ライフサイエンスコーパス: inhibitory postsynaptic current

1 urs during DSI of the temporally overlapping inhibitory postsynaptic current.

2 showed an increased frequency of spontaneous inhibitory postsynaptic currents.

3 gic neurotransmission by recording miniature inhibitory postsynaptic currents.

4 ereas muscarinic agonists potently increased inhibitory postsynaptic currents.

5 ture inhibitory postsynaptic current; mIPSC) inhibitory postsynaptic currents.

6 t produced a decrease in amplitude of evoked inhibitory postsynaptic currents.

7 s from the precise interaction of excitatory-inhibitory postsynaptic currents.

8 ced PPD, suggesting the contribution of slow inhibitory postsynaptic currents.

9 ly to influence the kinetics and efficacy of inhibitory postsynaptic currents.

10 imetics did not affect bicuculline-sensitive inhibitory postsynaptic currents.

11 enced by an increased amplitude of miniature inhibitory postsynaptic currents.

12 togenic tissue exhibit decreased spontaneous inhibitory postsynaptic currents.

13 icant increase in the amplitude of miniature inhibitory postsynaptic currents.

14 tion of GABA(A) receptor (GABA(A)R)-mediated inhibitory postsynaptic currents.

15 elevated frequency of GABAergic spontaneous inhibitory postsynaptic currents.

16 GABA(A)R number and reduces the amplitude of inhibitory postsynaptic currents.

17 litude of subsequent GABAA receptor-mediated inhibitory postsynaptic currents.

18 ing that they were GABA(A) receptor-mediated inhibitory postsynaptic currents.

19 slices by recording 5-HT1A receptor-mediated inhibitory postsynaptic currents (5-HT1A -IPSCs) generat

20 es and a lower rate of spontaneous miniature inhibitory postsynaptic current activity.

21 NPY also depressed the GABAergic inhibitory postsynaptic current, although leptin had no

22 me in null mice, with no change in miniature inhibitory postsynaptic current amplitude or frequency.

23 vels, flurazepam treatment reduced miniature inhibitory postsynaptic current amplitude, which returne

24 ynaptic gephyrin clusters and mean miniature inhibitory postsynaptic current amplitudes, whereas a do

25 aptic current kinetics and reduced miniature inhibitory postsynaptic current amplitudes.

26 avioral phenotypes, decreased excitatory and inhibitory postsynaptic current and reduced c-Fos immuno

27 GABA-mediated inhibitory postsynaptic currents and potentials decayed

28 entration that abolished miniature GABAergic inhibitory postsynaptic currents and responses to exogen

29 both action potential-dependent (spontaneous inhibitory postsynaptic current) and -independent (minia

30 ude of spontaneous GABA(A) receptor-mediated inhibitory postsynaptic currents, and are blocked by gab

31 Cl(-) influx into the cytosol, a decrease of inhibitory postsynaptic currents, and ultimately a shift

32 rsibly inhibited both evoked and spontaneous inhibitory postsynaptic currents, as well as GABA applic

33 e glycine receptor enables the generation of inhibitory postsynaptic currents at synapses via neurotr

34 release site opened on the timescale of the inhibitory postsynaptic currents before desensitizing.

35 BDNF had no effect on spontaneous inhibitory postsynaptic currents but produced a decrease

36 in the amplitude and frequency of miniature inhibitory postsynaptic currents compared with those in

37 es revealed a significantly faster miniature inhibitory postsynaptic current decay time in null mice,

38 The IL-6-induced inhibitory postsynaptic currents decrease was inhibited

39 ntials were inhibited by tetrodotoxin (TTX), inhibitory postsynaptic currents decreased and currents

40 Furthermore, the duration of inhibitory postsynaptic current depression mirrors the t

41 of the NTS resulted in evoked excitatory and inhibitory postsynaptic currents (eEPSCs and eIPSCs) in

42 antly inhibited the peak amplitude of evoked inhibitory postsynaptic currents (eIPSCs) in all 11 labe

43 transient suppression of evoked GABAA ergic inhibitory postsynaptic currents (eIPSCs) that follows b

44 Inhibitory postsynaptic currents (eIPSCs) were evoked af

45 The medial NTS was stimulated to evoke GABA inhibitory postsynaptic currents (eIPSCs).

46 eased the amplitude of GABAA-mediated evoked inhibitory postsynaptic currents (eIPSCs).

47 rding was performed to obtain excitatory and inhibitory postsynaptic currents (EPSCs and IPSCs) of th

48 rding was performed to obtain excitatory and inhibitory postsynaptic currents (EPSCs and IPSCs) of th

49 y the frequency dependence of excitatory and inhibitory postsynaptic currents (EPSCs, IPSCs) elicited

50 We recorded excitatory and inhibitory postsynaptic currents (EPSCs/IPSCs) in TC neu

51 onists selectively decreased the spontaneous inhibitory postsynaptic current frequency in the head-in

52 Inhibitory postsynaptic current frequency, measured on p

53 hat ethanol enhances GABAA receptor-mediated inhibitory postsynaptic currents (GABAA IPSCs) and reduc

54 DPDPE inhibition of GABAA receptor-mediated inhibitory postsynaptic currents (GABAAR IPSCs) is assoc

55 Normal-appearing inhibitory postsynaptic currents (human) and intrinsic n

56 associated with a reduction of GABA-mediated inhibitory postsynaptic current in neocortex and hippoca

57 d the frequency of spontaneous excitatory or inhibitory postsynaptic currents in a concentration-rela

58 cordings revealed spontaneous excitatory and inhibitory postsynaptic currents in all neurons, regardl

59 mulation of ventrobasal complex cells evoked inhibitory postsynaptic currents in cells of the medial

60 ing increase in the frequency of spontaneous inhibitory postsynaptic currents in dentate granule cell

61 ecordings revealed a diminished frequency of inhibitory postsynaptic currents in dentate granule cell

62 tribution in pyramidal neurons and supported inhibitory postsynaptic currents in distal dendrites bet

63 t1 extended glutamatergic interneuron-evoked inhibitory postsynaptic currents in motor neurons.

64 ncreased action potential-driven spontaneous inhibitory postsynaptic currents in nearby interneurons

65 7 nAChRs on feedforward interneurons induced inhibitory postsynaptic currents in pyramidal cells that

66 ated with reduced frequency of GABA-mediated inhibitory postsynaptic currents in pyramidal neurons, a

67 firing, which robustly increases spontaneous inhibitory postsynaptic currents in pyramidal neurons.

68 reased efficacy of spontaneous and miniature inhibitory postsynaptic currents in pyramidal neurons.

69 ase in the number and amplitude of miniature inhibitory postsynaptic currents in these cells in a TTX

70 GABAA receptor (GABAAR)-mediated spontaneous inhibitory postsynaptic currents in WT mice, indicating

71 oth the amplitude and frequency of miniature inhibitory postsynaptic currents increased in neurons de

72 c currents decreased, while the frequency of inhibitory postsynaptic currents increased.

73 This inhibitory postsynaptic current (IPSC) erosion resulted

74 role of nitric oxide (NO) in modulating the inhibitory postsynaptic current (IPSC) evoked by focal s

75 cell evoked a GABA(A) receptor-mediated slow inhibitory postsynaptic current (IPSC) in a PN and an au

76 apses in olfactory bulb slices evokes a slow inhibitory postsynaptic current (IPSC) in granule cells

77 ease in the amplitude of evoked monosynaptic inhibitory postsynaptic current (IPSC) in layer V pyrami

78 endent decrease of GABA(A) receptor-mediated inhibitory postsynaptic current (IPSC) is explained by t

79 release produced a 5-HT1A receptor-mediated inhibitory postsynaptic current (IPSC) that resulted in

80 This report describes an inhibitory postsynaptic current (IPSC) that was elicited

81 or activation depresses the stimulus-evoked, inhibitory postsynaptic current (IPSC).

82 ea of the pharmacologically isolated GABA/B/-inhibitory postsynaptic current (IPSC).

83 OF increased depression of inhibitory postsynaptic currents (IPSCs) along IPSC trai

84 n with PPs had no effect on the amplitude of inhibitory postsynaptic currents (IPSCs) and a minimal e

85 ocaine exposure in vivo caused a decrease in inhibitory postsynaptic currents (IPSCs) and an increase

86 show that all mutations prolong the decay of inhibitory postsynaptic currents (IPSCs) and induce spon

87 ld each depress the frequency of spontaneous inhibitory postsynaptic currents (IPSCs) and the amplitu

88 ynaptic inhibition was recorded by isolating inhibitory postsynaptic currents (IPSCs) at a membrane p

89 1 Hz) electrical stimulation produced evoked inhibitory postsynaptic currents (IPSCs) at a relatively

90 cuculline-sensitive and strychnine-sensitive inhibitory postsynaptic currents (IPSCs) could be evoked

91 rge increase in the frequency of spontaneous inhibitory postsynaptic currents (IPSCs) driven by actio

92 ate the effects of 5-HT and TRH on GABAergic inhibitory postsynaptic currents (IPSCs) evoked by stimu

93 Inhibitory postsynaptic currents (IPSCs) evoked in CA1 p

94 we obtained whole-cell recordings of unitary inhibitory postsynaptic currents (IPSCs) evoked in ventr

95 nduced more pronounced initial depression of inhibitory postsynaptic currents (IPSCs) followed by mod

96 Inhibitory postsynaptic currents (IPSCs) generated by GA

97 ation-dependent increase in the frequency of inhibitory postsynaptic currents (IPSCs) in 5-HT neurons

98 ethanol increases GABA(A) receptor-mediated inhibitory postsynaptic currents (IPSCs) in CeA neurons

99 ession, we evoked 100 Hz trains of GABAergic inhibitory postsynaptic currents (IPSCs) in cerebellar n

100 ordings of intrinsic membrane properties and inhibitory postsynaptic currents (IPSCs) in dentate gran

101 Stimulation of the NTS evoked inhibitory postsynaptic currents (IPSCs) in DMV neurones

102 dopamine terminals evoked robust D2-receptor inhibitory postsynaptic currents (IPSCs) in GIRK2-expres

103 of a study of the frequency potentiation of inhibitory postsynaptic currents (IPSCs) in hypoglossal

104 duced monosynaptically evoked GABAA-mediated inhibitory postsynaptic currents (IPSCs) in NRT and soma

105 re (SL-M) stimuli that activate GABA(A,slow) inhibitory postsynaptic currents (IPSCs) in pyramidal ce

106 afferents induces rhythmic, theta-frequency inhibitory postsynaptic currents (IPSCs) in pyramidal ce

107 Inhibitory postsynaptic currents (IPSCs) in RTN neurons

108 ion, we found that GABA(A)-receptor-mediated inhibitory postsynaptic currents (IPSCs) in the inhibito

109 eratrol potentiated GABAA and GABAB-mediated inhibitory postsynaptic currents (IPSCs) in VTA dopamine

110 0-1724 blocked I-LTD and acute depression of inhibitory postsynaptic currents (IPSCs) induced by D(2)

111 postsynaptic currents (EPSCs) and GABAergic inhibitory postsynaptic currents (IPSCs) induced by post

112 In addition, retigabine potentiated inhibitory postsynaptic currents (IPSCs) mediated by act

113 roduces a long-lasting enhancement of evoked inhibitory postsynaptic currents (IPSCs) mediated by D1-

114 ncy of glycinergic spontaneous and miniature inhibitory postsynaptic currents (IPSCs) of lamina II ne

115 c in the inhibition by morphine of GABAergic inhibitory postsynaptic currents (IPSCs) recorded from n

116 The time course of inhibitory postsynaptic currents (IPSCs) reflects GABA(A

117 uency and amplitude of spontaneous GABAergic inhibitory postsynaptic currents (IPSCs) than did wild t

118 Vagal stimulation also evoked inhibitory postsynaptic currents (IPSCs) that were rever

119 T(4) receptors on the amplitude of GABAergic inhibitory postsynaptic currents (IPSCs) to a reduction.

120 y drive onto VB neurons from multiple peaked inhibitory postsynaptic currents (IPSCs) to single peake

121 Here the duration of inhibitory postsynaptic currents (IPSCs) was dependent o

122 Evoked inhibitory postsynaptic currents (IPSCs) were induced in

123 r the frequency of GABA-mediated spontaneous inhibitory postsynaptic currents (IPSCs) were reduced in

124 Evoked GABAergic inhibitory postsynaptic currents (IPSCs) were reversibly

125 oltage-clamped GABA(A fast) and GABA(A slow) inhibitory postsynaptic currents (IPSCs) were selectivel

126 Inhibitory postsynaptic currents (IPSCs) were twice as p

127 eurons mediating fast GABA(A) (GABA(A,fast)) inhibitory postsynaptic currents (IPSCs), whereas theta

128 pharmacologically distinct from spontaneous inhibitory postsynaptic currents (IPSCs).

129 concentrations that barely affect GABAergic inhibitory postsynaptic currents (IPSCs).

130 ete, strychnine-sensitive, chloride-mediated inhibitory postsynaptic currents (IPSCs).

131 show that besides blocking GABA(A)-mediated inhibitory postsynaptic currents (IPSCs, I(phasic)), the

132 creased spontaneous inhibitory transmission (inhibitory postsynaptic currents, IPSCs) in the DR.

133 The magnitude of the depression of the inhibitory postsynaptic current is dependent on the freq

134 nvergence ratios ( approximately 40:1), fast inhibitory postsynaptic current kinetics (tau(decay) = 2

135 subunit altered the time course of miniature inhibitory postsynaptic current kinetics and reduced min

136 -evoked inhibition, we recorded light-evoked inhibitory postsynaptic currents (L-IPSCs) from rod bipo

137 ted inputs to rod BCs prolonged light-evoked inhibitory postsynaptic currents (L-IPSCs), while smalle

138 DCG IV (1 microM) had no effect on inhibitory postsynaptic currents mediated by GABA.

139 e frequency and the amplitude of spontaneous inhibitory postsynaptic currents mediated by gamma-amino

140 This study describes spontaneous miniature inhibitory postsynaptic currents mediated by vesicular d

141 apses, causing clear reductions in miniature inhibitory postsynaptic current (mIPSC) amplitude and fr

142 tion of Gad1 significantly reduced miniature inhibitory postsynaptic current (mIPSC) amplitudes and G

143 on did not exhibit increased basal miniature inhibitory postsynaptic current (mIPSC) frequency but sh

144 minimally altered sIPSC amplitude, miniature inhibitory postsynaptic current (mIPSC) frequency, and m

145 tion of transmitter fluctuation to miniature inhibitory postsynaptic current (mIPSC) variability.

146 ynaptic current) and -independent (miniature inhibitory postsynaptic current; mIPSC) inhibitory posts

147 ptible to DSI, while TTX-resistant miniature inhibitory postsynaptic current (mIPSCs) were not.

148 e activates rapid GABA(A) receptor miniature inhibitory postsynaptic currents (mIPSCs) (predominant d

149 We recorded miniature inhibitory postsynaptic currents (mIPSCs) from hippocamp

150 sed the frequency of GABA-mediated miniature inhibitory postsynaptic currents (mIPSCs) in all nine la

151 t the decay time of GABAR-mediated miniature inhibitory postsynaptic currents (mIPSCs) in CA1 pyramid

152 uency of glycinergic and GABAergic miniature inhibitory postsynaptic currents (mIPSCs) in cardiac vag

153 endent increase in the frequency ofminiature inhibitory postsynaptic currents (mIPSCs) in primary cul

154 ptor agonist baclofen, potentiated miniature inhibitory postsynaptic currents (mIPSCs) in pyramidal n

155 re excitatory postsynaptic currents (mEPSCs)/inhibitory postsynaptic currents (mIPSCs) in the Mecp2-m

156 brain development the duration of miniature inhibitory postsynaptic currents (mIPSCs) mediated by GA

157 brain development the duration of miniature inhibitory postsynaptic currents (mIPSCs) mediated by GA

158 rites in macaque monkeys, measured miniature inhibitory postsynaptic currents (mIPSCs) of granule cel

159 pare the properties of 'tonic' and miniature inhibitory postsynaptic currents (mIPSCs) recorded from

160 neous action potential independent miniature inhibitory postsynaptic currents (mIPSCs) was significan

161 postsynaptic currents (mEPSCs) and miniature inhibitory postsynaptic currents (mIPSCs) were recorded

162 croscopically, and the spontaneous miniature inhibitory postsynaptic currents (mIPSCs) were recorded

163 plitude and frequency of GABAergic miniature inhibitory postsynaptic currents (mIPSCs) were reduced i

164 n frequency and charge transfer of miniature inhibitory postsynaptic currents (mIPSCs) were significa

165 creased the frequency of miniature GABAergic inhibitory postsynaptic currents (mIPSCs) without changi

166 ynaptic currents (mEPSCs), but not miniature inhibitory postsynaptic currents (mIPSCs), increase in a

167 A currents and in the frequency of miniature inhibitory postsynaptic currents (mIPSCs).

168 as the frequency and amplitude of miniature inhibitory postsynaptic currents (mIPSCs).

169 or GABAergic markers and recording miniature inhibitory postsynaptic currents (mIPSCs).

170 tials (IPSPs) and the frequency of miniature inhibitory postsynaptic currents (mIPSCs).

171 g the neurons lost, and a marked decrease in inhibitory postsynaptic currents of lamina II neurons co

172 amplitude but not the frequency of miniature inhibitory postsynaptic currents or expression of the gl

173 layer VI cortical neurons exhibited reduced inhibitory postsynaptic current peak amplitudes, prolong

174 blunted membrane excitability and divergent inhibitory postsynaptic current responses to CRF applica

175 Analysis of the kinetics of inhibitory postsynaptic current revealed no modification

176 aracteristics of sound-evoked excitatory and inhibitory postsynaptic currents (seEPSCs and seIPSCs, r

177 the frequency of spontaneous excitatory and inhibitory postsynaptic currents (sEPSCs and sIPSCs).

178 pmol/L GLP-1 increased both the spontaneous inhibitory postsynaptic current (sIPSC) amplitudes and f

179 chol significantly increased the spontaneous inhibitory postsynaptic current (sIPSC) frequency and am

180 mutation had no clear effect on spontaneous inhibitory postsynaptic current (sIPSC) frequency at IN-

181 ngs revealed large reductions in spontaneous inhibitory postsynaptic current (sIPSC) frequency in bot

182 cotine (mean increased spontaneous GABAergic inhibitory postsynaptic current (sIPSC) frequency was ap

183 pyramidal neuron also causes LTP of the slow inhibitory postsynaptic current (sIPSC) mediated by meta

184 Spontaneous and miniature inhibitory postsynaptic currents (sIPSCS and mIPSCs) wer

185 ontrol of synaptic GABA release, spontaneous inhibitory postsynaptic currents (sIPSCs) and miniature

186 neurons presented frequencies of spontaneous inhibitory postsynaptic currents (sIPSCs) and spontaneou

187 fects on GABAA receptor-mediated spontaneous inhibitory postsynaptic currents (sIPSCs) and spontaneou

188 aclofen reduced the frequency of spontaneous inhibitory postsynaptic currents (sIPSCs) but did not al

189 sEPSCs were isolated from the spontaneous inhibitory postsynaptic currents (sIPSCs) by application

190 AA receptor-mediated spontaneous monoquantal inhibitory postsynaptic currents (sIPSCs) from rat supra

191 1% of recordings and facilitated spontaneous inhibitory postsynaptic currents (sIPSCs) in 20% of reco

192 ed amplitudes and frequencies of spontaneous inhibitory postsynaptic currents (sIPSCs) in CA1 pyramid

193 and analogs on the properties of spontaneous inhibitory postsynaptic currents (sIPSCs) in cultured ra

194 ) subunit expression and reduced spontaneous inhibitory postsynaptic currents (sIPSCs) in D1-type, bu

195 conductance, but did not affect spontaneous inhibitory postsynaptic currents (sIPSCs) in dentate gra

196 ncy of GABA(A) receptor-mediated spontaneous inhibitory postsynaptic currents (sIPSCs) in dlBnST targ

197 lease increased the frequency of spontaneous inhibitory postsynaptic currents (sIPSCs) in downstream

198 of action potential-dependent, spontaneous, inhibitory postsynaptic currents (sIPSCs) in hippocampus

199 uency of spontaneous GABAA receptor-mediated inhibitory postsynaptic currents (sIPSCs) in RT (by 60 +

200 croM) increased the frequency of spontaneous inhibitory postsynaptic currents (sIPSCs) in the majorit

201 rate and net charge transfer of spontaneous inhibitory postsynaptic currents (sIPSCs) recorded from

202 The frequency of spontaneous inhibitory postsynaptic currents (sIPSCs) was similar in

203 Spontaneous inhibitory postsynaptic currents (sIPSCs) were not diffe

204 Under these conditions spontaneous GABAergic inhibitory postsynaptic currents (sIPSCs) were seen as i

205 h the frequency and amplitude of spontaneous inhibitory postsynaptic currents (sIPSCs) were significa

206 res the amplitude and frequency of miniature inhibitory postsynaptic currents to wild-type levels.

207 By recording unitary inhibitory postsynaptic currents (uIPSCs) from cell pair

208 Unitary inhibitory postsynaptic currents (uIPSCs) were recorded

209 ability of synaptic failures in spike-evoked inhibitory postsynaptic currents (unitary IPSCs) in CA1

210 er, the frequency of GABAA receptor-mediated inhibitory postsynaptic currents was depressed in medium

211 of opioids because alpha(2)-AR-mediated slow inhibitory postsynaptic currents were depressed in wt bu

212 ss, and whole-cell patch clamp recordings of inhibitory postsynaptic currents were performed from ven

213 The effects of PS on GABAA receptor-mediated inhibitory postsynaptic currents were studied in culture

214 small decrease in the frequency of miniature inhibitory postsynaptic currents, which was not affected

215 duced the frequency of spontaneous miniature inhibitory postsynaptic currents while having little eff

216 vely and reversibly reduced the amplitude of inhibitory postsynaptic currents with a postsynaptic eff

217 ctively increases the frequency of miniature inhibitory postsynaptic currents with no effect on ampli

218 eveal a >20-fold increase in nicotine-evoked inhibitory postsynaptic currents with no effect on excit

219 Here we combine quantal analysis of evoked inhibitory postsynaptic currents with quantitative immun