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1 also exhibited 3-phosphatase activity toward inositol 1,3,4,5-tetrakisphosphate.
2 ert the second messenger Ins(1,4,5)P(3) into inositol 1,3,4,5-tetrakisphosphate.
3                            Microinjection of inositol 1,3,4,5-tetrakisphosphate ((1,3,4,5)IP4), the i
4 sitol-3,4,5-trisphosphate 5'-phosphatase and inositol-1,3,4,5-tetrakisphosphate 5'-phosphatase activi
5 Minpp1 fusion protein cleaved phosphate from inositol (1,3,4,5)-tetrakisphosphate and para-nitropheny
6 lso show how IP(5) 2-K discriminates between inositol 1,3,4,5-tetrakisphosphate and 3,4,5,6-tetrakisp
7 sphate from inositol 1,4,5-trisphosphate and inositol 1,3,4,5-tetrakisphosphate and thus serves as a
8  both inositol 1,3,4,6-tetrakisphosphate and inositol 1,3,4,5-tetrakisphosphate as products in a rati
9 o synaptotagmins, JFC1 was unable to bind to inositol 1,3,4,5-tetrakisphosphate but did bind to phosp
10 th their relative ability to dephosphorylate inositol (1,3,4,5)-tetrakisphosphate in vitro, and to su
11 o refutes the second messenger role of D-myo-inositol 1,3,4,5-tetrakisphosphate in regulating Fcepsil
12 tivity yet is defective in dephosphorylating inositol 1,3,4,5-tetrakisphosphate in vitro and fails to
13                       Fourth, although D-myo-inositol 1,3,4,5-tetrakisphosphate (Ins(1,3,4, 5)P(4)) m
14 4,5)P3) binding) binds specifically to D-myo-inositol 1,3,4,5-tetrakisphosphate (Ins(1,3,4,5)P4) (the
15                            Here we show that inositol 1,3,4,5-tetrakisphosphate (Ins(1,3,4,5)P4), a c
16 1,4,5-trisphosphate (Ins(1,4,5)P3 or IP3) to inositol 1,3,4,5-tetrakisphosphate (Ins(1,3,4,5)P4).
17 nity than to PtdIns-4,5-P2, PtdIns-3,4-P2 or inositol 1,3,4,5-tetrakisphosphate (Ins-1,3,4,5-P4).
18 se B (Itpkb), which converts Ins(1,4,5)P3 to inositol-1,3,4,5-tetrakisphosphate (Ins(1,3,4,5)P4), had
19 3-kinase B (Itpkb) via its enzymatic product inositol 1,3,4,5-tetrakisphosphate (InsP4) plays an esse
20 hatase and 3-kinase, yielding Ins(1,4)P2 and inositol 1,3,4,5-tetrakisphosphate (InsP4), respectively
21 inity label based on phospholipid analogs of inositol 1,3,4,5-tetrakisphosphate (InsP4), we have isol
22 role for the phosphorylated IP(3) metabolite inositol (1,3,4,5)tetrakisphosphate (IP(4)) in NK cells.
23 sitol 1,4,5-trisphosphate (IP(3)) to produce inositol 1,3,4,5-tetrakisphosphate (IP(4)) via the actio
24 ns of inositol 1,4, 5-trisphosphate (IP(3)), inositol 1,3,4,5-tetrakisphosphate (IP(4)), and phosphat
25          In vitro, it has been shown to bind inositol 1, 3,4,5-tetrakisphosphate (IP4), the water-sol
26                          In contrast to IP3, inositol 1,3,4, 5-tetrakisphosphate (IP4) was metabolize
27 at liver supernatant that is up-regulated by inositol 1,3,4,5-tetrakisphosphate (IP4) and inositol he
28 nger inositol 1,4,5-trisphosphate (IP3) into inositol 1,3,4,5-tetrakisphosphate (IP4) establishes ano
29                                              Inositol 1,3,4,5-tetrakisphosphate (IP4), is a ubiquitou
30 se substrates: inositol 1,4,5-trisphosphate, inositol 1,3,4,5-tetrakisphosphate, phosphatidylinositol
31 b converts inositol (1,4,5) trisphosphate to inositol (1,3,4,5) tetrakisphosphate, soluble second mes
32 kb) converts inositol 1,4,5-trisphosphate to inositol 1,3,4,5-tetrakisphosphate upon Ag receptor acti
33 gh GRP1 is selectively blocked by 100 microM inositol 1,3,4,5-tetrakisphosphate, which also binds the

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