ライフサイエンスコーパス: inhibitory current

1 recordings in knockout mice devoid of tonic inhibitory current.

2 frequencies above 10 Hz evoked steady-state inhibitory currents.

3 hat contain delta subunits and mediate tonic inhibitory currents.

4 sponses comprised clusters of excitatory and inhibitory currents.

5 GABAergic interneurons and the reduction of inhibitory currents.

6 larizing sodium currents and hyperpolarizing inhibitory currents.

7 play of somatic sodium currents and synaptic inhibitory currents.

8 rs results in a loss of alpha1-mediated fast inhibitory currents and a marked reduction in density of

9 t in hippocampal interneurons, mediate tonic inhibitory currents and are highly sensitive to low conc

10 we simultaneously measured phasic and tonic inhibitory currents and assessed their correlation.

11 Tau reduction increased inhibitory currents and normalized excitation/inhibition

12 First, possible contributions of inhibitory currents and postsynaptic spiking to the anir

13 ry sites, reduced the frequency of miniature inhibitory currents, and increased excitability in the h

14 eous and evoked excitatory currents, reduced inhibitory currents, and NMDA receptor dysfunction.

15 When the inhibitory currents are activated or blocked, the model

16 Despite this, the inhibitory currents are potent in modifying the firing p

17 duced numbers of Pv(+) neurons and decreased inhibitory currents, as well as alterations in event-rel

18 equency and amplitude of phasic IPSCs, tonic inhibitory currents, as well as in the number of inhibit

19 atal rat ventricular cardiomyocytes produced inhibitory currents at less than one-thousandth of the l

20 re consistent with most of hippocampal tonic inhibitory current being mediated by GABA released from

21 cker rTertiapin-Q diminished the NECA-evoked inhibitory current by 56 +/- 12%, whereas the SK channel

22 and that the balance between excitatory and inhibitory currents controls network stability and sensi

23 Voltage-clamp analysis of inhibitory currents demonstrated that activity blockade

24 We measured inhibitory currents evoked by the tutor song throughout

25 he guinea pig (Cavia porcellus) by recording inhibitory currents from RGCs in the presence of ionotro

26 table anesthetic etomidate increased a tonic inhibitory current generated by alpha5 subunit-containin

27 regimes that produce balanced excitatory and inhibitory currents have specific advantages over other

28 w that GABA fails to evoke fast postsynaptic inhibitory currents in apical developing inner and outer

29 ology to record post-synaptic excitatory and inhibitory currents in complex horizontal connections in

30 onal application of GABA evoked large, rapid inhibitory currents in concordance with electron microsc

31 er was correlated with increased spontaneous inhibitory currents in excitatory neurons, suggesting th

32 ing channelrhodopsin-2 evoked fast GABAergic inhibitory currents in mitral cells but failed to activa

33 Following removal of PNNs, the frequency of inhibitory currents in mPFC pyramidal neurons was decrea

34 onset and termination of "slow" postsynaptic inhibitory currents in neurons and atrial cells, RGS pro

35 ich contain delta subunits and mediate tonic inhibitory currents in neurons.

36 The data revealed that perisomatic inhibitory currents in pyramidal cells generated the maj

37 hypothesized that these features potentiate inhibitory currents in RCs.

38 ivation of cholinergic interneurons produced inhibitory currents in SPNs.

39 cts of isoflurane on properties of GABAergic inhibitory currents in the reticular thalamic nucleus (n

40 but reveal both excitatory and monosynaptic inhibitory currents in the ventral pallidum and lateral

41 Inhibitory currents in XII motoneurones were potentiated

42 demonstrate that pharmacologically reducing inhibitory currents increases correlated excitatory acti

43 he mIPSC frequency, NA also potentiated GABA inhibitory currents induced by direct stimulation of gra

44 s from nearby pyramidal cells, we found that inhibitory currents (IPSCs) are more correlated than exc

45 ysiological findings suggest a disruption in inhibitory current may play a role in these changes.

46 Phasic and tonic inhibitory currents of hippocampal pyramidal neurons exh

47 ed in part by weaker excitatory and stronger inhibitory currents onto MCs than onto TCs.

48 aneous inhibitory inputs and enhanced evoked inhibitory currents recorded from principal neurons of t

49 cytosis and protein kinase C to reduce GABAR inhibitory currents, surface GABAR alpha1 expression, an

50 citatory cells, overlapping with feedforward inhibitory currents that suppress action potentials.

51 ferential desensitization causing shift from inhibitory current to excitatory current with repeated c

52 individual contributions of AMPA, NMDA, and inhibitory currents to light responses of each cell type

53 ting a dynamic rebalancing of excitatory and inhibitory currents to maximize SNR.SIGNIFICANCE STATEME

54 on trains of ten of these excitatory and ten inhibitory current transients were combined with an addi

55 The mean reversal potential of the inhibitory current was -78 +/- 7 mV and inhibitions were

56 induced CPP, both frequency and amplitude of inhibitory currents were decreased.

57 Patch-clamp recordings of excitatory and inhibitory currents were performed, then dendritic struc

58 rs to be driven by presynaptic activation of inhibitory currents, whereas the hSyn-HM4D-mediated incr

59 rin clusters and mixed glycinergic/GABAergic inhibitory currents with large peak amplitudes and long