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1 rfusion and multiple organ dysfunction after initial resuscitation.
2 tal single-rescuer bystander CPR, successful initial resuscitation, 24-hour survival, and neurologica
3 rise of early goal-directed therapy for the initial resuscitation and management of severe sepsis an
4 d external defibrillations, would not impair initial resuscitation and outcomes of cardiopulmonary re
5 and alpha1-adrenergic blockade would improve initial resuscitation and postresuscitation myocardial a
6 onary resuscitation with the hypothesis that initial resuscitation and postresuscitation survival wou
9 et volume loading can be reduced markedly by initial resuscitation of large body surface area burn in
10 rterial pressure of at least 65 mm Hg during initial resuscitation of patients with septic shock.
11 her in survivors than in nonsurvivors in the initial resuscitation period and at the hemodynamic nadi
12 ent that attention is being refocused on the initial resuscitation period as a time when significant
13 fluid should be avoided and that, after the initial resuscitation phase, efforts should be made to k
15 -hr infusion of lisofylline was added to the initial resuscitation regimen, the 72-hr survival rate i
19 was able to improve myocardial function and initial resuscitation success after experimental cardiac
20 50% reduction of cardiac output (T1), after initial resuscitation to baseline (T2), and after optimi
21 gh-dose piglets died in the ICU period after initial resuscitation vs. 0 of ten standard-dose piglets
23 ts with severe TBI not in hypovolemic shock, initial resuscitation with either hypertonic saline or h
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