ライフサイエンスコーパス: inositol hexaphosphate

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1 w oxygen affinity state (i.e. treatment with inositol hexaphosphate).

2 ate upon addition of an allosteric effector, inositol hexaphosphate.

3 such as protons, 2,3-biphosphoglycerate, and inositol hexaphosphate.

4 Inositol-trisphosphate and inositol-hexaphosphate also partially supported activati

5 The addition of inositol hexaphosphate, an allosteric effector, causes r

6 In the presence of inositol hexaphosphate and bezafibrate (or derivatives),

7 ons, semihemoglobins responded to effectors (inositol hexaphosphate and L35) by decreasing the affini

8 on state upon adding an allosteric effector, inositol hexaphosphate, and reducing the temperature.

9 upon the addition of an allosteric effector, inositol hexaphosphate, and/or by reducing the temperatu

10 exhibits a stronger response to 2,3-BPG and inositol hexaphosphate as compared to Hb F.

11 T binds to a single site and is displaced by inositol hexaphosphate at a 1:1 mol ratio, indicating th

12 Since inositol hexaphosphate binding to syndecan-4 does not pr

13 ophosphate diesters by P NMR and accumulated inositol hexaphosphate by XANES were observed in surface

14 tions of low pH (pH 6.35) in the presence of inositol-hexaphosphate, COHb assumes an altered R-state.

15 nto a sample of deoxy-HbA with the effector, inositol hexaphosphate, encapsulated in a porous sol-gel

16 indicate a differential allosteric effect by inositol hexaphosphate for HbC approximately HbS > HbA.

17 Blocking the 2, 3-DPG binding site with inositol hexaphosphate (IHP) resulted in a selective red

18 ctors, 2,3-bisphosphoglycerate (2,3-BPG) and inositol hexaphosphate (IHP), is decreased relative to t

19 The allosteric effector, inositol hexaphosphate (IHP), promotes fiber formation,

20 ence and presence of an allosteric effector, inositol hexaphosphate (IHP), using 15N-1H residual dipo

21 ence and presence of an allosteric effector, inositol hexaphosphate (IHP), using a stretched polyacry

22 ave been measured at pH 7 in the presence of inositol hexaphosphate (IHP).

23 pH 6 and pH 8 in the presence and absence of inositol hexaphosphate (IHP).

24 nd in the absence of an allosteric effector, inositol hexaphosphate (IHP).

25 nding of a heterotropic allosteric effector, inositol hexaphosphate (IHP).

26 In the presence of inositol hexaphosphate, IHP, none of them exhibits coope

27 resence and absence of the organic phosphate inositol hexaphosphate, IHP.

28 ow-concentration Hb solution when induced by inositol-hexaphosphate in the presence of polyethylene g

29 Hydrolysis of inositol hexaphosphate (InsP6 , the substrate for phytas

30 Inositol hexaphosphate (InsP6) is the most abundant inos

31 ogen Ralstonia solanacearum, in complex with inositol hexaphosphate (InsP6), acetyl-coenzyme A (AcCoA

32 hat the Dbp5 ATPase is activated by Gle1 and inositol hexaphosphate (IP(6)).

33 Here, we assessed the efficacy of inositol hexaphosphate (IP6) against invasive human pros

34 Inositol hexaphosphate (IP6) causes G(1) arrest and incr

35 The total phenolic compounds (TPC), inositol hexaphosphate (IP6), reduced (GSH) and oxidised

36 ast Ino80 complexes and is also inhibited by inositol hexaphosphate (IP6).

37 possess enhanced cooperativity in vitro when inositol hexaphosphate is present.

38 strengthened when the effector molecule IHP (inositol hexaphosphate) is added to deoxy-desArgalpha141

39 4/beta15) of both chains, 3) displacement by inositol hexaphosphate of the Hb-bound 8-hydroxy-1,3,6-p

40 decreased capacity for the synthesis of myo-inositol hexaphosphate (phytic acid) and a concomitant i

41 nd to a novel peptide-based inhibitor and to inositol hexaphosphate suggests a molecular basis of sub

42 Addition of the allosteric modulator inositol hexaphosphate to increase Hb P50 and the thiol

43 al change by adding the allosteric effector, inositol hexaphosphate, to the fluoromet-Hb sample.

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