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1 49 men (26 without diabetes and 23 with non-insulin-dependent diabetes mellitus).
2 cause of both morbidity and mortality in non-insulin dependent diabetes mellitus.
3 y reduce the risk of vascular disease in non-insulin dependent diabetes mellitus.
4 who, at baseline, were free of dementia and insulin-dependent diabetes mellitus.
5 rheumatoid arthritis, multiple sclerosis and insulin-dependent diabetes mellitus.
6 abetic mice are a well-known model for human insulin-dependent diabetes mellitus.
7 besity, including insulin resistance and non-insulin-dependent diabetes mellitus.
8 c T cells play a critical role in initiating insulin-dependent diabetes mellitus.
9 oducing beta cells in the pancreas can cause insulin-dependent diabetes mellitus.
10 sents a hallmark event in the development of insulin-dependent diabetes mellitus.
11 OD mice (and in humans) in susceptibility to insulin-dependent diabetes mellitus.
12 insulin resistance syndrome and risk of non-insulin-dependent diabetes mellitus.
13 ts a phenotype that includes obesity and non-insulin-dependent diabetes mellitus.
14 the most common complication of obesity, non-insulin-dependent diabetes mellitus.
15 li had less N-sulfation of HS as a result of insulin-dependent diabetes mellitus.
16 nificantly reduce the risk of developing non-insulin-dependent diabetes mellitus.
17 forearm resistance vessels in patients with insulin-dependent diabetes mellitus.
18 abnormal vascular reactivity in humans with insulin-dependent diabetes mellitus.
19 non-insulin-dependent diabetes mellitus and insulin-dependent diabetes mellitus.
20 forearm resistance vessels of patients with insulin-dependent diabetes mellitus.
21 oendocrine cells, and a major autoantigen in insulin-dependent diabetes mellitus.
22 e impaired insulin secretion observed in non-insulin-dependent diabetes mellitus.
23 of the insulin resistance of obesity and non-insulin-dependent diabetes mellitus.
24 important role in the pathophysiology of non-insulin-dependent diabetes mellitus.
25 genes result in genetic diseases such as non-insulin-dependent diabetes mellitus.
26 ition similar to adult-onset obesity and non-insulin-dependent diabetes mellitus.
27 in the insulin resistance of obesity and non-insulin-dependent diabetes mellitus.
28 oduction characteristic of patients with non-insulin-dependent diabetes mellitus.
29 and its levels are increased in serum in non-insulin-dependent diabetes mellitus.
30 -full sibs in a genetic linkage study of non-insulin-dependent diabetes mellitus.
31 may be modulated in disease states like non-insulin-dependent diabetes mellitus.
32 duals with impaired glucose tolerance or non-insulin-dependent diabetes mellitus.
33 implicated in the autoimmune cytotoxicity of insulin-dependent diabetes mellitus.
34 tance and is the biggest risk factor for non-insulin-dependent diabetes mellitus.
35 one could provide in vivo protection against insulin-dependent diabetes mellitus.
36 hip of ob gene expression to obesity and non-insulin-dependent diabetes mellitus.
37 ide-mediated vasodilation is impaired in non-insulin-dependent diabetes mellitus.
38 ts of IRS-1, especially in patients with non-insulin-dependent diabetes mellitus.
39 (ZDF/Gmifa) rats were used as models of non-insulin-dependent diabetes mellitus.
40 herapy, and (3) a very low rate of new-onset insulin-dependent diabetes mellitus.
41 IIP and MDI is effective in controlling non-insulin-dependent diabetes mellitus.
42 th for any MPS result than patients with non-insulin-dependent diabetes mellitus.
43 imal-medial thickness (IMT) in children with insulin-dependent diabetes mellitus.
44 tes, tropical calcific pancreatitis, and non-insulin-dependent diabetes mellitus.
45 ing protection from cyclophosphamide-induced insulin-dependent diabetes mellitus.
46 nued to destroy their beta cells and develop insulin-dependent diabetes mellitus.
47 notype and correlate with the progression of insulin-dependent diabetes mellitus.
48 eficiency of NK T cells and the induction of insulin-dependent diabetes mellitus.
49 link I-Ag7 with autoimmune diseases, such as insulin-dependent diabetes mellitus.
50 of which is shared with DQ alleles linked to insulin-dependent diabetes mellitus.
51 application to real data from families with insulin-dependent diabetes mellitus.
52 seases, stiff-man syndrome (SMS) and type 1 (insulin-dependent) diabetes mellitus.
53 al symmetric polyneuropathy (DSP) in type 1 (insulin-dependent) diabetes mellitus.
54 type I (insulin-dependent) and type II (non-insulin-dependent) diabetes mellitus.
55 regions of HLA-DRB1 and HLA-DQB1 within the insulin-dependent diabetes mellitus 1 locus in T1D monoc
56 H3K4me3, H3K9me2, H3K9Ac, and H4K16Ac at the insulin-dependent diabetes mellitus 1 region in monocyte
57 2.0 versus 1.0 or lower: 1.37 [1.32-1.43]), insulin-dependent diabetes mellitus (1.45 [1.39-1.51]),
58 ratio, 11.2; P = 0.04), and 6 of 43 with non-insulin-dependent diabetes mellitus (14%; odds ratio, 11
60 The study group included 10 patients with insulin-dependent diabetes mellitus and 10 age-matched c
61 sociated with greatly increased risks of non-insulin-dependent diabetes mellitus and cardiovascular d
62 wn linkage of both HLA-DQ2.3 and -DQ3.2 with insulin-dependent diabetes mellitus and celiac disease,
63 e a characteristic pathologic feature of non-insulin-dependent diabetes mellitus and contain islet am
64 tion (PTx) is the most logical treatment for insulin-dependent diabetes mellitus and for amelioration
65 e of such complications in patients with non-insulin-dependent diabetes mellitus and hypertension who
66 portant adjunct in the treatment of both non-insulin-dependent diabetes mellitus and insulin-dependen
67 ne loss is multifactorial, and patients with insulin-dependent diabetes mellitus and renal failure ma
68 d in patients with insulin-dependent and non-insulin-dependent diabetes mellitus and restored by vita
69 ient with primary sclerosing cholangitis and insulin-dependent diabetes mellitus and review the liter
70 r treatment of autoimmune diseases including insulin-dependent diabetes mellitus and Sjogren's syndro
72 associated with end-stage renal disease and insulin-dependent diabetes mellitus and the prolonged, v
73 nity to screen the gene in patients with non-insulin-dependent diabetes mellitus and/or obesity for s
74 diseases, including dilated cardiomyopathy, insulin-dependent diabetes mellitus, and chronic inflamm
75 ence of adult hypertension, incidence of non-insulin-dependent diabetes mellitus, and prevalence of o
76 ase that accompanies obesity and related non-insulin-dependent diabetes mellitus, and that the adipoc
77 slet beta-cell, selectively destroyed during insulin-dependent diabetes mellitus, appears to be one i
79 sity, hypertension, hyperlipidaemia, and non-insulin-dependent diabetes mellitus are set against asso
80 Type I (insulin-dependent) and type II (non-insulin-dependent) diabetes mellitus are different disea
81 lants that synthesize human insulin, a major insulin-dependent diabetes mellitus autoantigen, at leve
82 DM in HLA-DR4-restricted presentation of an insulin-dependent diabetes mellitus autoantigen, glutama
83 e binding motif for DQ0602 and peptides from insulin-dependent diabetes mellitus autoantigens that bi
84 ndividuals already at increased risk for non-insulin-dependent diabetes mellitus because of obesity,
85 ilk has been implicated in the occurrence of insulin-dependent diabetes mellitus but there is little
86 We show that the Ab inhibits transfer of insulin-dependent diabetes mellitus by the CD4+ Th1 clon
87 ats have normal numbers of RT6+ T cells, and insulin-dependent diabetes mellitus can be induced in th
88 utosomal-recessive disorder characterized by insulin-dependent diabetes mellitus, caused by nonautoim
89 ctors (eg, dialysis-dependent renal failure, insulin-dependent diabetes mellitus) continued to increa
91 ansfer into young NOD mice and could inhibit insulin-dependent diabetes mellitus development, althoug
94 diabetic (NOD) mice, a spontaneous model for insulin-dependent diabetes mellitus, exhibit elevated le
95 e Finland-United States Investigation of Non-Insulin-Dependent Diabetes Mellitus (FUSION) Genetics st
97 e Finland-United States Investigation of Non-Insulin-Dependent Diabetes Mellitus Genetics study and p
98 Nonobese diabetic (NOD) mice, a model of insulin-dependent diabetes mellitus, have a defect in na
99 in patients with a history of long-standing insulin-dependent diabetes mellitus, have been reported
100 ineural hearing loss, childhood obesity, non-insulin-dependent diabetes mellitus, hyperlipidemia and
102 ested: 50 from patients with newly diagnosed insulin-dependent diabetes mellitus (IDDM) and 50 from a
103 utoimmune response during the development of insulin-dependent diabetes mellitus (IDDM) and after imm
104 and other autoimmune diseases, in particular insulin-dependent diabetes mellitus (IDDM) and autoimmun
105 bese diabetic (NOD) mice before the onset of insulin-dependent diabetes mellitus (IDDM) and may be cr
106 retinal blood flow changes in patients with insulin-dependent diabetes mellitus (IDDM) and no diabet
108 n effective therapy that enables people with insulin-dependent diabetes mellitus (IDDM) and renal fai
109 IA-2 and IA-2 beta are major autoantigens in insulin-dependent diabetes mellitus (IDDM) and the precu
110 -2) in nonactivated monocytes of humans with insulin-dependent diabetes mellitus (IDDM) and those wit
111 lar attention has been focused on autoimmune insulin-dependent diabetes mellitus (IDDM) because nonob
112 ciated with susceptibility and resistance to insulin-dependent diabetes mellitus (IDDM) but the immun
114 s long been implicated in the development of insulin-dependent diabetes mellitus (IDDM) caused by vir
115 roiditis (AIT), multiple sclerosis (MS), and insulin-dependent diabetes mellitus (IDDM) during 1990-1
116 Also, after risk adjustment, patients with insulin-dependent diabetes mellitus (IDDM) had higher ri
118 one-half of Caucasians with newly diagnosed insulin-dependent diabetes mellitus (IDDM) have autoanti
121 been reported to have a 10-20% incidence of insulin-dependent diabetes mellitus (IDDM) in adults, bu
122 target of T cells that initiate and sustain insulin-dependent diabetes mellitus (IDDM) in humans and
123 sulted in delay or transient protection from insulin-dependent diabetes mellitus (IDDM) in NOD mice.
124 rvival of NZB/W F1 mice, and suppress type I insulin-dependent diabetes mellitus (IDDM) in non-obese
125 T cell-mediated autoimmune diseases such as insulin-dependent diabetes mellitus (IDDM) in nonobese d
126 ic component of susceptibility to autoimmune insulin-dependent diabetes mellitus (IDDM) in nonobese d
127 in an Ag-specific manner and in turn prevent insulin-dependent diabetes mellitus (IDDM) in nonobese d
128 nduces GAD65-specific Th2 cells and prevents insulin-dependent diabetes mellitus (IDDM) in nonobese d
129 t component for the initiation of autoimmune insulin-dependent diabetes mellitus (IDDM) in the NOD mo
130 uencies of >1/1, 000 are sufficient to cause insulin-dependent diabetes mellitus (IDDM) in transgenic
135 ci responsible for genetic susceptibility to insulin-dependent diabetes mellitus (IDDM) is the insuli
137 entified by DNA sequence analysis within the insulin-dependent diabetes mellitus (IDDM) locus IDDM4 o
138 n the nonobese diabetic (NOD) mouse model of insulin-dependent diabetes mellitus (IDDM) may be genera
140 ) as a transgene in their beta cells develop insulin-dependent diabetes mellitus (IDDM) only after LC
141 a (IL-1 beta) were measured in a group of 39 insulin-dependent diabetes mellitus (IDDM) patients and
142 cells from the pancreata of newly diagnosed insulin-dependent diabetes mellitus (IDDM) patients expr
144 In mice, coxsackievirus B4 (CB4) induces insulin-dependent diabetes mellitus (IDDM) resembling th
145 ability of MHC class II alphabeta dimers and insulin-dependent diabetes mellitus (IDDM) susceptibilit
147 us-induced CTL-mediated autoimmune diabetes (insulin-dependent diabetes mellitus (IDDM)) in vivo with
148 tic duct secretion is frequently observed in insulin-dependent diabetes mellitus (IDDM), although the
149 ated microenvironment in the pathogenesis of insulin-dependent diabetes mellitus (IDDM), an LTbeta re
151 etabolism in patients with poorly controlled insulin-dependent diabetes mellitus (IDDM), we used 13C-
168 ing the past decade, the genetics of type 1 (insulin-dependent) diabetes mellitus (IDDM) has been stu
169 complex (MHC)-associated autoimmune type 1 (insulin-dependent) diabetes mellitus (IDDM) has shown th
171 genetic factors involved in type 1 diabetes (insulin-dependent diabetes mellitus [IDDM]) in the past
173 ry of peptides bound by the type I diabetes (insulin-dependent diabetes mellitus, IDDM)-associated HL
174 mprove insulin resistance in obesity and non-insulin-dependent diabetes mellitus in both rodents and
175 602) is observed at a decreased frequency in insulin-dependent diabetes mellitus in different ethnic
176 strongly associated with the development of insulin-dependent diabetes mellitus in humans and shares
178 e production of CGRP by beta cells prevented insulin-dependent diabetes mellitus in male and reduced
179 tigation of agouti's role in obesity and non-insulin-dependent diabetes mellitus in mice holds signif
180 or cells because it prevented BDC2.5-induced insulin-dependent diabetes mellitus in nonobese diabetic
181 critical in the long-term management of non-insulin-dependent diabetes mellitus in obese patients.
182 and attractive approach to prevent clinical insulin-dependent diabetes mellitus in prediabetic indiv
183 induced beta cell destruction and subsequent insulin-dependent diabetes mellitus in RIP-mCD80(+) mice
184 egion linked with insulin resistance and non-insulin-dependent diabetes mellitus in the Pima Indians,
185 2 in vivo ameliorates streptozotocin-induced insulin-dependent diabetes mellitus in transgenic mice.
186 e role of CD28/B7 interactions in a model of insulin-dependent diabetes mellitus in which T cell-depe
187 th increased risk of type 2 diabetes, or non-insulin-dependent diabetes mellitus, in Mexican American
189 bnormalities associated with early stage non-insulin-dependent diabetes mellitus, including glucose i
190 h type 1 (insulin-dependent) and type 2 (non-insulin-dependent) diabetes mellitus, including islet tr
191 tis, Graves' disease, Hashimoto thyroiditis, insulin-dependent diabetes mellitus, inflammatory bowel
192 D spleen or salivary glands did not transfer insulin-dependent diabetes mellitus into similar CD8-rec
193 pothesis that endothelial dysfunction in non-insulin dependent diabetes mellitus is the consequence o
202 Insulin resistance, the hallmark of non-insulin dependent diabetes mellitus, is characterized by
203 autosomal dominant, early-onset form of non-insulin-dependent diabetes mellitus (maturity-onset diab
205 development of one form of early-onset, non-insulin-dependent diabetes mellitus, MODY1, as well as a
206 ical calcific pancreatitis (n = 15), and non-insulin-dependent diabetes mellitus (n = 43) and control
207 nt of several chronic diseases including non-insulin dependent diabetes mellitus (NIDDM) and coronary
208 fetal thinness have been associated with non-insulin dependent diabetes mellitus (NIDDM) and insulin
209 tested the hypothesis that persons with non-insulin dependent diabetes mellitus (NIDDM) have greater
210 ) suffering from periodontal disease and non-insulin dependent diabetes mellitus (NIDDM) were randomi
216 ibuting to postprandial hyperglycemia in non-insulin-dependent diabetes mellitus (NIDDM) has not been
217 First-degree relatives of patients with non-insulin-dependent diabetes mellitus (NIDDM) have an incr
218 the pathophysiology of renal disease in non-insulin-dependent diabetes mellitus (NIDDM) have been hi
219 und troglitazone, which is used to treat non-insulin-dependent diabetes mellitus (NIDDM) in man, is a
225 Hypertension is a common finding in non-insulin-dependent diabetes mellitus (NIDDM) nephropathy.
226 ure of insulin sensitivity in studies of non-insulin-dependent diabetes mellitus (NIDDM) risk, but th
228 d the role of MIF in the pathogenesis of non-insulin-dependent diabetes mellitus (NIDDM) using MIF(-/
229 n-dependent diabetes mellitus (IDDM) and non-insulin-dependent diabetes mellitus (NIDDM) were randoml
230 s 5.4 micrograms/minute for persons with non-insulin-dependent diabetes mellitus (NIDDM) who were not
231 sal is a common finding in patients with non-insulin-dependent diabetes mellitus (NIDDM), as well as
232 sitivity in obesity and in patients with non-insulin-dependent diabetes mellitus (NIDDM), by activati
233 d positive result from a genome scan for non-insulin-dependent diabetes mellitus (NIDDM), Hanis et al
234 hizophrenia, bipolar affective disorder, non-insulin-dependent diabetes mellitus (NIDDM), inflammator
235 lin resistance have been associated with non-insulin-dependent diabetes mellitus (NIDDM), obesity, at
249 proved to be caused by mtDNA mutations; non-insulin-dependent diabetes mellitus (NIDDM); and hyperte
252 by mutations in WFS1 and is characterized by insulin-dependent diabetes mellitus, optic atrophy, and
253 stantial negative effect similar to that for insulin-dependent diabetes mellitus or smoking in adults
255 se-causing leukocytes that are a hallmark of insulin-dependent diabetes mellitus produce and respond
256 lycogen synthase activation in the mice with insulin-dependent diabetes mellitus, providing a novel t
257 diseases with late age at onset such as non-insulin-dependent diabetes mellitus, psychiatric disorde
261 refore, nondiabetic (lean and obese) and non-insulin dependent diabetes mellitus subjects were studie
262 islets from rodent models of obesity and non-insulin-dependent diabetes mellitus, suggesting the invo
263 ve suggested linkage of Type 2 diabetes (non-insulin-dependent diabetes mellitus) susceptibility to a
264 imic events and molecules involved in type 1 insulin-dependent diabetes mellitus (T1D), we previously
265 he presence of autoimmune conditions such as insulin-dependent diabetes mellitus (T1DM) or a family h
267 natives of Alaska, have a prevalence of non-insulin-dependent diabetes mellitus that is two to six t
268 nsplantation for patients with long-standing insulin-dependent diabetes mellitus that progresses to r
269 in a model of type 2 diabetes mellitus (non-insulin-dependent diabetes mellitus), the ob/ob mouse.
270 affect the risk for diabetes, especially non-insulin-dependent diabetes mellitus, the authors examine
271 e mutation was diagnosed at age 3 years with insulin-dependent diabetes mellitus, the central feature
274 d experimental procedure in the treatment of insulin-dependent diabetes mellitus, this operation has
275 IPEX manifests most commonly with diarrhea, insulin-dependent diabetes mellitus, thyroid disorders,
276 islet-infiltrating CD4+ T cells transferred insulin-dependent diabetes mellitus to CD8 reconstituted
277 (lean to very obese) and with or without non-insulin-dependent diabetes mellitus to examine the relat
278 shed association between coeliac disease and insulin dependent diabetes mellitus, together with the m
279 range of mouse and human conditions such as insulin-dependent diabetes mellitus, tumor rejection, an
280 dependent diabetes mellitus (type 1) and non-insulin dependent diabetes mellitus (type 2) after liver
282 er there was a difference in outcome between insulin-dependent diabetes mellitus (type 1) and non-ins
283 uccessful in correcting the hyperglycemia of insulin-dependent diabetes mellitus (type 1), the result
284 cular morphology and function in type 2 (non-insulin-dependent) diabetes mellitus (type 2D), small ar
285 of posttransplant fractures in patients with insulin-dependent diabetes mellitus undergoing combined
286 tocin treatment in vivo generates a model of insulin-dependent diabetes mellitus via destruction of t
290 reatic diseases in Bangladesh, including non-insulin-dependent diabetes mellitus, was undertaken.
291 itial and final incidences of posttransplant insulin-dependent diabetes mellitus were 7.0% and 2.9%;
292 with mixed connective tissue disease and non-insulin-dependent diabetes mellitus who developed an unu
293 of pancreas transplantation in patients with insulin-dependent diabetes mellitus who have received a
294 Data were gathered on 478 US patients with insulin-dependent diabetes mellitus who participated in
296 portend an increase in the incidence of non-insulin-dependent diabetes mellitus with important publi
297 tween 1986 and 1992, 37 patients with type 1 insulin-dependent diabetes mellitus with renal failure u
298 is is an uncommon but severe complication of insulin-dependent diabetes mellitus with unclear pathoph
299 presence of enhanced oxidative stress in non-insulin dependent diabetes mellitus, with recent data im
300 the genetic basis for susceptibility to non-insulin-dependent diabetes mellitus within the context o
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