).

291 racterized its interaction with the X-linked inhibitor of apoptosis protein (XIAP).

292 exposure, stabilized the X-chromosome-linked inhibitor of apoptosis protein (XIAP).

293 of apoptosis protein 1 (cIAP1) and X-linked inhibitor of apoptosis protein (XIAP).

294 itory protein (FLIP) and X-chromosome-linked inhibitor of apoptosis protein (XIAP).

295 e identification of peptides targeting the X-inhibitor of apoptosis protein (XIAP).

296 n as well as down-regulation of the X-linked inhibitor of apoptosis protein (XIAP).

297 on, we demonstrated that X chromosome-linked inhibitor-of-apoptosis protein (XIAP) coimmunoprecipitat

298 nd caspase-3 activation and reduced X-linked inhibitor-of-apoptosis protein (XIAP) levels in these ce

299 expression of Bcl-2, A1, X-chromosome-linked inhibitor-of-apoptosis protein (XIAP), cellular inhibito

300 Compound 2 binds to X-linked inhibitors of apoptosis proteins (XIAP) with a Ki of 61