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1 al horn neurons by activating the descending inhibitory system.
2 ts GTPCH by engaging the endogenous feedback inhibitory system.
3 modulation system from the LC and the opioid inhibitory system.
4 in the cortex may be especially dependent on inhibitory systems.
5 ation of central descending facilitatory and inhibitory systems.
6 key neurotransmitter of the endogenous pain inhibitory system, acutely inhibits nociceptive transmis
8 otopic synapses is altered and suggests that inhibitory systems are enhanced in the malformed brain.
9 es in primates, we wanted to see whether the inhibitory systems are influenced by changes in cytoarch
10 nted that both the GABAergic and glycinergic inhibitory systems are up-regulated following complete s
11 BH4 and engages the GFRP-dependent feedback inhibitory system; at higher concentrations, DAHP compet
13 emarkably robust and fast-acting glycinergic inhibitory system capable of suppressing calyceal transm
16 gated the role of the vestibular commissural inhibitory system in vestibular compensation (VC, the be
19 to examine the excitability of two different inhibitory systems in the human motor cortex: short inte
21 ings indicate that an imbalanced commissural inhibitory system is a root cause of the severe oculomot
22 function during activation of the brainstem inhibitory system is related to both the descending inhi
25 -apoptotic effects via an endogenous caspase inhibitory system mediated by cellular inhibitor of apop
26 rs may enhance gamma-aminobutyric acid-ergic inhibitory systems or inhibit glutamatergic excitatory n
27 n inhibited by them, thus forming a mutually inhibitory system resembling what electrical engineers c
28 n the calyx of Held by a powerful, precision inhibitory system suggests that the relay function of th
29 s have highlighted an apparent difference in inhibitory systems; synaptic inhibition is thought to be
30 he data in this report, we conclude that the inhibitory system that mediates atonia during the state
31 e key elements of a brainstem-reticulospinal inhibitory system that participates in rapid eye movemen
32 of premotor cortex is weak, possibly due to inhibitory systems that can shape the flow of informatio
34 Thalamic circuits are targeted by two major inhibitory systems: the thalamic reticular nucleus (TRN)
35 in, we explored the contribution of the GABA inhibitory system to A3AR-mediated analgesia using well-
36 The co-ordination of these excitatory and inhibitory systems, together with intrinsic voltage-gate
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