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1 eratinize as they move up the hair shaft and inner root sheath.
2 es of the hair follicle bud, and then in the inner root sheath.
3 itotic precursor cells of the hair shaft and inner root sheath.
4 he hair follicle gives rise to the fiber and inner root sheath.
5 ntrast, hair follicles lost integrity of the inner root sheath and hair cuticle without apparent elev
6 in the upper epidermis, the differentiating inner root sheath and hair shaft and in the most mature
8 cross-bridging protein that functions in the inner root sheath and perhaps in other specialized epith
9 agen, NF-kappaB activity was observed in the inner root sheath and unilaterally clustered in the HF m
10 Edar protein were expressed in the outer and inner root sheaths and later in the secondary hair germ.
11 pressed in the hair follicle, especially the inner root sheath, and that the Q30R substitution affect
12 expressed in epidermal suprabasal cells, the inner root sheath, and the innermost layers of the outer
13 (cuticle, cortex, and medulla), one for the inner root sheath, and the third for the outer root shea
15 n (THH) is a major structural protein of the inner root sheath cells and medulla layer of the hair fo
16 rotein of the cornified cell envelope of the inner root sheath cells by becoming cross-linked to seve
19 ession and induce expression in the channel (inner root sheath) cells surrounding the hair shaft.
21 , abnormally long anagen follicles, retained inner root sheath, hair fiber perforation of the anagen
23 n hair matrix; as a consequence, medulla and inner root sheath (IRS) differentiation is impaired.
25 find that mK75 regulation parallels that of inner root sheath (IRS) markers, with expression onset a
26 gene expression to the keratinocytes of the inner root sheath (IRS) of the hair follicle, whereas a
27 how the equally important hair channel, the inner root sheath (IRS), develops in concert to shape an
28 F infundibulum, in the HF matrix, and in the inner root sheath (IRS), whereas the dermal papilla (DP)
30 ifferentiation of the hair shaft but not the inner root sheath is severely impaired in Msx2-Noggin tr
31 and to the head and tail end domains of the inner root sheath keratin intermediate filament chains.
32 ne glands and was stronger than DAX-1 in the inner root sheath, matrix cells, and dermal papilla cell
33 hair follicle with weaker expression in the inner root sheath, matrix cells, and dermal papilla cell
34 separation between the outer root sheath and inner root sheath of the hair follicle, at the plane of
35 promoter activity in the companion layer and inner root sheath of the hair follicle, SG, and epidermi
37 ibulum, isthmus, and of Henle's layer of the inner root sheath of the lower anagen hair follicle and
40 icles and regrowth of the hair shaft and the inner root sheath resulted in subsequent severe hair los
41 pecia owing to failure of the hair shaft and inner root sheath to form, which is caused by the abnorm
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