ライフサイエンスコーパス: insulin sensitivity

1 e effects on substrate handling and improves insulin sensitivity.

2 he ability of muscle contraction to increase insulin sensitivity.

3 e accumulation in association with increased insulin sensitivity.

4 influence whole-body glucose homeostasis and insulin sensitivity.

5 ernal adiponectin deficiency does not reduce insulin sensitivity.

6 predict metabolic phenotypes like whole-body insulin sensitivity.

7 the maintenance of euglycemia and peripheral insulin sensitivity.

8 egulating the expression of genes related to insulin sensitivity.

9 hways, glucose tolerance and utilization and insulin sensitivity.

10 hether TRPC1 is involved in exercise-induced insulin sensitivity.

11 olic alterations indicative of modulation of insulin sensitivity.

12 atherosclerosis progression and increases in insulin sensitivity.

13 to reduced muscle, liver, and adipose tissue insulin sensitivity.

14 lipid storage is negatively associated with insulin sensitivity.

15 ociated with oxidative capacity but not with insulin sensitivity.

16 e intolerance and diabetes without affecting insulin sensitivity.

17 from obesity but retained exquisite hepatic insulin sensitivity.

18 ms to be involved, and we observed a link to insulin sensitivity.

19 ased metabolic rates and improved whole-body insulin sensitivity.

20 rance did not change, owing to a doubling of insulin sensitivity.

21 nal glucose stimulation, which downregulates insulin sensitivity.

22 sed approaches demonstrated increased muscle insulin sensitivity.

23 e of fibroblast growth factor 21, or improve insulin sensitivity.

24 inemia, indicating enhancement of peripheral insulin sensitivity.

25 lamps were performed to determine whole-body insulin sensitivity.

26 ce the risk of type 2 diabetes by increasing insulin sensitivity.

27 ng and contribute to the failure to increase insulin sensitivity.

28 of CTRP6 in modulating both inflammation and insulin sensitivity.

29 ophage chemokine Mcp1, resulting in improved insulin sensitivity.

30 variants that influence fasting measures of insulin sensitivity.

31 at oxidation, lipolysis, and tissue-specific insulin sensitivity.

32 nd replicated known variants associated with insulin sensitivity.

33 nt means with little effect upon physiologic insulin sensitivity.

34 ecretion, and improved glucose tolerance and insulin sensitivity.

35 n activation on BAT-myostatin expression and insulin sensitivity.

36 would, which might account for the change in insulin sensitivity.

37 hich reduces DNL in WAT, and impairs hepatic insulin sensitivity.

38 beta cell stress and death independently of insulin sensitivity.

39 VNS) therapy was shown to improve peripheral insulin sensitivity.

40 of hepatic insulin extraction in regulating insulin sensitivity.

41 howed an improvement in peripheral and brain insulin sensitivity.

42 acological target to improve skeletal muscle insulin sensitivity.

43 STRA6 is necessary for diurnal variations in insulin sensitivity.

44 Iron removal could improve insulin sensitivity.

45 nd improves whole-body glucose tolerance and insulin sensitivity.

46 ced by reduced fat accumulation and improved insulin sensitivity.

47 bited increased fat accumulation and reduced insulin sensitivity.

48 C57BL/6 mice improved glucose clearance and insulin sensitivity.

49 gatively correlated with body-mass index and insulin sensitivity.

50 energy expenditure (EE), and enhancement of insulin sensitivity.

51 secretion and improves glucose tolerance and insulin sensitivity.

52 nt weight loss and improvement of peripheral insulin sensitivity.

53 y a decrease in insulin secretion but not in insulin sensitivity.

54 d placebo groups did not differ in change in insulin sensitivity (0.02 +/- 2.0 compared with -0.03 +/

55 the changes in whole-body and organ-specific insulin sensitivities 12 weeks after permanent, bilatera

56 abolites that accurately predicts whole-body insulin sensitivity across three mouse strains.

57 paralleled by a decrease in genes related to insulin sensitivity (ADIPOQ, GLUT4, PPARG2, and SIRT1) a

58 gest that dietary soy ameliorates adiposity, insulin sensitivity, adipose tissue inflammation, and ar

59 artially attenuated associations of CKD with insulin sensitivity (adjusted difference, -0.7; 95% conf

60 axis is likely mediating the improved muscle insulin sensitivity after contraction/exercise and illum

61 P in mediating the improvement in peripheral insulin sensitivity after CR.

62 The failure to increase insulin sensitivity after training correlates with impai

63 TRACT: Intramyocellular lipid (IMCL) hampers insulin sensitivity, albeit not in endurance-trained ath

64 ose tolerance and markedly increased hepatic insulin sensitivity, an effect that is dependent upon VD

65 to a significant weight loss with increased insulin sensitivity and a marked reduction in serum Resi

66 DIO mice resulted in significantly increased insulin sensitivity and a moderate but significant reduc

67 n clearance were correlated with measures of insulin sensitivity and acute insulin response to glucos

68 mal aerobic capacity (VO2 peak ), whole body insulin sensitivity and arterial stiffness were also ass

69 ring the pre-diabetic phase and assessed for insulin sensitivity and beta cell function relative to c

70 ificant heritability for measures of fasting insulin sensitivity and beta-cell function, for time spe

71 enetic control of metabolic traits including insulin sensitivity and beta-cell function.

72 or physical activity, significantly improved insulin sensitivity and body composition of OVX rats bre

73 osed to low palmitate concentrations reduced insulin sensitivity and cardiomyocyte contractility, whi

74 ate-severe CKD associated with reductions in insulin sensitivity and clearance that are explained, in

75 ing, glucose absorption, beta-cell function, insulin sensitivity and clearance, and the portal insuli

76 ER stress, and a significant improvement in insulin sensitivity and consequently in glucose homeosta

77 nsin II, aldosterone, and neprilysin) impair insulin sensitivity and contribute to microvascular dise

78 ts (LDs) is a determinant of skeletal muscle insulin sensitivity and contributes to the athlete's par

79 rate alcohol use is associated with improved insulin sensitivity and decreased cardiovascular mortali

80 umans, surgery-induced weight loss increased insulin sensitivity and decreased skeletal muscle CEPT1

81 se activity by a specific inhibitor improves insulin sensitivity and decreases hyperglycemia in obese

82 ce less fat and more lean mass, and enhances insulin sensitivity and energy expenditure compared with

83 The relationship between insulin sensitivity and fasting glucagon concentrations

84 nspired oxygen 21-5%, 8 h/day) on whole-body insulin sensitivity and glucose tolerance in lean mice.

85 are healthy and display a robustly enhanced insulin sensitivity and glucose tolerance, phenotypes mi

86 the metabolic profile reversed with impaired insulin sensitivity and glucose tolerance, reduced insul

87 s and act locally or systemically to promote insulin sensitivity and glucose tolerance; as a class, t

88 ha or FFA, we demonstrate that 2-AG improves insulin sensitivity and glucose uptake.

89 Because adiponectin significantly improves insulin sensitivity and has potent anti-inflammatory eff

90 dent of body weight loss or improved hepatic insulin sensitivity and importantly does not induce unhe

91 , and changes in systemic and organ-specific insulin sensitivity and inflammation were measured.

92 kinase, and integrin-linked kinase, enhanced insulin sensitivity and insulin secretion in C2C12 myotu

93 Type 2 diabetes results from defects in both insulin sensitivity and insulin secretion.

94 tochondrial network fragmentation, improving insulin sensitivity and limiting oxidative stress.

95 Plasma FA, blood pressure, insulin sensitivity and lipid concentrations were determ

96 Restoration of IGF-1 improved overall insulin sensitivity and lipid profile in serum and reduc

97 and adipocyte Jak2 on whole-body and tissue insulin sensitivity and liver metabolism.

98 mals, and improved glucose tolerance, better insulin sensitivity and markedly diminished adipose tiss

99 ngolipids in obesity leads to impairments in insulin sensitivity and mitochondrial metabolism, but th

100 rnative to MICT to improve aerobic capacity, insulin sensitivity and muscle capillarisation and endot

101 bile acid transporter, appear to affect both insulin sensitivity and NAFLD/NASH pathogenesis at multi

102 Insulin sensitivity and Nlrp3 inflammasome activation we

103 mulation of AgRP --> LHA projections impairs insulin sensitivity and promotes feeding while activatio

104 macrophage JAK2 deficiency improves systemic insulin sensitivity and reduces inflammation in VAT and

105 promote diabetes in db/db mice, but improves insulin sensitivity and reduces pancreatic beta-cell apo

106 Caloric restriction (CR) improves insulin sensitivity and reduces the incidence of diabete

107 tissue is epistatic to liver with regard to insulin sensitivity and responsiveness, despite fatty li

108 that vitamin D supplementation would improve insulin sensitivity and secretion compared with placebo.

109 for type 2 diabetes, resulting from reduced insulin sensitivity and secretion.

110 of a HFHS diet during pregnancy on maternal insulin sensitivity and signalling in relation to feto-p

111 form, is characterised initially by impaired insulin sensitivity and subsequently by an inadequate co

112 ult in significant benefits in liver fat and insulin sensitivity and that these changes are sustained

113 able association of high IGFBP-1 levels with insulin sensitivity and whether these could be exploited

114 l nutrients should also cause a reduction of insulin sensitivity and/or secretion (anti-incretin effe

115 diet (LCD) reduces fat mass excess, improves insulin sensitivity, and alters adipose tissue (AT) gene

116 eride clearance, liver triglyceride content, insulin sensitivity, and atherosclerosis in mice.

117 d levels of circulating adipokines, improved insulin sensitivity, and better glucose tolerance.

118 fore, miR-155 deletion increases adipogenic, insulin sensitivity, and energy uncoupling machinery, wh

119 es blood glucose, strongly increases hepatic insulin sensitivity, and improves glucose homeostasis in

120 ent intake on blood glucose, insulin, HbA1c, insulin sensitivity, and insulin secretion in adults age

121 to influence processes such as food intake, insulin sensitivity, and insulin secretion.

122 e of surrogate measures of body composition, insulin sensitivity, and insulin secretion.To address ex

123 ep subcutaneous adipose tissue with improved insulin sensitivity, and losing superficial subcutaneous

124 cy (GHRD) results in short stature, enhanced insulin sensitivity, and low circulating levels of insul

125 , CR ameliorates glomerular hyperfiltration, insulin sensitivity, and other cardiovascular risk facto

126 effects on cellular insulin action, in vivo insulin sensitivity, and overall glucose homeostasis.

127 Gender differences in WD-induced steatosis, insulin sensitivity, and predicted microbiota functions

128 ake, increased energy expenditure, increased insulin sensitivity, and reduced linear growth.

129 o improves cardiovascular function, enhances insulin sensitivity, and reduces frailty, targeting this

130 c circuitry mapping reveals that feeding and insulin sensitivity are controlled by both distinct and

131 r, in adult patients, glucose metabolism and insulin sensitivity are essentially unknown.

132 sms responsible for the diurnal variation in insulin sensitivity are unclear.

133 Surrogate indexes of insulin resistance and insulin sensitivity are widely used in nonalcoholic fatt

134 tes, the relationship between adipose tissue insulin sensitivity (ATIS) and beta-cell function remain

135 alterations in peripheral and adipose tissue insulin sensitivity, body composition, and energy and su

136 neuroplasticity pathways and also influence insulin sensitivity, bone metabolism and sympathetic out

137 nitive function, metabolic parameters, brain insulin sensitivity, brain mitochondrial function, brain

138 y MR on EE, remodeling of WAT, and increased insulin sensitivity but not of its effects on hepatic ge

139 improved steatohepatitis, dyslipidemia, and insulin sensitivity, but also ameliorated significant ao

140 ased whole-body, hepatic, and adipose tissue insulin sensitivity by 25%, 15%, and 34%, respectively.

141 reported to lower blood glucose and increase insulin sensitivity by altering bile acid signaling path

142 of the insulin-signaling pathway, modulating insulin sensitivity by limiting p85alpha abundance.

143 a potential therapeutic strategy to restore insulin sensitivity by modulating Atgl levels in adipocy

144 d the effect of 16 weeks of CR on whole-body insulin sensitivity by pancreatic clamp before and after

145 have investigated this association measuring insulin sensitivity by the hyperglycemic clamp technique

146 etal muscle (gastrocnemius) was analyzed for insulin sensitivity, ceramide accumulation and the post

147 d beta-cell function, including quantitative insulin sensitivity check index, homeostasis model asses

148 lucose and insulin during OGTT, Quantitative Insulin Sensitivity Check Index, Simple Index Assessing

149 d 39 healthy control subjects, we quantified insulin sensitivity, clearance, and secretion and glucos

150 ical weight gain, body fat distribution, and insulin sensitivity compared with controls.

151 ) mice showed improved glucose tolerance and insulin sensitivity compared with controls.

152 diet that was high in low-fat dairy reduced insulin sensitivity compared with the effect of a diet t

153 t mice showed impaired glucose clearance and insulin sensitivity (compared with littermate controls)

154 The levels of insulin sensitivity correlated with the magnitudes of he

155 transgenic animals display improved systemic insulin sensitivity, decreased collagen deposition and i

156 n mitochondrial dysfunction, improving brain insulin sensitivity, decreasing cell apoptosis, and incr

157 the effects of chronic VNS therapy on brain insulin sensitivity, dendritic spine density, brain mito

158 ce, thereby maintaining normoinsulinemia and insulin sensitivity despite continuous high fat intake.

159 to-fat ratio, and show dramatically improved insulin sensitivity despite prolonged high-fat diet feed

160 function measured with the insulin secretion/insulin sensitivity (disposition) index increased signif

161 a partial agonist of PPARgamma with improved insulin sensitivity due to its ability to bind PPARgamma

162 Insulin sensitivity during the hyperglycemic clamp was n

163 ement in mitochondrial fatty acid oxidation, insulin sensitivity, dyslipidemia and aortic streaking i

164 dy weight, reduced total adiposity, improved insulin sensitivity, enhanced energy expenditure, and fa

165 wo separate occasions, whole-body and muscle insulin sensitivity (euglycemic-hyperinsulinemic clamp w

166 lected data on peripheral and adipose tissue insulin sensitivity, fecal microbiota composition, plasm

167 Elafibranor improves insulin sensitivity, glucose homeostasis, and lipid meta

168 fects of a single oral saturated fat load on insulin sensitivity, hepatic glucose metabolism, and lip

169 ll function, homeostasis model assessment of insulin sensitivity, homeostasis model assessment of ins

170 We assessed whether insulin sensitivity improved after renal denervation (RD

171 ion during lipid overload, along with muscle insulin sensitivity, improved with weight loss.

172 low in unsaturated FAs may adversely affect insulin sensitivity in a Hispanic/Latino cohort.

173 4 (Map4k4) acted as a negative regulator of insulin sensitivity in chronically obese mice, yet syste

174 ed whole-body glucose metabolism and hepatic insulin sensitivity in comparison to the control diet.

175 ccumulation in liver and muscle and restored insulin sensitivity in glycolytic muscles from LCR rats.

176 tically enhancing autophagy improves hepatic insulin sensitivity in GSNOR-deficient hepatocytes.

177 ed form (UnAG) is associated with whole-body insulin sensitivity in humans and may reduce oxidative s

178 Furthermore, improved insulin sensitivity in IRF3-deficient mice was associate

179 t compromised maternal glucose tolerance and insulin sensitivity in late pregnancy in association wit

180 HFHS feeding perturbed maternal insulin sensitivity in late pregnancy; hepatic insulin s

181 Prior in situ contraction did not increase insulin sensitivity in m.

182 Prior in situ contraction did not increase insulin sensitivity in m. soleus from either genotype.

183 m and diet induced dyslipidaemia, as well as insulin sensitivity in metabolic dysfunction.

184 However, the mechanisms that dictate diurnal insulin sensitivity in metabolic tissues are not well un

185 he visceral adipose tissue mass and enhanced insulin sensitivity in mice fed a high-fat diet (HFD).

186 This study measured insulin sensitivity in mice in late pregnancy at day 16

187 e tissue inflammation in addition to altered insulin sensitivity in mice, most likely via enhanced co

188 BUBR1 deficiency enhances insulin sensitivity in mice.

189 activation of AMPK is sufficient to increase insulin sensitivity in mouse skeletal muscle.

190 aired glucose effectiveness in the liver and insulin sensitivity in muscle by eliminating glucotoxici

191 have investigated the effect of fructose on insulin sensitivity in nondiabetic subjects.

192 ignificance of this mechanism for regulating insulin sensitivity in normal tissue remains unclear.

193 r 5 (TGR5), are known to improve glucose and insulin sensitivity in obese and diabetic mice.

194 Rosiglitazone treatment restored insulin sensitivity in obese B6 mice, yet, surprisingly,

195 inhibitor enalapril, and improves peripheral insulin sensitivity in obese hypertensive patients.

196 or pharmacologic loss of function, improved insulin sensitivity in obese mice.

197 not translate into increased tissue-specific insulin sensitivity in overweight and obese subjects.

198 l processes related to diabetes and obesity (insulin sensitivity in peripheral tissue, pancreatic isl

199 2 groups, but there was a trend for improved insulin sensitivity in potassium-treated participants.In

200 P) whose mRNA levels correlate with improved insulin sensitivity in primary adipose cells carrying th

201 for initially improved glucose tolerance and insulin sensitivity in response to 2 weeks transverse ao

202 ng-term benefits for body weight control and insulin sensitivity in the obese insulin resistant state

203 ill exercise increased muscle and whole-body insulin sensitivity in wild-type (WT) mice, respectively

204 ses adipose tissue ChREBPbeta expression and insulin sensitivity in wild-type mice, and does not furt

205 cretion and maintain glucose homeostasis and insulin sensitivity in wild-type mice.

206 duced a more significant increase in in vivo insulin sensitivity in wild-type than in Fgf21(-/-) mice

207 n levels, larger adipocyte size, and reduced insulin sensitivity in WTs.

208 youth with IGT manifest a global decline in insulin sensitivity, including impaired insulin action i

209 weight change and most health outcomes, but insulin sensitivity increased with breakfast relative to

210 zymes and markers of inflammation decreased, insulin sensitivity increased, and serum level of kerati

211 mice, diet-induced obesity, which decreases insulin sensitivity, increased muscle CEPT1 expression.

212 0.03, respectively), increased oral glucose insulin sensitivity index (42 mL min(-1) m(-2), P < 0.02

213 associations between the weighted IR-GRS and insulin sensitivity index (ISI) at baseline in all parti

214 We performed a GWAS of the modified Stumvoll Insulin Sensitivity Index (ISI) within the Meta-Analyses

215 tisole also caused a slight reduction in the insulin sensitivity index independent of prior saccharin

216 glucose tolerance test-based index (Matsuda insulin sensitivity index).

217 lin resistance, and the composite whole-body insulin sensitivity index.

218 d hormone that increases energy expenditure, insulin sensitivity, insulin secretion, and glucose tole

219 the effects of IGFBP-1 and its RGD domain on insulin sensitivity, insulin secretion, and whole-body g

220 ice were phenotyped for glucose homeostasis, insulin sensitivity, insulin secretion, steatosis, metab

221 the putative relationship between PLIN5 and insulin sensitivity is at best indirect and is apparent

222 The interaction between mitochondria and insulin sensitivity is bidirectional and varies dependin

223 ffect of dietary fish oil on weight gain and insulin sensitivity is dependent on APOE genotype in hum

224 ulated insulin secretion in MENX rats, while insulin sensitivity is improved.

225 or the first time that increased endothelial insulin sensitivity leads to a proatherosclerotic imbala

226 Increased EC insulin sensitivity leads to a proatherosclerotic imbala

227 Validated indices of insulin sensitivity (Matsuda index), beta-cell function

228 hypoglycemia when coupled with the levels of insulin sensitivity measured during IGIV.

229 outcome was the effect of GOS on peripheral insulin sensitivity, measured by the hyperinsulinemic-eu

230 ossibility that pioglitazone, which improves insulin sensitivity, might benefit patients with cerebro

231 e observed for 2 h post-challenge glucose or insulin sensitivity (minimal-model index).

232 one signature, they classified mice based on insulin sensitivity more accurately than each metabolite

233 There was an overall difference in insulin sensitivity of -2.1 mg . kg(-1) . min(-1) betwee

234 Therefore, acute exercise increases insulin sensitivity of muscle by a coordinated increase

235 The effect of enhancing endothelial insulin sensitivity on NO availability is unclear.

236 arrestin 2 deficiency did not affect hepatic insulin sensitivity or beta-adrenergic signaling.

237 D, eGFR did not significantly correlate with insulin sensitivity or clearance.

238 this did not produce significant changes in insulin sensitivity or related substrate and energy meta

239 itamin D-deficient individuals would improve insulin sensitivity or secretion as measured with the us

240 ).Vitamin D supplementation does not improve insulin sensitivity or secretion in vitamin D-deficient,

241 o show clear improvements in blood pressure, insulin sensitivity, or lipid parameters, thus suggestin

242 sitivity Check Index, Simple Index Assessing Insulin Sensitivity Oral Glucose Tolerance, and HOMA-IR

243 t induced a 29 +/- 5% decrease in whole-body insulin sensitivity (P < 0.01).

244 increased VO2 peak (P < 0.05) and whole body insulin sensitivity (P < 0.05), and reduced central arte

245 n120/0 was associated with an improvement in insulin sensitivity (P = 0.003).

246 he anti-inflammatory cytokine IL-10 improves insulin sensitivity, protects against diet-induced obesi

247 Insulin clearance correlated with insulin sensitivity (r=0.72; P<0.001) and was also lower

248 Insulin secretion, insulin sensitivity, Ra, and disposition index were meas

249 influx rates, tissue depots, and whole-body insulin sensitivity (referred to as the M value).

250 ition of BRD4 reduced the expression of many insulin sensitivity-related genes, including genes relat

251 The primary end point is improved insulin sensitivity scores at the 12 week time point.

252 Skeletal muscle insulin sensitivity showed a gradual impairment at low d

253 y using renal denervation (RDN) will improve insulin sensitivity (SI) in a nonhypertensive obese cani

254 adverse effects on longitudinal measures of insulin sensitivity (SI), beta-cell function, and obesit

255 Enhancing insulin sensitivity specifically in EC leads to a parado

256 lude that chronic vagal stimulation improves insulin sensitivity substantially in diet-induced obesit

257 Hepatic insulin sensitivity (suppression of endogenous glucose p

258 In contrast, adipose tissue insulin sensitivity (suppression of free fatty acids by

259 ormal hepatic G6PT activity exhibited higher insulin sensitivity than mice expressing 44-62%.

260 Participants with CKD had lower insulin sensitivity than participants without CKD (mean[

261 reciprocal changes of insulin secretion and insulin sensitivity that preserve glucose homeostasis in

262 sulin secretion to compensate for changes of insulin sensitivity that result from alteration of nutri

263 gh exercise clearly improves skeletal muscle insulin sensitivity, the mechanisms are unclear.

264 Adipose tissue (AT) regulates systemic insulin sensitivity through multiple mechanisms, and alt

265 ry BCAAs also restores glucose tolerance and insulin sensitivity to obese mice, even as they continue

266 arlier studies have demonstrated that muscle insulin sensitivity to stimulate glucose uptake is enhan

267 e traditional Mexican diet modestly improved insulin sensitivity under conditions of weight stability

268 ting, glucose tolerance and utilization, and insulin sensitivity using acute insulin administration a

269 composition (via dual X-ray absorptiometry), insulin sensitivity (via hyperinsulinemic-euglycemic cla

270 ne activity and ECM remodeling-and a link to insulin sensitivity was also apparent.Our present findin

271 Tissue-specific insulin sensitivity was assessed by a hyperinsulinemic-e

272 On a different day, brain insulin sensitivity was assessed by functional MRI.

273 fference in glucose homeostasis, whereas the insulin sensitivity was higher in DHT-exposed dams.

274 sulin sensitivity in late pregnancy; hepatic insulin sensitivity was higher, whereas sensitivity of t

275 Mean whole-body insulin sensitivity was lower by 34% (P < 0.01) and the

276 Despite isoglycemic conditions, insulin sensitivity was lower during oral than during IV

277 Insulin sensitivity was measured by the homeostatic mode

278 This improvement in insulin sensitivity was not accompanied by changes in sk

279 Improvement in muscle insulin sensitivity was not associated with enhanced gly

280 Insulin sensitivity was not significantly perturbed unti

281 machine learning to segregate mice based on insulin sensitivity, we identified C22:1-CoA, C2-carniti

282 er consumption and has been shown to improve insulin sensitivity.We hypothesized that OCFAs are produ

283 elve weeks after surgery, glucose uptake and insulin sensitivity were measured using positron emissio

284 ic and glucose intolerant, but adiposity and insulin sensitivity were not different between HFD-fed S

285 ions showed that hypoglycemia would occur if insulin sensitivity were not reduced by oral glucose sti

286 mprehensive measures of glucose turnover and insulin sensitivity were performed during euglycemic pan

287 hough food intake, body weight, and systemic insulin sensitivity were still affected, the improvement

288 from lean mice improve glucose tolerance and insulin sensitivity when administered to obese recipient

289 n of adipose tissue mass and preservation of insulin sensitivity when compared to MIP-Cre controls.

290 hers showed increased energy expenditure and insulin sensitivity when on an obesogenic diet compared

291 We conclude that cIH impairs insulin sensitivity while improving whole-body glucose t

292 nd adipocytes (JAK2LA) completely normalized insulin sensitivity while reducing liver lipid content.

293 CHO increased whole-body and leg insulin sensitivity, while increasing hepatic glucose pr

294 , any observations of altered adipose tissue insulin sensitivity with extended morning fasting do not

295 Secondly, altered SCAT insulin sensitivity with morning fasting is unlikely to

296 t improved glucose tolerance and/or improved insulin sensitivity, with an exaggerated counter-regulat

297 amine restored IgG sialylation and preserved insulin sensitivity without affecting weight gain.

298 exercise intervention that improved maternal insulin sensitivity without changing maternal body weigh

299 s skeletal muscle mass and lowers whole-body insulin sensitivity, without affecting mechanisms implic

300 ssessment of fasting and dynamic measures of insulin sensitivity would detect novel common variants.