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1  affordable products (diagnostics, drugs and insecticides).
2 economic losses and increased application of insecticide.
3 esticide risk, which was driven primarily by insecticides.
4 ary exposure (e.g., via leaves) for systemic insecticides.
5 entalis on plants that were not treated with insecticides.
6 target-site of pyrethroid and organochlorine insecticides.
7 nce appreciable exposures to a wide range of insecticides.
8 des and binary mixtures of cyanobacteria and insecticides.
9 of resistance development to a wide range of insecticides.
10 were showing significant resistance to these insecticides.
11  to preserve the effectiveness of pyrethroid insecticides.
12 tly less susceptible than T. tabaci to these insecticides.
13 on mosquitoes to develop resistance to these insecticides.
14  a mix of insecticides, or a mix of all five insecticides.
15  the pervasive use of systemic neonicotinoid insecticides.
16 llinator exposure to neonicotinoid and other insecticides.
17 l background for rational development of new insecticides.
18  have been ascribed in part to neonicotinoid insecticides.
19 ocesses they are major targets for effective insecticides.
20 era, has developed strong resistance to many insecticides.
21 , it is also possible to apply nonpyrethroid insecticides.
22  sites, which could be potential targets for insecticides.
23 , respectively type I and type II pyrethroid insecticides.
24 n of mating disruption and use of biological insecticides.
25  by allowing resistant insects to metabolize insecticides.
26 gnostics and production of steroid drugs and insecticides.
27 ferential susceptibilities of the species to insecticides.
28 ls and strategies to restore the efficacy of insecticides.
29  highest exceedances found for neonicotinoid insecticides.
30 ganophosphate, pyrethroid, and neonicotinoid insecticides.
31      The PRs included 16 fungicides (52%), 8 insecticides (26%), 2 herbicides (6%), 3 pesticide deriv
32 8), benzimidazoles (20), coccidiostats (14), insecticides (3), dyes (12) and others (3).
33 n the setting of indoor residual spraying of insecticide, adding monthly DP to daily TMP-SMX did not
34 vector population and in turn protecting the insecticide against the spread of insecticide resistance
35 s a powerful tool for developing tailor-made insecticides against new target insects.
36 cide, propiconazole (the synergist), and the insecticide, alpha-cypermethrin, on the mortality of the
37                             Resistance to an insecticide always spreads slower if that insecticide is
38  the major component of spinosad, an organic insecticide and an FDA-approved agent used worldwide.
39                      After administration of insecticide and fungus, activities of glutathione-S-tran
40 ntinuous presence of agricultural chemicals (insecticides and a fungicide) over the last 40 years.
41 ere, provide valuable starting points toward insecticides and are complementary to existing and new c
42 posure to different cyanobacteria, different insecticides and binary mixtures of cyanobacteria and in
43 ion of organophosphorous chemicals including insecticides and chemical warfare agents.
44 ement has relied on multiple applications of insecticides and cultural practices such as removal of w
45 012 to characterize prenatal exposure to IRS insecticides and exposures' impacts on child health and
46  lepidopteran insecticides, non-lepidopteran insecticides and fungicides.
47 r bond found in a variety of organophosphate insecticides and nerve agents.
48 chemicals and suggests a possible role of OC insecticides and PCBs in the etiology of metastatic pros
49  organophosphate [OR=2.0 (95% CI: 1.3, 3.0)] insecticides and phenoxyacetate herbicides [OR=1.9 (95%
50  was carried out on the dissipation of three insecticides and three fungicides during the freezing of
51 t of new methods of prevention (vaccines and insecticides) and treatment (single dose drugs) of the d
52 m to encounter high or low concentrations of insecticide, and allows selection pressures and dominanc
53 henyl)ethane (DDT), the first organochlorine insecticide, and pyrethroid insecticides are sodium chan
54 low-up, 39% of the women reported ever using insecticides, and 1,081 were diagnosed with breast cance
55 onitoring systems, threshold-based selective insecticides, and biological control.
56 s that could potentially reduce or eliminate insecticide application for control of Lygus and the ass
57 agement of mosquito-associated diseases, and insecticide application is the most important component
58  the importance of external factors, such as insecticide applications, in mediating the outcome of in
59                       Systemic neonicotinoid insecticides are increasingly used as a crop protection
60         Several pyrethroid and neonicotinoid insecticides are most effective for controlling the inse
61 miologic and laboratory studies suggest that insecticides are related to increased breast cancer risk
62 t organochlorine insecticide, and pyrethroid insecticides are sodium channel agonists.
63                                    Pesticide insecticides are used on wheat grains in storage units b
64 door-resting mosquitoes, such as bednets and insecticides, are currently the cornerstone of malaria c
65 on's Silent Spring, pesticides, particularly insecticides, arguably remain the most influential pest
66 as restricted the use of three neonicotinoid insecticides as seed dressings on bee-attractive crops.
67     Here, we used fipronil, a broad-spectrum insecticide, as a model chemical to probe its enantiosel
68  to strong selection with glucose-containing insecticide baits.
69  technology that is compatible with existing insecticide-based control methods.
70                                              Insecticide-based interventions have contributed to appr
71 s detected here as increasing selection from insecticide-based interventions may change the dynamic i
72 s to the future to highlight some of the new insecticide-based tools under development and the challe
73 en largely attributable to the deployment of insecticide-based vector control tools such as bed nets
74                                  Traditional insecticide-based vector-control programmes have limited
75 direct ecological effects of the widely used insecticide bifenthrin on stream ecosystems are largely
76 ed to environmental levels of the estrogenic insecticide bifenthrin or ethinylestradiol (EE2) at 22 d
77   Sorption and degradation parameters of the insecticide bifenthrin were measured in two soils for (i
78           In H4IIEcells, four organochlorine insecticides, bifenthrin, and 3-PBA decreased cortisol-i
79 glyphosate herbicides, chemical lepidopteran insecticides, biological lepidopteran insecticides, non-
80                                   Individual insecticides but not herbicides reduced the microbial di
81  They are used in commercial formulations of insecticides, but are also frequently used in the elucid
82               Neonicotinoids are widely used insecticides, but their use is subject of debate because
83           Multiple pest resistances to these insecticides by the 1990s, and increasing consumer deman
84 the microbial diversity and richness and two insecticides, carbaryl and permethrin, also altered the
85  pesticides (herbicides: 2,4-D and simazine; insecticides: carbaryl, dimethoate, disulfoton, and zeta
86 shoppers with simultaneous doses of both the insecticide chlorantraniliprole and the fungal pathogen,
87 rethroids could improve the efficacy of this insecticide class and overcome resistance in field popul
88 eased the efficacy of the most commonly used insecticide class of pyrethroids.
89          Resistance to pyrethroids, the sole insecticide class recommended for treating bed nets, thr
90 he current status of resistance to the major insecticide classes in African malaria vectors, consider
91             Honeybees were most sensitive to insecticides (clothianidin > dimethoate >> tau-fluvalina
92  studies have focused on three neonicotinoid insecticides (clothianidin, imidacloprid, and thiamethox
93 of residual bifenthrin and pirimiphos-methyl insecticides commonly used in storage wheat grains, as w
94                                  The contact insecticide DDT has been reappraised as a safe, life-sav
95 unds and contrasted resistance levels to the insecticide deltamethrin.
96 le, a mixture may last 5 years while the two insecticides deployed individually may last 3 and 4 year
97 ene expression variation at loci involved in insecticide detoxification.
98 rget sites and target-specific compounds for insecticide development.
99  identifying channel blockers of interest to insecticide discovery or biosecurity.
100                                           If insecticides do not reliably kill homozygous sensitive g
101 istant malaria vectors following exposure to insecticides does not diminish the threat of growing res
102                   Mixtures are favoured when insecticide effectiveness (their ability to kill homozyg
103  interactively impact individual fitness and insecticide efficacy.
104  to differ depending on the concentration of insecticide encountered.
105 malaria control, its use contributes to high insecticide exposure in sprayed communities and raises c
106                        We found no impact of insecticide exposure on colony weight gain, or the numbe
107                                    In total, insecticide exposure was predicted to reduce the lifetim
108  should not discount mosquitoes that survive insecticide exposure with fewer than six legs, as they m
109 tance and elevate the detrimental effects of insecticide exposure, demonstrating how environmental co
110 the relationship, if any, between individual insecticide exposures and breast cancer risk.
111 mental concentrations (PEC) for 466 cases of insecticide field concentrations measured in European su
112            Topical application of an acetone insecticide formulation to circumvent lipid-based uptake
113                                  New topical insecticide formulations have proven to be effective in
114 pyrethroid resistance, IRS using alternative insecticide formulations may be needed to achieve substa
115  (IMD) is the most widely used neonicotinoid insecticide found on environmental surfaces and in water
116 tness relative to susceptible populations in insecticide free environments.
117                      The older generation of insecticide groups has been largely replaced by a pletho
118  of Uganda where indoor residual spraying of insecticide had recently been implemented.
119 cline in the armament of registered chemical insecticides has stimulated research into environmentall
120 cultural use of diazinon, an organophosphate insecticide, has raised serious environmental health con
121  ingredients in residential and agricultural insecticides have changed over time, due in part to regu
122 rees treated with one of three neonicotinoid insecticides (imidacloprid, thiacloprid, or acetamiprid)
123 ally used as a model organism to investigate insecticide impacts on bees, but relatively little is kn
124 aying (IRS) with a pyrethroid (deltamethrin) insecticide in the first year and a carbamate (bendiocar
125  the first year and a carbamate (bendiocarb) insecticide in the two subsequent years.
126  are the most frequently and largely applied insecticide in the world due to their biodegradable natu
127        In adjusted models, concentrations of insecticides in carpet dust decreased for three of four
128  potential determinants of concentrations of insecticides in carpet dust, such as home and garden use
129 channel is the primary target for pyrethroid insecticides in insect pests.
130                We measured concentrations of insecticides in one carpet-dust sample from each of 434
131 n and quantification of (five) neonicotinoid insecticides in sugarcane juice.
132          These experiments demonstrated that insecticide-induced leg loss had no significant effect u
133 idespread and intensive use of neonicotinoid insecticides induces negative cascading effects on ecosy
134 ral landscape is most beneficial to reducing insecticide inputs; reality is far more complex.
135 nets (LLINs) and indoor residual spraying of insecticide (IRS) are the primary vector control interve
136 men protected by indoor residual spraying of insecticide (IRS).
137 an insecticide always spreads slower if that insecticide is used in a mixture although this may be in
138            Defining the molecular targets of insecticides is crucial for assessing their selectivity
139  proportion of mosquitoes that encounter the insecticide) is low.
140  changes in active ingredients have impacted insecticide levels measured in homes.
141 herbicides (atrazine, glyphosate), and three insecticides (malathion, carbaryl, permethrin) on microb
142  gene families for chemoreception, toxin and insecticide metabolism, cuticle proteins, opsins, and aq
143 nutafailed to grow using the organophosphate insecticide methyl parathion as sole source of phosphate
144             The use of chemical lepidopteran insecticides might be associated with hepatic dysfunctio
145 rol, ethanol vehicle, herbicide mixture, and insecticide mixture) for 14 d to quantify survival and i
146               Radical oxidation of carbamate insecticides, namely carbaryl and carbofuran, was invest
147 s in carpet dust decreased for three of four insecticides no longer sold for residential use: chlorda
148 pteran insecticides, biological lepidopteran insecticides, non-lepidopteran insecticides and fungicid
149 mine the combined benefit (forage) and cost (insecticide) of oilseed rape grown from thiamethoxam-tre
150 and delayed (>24 h after exposure) impact of insecticides on daily survival and malaria transmission
151 bial diversity or structure but a mixture of insecticides or all five pesticides reduced microbial di
152 oci (e.g. the Ace-1 target site of carbamate insecticides) or detoxification genes has been implicate
153 sticide alone, a mix of herbicides, a mix of insecticides, or a mix of all five insecticides.
154 of herbivore densities by aerial spraying of insecticide over 20 hectares.
155                         Although ever use of insecticides overall was not associated with breast canc
156 ding to pNP production from hydrolysis of an insecticide, paraoxon, in a coupled assay involving phos
157 rcentage change in the concentration of each insecticide per year, adjusting for significant determin
158  of stressors (e.g., either cyanobacteria or insecticides) performed better than general models devel
159 ly used herbicides 2,4-D and glyphosate, the insecticides permethrin and carbaryl, and the rodenticid
160 interplay between evolutionary adaptation to insecticide pressure and variable environmental conditio
161 , imidacloprid, acetamiprid and thiacloprid) insecticides previously extracted from fruit and vegetab
162 s was dominated by intensive organophosphate insecticide regimes to control tortricid leafrollers.
163          All samples had very low amounts of insecticide residues (37 compounds), cyanide, and trypsi
164 etermined nutrients, chemical contaminants, (insecticide residues and heavy metals), and natural toxi
165 osquitoes is threatened by the appearance of insecticide resistance and therefore new control chemica
166  flexible, two-locus model for the spread of insecticide resistance applicable to mosquito species th
167  P450 monooxygenases play a critical role in insecticide resistance by allowing resistant insects to
168          Synergists can counteract metabolic insecticide resistance by inhibiting detoxification enzy
169                                              Insecticide resistance can arise from a variety of mecha
170                                              Insecticide resistance has decreased the efficacy of the
171            Fitness-related costs of evolving insecticide resistance have been reported in a number of
172                                              Insecticide resistance in malaria vectors could presage
173 is study investigated the molecular basis of insecticide resistance in Malaysian populations of Ae. a
174               The role of cuticle changes in insecticide resistance in the major malaria vector Anoph
175 elling strategy to investigate the spread of insecticide resistance in vector populations and demonst
176                                              Insecticide resistance is a growing threat to mosquito c
177                                              Insecticide resistance is a major obstacle to control of
178 nagement enables potato production, although insecticide resistance is becoming an issue.
179         Understanding the molecular basis of insecticide resistance is key to improve the surveillanc
180                                              Insecticide resistance is typically associated with alte
181  burden and the continued spread of drug and insecticide resistance make malaria elimination both via
182                          The implications of insecticide resistance management of B. tabaci on Indian
183  A major impediment to the implementation of insecticide resistance management strategies is that evi
184                          An understanding of insecticide resistance mechanisms would help to develop
185 ed our understanding of the genetic basis of insecticide resistance over the last decade, we still kn
186 f the environmental background in developing insecticide resistance phenotypes, and caution for the i
187                                              Insecticide resistance poses a significant and increasin
188                                              Insecticide resistance threatens the success achieved th
189                 However, increasing drug and insecticide resistance threatens the successes made with
190 improve efficacy, an urgent need as emerging insecticide resistance threatens their future.
191 udy is a summary of the current level of the insecticide resistance to selected organophosphates, pyr
192                    However, the emergence of insecticide resistance within mosquito vectors risks jeo
193 ndation for future research on mechanisms of insecticide resistance, human-bed bug and symbiont-bed b
194 tigating the possible association of CN with insecticide resistance, three assays were compared for t
195 ersal, extreme polyphagy, and development of insecticide resistance, together with human activities,
196 germanica L.) populations, which differed in insecticide resistance, with either nutritionally rich o
197 tegies most likely to minimise the spread of insecticide resistance.
198 ation of association between the Vgsc CN and insecticide resistance.
199 y undermines vector control programs through insecticide resistance.
200 d LLIN effectiveness in the presence of high insecticide resistance.
201 iont interactions, and several mechanisms of insecticide resistance.
202 ave important implications for the spread of insecticide resistance.
203 ecting the insecticide against the spread of insecticide resistance.
204 nsporters have previously been implicated in insecticide resistance.
205 ravel and commerce in addition to widespread insecticide resistance.
206 ya vector, is threatened by growing cases of insecticide resistance.
207 entation of new technologies that circumvent insecticide resistance.
208                             The emergence of insecticide resistant insect pests is of significant con
209                                              Insecticide resistant populations of insects often have
210 d by drug-resistant Plasmodium parasites and insecticide-resistant Anopheles mosquitoes, and first ge
211 efficacy of Metarhizium against wild-caught, insecticide-resistant anophelines.
212 loping new classes of insecticides to combat insecticide-resistant mosquitoes and the transmission of
213 -regulation of AMPs in experimental lines of insecticide-resistant P. xylostella.
214 e lifetime malaria transmission potential of insecticide-resistant vectors by two-thirds, with delaye
215 ed to be 2 and 5microgkg(-1) for the studied insecticides, respectively, with the exception of imidac
216 nagement programs largely depend on chemical insecticides, resulting in high economic and environment
217 osts associated with resistance suggest that insecticide rotation or utilization of non-insecticidal
218 mosquitoes that enter treated properties the insecticide selects for vector phenotypes deflected by t
219 phosphates and hence that the usage of these insecticides should be reconsidered.
220 s for the women's and husbands' use of these insecticides showed limited concordance.
221                       Analytes included four insecticides sold for indoor home use during our study p
222                      Concentrations of other insecticides sold for indoor use (carbaryl, cypermethrin
223                                Neonicotinoid insecticides specifically have been detected in surface
224 yme SpnF involved in the biosynthesis of the insecticide spinosyn A in Saccharopolyspora spinosa is t
225  95% CI 0.04-0.96, p=0.045), as did residual insecticide spraying of household walls (0.52, 0.31-0.87
226 ol policy in India restricts use of residual insecticide sprays to domestic dwellings.
227              Weight is a strong predictor of insecticide susceptibility and bigger mosquitoes are mor
228  for the interpretation of data generated by insecticide susceptibility assays.
229 ions in larval rearing conditions change the insecticide susceptibility phenotype of adult Anopheles
230                                              Insecticide susceptibility to 4% DDT and 0.05% deltameth
231  adult female mosquitoes from representative insecticide-susceptible and -resistant strains of An. ga
232                        One population was an insecticide-susceptible laboratory strain; the other two
233 ant species, while in plots not treated with insecticides, T. tabaci remained the predominant species
234 sistent levels of resistance to fipronil, an insecticide targeting on gamma-aminobytyric acid (GABA)
235 ls of contamination in pollen are pyrethroid insecticides targeting mosquitoes and other nuisance pes
236                                     In turn, insecticides targeting this pathway suppress vitellogene
237 nformation for development of sodium channel insecticides that target key insect pests without harmin
238  difficulty in combating thrips vectors with insecticides, the best way to limit/prevent tospovirus-i
239 r their sensitivity toward the neonicotinoid insecticide thiacloprid using their feeding rate as resp
240 periment with pulsed contaminations with the insecticide thiacloprid.
241 chemical sensors for detection of two common insecticides - thiamethoxam and imidacloprid.
242         The capacity of mosquitoes to resist insecticides threatens the control of diseases such as d
243 ical scaffolds for developing new classes of insecticides to combat insecticide-resistant mosquitoes
244      Pyrethroids remain one of the most used insecticides to control Aedes mosquitoes, despite the de
245                Malaria control is reliant on insecticides to control the mosquito vector.
246 s, there is no strong evidence linking these insecticides to losses of the majority of wild bee speci
247 ctin and beta-cypermethrin are commonly used insecticides to manage thrips in China, and laboratory b
248 exposure during the registration of systemic insecticides to safeguard ecosystem integrity.
249 o evaluate the contribution of neonicotinoid insecticides to worldwide pollinator declines have focus
250 arriers, to the formulation of cosmetics and insecticides, to the fabrication of nanostructured mater
251 ity, we observed an up to 8-fold increase in insecticide tolerance in a total of 17 macroinvertebrate
252 trials in which both species were exposed to insecticide treated cabbage plants, F. occidentalis beca
253 malaria control via funding for long-lasting insecticide treated nets (LLINs), indoor residual sprayi
254                Current control tools such as insecticide treated nets or indoor residual sprays targe
255  (>10 per 10,000) and low coverage (<50%) of insecticide-treated bed nets and antimalarial drugs incl
256 , 2 years after distribution of long-lasting insecticide-treated bed nets, the prevalence of malaria
257 361 (72%) were women, and 442 (88%) reported insecticide-treated bednet use.
258 for diarrhoea, pneumonia, and malaria (23%), insecticide-treated bednets (20%), vaccines (17%), reduc
259                                              Insecticide-treated bednets (ITNs) are effective in prev
260 Mass drug administration and distribution of insecticide-treated bednets timed to coincide with the 2
261 onutrients) and other health products (e.g., insecticide-treated bednets, soap, and water disinfectan
262 ldren aged 0-5 y, adjusting for age, gender, insecticide-treated net (ITN) use, indoor residual spray
263 Kenya and linked it to data on residence and insecticide-treated net (ITN) use.
264 r markers of drug resistance, and to measure insecticide-treated net (ITN) use.
265 cation; OR 1.31, 1.22-1.41), had a household insecticide-treated net (ITN; vs no ITN; OR 1.21, 1.13-1
266 nce survey data, with case management rates, insecticide-treated net usage, and drug campaign coverag
267         This association was not modified by insecticide-treated net use or gravidity, and remained s
268 tween gestation weeks 12 and 28 and given an insecticide-treated net.
269 protected pregnancies (i.e., those not using insecticide-treated nets [ITNs]) leading to live births
270                                 Long-lasting insecticide-treated nets are generally the most cost-eff
271                       Population coverage of insecticide-treated nets increased by 8.34 percentage po
272  trimethoprim-sulfamethoxazole (TMP-SMX) and insecticide-treated nets remain the main interventions f
273 th larviciding, distribution of long-lasting insecticide-treated nets, indoor residual spraying, and
274 bigger mosquitoes are more likely to survive insecticide treatment.
275 ls, the species co-existed on cabbage before insecticide treatments began, but with T. tabaci being t
276  retinoblastoma was associated with parental insecticide use (odds ratio [OR], 2.8; confidence interv
277             We examined associations between insecticide use and breast cancer incidence among wives
278 his substantially eliminated organophosphate insecticide use by 2001, replacing it with pest monitori
279     Here, we use data on crop production and insecticide use from over 100,000 field-level observatio
280 sity, field size, and cropland extent affect insecticide use in practice.
281                             Further, we find insecticide use increases with increasing field size.
282 grated pest management the myriad effects of insecticide use on arthropod pest species.
283 onents of landscape simplification influence insecticide use over space and for different crops.
284  relation to the women's and their husbands' insecticide use using Cox proportional hazards regressio
285  find that higher crop diversity does reduce insecticide use, but the relationship is strongly influe
286 mers and their wives provided information on insecticide use, demographics, and reproductive history
287 e-dependent, with nearby cropland decreasing insecticide use, whereas cropland further away increases
288 use, whereas cropland further away increases insecticide use.
289 insect-resistant crops significantly reduces insecticide use.
290 and contributed to major reductions in total insecticide use.
291 ing the relative merits of sequential use of insecticides versus their deployment as a mixture to min
292               Use of several organophosphate insecticides was associated with elevated breast cancer
293 ive ingredients (herbicides, fungicides, and insecticides); we then selected fludioxonil, a halogenat
294 nomic losses and an increased application of insecticide, which eventually may compromise the future
295      Despite intensive research efforts, new insecticides will not reach the market for at least 5 ye
296  new repellents by combining a high-toxicity insecticide with a candidate repellent initially effecti
297 ro-2,2-bis(4-chlorophenyl)ethane), a contact insecticide with a rich and controversial history since
298 sent viable molecular targets for developing insecticides with new mechanisms of action.
299 s long been dominated by the use of chemical insecticides, with the aim of instantaneously killing en
300 hroids are now the fourth most used group of insecticides worldwide.

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