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1 in valproate leads to decreased synthesis of inositol monophosphate.
2 erase that cleaves the cyclic bond of cyclic inositol monophosphate.
3 selective kinetic resolution of a protected inositol monophosphate.
4 and LTB(4) induced Ca(2+) and intracellular inositol monophosphate accumulation, respectively, highl
5 es that, by trapping inositol in the form of inositol monophosphates and certain inositol polyphospha
6 al phosphatase that has dual activity toward inositol monophosphates and fructose 1,6-bisphosphate.
7 yme possessing the ability to hydrolyze both inositol monophosphates and Gal-1-P with equal efficienc
9 s capacity to release free myo-inositol from inositol monophosphates generated from receptor-linked a
10 d for hormone binding and induction of cAMP, inositol monophosphate, inositol bisphosphate, and inosi
11 valproate does not increase accumulation of inositol monophosphates, inositol bisphosphates, or inos
12 rylates myo-inositol to produce multiple myo-inositol monophosphates, Ins1/3P, Ins4/6P and possibly I
13 cid resulted in a marked accumulation of [3H]inositol monophosphate (InsP1) and inositol-1,4,5-trisph
14 activation by measuring accumulation of [3H] inositol monophosphates (IP) in rats pre-labeled with [3
16 y-EM293 cells with TSH and measuring cAMP or inositol monophosphate (IP1) production, a measure of ph
18 cAMP, KB = 4.9 and 5.9 nM, respectively) and inositol monophosphate (IP1, KB = 0.6 and 16 nM, respect
19 ol-1,4,5-trisphosphate, which is degraded to inositol monophosphate (IP1; phosphoinositide signaling)
20 the synthesis of free inositol from various inositol monophosphates, is encoded by a small multigene
22 lso effective in stimulating basal tritiated inositol monophosphate production in the neonatal rat ce
24 overlay assays suggested the PH domain binds inositol monophosphates, surface plasmon resonance demon
25 phatase (IMPase) catalyzes the hydrolysis of inositol monophosphate to inorganic phosphate and inosit
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