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3 circulating monoamine oxidase activity, has insulin-like effects, and can initiate oxidative stress.
13 stoma, is driven by Sonic hedgehog (Shh) and insulin-like growth factor (IGF) in the developing cereb
17 the sirtuin family of proteins, the insulin/insulin-like growth factor (IGF) signaling (IIS) pathway
18 D) progression is significantly delayed when insulin-like growth factor (IGF) signaling is blocked in
21 d plasma protein-A (PAPP-A), which modulates insulin-like growth factor (IGF) signaling through prote
22 study is the identification of mutations in insulin-like growth factor (IGF) signalling genes in 8/1
24 .0001), insulin (r(2) = 0.40, P < 0.005) and insulin-like growth factor (IGF)-1 (r(2) = 0.80, P < 0.0
26 AT3 phosphorylation by interleukin-6 (IL-6), insulin-like growth factor (IGF)-1, IGF-2, and leukemia
27 notype were accompanied by altered placental insulin-like growth factor (IGF)-2 expression and insuli
30 A focus on reversible mechanisms identified Insulin-like growth factor (IGF1) deficiency with inadeq
36 mones and neuropeptides, such as insulin and insulin-like growth factor 1 (IGF-1) likely modulate gly
37 spective case-control studies suggested that insulin-like growth factor 1 (IGF-1) or insulin-like gro
38 t or energy levels-most notably, the insulin/insulin-like growth factor 1 (IGF-1) signaling pathway,
39 nction through the regulation of the insulin/insulin-like growth factor 1 (IGF-1) signaling pathway.
40 liferation by modulating a decrease in serum insulin-like growth factor 1 (IGF-1) that allows GH rele
41 f the pro-osteoclastogenic factors RANKL and insulin-like growth factor 1 (IGF-1) to induce osteoclas
43 anismal level as shown by down-regulation of insulin-like growth factor 1 (IGF-1), a hallmark of prem
45 pe 2 diabetes mellitus (T2DM) via binding of insulin-like growth factor 1 (IGF-1), an insulin-like ho
46 ng serum concentrations of glucose, insulin, insulin-like growth factor 1 (IGF-1), insulin-like growt
52 +DE patient serum revealed altered levels of insulin-like growth factor 1 (IGF-I) and its binding pro
53 o-expression of two soluble proteins, namely insulin-like growth factor 1 (IGF1) and osteopontin (OPN
55 ingly, treatment of RTT-derived neurons with Insulin-like Growth Factor 1 (IGF1) could rescue some of
59 receptor tyrosine kinases by neuregulin and insulin-like growth factor 1 (IGF1) leads to the phospho
61 ffects of (56)Fe radiation on adipokines and insulin-like growth factor 1 (IGF1) signaling axis in mo
62 toxic inflammation, increases cytoprotective insulin-like growth factor 1 (IGF1) signaling, and preve
63 Human fibroblast growth factor 2 (FGF2) and insulin-like growth factor 1 (IGF1), are canonical proto
64 reased cellular sensitivity to estradiol and insulin-like growth factor 1 (IGF1), as measured in grow
65 or muscle growth, including the induction of insulin-like growth factor 1 (IGF1), as well as concomit
66 ted sites cluster on the binding surfaces of insulin-like growth factor 1 (IGF1), IGF1 receptor (IGF1
67 n, or co-overexpression of osteopontin (OPN)/insulin-like growth factor 1 (IGF1)/ciliary neurotrophic
68 cer pinpoint to a potential new function for insulin-like growth factor 1 (Igf1r) in the basal epithe
69 ands, and feet and are diagnosed by elevated insulin-like growth factor 1 levels and growth hormone l
71 the insulin receptor (F-IRKO) or both IR and insulin-like growth factor 1 receptor (F-IR/IGFRKO).
72 atric skin display reduced activation of the insulin-like growth factor 1 receptor (IGF-1R) and alter
73 iates for the activation of PI3K by both the insulin-like growth factor 1 receptor (IGF-1R) and its c
75 rs revealed 6ha as preferential inhibitor of insulin-like growth factor 1 receptor (IGF-1R) in a pane
77 tor 2 (IGF2) gene, encoding a ligand for the insulin-like growth factor 1 receptor (IGF-1R), in a sub
78 ession in this lineage and was stimulated by insulin-like growth factor 1 receptor (IGF1R) activation
79 in asthma, but little is known about how the insulin-like growth factor 1 receptor (IGF1R) affects as
81 ang et al that describes a critical role for insulin-like growth factor 1 receptor in the progression
82 s as an autocrine activator of the beta-cell insulin-like growth factor 1 receptor signaling pathway.
83 2 (Cyclin D2), CCND1 (Cyclin D1), and IGF1R (insulin-like growth factor 1 receptor) genes involved in
84 t-mammalian target of rapamycin pathway, and insulin-like growth factor 1 receptor-targeted therapy h
88 story, medications, and laboratory findings (insulin-like growth factor 1, follicle-stimulating hormo
89 eased expression of pro-regenerative factors insulin-like growth factor 1, leukemia inhibitory factor
92 ation but instead sustained signaling of the insulin-like growth factor 1/phosphatidylinositol 3-kina
94 mouse hippocampus, induces the expression of insulin-like growth factor 2 (IGF2) for the stabilizatio
95 s Network detected the overexpression of the insulin-like growth factor 2 (IGF2) gene, encoding a lig
99 t that Hh markedly induces the expression of insulin-like growth factor 2 (Igf2) that activates the m
100 activating enzyme, deiodinase-III (Dio3) and insulin-like growth factor 2 (Igf2), genes that are know
102 he expression of several myokines, including insulin-like growth factor 2 (IGF2), whereas RNAi and do
105 f the heterochronic let-7 miRNA pathway, the insulin-like growth factor 2 mRNA-binding protein 1 (IGF
108 iation [RalGDS/AF-6] domain family member 1, insulin-like growth factor 2, and adenomatous polyposis
111 ts known ability to promote the abundance of Insulin-like Growth Factor 2/IGF2, we find that IMP2 str
112 ing indicated substantial down-regulation of insulin-like growth factor binding protein (Igfbp) genes
114 tor (PDGF) isoforms (PDGF-AA, -BB, and -AB), insulin-like growth factor binding protein (IGFBP)-2, an
118 that insulin-like growth factor 1 (IGF-1) or insulin-like growth factor binding protein 3 (IGFBP-3) w
119 sulin, insulin-like growth factor 1 (IGF-1), insulin-like growth factor binding protein 3 (IGFBP-3),
120 K regulates Runx1-dependent transcription of insulin-like growth factor binding protein 3 (IGFBP3), a
121 -1, cartilage oligomeric matrix protein, and insulin-like growth factor binding protein 7 concentrati
124 ide, interleukin-6, soluble CD40 ligand, and insulin-like growth factor binding protein-2) and 5 clin
127 atocyte growth factor, endocrine gland VEGF, insulin-like growth factor binding proteins, or endostat
130 AD in mice responds positively to decreased insulin-like growth factor I (IGF-I) signaling, a pathwa
131 mini nutritional assessment (P = 0.003), and insulin-like growth factor I (P = 0.002), and lowered C-
135 ved growth factor receptor beta (Pdgfrb) and insulin-like growth factor I receptor (Igf1r) on T-ALL c
137 human epidermal growth factor, insulin, and insulin-like growth factor I, particularly at the parasi
138 bolic variables, including glucose, insulin, insulin-like growth factor I, triglycerides, cholesterol
139 e gene that experiences LOI is the paracrine insulin-like growth factor IGF2, which occurs commonly i
140 ancer (insulinoma) cells that do not produce insulin-like growth factor II (IGF-II) grow slowly in pu
142 a systemic administration of the polypeptide insulin-like growth factor II (IGF-II) reverses all thes
143 st the effects of systemic administration of insulin-like growth factor II (IGF-II), a polypeptide th
144 antagomir-352 up-regulated the expression of insulin-like growth factor II receptor (IGF2R), which ma
146 the MAPK pathway, PI3Kbeta, and PI3Kalpha or insulin-like growth factor receptor 1 (IGF1R) synergisti
147 receptor (EGFR) and insulin receptor (InsR)/insulin-like growth factor receptor 1 (IGF1R), as well a
155 o DNA damage repair, cell cycle progression, insulin-like growth factor signaling, innate immunity, a
158 a key role in regulating the growth hormone/insulin-like growth factor type 1 (GH/IGF1) axis, whose
159 inases, including the highly cancer relevant insulin-like growth factor type 1 receptor (IGF-1R).
161 eceptor isoforms A and B (IR-A and IR-B) and insulin-like growth factor type I receptor (IGF-1R).
163 nockdown cells was associated with increased insulin-like growth factor- 1Rbeta (IGF-1Rbeta) levels.
165 entified dysregulation of the Growth Hormone/Insulin-like Growth Factor-1 (GH/IGF-1) pathway as well
166 In addition to IRBP, the IPM also contains insulin-like growth factor-1 (IGF-1) and its associated
167 scular endothelial growth factor-A (VEGF-A), insulin-like growth factor-1 (IGF-1) and Klotho, in the
169 recurrent GBM, driven by macrophage-derived insulin-like growth factor-1 (IGF-1) and tumor cell IGF-
170 e previously shown that systemic infusion of insulin-like growth factor-1 (IGF-1) exerts anti-inflamm
173 eurons, have triggered a renewed interest in insulin-like growth factor-1 (IGF-1) pathway activation
174 e MC4R, we assessed pulsatile GH release and insulin-like growth factor-1 (IGF-1) production and/or r
177 atory pathway for E2F-2 production involving insulin-like growth factor-1 (IGF-1) signaling is simult
178 mediates adipogenesis and show a link to the insulin-like growth factor-1 (IGF-1) signaling pathway.
179 ells secrete a subthreshold concentration of insulin-like growth factor-1 (IGF-1) that primes the IGF
180 ce (95% CI) was computed for levels of serum insulin-like growth factor-1 (IGF-1), leptin, and adipon
181 elaborates the cardioprotective polypeptide, insulin-like growth factor-1 (IGF-1), which activates IG
182 ical stimulation that significantly promoted insulin-like growth factor-1 (IGF1) production and elici
183 ion and proliferation as well as insulin and insulin-like growth factor-1 (IGF1) receptor activation
184 eration, the expression of cellular insulin, insulin-like growth factor-1 (IGF1), and GHRH receptor,
187 ained only a modest amount of variability in insulin-like growth factor-1 and insulin-like growth fac
188 tress syndrome were strongly associated with insulin-like growth factor-1 and insulin-like growth fac
190 ignificantly influence circulating levels of insulin-like growth factor-1 and insulin-like growth fac
191 , SP and its metabolites in combination with insulin-like growth factor-1 are shown to promote the co
192 Perfluoroalkyl substances, sex hormones, and insulin-like growth factor-1 at 6-9 years of age: a cros
193 Evaluation III was strongly associated with insulin-like growth factor-1 levels; and age was strongl
194 TRPC6 complementation and by treatment with insulin-like growth factor-1 or hyperforin, a TRPC6-spec
198 Increased expression or signaling from the insulin-like growth factor-1 receptor (IGF-1R) has been
199 abolished these effects of insulin, whereas insulin-like growth factor-1 receptor (IGF-1R) silencing
203 mmunohistochemical positivity for p4E-BP1 or insulin-like growth factor-1 receptor was statistically
206 2 saturation and plasma glucose, insulin and insulin-like growth factor-1 were positively associated
207 ccumulation, which was confirmed in vitro as insulin-like growth factor-1, a known mediator of physio
208 ich was amplified by MCL-specific cytokines (insulin-like growth factor-1, B-cell activating factor,
209 as well as four long-lived mutants (insulin/insulin-like growth factor-1, dietary restriction, prote
210 changes including altered levels of insulin, insulin-like growth factor-1, leptin, adiponectin, stero
211 -1 and AKT following stimulation by insulin, insulin-like growth factor-1, or the neurotrophins (NGF
214 w this control is abrogated by inhibition of insulin-like growth factor-1R/INSR-driven phosphoinositi
218 acellular domains of the mannose 6-phosphate/insulin-like growth factor-2 receptor (M6P/IGF2R), domai
219 xpression of the trophoblast survival factor insulin-like growth factor-2 was significantly lower.
220 d hepatocyte nuclear factor (HNF) 4alpha and insulin-like growth factor-binding protein (IGFBP) 1 mRN
221 Binding of STC2 prevents PAPP-A cleavage of insulin-like growth factor-binding protein (IGFBP)-4 and
222 esponse elements located in the promoters of insulin-like growth factor-binding protein 1 (IGFBP1) an
223 anges in microglial secretome and identified insulin-like growth factor-binding protein 1 (IGFBP1) as
224 everal genes, e.g., Sonic hedgehog (Shh) and Insulin-like growth factor-binding protein 2 (Igfbp2), t
227 tissue inhibitor of metalloproteinase-2 and insulin-like growth factor-binding protein 7 accurately
228 tissue inhibitor of metalloproteinase-2 and insulin-like growth factor-binding protein 7 in patients
229 tissue inhibitor of metalloproteinase-2 and insulin-like growth factor-binding protein 7 in this pop
230 tissue inhibitor of metalloproteinase-2 and insulin-like growth factor-binding protein 7 results in
231 tissue inhibitor of metalloproteinase-2 and insulin-like growth factor-binding protein 7 significant
232 tissue inhibitor of metalloproteinase-2 and insulin-like growth factor-binding protein 7 test was no
233 tissue inhibitor of metalloproteinase-2 and insulin-like growth factor-binding protein 7 was 0.84 (0
234 owed by a marker of cell cycle arrest (urine insulin-like growth factor-binding protein 7) and, final
236 g levels of insulin-like growth factor-1 and insulin-like growth factor-binding protein-3 early in cr
237 854746 is also significantly associated with insulin-like growth factor-binding protein-3 levels in t
238 ciated with insulin-like growth factor-1 and insulin-like growth factor-binding protein-3 levels; Acu
243 cancer model and decreased the expression of insulin-like growth factor-binding protein-5 (IGFBP5).
244 GFBP)-1 influences fetal growth by modifying insulin-like growth factor-I (IGF-I) bioavailability.
245 l and neuropsychological recovery, and serum insulin-like growth factor-I (IGF-I) may mediate this ef
247 ile hormone, 20-hydroxyecdysone, and insulin/insulin-like growth factor-like signaling in regulating
250 ance of pancreatic cancer cells by secreting insulin-like growth factors (IGF) 1 and 2, which activat
251 oming a key regulator of metabolism, whereas insulin-like growth factors (IGF)-I/II are major growth
257 s of the transforming growth factor-beta and insulin-like growth factors pathways, and extracellular
261 of insulin-like growth factor 1 (IGF-1), an insulin-like hormone that is involved in glucose homeost
262 ulin-like peptide 8 (Dilp8) as a relaxin and insulin-like molecule secreted from growing tissues that
263 nked ins-9 gene, which encodes a new agonist insulin-like peptide (ILP) expressed specifically in the
265 thway revealed elevated levels of Drosophila insulin-like peptide 2 (Dilp2) in the IPCs and elevated
266 no acids promote the secretion of Drosophila insulin-like peptide 2 (Dilp2), circulating sugars promo
270 we showed that the expression of Drosophila insulin-like peptide 8 (dilp8), which encodes a paracrin
272 n identified distinct peptides that regulate insulin-like peptide expression, feeding behavior, or bo
274 aling leads to insufficient expression of an insulin-like peptide, dILP8, which is required for the d
275 ong-lived dietary restricted fruit flies and insulin-like-peptide mutants exhibit small nucleoli and
276 IIS models, namely Drosophila lacking three insulin-like peptides (dilp2-3,5(-/-)) and mice lacking
278 male-female differences in the production of insulin-like peptides (Ilps) from the IPCs do not appear
280 s, ovary ecdysteroidogenic hormone (OEH) and insulin-like peptides (ILPs), which activate multiple pr
281 In silico target prediction identified that insulin-like peptides 7 and 8 (ilp7 and ilp8) are putati
282 pendent signals that act systemically (e.g., Insulin-like peptides [ILPs] transduced by the Target of
283 the food odor benzaldehyde (BZ) and release insulin-like peptides and acetylcholine, respectively, w
285 in, we show that in Drosophila expression of insulin-like peptides is regulated by neprilysin activit
286 response to photoreceptor-EGF, glia produce insulin-like peptides, which induce lamina neuronal diff
290 ecipitation of Lin-28-bound mRNAs identified Insulin-like Receptor (InR), forced expression of which
293 espan extension via the suppression of IGF-1/insulin-like signaling (IIS) offers a possibility to ret
294 n as dauer by inhibiting the conserved DAF-2 insulin-like signaling (ILS) pathway through incompletel
296 hese pathways to extend life span, including insulin-like signaling and the response to dietary restr
297 ynthesis, and upstream components of insulin/insulin-like signaling are activated in their discs.
300 lationship between components of the insulin/insulin-like/Tor and ecdysone pathways in the control of
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