ライフサイエンスコーパス: innate lymphoid cell

1 sets of T cells, NK cells, and other group 1 innate lymphoid cells.

2 st cells, basophils, eosinophils, and type 2 innate lymphoid cells.

3 on by promoting interleukin-13 production by innate lymphoid cells.

4 cutaneous expansion of IL-5-producing type 2 innate lymphoid cells.

5 y increases the frequency of IL-17-producing innate lymphoid cells.

6 lower levels by gamma-delta T cells, NKT and innate lymphoid cells.

7 f proinflammatory cytokines from myeloid and innate lymphoid cells.

8 ene 2 KO mice, which lack T cells but retain innate lymphoid cells.

9 ression of IL-22 and the presence of group 3 innate lymphoid cells.

10 okine that stimulates production of IL-22 by innate lymphoid cells.

11 -CCR2 chemokine axis and by IL-13 expressing innate lymphoid cells.

12 volving eosinophils, mast cells, and group 3 innate lymphoid cells.

13 omato-C3aR(-), except some LP-derived type 3 innate lymphoid cells.

14 for proper development of adaptive, but not innate, lymphoid cells.

15 ler (NK), lymphoid tissue inducer (LTi), and innate lymphoid cell 1 (ILC1) cells, but not ILC2 or ILC

16 killer T cells, mast cells, eosinophils, and innate lymphoid cells, although the cells required for m

17 Type 2 innate lymphoid cells and basophils were scarce in BAL f

18 in) by colon tissues, which activated type 2 innate lymphoid cells and dendritic cells to promote dif

19 ciency in the TH17 subset of helper T cells, innate lymphoid cells and gammadelta T cells resulted in

20 mmarize the studies that formally identified innate lymphoid cells and highlight their emerging roles

21 the numbers of eosinophils, IL-13(+) type 2 innate lymphoid cells and IL-13(+)CD4(+) T cells and IL-

22 , including eosinophils, mast cells, group 2 innate lymphoid cells and lymphocytes, which participate

23 helial cells and antigen-presenting cells to innate lymphoid cells and regulatory T cells.

24 lls resulted in loss of AHR-dependent type 3 innate lymphoid cells and T helper 17 cells and increase

25 Host & Microbe, Abt et al. demonstrated that innate lymphoid cells and the effector cytokine IFN-gamm

26 nism that engages unconventional type-1-like innate lymphoid cells and type 1 innate-like T cells.

27 important role for vitamin A in controlling innate lymphoid cells and, consequently, postnatal forme

28 d by recruitment of Th2 lymphocytes, group 2 innate lymphoid cells, and eosinophils to the lung.

29 (ER) stress, required extrinsic signals from innate lymphoid cells, and limited bacterial disseminati

30 Conversely, eosinophils, Th2 T cells, type 2 innate lymphoid cells, and possibly Foxp3+ Tregs protect

31 IL-5 and IL-13 coming from Th2 cells, type 2 innate lymphoid cells, and probably mast cells.

32 airway epithelial cells, macrophages, type 2 innate lymphoid cells, and TH2 cells along with increase

33 NK cell developmental intermediates, non-NK innate lymphoid cells, and the capacity for NK cells to

34 Finally, we provide evidence that group 2 innate lymphoid cells are a source of IL-13, which promo

35 h abundant IL-22 production, whereas group 1 innate lymphoid cells are accumulated in chronic inflamm

36 We further demonstrate that group 3 innate lymphoid cells are an innate immune source of IL-

37 Innate lymphoid cells are functionally diverse subsets o

38 Group 3 innate lymphoid cells are implicated in intestinal homeo

39 Innate lymphoid cells are the most recently identified c

40 ng cells, including Th17, gamma/delta T, and innate lymphoid cells, are differentially distributed al

41 ansion of eosinophils, Th2 cells, and type 2 innate lymphoid cells, associated with an increase in ty

42 Adipose type 1 innate lymphoid cells (AT1-ILCs) promote pro-inflammator

43 , ST2 deletion led to an overall increase in innate lymphoid cells (CD45(+)lin(-)CD25(+) cells) and I

44 ctivated myeloid dendritic cells and group 2 innate lymphoid cells compared with mature TSLP.

45 hocytes, NK T cells, macrophages, and type 2 innate lymphoid cells compared with wild-type control mi

46 Natural killer (NK) cells, a subset of the innate lymphoid cell compartment, are protective in vira

47 aluated whether AHR antagonism could promote innate lymphoid cell differentiation from hESCs.

48 ase deficiency reduced the number of group 2 innate lymphoid cells during allergen challenge but was

49 cells, goblet cells, eosinophils, and type 2 innate lymphoid cells during parasite colonization.

50 teins, damage-associated molecular patterns, innate lymphoid cells, epithelial-derived cytokines and

51 Type 2 innate lymphoid cells from polyps produce IL-5 and IL-13

52 opmentally divergent lineages, type 1 helper innate lymphoid cells (hILC1s) and conventional NK cells

53 cells and in ex vivo CD27(-)CD4(+) cells and innate lymphoid cell (ILC) 2 from patients with grass po

54 levels, pulmonary IL-33 levels, and IL-13(+) innate lymphoid cell (ILC) and TH2 cell numbers but simi

55 The current model of murine innate lymphoid cell (ILC) development holds that mouse

56 s regulate early T cell progenitor (ETP) and innate lymphoid cell (ILC) development remains unclear.

57 Commitment to the innate lymphoid cell (ILC) lineage is determined by Id2,

58 ntial progenitor population specified toward innate lymphoid cell (ILC) lineages, but their relations

59 owledge on the different effector T-cell and innate lymphoid cell (ILC) lineages, it is clear that th

60 Little is known about innate lymphoid cell (ILC) populations in the human gut,

61 scriptional regulator for the development of innate lymphoid cell (ILC) progenitors, remains highly e

62 promoted the development of tissue-resident innate lymphoid cell (ILC) subsets.

63 Diverse innate lymphoid cell (ILC) subtypes have been defined on

64 s) and NK cells, but also several additional innate lymphoid cell (ILC) types.

65 Innate lymphoid cells (ILC) are a heterogeneous group of

66 The signals that maintain tissue-resident innate lymphoid cells (ILC) in different microenvironmen

67 Innate lymphoid cells (ILC) play an important role in ma

68 ecirculating classical NK (cNK) cells, liver innate lymphoid cells (ILC) type 1 (ILC1s) have been sho

69 ing viral infection regulates the balance of innate lymphoid cells (ILC), a diverse class of lymphocy

70 ng other lymphocytes, specifically Th cells, innate lymphoid cells (ILC), and gammadelta T cells.

71 Recently, several groups of innate lymphoid cells (ILC), distinct from NK cells in d

72 Group 2 innate lymphoid cells (ILC-2s) regulate immune responses

73 as a critical negative regulator of group 2 innate lymphoid cells (ILC-2s).

74 Here, we show that tissue-resident type 1 innate lymphoid cells (ILC1) serve an essential early ro

75 Among the features that distinguish type 1 innate lymphoid cells (ILC1s) from natural killer (NK) c

76 , recombinant HpARI abrogated IL-33, group 2 innate lymphoid cell (ILC2) and eosinophilic responses t

77 that Bcl11b has a role in specifying type II innate lymphoid cell (ILC2) identity and blocks their co

78 ammatory gene expression, and TH and group 2 innate lymphoid cell (ILC2) responses.

79 rs of eosinophils and both type 2 and type 3 innate lymphoid cells (ILC2 and ILC3), specifically in t

80 tor led to deregulated activation of group 2 innate lymphoid cells (ILC2 cells) and infection-associa

81 Group 2 innate lymphoid cells (ILC2 cells) are important for typ

82 While type 2 innate lymphoid cells (ILC2 cells) are the dominant inna

83 Type-2 innate lymphoid cells (ILC2) are a prominent source of t

84 Group 2 innate lymphoid cells (ILC2) are important in effector f

85 The recent discovery of group 2 innate lymphoid cells (ILC2) has increased our understan

86 Group 2 innate lymphoid cells (ILC2) have been implicated in the

87 L233 increased the proportion of ST2-bearing innate lymphoid cells (ILC2) in blood and kidneys, and a

88 IL-5 production by type 2 cytokine-producing innate lymphoid cells (ILC2) in the FALC.

89 Group 2 innate lymphoid cells (ILC2) include IL-5- and IL-13-pro

90 Type 2 innate lymphoid cells (ILC2) mediate inflammatory immune

91 Type 2 innate lymphoid cells (ILC2) share cytokine and transcri

92 6.C3(Cg)-Rorasg/sg mice deficient in group 2 innate lymphoid cells (ILC2), and C57BL/6 wild-type mice

93 nd IL-13, secreted by CD4(+) Th2 and group 2 innate lymphoid cells (ILC2), but whether certain metabo

94 Here we show that type 2 innate lymphoid cells (ILC2), important mediators of bar

95 tural helper (NH) cells, a member of group 2 innate lymphoid cells (ILC2), to secrete Th2 type-cytoki

96 ived suppressor cells (M-MDSCs), and group 2 innate lymphoid cells (ILC2).

97 It also caused a reduction in innate lymphoid cell, ILC2, and IL-9(+) and IL-13(+) ILC

98 associated with the expansion of lung type 2 innate lymphoid cells (ILC2s) and are dependent on IL-13

99 Group 2 innate lymphoid cells (ILC2s) and CD4(+) type 2 helper T

100 IL)-13 production by tissue-resident group 2 innate lymphoid cells (ILC2s) and recruited type 2 helpe

101 Group 2 innate lymphoid cells (ILC2s) and regulatory T (Treg) ce

102 or ST2, which is highly expressed on group 2 innate lymphoid cells (ILC2s) and T helper 2 (Th2) cells

103 Type-2 innate lymphoid cells (ILC2s) and the acquired CD4(+) Th

104 pacity of RSV infection to stimulate group 2 innate lymphoid cells (ILC2s) and the associated mechani

105 Group 2 innate lymphoid cells (ILC2s) and type 2 helper T cells

106 Type 2 innate lymphoid cells (ILC2s) and type 3 innate lymphoid

107 Type 2 innate lymphoid cells (ILC2s) are a newly identified sub

108 Group 2 innate lymphoid cells (ILC2s) are a potential innate sou

109 Group 2 innate lymphoid cells (ILC2s) are a recently identified

110 Group 2 innate lymphoid cells (ILC2s) are a subset of ILCs that

111 Group 2 innate lymphoid cells (ILC2s) are effector cells within

112 Interleukin (IL)-33-dependent group 2 innate lymphoid cells (ILC2s) are enriched at barrier su

113 Group 2 innate lymphoid cells (ILC2s) are important effector cel

114 Group 2 innate lymphoid cells (ILC2s) are involved in human dise

115 Group 2 innate lymphoid cells (ILC2s) are involved in the initia

116 Group 2 innate lymphoid cells (ILC2s) are key regulators of type

117 Group 2 innate lymphoid cells (ILC2s) are often found associated

118 Type 2 innate lymphoid cells (ILC2s) are tissue sentinel mediat

119 entified interleukin (IL)-9-producing type 2 innate lymphoid cells (ILC2s) as the mediators of a mole

120 Type 2 innate lymphoid cells (ILC2s) both contribute to mucosal

121 ently expressed during development of type 2 innate lymphoid cells (ILC2s) but is not present at the

122 ell established, the newly identified type 2 innate lymphoid cells (ILC2s) can also contribute to orc

123 Group 2 innate lymphoid cells (ILC2s) can regulate adaptive immu

124 RATIONALE: Newly characterized type 2 innate lymphoid cells (ILC2s) display potent type 2 effe

125 Group 2 innate lymphoid cells (ILC2s) expand in the lungs of mic

126 versely associated with the number of type 2 innate lymphoid cells (ILC2s) expressing IL-33Ralpha and

127 Group 2 innate lymphoid cells (ILC2s) have recently been shown t

128 Ts induced eosinophilia and expanded group 2 innate lymphoid cells (ILC2s) in aspirin-exacerbated res

129 SA, histology, and real-time PCR; and type 2 innate lymphoid cells (ILC2s) in lung single-cell prepar

130 murine models of allogeneic BMT, that type 2 innate lymphoid cells (ILC2s) in the lower GI tract are

131 Finally, group 2 innate lymphoid cells (ILC2s) in the lungs showed robust

132 ously undescribed deficit in c-Kit(+) type 2 innate lymphoid cells (ILC2s) in W/W(v) mice.

133 our observations in prostate cancer.Group 2 innate lymphoid cells (ILC2s) modulate inflammatory and

134 The recently identified type 2 innate lymphoid cells (ILC2s) play significant roles in

135 Type 2 innate lymphoid cells (ILC2s) produce large amounts of T

136 Group 2 innate lymphoid cells (ILC2s) produce type 2 cytokines,

137 Group 2 innate lymphoid cells (ILC2s) promote allergic inflammat

138 Type 2 innate lymphoid cells (ILC2s) promote anti-helminth resp

139 In innate immunity, IL-33 and group 2 innate lymphoid cells (ILC2s) provide an essential axis

140 Group 2 innate lymphoid cells (ILC2s) regulate tissue inflammati

141 Type 2 innate lymphoid cells (ILC2s) represent an important typ

142 Type 2 innate lymphoid cells (ILC2s) resemble TH2 cells and pro

143 Type 2 innate lymphoid cells (ILC2s) resemble type 2 helper (Th

144 Group 2 innate lymphoid cells (ILC2s) reside in multiple organs

145 Group 2 innate lymphoid cells (ILC2s) robustly produce IL-5 and

146 Elimination of T cells and type 2 innate lymphoid cells (ILC2s) through lethal irradiation

147 Recently, a key role for type 2 innate lymphoid cells (ILC2s) was linked to asthma patho

148 Resident intestinal type 2 innate lymphoid cells (ILC2s) were identified as the maj

149 y, smoke decreased ST2 expression on group 2 innate lymphoid cells (ILC2s) while elevating ST2 expres

150 Type 2 innate lymphoid cells (ILC2s), an innate source of the t

151 ons including mast cells, basophils, group 2 innate lymphoid cells (ILC2s), and a subset of tissue-re

152 IL-33 initiates the accumulation of group 2 innate lymphoid cells (ILC2s), eosinophils, and alternat

153 nd IL-13 are prominently secreted by group 2 innate lymphoid cells (ILC2s), which are stimulated by t

154 Group 2 innate lymphoid cells (ILC2s), which promote tissue eosi

155 by TH2 cells and innate responses by group 2 innate lymphoid cells (ILC2s), with these latter being w

156 e correlated with reduced numbers of group 2 innate lymphoid cells (ILC2s).

157 the role of the recently discovered group 2 innate lymphoid cells (ILC2s).

158 atory T (Treg) cells and the emerging type 2 innate lymphoid cells (ILC2s).

159 cytokine (IL-5, IL-13) production by type 2 innate lymphoid cells (ILC2s).

160 via the activity of islet-associated group 2 innate lymphoid cells (ILC2s).

161 type 2 inflammation, eosinophils and group 2 innate lymphoid cells (ILC2s).

162 locked cNK development and supported group 3 innate lymphoid cell (ILC3) differentiation.

163 promoting IL-22 production from the group 3 innate lymphoid cell (ILC3) in an aryl hydrocarbon recep

164 the right vagus decreased peritoneal group 3 innate lymphoid cell (ILC3) numbers and altered peritone

165 ing dendritic-cell-derived IL-10 and group 3 innate lymphoid cell (ILC3)-derived IL-22.

166 Here, we demonstrate that group 3 innate lymphoid cell (ILC3)-intrinsic expression of majo

167 Intestinal T cells and group 3 innate lymphoid cells (ILC3 cells) control the compositi

168 of increased numbers of RORgammat(+) group 3 innate lymphoid cells (ILC3) correlates with an increase

169 licit the activation of RORgammat(+) group 3 innate lymphoid cells (ILC3) in an IL-1beta-dependent ma

170 Lymphoid organs are also home to type 3 innate lymphoid cells (ILC3), innate effector cells esse

171 e identified an SFB-dependent role of type 3 innate lymphoid cells (ILC3), which secreted IL-22 that

172 However, ROR-gammat-dependent group 3 innate lymphoid cells ILC3s provide essential immunity a

173 Type 3 innate lymphoid cells (ILC3s) and enteric glia, an essen

174 ells (preFDCs) that require specific group 3 innate lymphoid cells (ILC3s) and LTbeta for their expan

175 The recently defined group 3 innate lymphoid cells (ILC3s) are critical for maintenan

176 Group 3 innate lymphoid cells (ILC3s) are important for intestin

177 Group 3 innate lymphoid cells (ILC3s) are important regulators o

178 Type 3 innate lymphoid cells (ILC3s) are involved in maintenanc

179 Type 3 innate lymphoid cells (ILC3s) are regulators of homeosta

180 genesis, but where these specialized group 3 innate lymphoid cells (ILC3s) develop remains unclear.

181 Group 3 innate lymphoid cells (ILC3s) expressing the transcripti

182 e 2 innate lymphoid cells (ILC2s) and type 3 innate lymphoid cells (ILC3s) have been implicated, resp

183 Group 3 innate lymphoid cells (ILC3s) have demonstrated roles in

184 results in depletion of intrathymic group 3 innate lymphoid cells (ILC3s) necessary for thymic regen

185 The IL-23-driven tissue-resident group 3 innate lymphoid cells (ILC3s) play essential roles in in

186 peptide:MHCII by RORgamma-expressing group 3 innate lymphoid cells (ILC3s), which are enriched within

187 se lung were the recently identified group 3 innate lymphoid cells (ILC3s).

188 Innate lymphoid cell (ILCs) subsets differentially popul

189 Innate lymphoid cells (ILCs) 'preferentially' localize i

190 essor Id2 is constitutively expressed in all innate lymphoid cells (ILCs) and is required for their d

191 The precise lineage relationship between innate lymphoid cells (ILCs) and lymphoid tissue-inducer

192 Innate lymphoid cells (ILCs) are a family of immune effe

193 Innate lymphoid cells (ILCs) are a family of innate immu

194 Innate lymphoid cells (ILCs) are a growing family of imm

195 Innate lymphoid cells (ILCs) are a heterogeneous group o

196 Innate lymphoid cells (ILCs) are a new family of immune

197 Innate lymphoid cells (ILCs) are a recently identified p

198 Innate lymphoid cells (ILCs) are critical for maintainin

199 Innate lymphoid cells (ILCs) are critical mediators of m

200 Innate lymphoid cells (ILCs) are early responders to ent

201 Innate lymphoid cells (ILCs) are effectors and regulator

202 Innate lymphoid cells (ILCs) are emerging as important r

203 Innate lymphoid cells (ILCs) are important regulators in

204 Innate lymphoid cells (ILCs) are increasingly acknowledg

205 Innate lymphoid cells (ILCs) are innate immune cells tha

206 Innate lymphoid cells (ILCs) are known as first responde

207 Innate lymphoid cells (ILCs) are lymphocyte-like cells t

208 Innate lymphoid cells (ILCs) are part of a heterogeneous

209 Innate lymphoid cells (ILCs) are rapidly-responding cell

210 Innate lymphoid cells (ILCs) are the most recently disco

211 Innate lymphoid cells (ILCs) are tissue-resident "first

212 Innate lymphoid cells (ILCs) are tuned to quickly respon

213 In this study, we show that IL-7R-dependent innate lymphoid cells (ILCs) block LIP of CD8(+) T cells

214 Innate lymphoid cells (ILCs) communicate with other haem

215 Innate lymphoid cells (ILCs) contribute to barrier immun

216 Innate lymphoid cells (ILCs) contribute to host defence

217 itic cells (DCs), but its role in regulating innate lymphoid cells (ILCs) during fetal and adult life

218 Innate lymphoid cells (ILCs) function to protect epithel

219 Innate lymphoid cells (ILCs) functionally resemble T lym

220 In particular, the discovery of innate lymphoid cells (ILCs) has opened entirely new ave

221 Innate lymphoid cells (ILCs) have an important role in t

222 Innate lymphoid cells (ILCs) have been classified into "

223 In recent years, innate lymphoid cells (ILCs) have emerged as innate corr

224 ht to determine the potential involvement of innate lymphoid cells (ILCs) in allergic airway disease

225 al models have highlighted the importance of innate lymphoid cells (ILCs) in multiple immune response

226 a cardinal role of T-bet-dependent NKp46(+) innate lymphoid cells (ILCs) in the initiation of CD4(+)

227 The recognized diversity of innate lymphoid cells (ILCs) is rapidly expanding.

228 Innate lymphoid cells (ILCs) patrol environmental interf

229 Innate lymphoid cells (ILCs) play a central role in the

230 Innate lymphoid cells (ILCs) play critical roles in immu

231 er cells (Th cells), and recently identified innate lymphoid cells (ILCs) play important roles in hos

232 Innate lymphoid cells (ILCs) play key roles in host defe

233 sented by a subset of fetal CD103(+) group 3 innate lymphoid cells (ILCs) producing high levels of IL

234 Recent findings that innate lymphoid cells (ILCs) regulate adaptive T cell re

235 r events that drive the early development of innate lymphoid cells (ILCs) remain poorly understood.

236 Innate lymphoid cells (ILCs) represent innate versions o

237 Subsets of innate lymphoid cells (ILCs) reside in the mucosa and re

238 Innate lymphoid cells (ILCs) serve as sentinels in mucos

239 Distinct groups of innate lymphoid cells (ILCs) such as ILC1, ILC2, and ILC

240 is indispensable for the development of all innate lymphoid cells (ILCs) that express the interleuki

241 59-induced, envelope (Env)-dependent mucosal innate lymphoid cells (ILCs) that produce interleukin (I

242 Innate lymphoid cells (ILCs) type 3, also known as lymph

243 was required for the expression of IL-22 in innate lymphoid cells (ILCs) upon T. gondii infection.

244 nd transcriptional characteristics of sorted innate lymphoid cells (ILCs) were defined by using quant

245 The signals guiding differentiation of innate lymphoid cells (ILCs) within tissues are not well

246 ell type of the innate immune system, termed innate lymphoid cells (ILCs), has been characterized in

247 Additionally, innate lymphoid cells (ILCs), important producers of cyt

248 Innate lymphoid cells (ILCs), including NK cells, contri

249 ears has led to the formal identification of innate lymphoid cells (ILCs), increased the understandin

250 t PGE2-EP4 signaling acts directly on type 3 innate lymphoid cells (ILCs), promoting their homeostasi

251 ed TGF-beta1 displayed a reduction in type 2 innate lymphoid cells (ILCs), resulting in suppression o

252 an the innate-adaptive continuum and include innate lymphoid cells (ILCs), unconventional T cells (e.

253 ng the GR is selectively deleted in NKp46(+) innate lymphoid cells (ILCs), we demonstrated a major ro

254 12(+) endothelial cells and Cxcr4(+) gastric innate lymphoid cells (ILCs).

255 of these vitamins on the recently identified innate lymphoid cells (ILCs).

256 elta T cells, NKT cells, and newly described innate lymphoid cells (ILCs).

257 mediastinal lymph nodes, comprised mainly of innate lymphoid cells (ILCs); approximately 90% of IL-17

258 NK cells are innate lymphoid cells important for immune surveillance,

259 Lee et al. (2015) demonstrate that resident innate lymphoid cells in subcutaneous fat generate and a

260 ations have an increased frequency of type 2 innate lymphoid cells in the skin in comparison with con

261 expression of IL-33, a stimulator of type II innate lymphoid cells, in lung epithelial cells was asso

262 cell-derived cytokines that activate group 2 innate lymphoid cells, induce migration and activation o

263 disease pathology, mucus production, group 2 innate lymphoid cell infiltration, IL-5 and IL-13 produc

264 e lineages, including natural killer T cell, innate lymphoid cell, mucosal-associated invariant T cel

265 sident population in other organs, including innate lymphoid cells, natural killer cells, natural kil

266 The discovery of tissue-resident innate lymphoid cell populations effecting different for

267 We discuss the identification of a common innate lymphoid cell precursor characterized by transien

268 PLZF expression at the innate lymphoid cell precursor stage has a long-range ef

269 lin-induced IL-22 expression, which required innate lymphoid cells, prevented microbiota encroachment

270 In patients with allergy and asthma, group 2 innate lymphoid cells produce high amounts of IL-5 and I

271 d a more recently described pathway in which innate lymphoid cells produce interleukin-22 (IL-22) in

272 Type 3 innate lymphoid cells producing predominantly GM-CSF are

273 common helper innate lymphoid progenitor and innate lymphoid cell progenitor compartments.

274 genitor that was essentially identical to an innate lymphoid cell progenitor.

275 d expressed on natural killer (NK) cells and innate lymphoid cells, recognizes PDGF-DD.

276 predominantly TC1, TH1, and TH17 cells), and innate lymphoid cells recruited from the circulation.

277 controlled by the microbiota through group 3 innate lymphoid cells, STAT3 (signal transducer and acti

278 PRKCQ gene expression was assessed in innate lymphoid cell subsets purified from human PBMCs a

279 kins derived from T-helper-2 (Th2) cells and innate lymphoid cells, such as interleukins 4, 5, and 13

280 ) T cells, but not natural killer T cells or innate lymphoid cells, suggesting a TH cell-dependent ph

281 tion of dendritic cell, mast cell, basophil, innate lymphoid cell, T-cell, and B-cell responses to al

282 owel in CD, we observed B cells and apparent innate lymphoid cells that had invaded the lymphatic ves

283 Natural killer cells constitute potent innate lymphoid cells that play a major role in both tum

284 ight be orchestrated by TH2 cells and type 2 innate lymphoid cells though release of IL-33 from epith

285 pithelial cells; it rapidly activates type 2 innate lymphoid cells to produce IL-13 and IL-5.

286 tween epithelial cells, dendritic cells, and innate lymphoid cells translates to T-cell outcomes, wit

287 PH are conventional natural killer cells and innate lymphoid cells type 1.

288 In obese mice, ozone increased lung IL-13+ innate lymphoid cells type 2 (ILC2) and IL-13+ gammadelt

289 Unexpectedly, innate lymphoid cells were found to have a potent influe

290 tract (vagina and cervix), whereas APCs and innate lymphoid cells were mainly located in the upper t

291 airway inflammation, mast cells, and group 3 innate lymphoid cells were more enriched in adult-onset

292 , IL-17A, and IL-22, all hallmarks of type 3 innate lymphoid cells, were expanded in the blood of pat

293 Natural killer (NK) cells are innate lymphoid cells which mediate resistance against p

294 functions of specific subsets of T cells and innate lymphoid cells, which are key drivers of inflamma

295 PP IgA class switching requires innate lymphoid cells, which promote lymphotoxin-beta re

296 ng tissue injury in sepsis, activates type 2 innate lymphoid cells, which promote polarization of M2

297 airways increased lung IL-5-producing type 2 innate lymphoid cells, which required protease-activated

298 ere the focus of the 2(nd)EMBO Conference on Innate Lymphoid Cells, which took place from November 30

299 actor PLZF, and the lineage relationships of innate lymphoid cells with conventional natural killer c

300 l sources of alloantigens, the cross talk of innate lymphoid cells with damaged epithelia and with th