ライフサイエンスコーパス: inhibitory protein

1 novel interacting partner Rkip (Raf-1 kinase inhibitory protein).

2 ver-activating protein), LAP, and LIP (liver-inhibitory protein).

3 ed to as LIP (liver-enriched transcriptional inhibitory protein).

4 cule c-FLIP (cellular homolog of viral FLICE inhibitory protein).

5 iapoptotic regulator cellular caspase-8-like inhibitory protein.

6 presence of increasing concentrations of the inhibitory protein.

7 n the epidermis RNases are complexed with an inhibitory protein.

8 that the HCMV UL38 gene encodes a cell death inhibitory protein.

9 nriched activator protein and liver-enriched inhibitory protein.

10 attenuated by the forced expression of FLICE-inhibitory protein.

11 slation factor, and 4EBP1 translation factor inhibitory protein.

12 hile a human FoxP2 mutant acts as a dominant-inhibitory protein.

13 interaction of Cbl with the Sprouty2 (Spry2) inhibitory protein.

14 complex is regulated by both activating and inhibitory proteins.

15 hich is predicated upon an arsenal of potent inhibitory proteins.

16 are sequestered in the cytoplasm by IkappaB inhibitory proteins.

17 bitor of apoptosis proteins, and viral FLICE-inhibitory proteins.

18 ct from that associated with differentiation-inhibitory proteins.

19 regulatory loops, involving an assortment of inhibitory proteins.

20 ate immunity by expressing a series of small inhibitory proteins.

21 oxO3a and FoxO-mediated expression of growth-inhibitory proteins.

22 mily zinc finger 1)] or MGE [Map3k12 binding inhibitory protein 1 (Mbip); zinc-finger, SWIM domain co

23 creased expression of modulatory calcineurin inhibitory protein 1 (MCIP1), a direct downstream target

24 d by 409 +/- 32%, and modulatory calcineurin inhibitory protein 1 by 315 +/- 28%, P < 0.01).

25 duces an up-regulation of neuronal apoptosis inhibitory protein-1 expression in neurons that promotes

26 osis proteins (IAPs), the neuronal apoptosis inhibitory protein-1.

27 Plasma macrophage inhibitory protein-1alpha and insulin-like growth factor

28 li; and vi) reduced expression of macrophage inhibitory protein-2 and transforming growth factor-beta

29 The invasion inhibitory protein 45 (IIp45) we recently identified was

30 ybrid system, we identified a gene, invasion inhibitory protein 45 (IIp45), whose protein product bou

31 NAIP5 (NLR family, apoptosis inhibitory protein 5) binds the bacterial protein flagel

32 logue of NLRC4, NAIP5 (NLR family, apoptosis inhibitory protein 5), has been implicated in activation

33 ligand for the NAIP5 (NLR family, apoptosis inhibitory protein 5)/NLRC4 (NLR family, CARD-domain-con

34 NAIP5/NLRC4 (neuronal apoptosis inhibitory protein 5/nucleotide oligomerization domain-l

35 wnregulates expression of the cellular FLICE inhibitory protein, a negative regulator of death recept

36 tion induced the synthesis of liver-enriched inhibitory protein, a potent mitogen and the dominant ne

37 We have shown that the NF-kappaB inhibitory protein A20 exerts vasculoprotective effects

38 The impact of NF-kappaB inhibitory protein A20 on IFNgamma signaling in vascular

39 ealed the differential mapping of Raf kinase inhibitory protein after T cell recall in vivo.

40 ory and active isoforms, i.e. liver-enriched inhibitory protein and liver-enriched activating protein

41 Liver-enriched inhibitory protein and liver-enriched activating protein

42 omoted the phosphorylation of the Raf kinase inhibitory protein and the downregulation of carabin, al

43 activity is critical for processing IkappaB inhibitory proteins and activating the NF-kappaB signali

44 in mapping the interactions between amyloid-inhibitory proteins and amyloidogenic peptides.

45 s that EZH2 inhibition stimulates cell cycle inhibitory proteins and enhances the production of extra

46 trix components, as well as several TGF-beta inhibitory proteins and pro-peptides.

47 f FLIP (FADD-like IL-1beta-converting enzyme-inhibitory protein) and Bcl-2.

48 ntify the conserved protein SciP (small CtrA inhibitory protein) and show that it accumulates during

49 echanisms: transcriptional regulation, small inhibitory proteins, and protein degradation.

50 ta-converting enzyme inhibitory protein-like inhibitory protein], and metastasis (vascular endothelia

51 facilitator (APOA5 [apolipoprotein A-V]) and inhibitory proteins (APOC3 [apolipoprotein C-III], ANGPT

52 actin assembly, the physiologic functions of inhibitory proteins are unclear.

53 In several well-studied cases the inhibitory proteins are water-soluble and bind at a chan

54 NAIPs (NLR family, apoptosis inhibitory proteins) are NLRs that appear to function as

55 In this study we identify Raf-1 kinase inhibitory protein as an essential mediator of ethanol-i

56 ation of CNS axons is the presence of growth-inhibitory proteins associated with myelin and the glial

57 ns cFLIP(L) (cellular caspase-8 (FLICE)-like inhibitory protein), Bcl-2, and Bcl-X(L) and inhibit the

58 beta-Lactamase inhibitory protein (BLIP) binds and inhibits a diverse c

59 Beta-lactamase inhibitory protein (BLIP) binds and inhibits a diverse c

60 beta-Lactamase inhibitory protein (BLIP) binds and inhibits a diverse c

61 ers for 20 alanine mutants in beta-lactamase inhibitory protein (BLIP) for binding to TEM-1 beta-lact

62 ted contact residue mutants ofbeta-lactamase-inhibitory protein (BLIP) using isothermal titration cal

63 The interactions between beta-lactamase inhibitory proteins (BLIPs) and beta-lactamases have bee

64 nt here structures of a bacterial complement inhibitory protein, both free and bound to its complemen

65 role for Spry involving sequestration of the inhibitory protein c-Cbl.

66 Cellular FLICE-inhibitory protein (c-FLIP(L)) is a key regulator of the

67 or, and reduces the levels of cellular FLICE-inhibitory protein (c-FLIP) (both the long and short for

68 ediated stabilization of cellular FLICE-like inhibitory protein (c-FLIP) and Mcl-1.

69 domain (FADD)-like apoptosis regulator-like inhibitory protein (c-FLIP) and myeloid cell leukemia 1

70 ke interleukin-1 beta-converting enzyme-like inhibitory protein (c-FLIP) expression was determined by

71 g the antiapoptotic gene cellular FLICE-like inhibitory protein (C-FLIP) in myeloid cells, we have ge

72 FLICE inhibitory protein (c-FLIP) is an important regulator of

73 or inhibitor cellular caspase 8 (FLICE)-like inhibitory protein (c-FLIP) is required for TNFalpha-ind

74 FAS-mediated apoptosis pathway by FLICE-like inhibitory protein (c-FLIP) may contribute to oncogenesi

75 ain-like interleukin-1beta-converting enzyme-inhibitory protein (c-FLIP) mediates the DISC assembly i

76 suggest that cellular caspase 8 (FLICE)-like inhibitory protein (c-FLIP) promotes cell survival in de

77 e show a novel mechanism by which FLICE-like inhibitory protein (c-FLIP) regulates apoptosis induced

78 osis was associated with cellular FLICE-like inhibitory protein (c-FLIP) turnover and that gamma-secr

79 n-regulated the expression of cellular FLICE-inhibitory protein (c-FLIP), a major negative regulator

80 ction based on degradation of cellular FLICE-inhibitory protein (c-FLIP), a major regulator of the de

81 We show that cellular FLICE-like inhibitory protein (c-FLIP), a procaspase-8-like apoptot

82 Cellular FLICE-inhibitory protein (c-FLIP), an antioxidant and an impor

83 lls led to down-regulation of cellular FLICE inhibitory protein (c-FLIP), an inhibitor of apoptosis.

84 the caspase-8 regulator cellular FLICE-like inhibitory protein (c-FLIP), targeting it for proteasome

85 ain-like interleukin-1beta-converting enzyme-inhibitory protein (c-FLIP), thus releasing their inhibi

86 ike interleukin-1beta-converting enzyme-like inhibitory protein (c-FLIP), were expressed in basal con

87 ain-like interleukin 1beta-converting enzyme inhibitory protein (c-FLIP), whose expression was coregu

88 ke interleukin-1beta-converting enzyme)-like inhibitory protein (c-FLIP).

89 he mRNA and protein levels of cellular FLICE-inhibitory protein (c-FLIP).

90 associated with a decrease in cellular FLICE-inhibitory protein (c-FLIP).

91 Overexpression of cellular FLICE-like inhibitory protein (c-FLIP-s) or knockdown of CD95 suppr

92 D95 or overexpression of cellular FLICE-like inhibitory protein (c-FLIP-s).

93 C/EBPbeta-liver-enriched inhibitory protein (C/EBPbeta-LIP), a truncated C/EBPbet

94 These inhibitory proteins can act both directly by targeting s

95 T cell expression of inhibitory proteins can be a critical component for the

96 uzumab binding, minimal change in complement inhibitory protein (CD46) expression and no complement-d

97 on endothelial cell expression of complement inhibitory proteins CD59 and decay-accelerating factor.

98 , but not collagen, expressed cellular FLICE inhibitory protein (cFLIP) and TNFalpha stimulation of l

99 ppaB activation, and cellular caspase-8-like inhibitory protein (cFLIP) inhibited activation of NF-ka

100 Overexpression of cellular FLICE-like inhibitory protein (cFLIP) is reported to confer chemore

101 The cellular FLICE-like inhibitory protein (cFLIP) is reported to inhibit IFNalp

102 r permanent) with deletion of cellular FLICE-inhibitory protein (cFlip) or caspase-8 in the intestina

103 Cellular FLICE-like inhibitory protein (cFLIP) overexpression and caspase-8

104 ceptors and the antiapoptotic cellular FLICE-inhibitory protein (cFLIP) was inconsistent across the c

105 uncovered down-regulation of cellular FLICE inhibitory protein (cFLIP).

106 of the antiapoptotic molecule cellular FLICE inhibitory protein (cFLIP).

107 conditions and in the presence of the growth-inhibitory proteins chondroitin sulfate proteoglycan, my

108 (Cdk), (iii) increased expression of the Cdk inhibitory proteins (Cip1/p21, Kip1/p27), enhanced bindi

109 ent expression levels of CD38 and complement-inhibitory proteins (CIPs) are associated with response

110 The epithelial complement inhibitory proteins (CIPs) cluster of differentiation 46

111 To determine whether stimulation by the inhibitory protein coevolved with resistance or whether

112 MC38-FasL/FLICE-like inhibitory protein colon cancer cells induce apoptosis i

113 Expression of FLICE-like inhibitory protein confers apoptosis resistance, increas

114 permitted infection of cells expressing the inhibitory protein CPSF6-358.

115 ement receptor 2 (CR2) to a mouse complement-inhibitory protein, Crry.

116 consequence of deficiency of the complement inhibitory proteins decay accelerating factor (DAF, CD55

117 th and caspase activation by promoting FLICE-inhibitory protein degradation and mitochondrial release

118 d in wild-type-infected cells expressing the inhibitory proteins (DeltaH).

119 Increases in liver-enriched inhibitory protein directly correlated with proliferatin

120 The expression of the inhibitory protein elevated the strain's fitness signifi

121 n kinase, RNA activated), and viperin (virus inhibitory protein, endoplasmic reticulum associated, in

122 Complement-inhibitory proteins expressed on cancer cells can provid

123 itors (Y-27632, LY294002, PF271 and PP2) and inhibitory protein expression (FAK-related nonkinase).

124 lls, suggesting that the modulation of Raf-1 inhibitory protein expression may have future therapeuti

125 show that either suppression of Raf-1 kinase inhibitory protein expression using short hairpin RNA or

126 over, the analog is not stabilized unless an inhibitory protein factor binds to the enzyme.

127 e recruit the alternative pathway complement inhibitory protein factor H (fH) to their surfaces to ev

128 lls by mediating the degradation of the TRAF inhibitory protein, Fbxl2.

129 he degradation of the long form of the FLICE-inhibitory protein (FLIP(L)), an inhibitor of death-indu

130 isoform of the caspase-8 inhibitor, c-FLICE inhibitory protein (FLIP(S)), and that FLIP(S) expressio

131 n-1-beta-converting enzyme [FLICE/caspase 8]-inhibitory protein (FLIP) activates NF-kappaB more poten

132 down-regulated cellular levels of FLICE-like inhibitory protein (FLIP) and X-chromosome-linked inhibi

133 FLICE-inhibitory protein (FLIP) blocks death receptor-mediated

134 O and concomitant decrease in cellular FLICE inhibitory protein (FLIP) expression without significant

135 The viral FLICE-like inhibitory protein (FLIP) protein from Kaposi sarcoma-as

136 ein-like interleukin-1beta-converting enzyme inhibitory protein (FLIP) were all down-regulated by ind

137 rleukin-1beta-converting enzyme (FLICE)-like inhibitory protein (FLIP), and reduced FLIP expression p

138 g the inhibitor of caspase activation, FLICE inhibitory protein (FLIP), appears to be a direct target

139 recepter 2) or antiapoptotic proteins (FLICE-inhibitory protein (FLIP), inhibitor of apoptosis (IAP),

140 feration when the caspase 8 inhibitor, FLICE-inhibitory protein (FLIP), is active.

141 interleukin-1beta-converting enzyme (FLICE) inhibitory protein (FLIP), leading to apoptosis.

142 in the presence of CDDP through a FLICE-like inhibitory protein (FLIP)-Itch interaction.

143 ates with decreased expression of FLICE-like inhibitory protein (FLIP).

144 ROS) generation and down-regulation of FLICE inhibitory protein (FLIP); however, the relationship bet

145 rylation of AKT and activation of FLICE-like-inhibitory-protein (FLIP).

146 FLICE-inhibitory proteins (FLIPs) are a family of viral (poxvi

147 Caspase-8/10 and FLICE/caspase-8 inhibitory proteins (FLIPs) that inhibit caspase activat

148 C20) but not of protein kinase C-potentiated inhibitory protein for myosin phosphatase of 17 kDa (CPI

149 Inhibitory proteins from the serum were determined as ne

150 These findings suggest a role for inhibitory proteins (G(i)/G(o)) within the nucleus acumb

151 atment caused a rapid abrogation of the ATGL inhibitory protein G0S2.

152 Here, we show that the GSK3 inhibitory protein GBP/Frat binds kinesin light chain (K

153 anio rerio) retinas, and a guanylate cyclase-inhibitory protein (GCIP) has been shown to be present i

154 Glucokinase (GK) and its inhibitory protein, GK regulatory protein (GKRP), were c

155 hus, the novel activity was termed GlcNAcT-I inhibitory protein (GnT1IP).

156 epelled by cornea, and presence of Robo-Slit inhibitory protein had no effect.

157 and therapy with various systemic complement-inhibitory proteins has been investigated in this model

158 atory and nuclear factor kappa B (NF-kappaB) inhibitory protein, has established pro-proliferative pr

159 beta-Lactamase inhibitory protein-II (BLIP-II) is a potent proteinaceou

160 (a degradation-resistant mutant of NF-kappaB inhibitory protein IkappaB alpha) in myotubes blocked th

161 nhibit NF-kappaB activity by stabilizing the inhibitory protein IkappaB alpha, raising the possibilit

162 nd by silencing and overexpressing NF-kappaB inhibitory protein IkappaB expression, we demonstrate th

163 t proceeded through specific isoforms of the inhibitory protein IkappaB mediated these diverse respon

164 Expression of the NF-kappaB inhibitory protein, IkappaB alpha, was increased in old

165 e phosphorylation and destabilization of its inhibitory protein, IkappaB-alpha.

166 d degradation, and reduced mRNA level of the inhibitory protein IkappaBA followed by the translationa

167 lB dissociates from its interaction with the inhibitory protein IkappaBalpha and binds to the promote

168 ctor NF-kappaB involves its release from the inhibitory protein IkappaBalpha in the cytoplasm and sub

169 ylation of nuclear factor-kappaB (NF-kappaB) inhibitory protein IkappaBalpha, (c) phosphorylation of

170 ociated with the enhanced degradation of the inhibitory proteins IkappaBalpha and IkappaBbeta but not

171 fect on the phosphorylation of the NF-kappaB inhibitory protein, IkappaBalpha; on the nuclear translo

172 Overexpression of either NF-kappaB inhibitory protein IkappaBalphaDeltaN (a degradation res

173 Finally, the specific role of the cRel/p65 inhibitory protein IkappaBbeta was evaluated.

174 e distinctive physiological roles of the two inhibitory proteins IkappaBbeta and IkappaBalpha.

175 r investigated the use of a novel complement inhibitory protein in a therapeutic paradigm.

176 Acm1 was reported to be a Cdh1-binding and -inhibitory protein in budding yeast.

177 otic effectors such as Bcl-2, p53, and FLICE inhibitory protein in cancer cell anoikis is also discus

178 main protein-like IL-1beta-converting enzyme-inhibitory protein in wild-type MEF but not in PKR-/- ce

179 lt mammalian CNS is limited in part by three inhibitory proteins in CNS myelin: Nogo-A, MAG, and OMgp

180 ve stress, the role of the IkappaB family of inhibitory proteins in modulating this activity remains

181 following injury; regeneration is blocked by inhibitory proteins in myelin, such as myelin-associated

182 saliva and support important roles for these inhibitory proteins in the modulation of PMN function in

183 ory (Th2i) cells that express high levels of inhibitory proteins including IL-10, CTLA-4, and granzym

184 An analysis of cell-cycle inhibitory proteins including p16, p21, and p27 showed t

185 mechanisms such as the repression of growth-inhibitory proteins, including JunD.

186 ed herpesvirus (KSHV) K13/vFLIP (viral Flice-inhibitory protein) induces transcription of numerous ge

187 Treatment with locostatin, an Raf kinase inhibitory protein inhibitor, induced T cell anergy by b

188 ovo H protein synthesis, suggesting that the inhibitory proteins interfere with the wild-type H prote

189 ype III secretion system to deliver a set of inhibitory proteins into the cytoplasm of immune cells.

190 stimulation induced expression of the IRAK-1 inhibitory protein IRAK-M.

191 Phosphorylation of the inhibitory proteins is mediated by an IkappaB kinase (IK

192 c-FLIP (cellular FLICE inhibitory protein) is an enzymatically inactive relativ

193 ain-like interleukin 1beta-converting enzyme inhibitory protein) is an inhibitor of death receptor-me

194 Cellular FLIP (Flice-like inhibitory protein) is critical for the protection again

195 Loss of the liver-enriched inhibitory protein isoform results in increased ATF4 mRN

196 C cells express primarily the liver-enriched inhibitory protein isoform.

197 iated herpesvirus (KSHV)-encoded viral FLICE inhibitory protein K13 interacts with a cytosolic Ikappa

198 main protein-like IL-1beta-converting enzyme inhibitory protein K13 is sufficient to induce spindle c

199 h the expression of KSHV-encoded viral FLICE inhibitory protein K13.

200 tion or on ectopic expression of viral FLICE inhibitory protein K13.

201 idence was provided that PP2A counteracts an inhibitory protein kinase that phosphorylates and inacti

202 (also called ITGB4BP or p27BBP), a ribosome inhibitory protein known to prevent productive assembly

203 ein-like interleukin-1beta-converting enzyme inhibitory protein-like inhibitory protein], and metasta

204 te gene expression, while the liver-enriched inhibitory protein (LIP) isoform negatively regulates la

205 esponse to UV stress, and the liver-enriched inhibitory protein (LIP) isoform of C/EBPbeta, but not t

206 Of the C/EBPbeta isoforms, liver inhibitory protein (LIP) was notably induced and liver-a

207 fied an isoform of C/EBPbeta, liver-enriched inhibitory protein (LIP), as a previously unrecognized t

208 eferred to as liver-enriched transcriptional inhibitory protein (LIP).

209 activating protein (LAP) and liver-enriched inhibitory protein (LIP).

210 e dominant-negative C/EBPbeta isoform, liver inhibitory protein (LIP).

211 oteolytically active complex with FLICE-like inhibitory protein long (FLIP(L), also known as CFLAR),

212 at the antiapoptotic molecule cellular-FLICE-inhibitory protein long isoform [c-FLIP(L)] is necessary

213 ation and proteasomal degradation of the Myc inhibitory protein Mad1.

214 y decreases the expression of the complement inhibitory protein MCP on quiescent EC, but does not ind

215 lways accompanied by upregulation of the p53-inhibitory protein MDM2 and/or phosphorylation of MDM2 a

216 nt phosphorylation of migration and invasion inhibitory protein (MIIP) at Ser303; this phosphorylatio

217 phil-activating peptide (ENA)-78, macrophage inhibitory protein (MIP)-1alpha, MIP-1beta, monocyte che

218 rophage-colony-stimulating factor, migratory inhibitory protein [MIP]-1 alpha , MIP-2, MIP-3 alpha ,

219 , only the phosphorylation of the cell cycle inhibitory proteins MOBKL1A/B is lost entirely in TCR-st

220 analyzed ligand recognition by NLR apoptosis inhibitory protein (NAIP) inflammasomes.

221 Murine NLR family, apoptosis inhibitory protein (Naip)1, Naip2, and Naip5/6 are host

222 ine-rich repeat-containing family, apoptosis inhibitory proteins (NAIPs) activate the nucleotide-bind

223 The NLR family apoptosis inhibitory proteins (NAIPs) bind conserved bacterial lig

224 P-2, -9, and -14 and decreased levels of the inhibitory proteins neurocan and CD44 within the retina.

225 ichiometrically complexed with the NF-kappaB inhibitory protein, NF-kappaB1 p105, and the ubiquitin-b

226 mes, such as the NAIP (NLR family, apoptosis inhibitory protein)/NLRC4 inflammasomes.

227 fter stroke is neutralization of the neurite inhibitory protein Nogo-A.

228 R1) is a promiscuous receptor for the myelin inhibitory proteins Nogo/Nogo-66, myelin-associated glyc

229 d phosphorylation of nuclear factor-kappaB's inhibitory protein, nuclear factor-kappaB translocation

230 Protein kinase C (PKC)-potentiated inhibitory protein of 17 kDa (CPI-17) inhibits myosin li

231 and IKKbeta) that phosphorylate IkappaB, an inhibitory protein of NF-kappaB.

232 hosphorylation of the 17-kDa PKC-potentiated inhibitory protein of type 1 protein phosphatase (CPI-17

233 Mitochondrial-targeted CaMKII inhibitory protein or cyclosporin A, an mPTP antagonist

234 such as the suppressed activity of apoptosis-inhibitory proteins or activation of other signal transd

235 ition of activin A by follistatin, a natural inhibitory protein, or by a specific blocking Ab, result

236 xpression of calpastatin, a specific calpain-inhibitory protein, or small interfering RNA-mediated kn

237 In agreement with MDM2 inhibition, MDM2-inhibitory protein p14ARF was increased in MCF+FIR cells

238 Phosphorylation of cell cycle-inhibitory protein p21(cip1), one of the downstream targ

239 ranscriptional stimulation of the cell-cycle inhibitory protein p21(Waf1/Cip1) Consistently, Foxp1 ac

240 the nucleus, and down-regulation of the CDK2 inhibitory protein p27 in the nucleus.

241 yampholytic C-terminal IDR of the cell cycle inhibitory protein p27(Kip1) (p27).

242 a tolerant state that is enforced by the CDK inhibitory protein p27kip1.

243 The MAD2 inhibitory protein p31(comet) promotes checkpoint inacti

244 ia mutated (ATM) as models to isolate growth inhibitory proteins, peptides and antisense RNAs, and te

245 increased after blocking Cdk2 activity with inhibitory proteins, peptides or small interfering RNA (

246 mic reticulum Ca(2+) ATPase-2 (SERCA-2) pump inhibitory protein phospholamban (PLB), and alters sarco

247 uces protein kinase C-dependent Raf-1 kinase inhibitory protein phosphorylation, sensitization of cho

248 Knockdown of cellular FLICE inhibitory protein potentiates the caspase-dependent pat

249 domain-like interleukin-1-converting enzyme inhibitory protein preventing Fas-mediated apoptosis.

250 in-like interleukin-1 beta-converting enzyme-inhibitory protein), proliferation (c-myc, cyclin D1, an

251 ocks the action of several myelin-associated inhibitory proteins promotes relatively unrestricted reg

252 IL R1, Fas-associated death domain and FLICE-inhibitory protein proteins.

253 its properties, we have re-named ORF145 RNAP Inhibitory Protein (RIP).

254 Raf kinase inhibitory protein (RKIP or PEBP) is an inhibitor of the

255 iously showed that the suppressor Raf kinase inhibitory protein (RKIP) inhibits breast tumour metasta

256 Raf kinase inhibitory protein (RKIP) is a physiologic inhibitor of

257 Raf kinase inhibitory protein (RKIP) negatively regulates the MAP k

258 of metastasis suppressors such as Raf kinase inhibitory protein (RKIP), which inhibits tumor invasive

259 urveillance cancer gene product Raf-1 kinase inhibitory protein (RKIP).

260 ork for the metastasis suppressor Raf kinase inhibitory protein (RKIP).

261 gered swap of protein partners by Raf Kinase Inhibitory Protein (RKIP).

262 Raf kinase inhibitory protein (RKIP/PEBP1), a member of the phospha

263 ts of the metastasis suppressor Raf-1 kinase inhibitory protein (RKIP/PEBP1), we utilized an integrat

264 Raf kinase inhibitory protein (RKIP; also known as phosphatidyletha

265 ar FLICE (FADD-like IL-1b-converting enzyme)-inhibitory protein short, protected cells from the drug

266 in)-like interleukin-1beta-converting enzyme inhibitory protein, short form (c-FLIP-s) blocked enhanc

267 Expression of cellular FLICE-like inhibitory protein-short did not significantly inhibit c

268 CS family genes encoding the STAT signalling inhibitory proteins SOCS1, SOCS3 and CISH were marked by

269 and overrides the accumulation of cell cycle inhibitory proteins, such as p16INK4a.

270 -kappaB and phosphorylation of IkappaB alpha inhibitory protein suggesting that PAP2 signaling involv

271 of a novel, relatively small, broad-acting C inhibitory protein (termed OmCI) from the soft tick Orni

272 ) genomes encode a homolog of cellular FLICE-inhibitory proteins (termed v-FLIP) that activates NF-ka

273 f mast cells by downregulating Srcin1, a Src-inhibitory protein that counteracts FcvarepsilonRI signa

274 ingly, these mutations convert FLASH into an inhibitory protein that reduces in vitro processing effi

275 NF-kappaB is restricted to the cytoplasm by inhibitory proteins that are degraded when specifically

276 its the critical period for learning through inhibitory proteins that suppress axon sprouting and syn

277 activation through increased binding of the inhibitory protein thioredoxin (TXN/TRX-1/Trx), resultin

278 NF-kappaB pathway involves degradation of an inhibitory protein, TNF receptor-associated factor 3 (TR

279 st that the wild-type protein transports the inhibitory protein to the pathway.

280 Here, we tested whether a defect in LYN, an inhibitory protein tyrosine kinase that is implicated in

281 is regulated by the KSHV-encoded viral FLICE inhibitory protein (vFLIP) and by viral IFN regulatory f

282 in-like interleukin-1beta-converting enzyme) inhibitory protein (vFLIP) caused efficient apoptosis in

283 iated herpesvirus (KSHV)-encoded viral FLICE inhibitory protein (vFLIP) enhances IRF4-mediated gene i

284 re, we have revealed the role of viral FLICE-inhibitory protein (vFLIP) in the initiation of PEL and

285 ain-like interleukin-1beta-converting enzyme-inhibitory protein (vFLIP) increased SQSTM1 expression a

286 ted death domain-like IL-1-converting enzyme inhibitory protein (vFLIP) is one of the few viral prote

287 terized the role of KSHV-encoded viral FLICE inhibitory protein (vFLIP) K13 in the modulation of anti

288 iated herpesvirus (KSHV)-encoded viral FLICE inhibitory protein (vFLIP) K13 is a potent activator of

289 a-associated herpesvirus-encoded viral FLICE inhibitory protein (vFLIP) K13 was originally believed t

290 ath domain-like IL- beta 1-converting enzyme inhibitory protein (vFLIP) K13.

291 feron converting enzyme or caspase 8 (FLICE) inhibitory protein (vFLIP) that prevents death receptor-

292 l interleukin-6 (vIL-6) and viral FLICE-like inhibitory protein (vFLIP) transcripts.

293 herpesvirus 8 (HHV-8) encodes a viral FLICE inhibitory protein (vFLIP), called K13, with homology to

294 of B lymphocytes by KSHV-encoded viral FLICE-inhibitory protein (vFLIP).

295 ,5'-oligoadenylate synthetase (OAS-1), Virus inhibitory protein (viperin), ISG15 and ISG56.

296 The level of Socs3, the endogenous Stat3 inhibitory protein, was higher in Gprc5a(+/+) than in Gp

297 eron induced mortality-19 (GRIM-19), a STAT3-inhibitory protein, was isolated as a growth-suppressive

298 change and size exclusion chromatography, an inhibitory protein which exhibits significant similarity

299 upted by Rho guanine nucleotide dissociation inhibitory proteins, which are negative regulators of Cd

300 w that increased expression of the apoptosis inhibitory protein XIAP contributes to anoikis resistanc