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4 sulin negatively regulates expression of the insulin-like growth factor binding protein 1 (IGFBP-1) g
5 ntrast, phage-derived peptide antagonists of insulin-like growth factor binding protein 1 (IGFBP-1) h
8 genes and proteins in regenerating liver is insulin-like growth factor binding protein 1 (IGFBP-1),
9 c-containing superoxide dismutase 1 (SOD-1), insulin-like growth factor binding protein 1 (IGFBP-1),
10 a 20-fold increase in hepatic expression of insulin-like growth factor binding protein 1 (IGFBP-1),
11 ession profiling revealed elevated levels of insulin-like growth factor binding protein 1 (IGFBP1) tr
12 (HGF), insulin-like growth factor I (IGF-I), insulin-like growth factor binding protein 1 (IGFBP1), m
13 phosphatase [G6Phase], and secreted factors (insulin-like growth factor binding protein 1 [GFBP-1].
15 iferator-activated receptor alpha), IGFBP-1 (insulin-like growth factor binding protein 1), HIF3alpha
16 factor-I, insulin-like growth factor-II, and insulin-like growth factor binding proteins 1, 2, and 3
17 esponse elements located in the promoters of insulin-like growth factor-binding protein 1 (IGFBP1) an
18 anges in microglial secretome and identified insulin-like growth factor-binding protein 1 (IGFBP1) as
19 the insulin-responsive sequence (IRS) in the insulin-like growth factor-binding protein 1 promoter an
20 able to bind to its cognate sites within the insulin-like growth factor-binding protein 1 promoter on
21 articles and chromatin arrays containing the insulin-like growth factor-binding protein 1 promoter.
24 ing globulin, estrone, estradiol, C-peptide, insulin-like growth factor-binding proteins 1 and 2, adi
25 ctor receptor-binding protein 10 (GRB10) and insulin-like growth factor-binding proteins 1 and 3 (IGF
26 ttern of the thyroglobulin type 1A domain of insulin-like growth factor-binding proteins 1 and 6.
27 on of multiple genes in the liver, including insulin-like growth factor binding protein-1 (IGFBP-1) a
29 sistance, accompanied by decreased levels of insulin-like growth factor binding protein-1 (IGFBP-1) m
31 patic gene expression using the model of the insulin-like growth factor binding protein-1 (IGFBP-1) p
32 serum insulin-like growth factor-I (IGF-I), insulin-like growth factor binding protein-1 and -3 (IGF
34 nts DAF-16 binding to its target site in the insulin-like growth factor binding protein-1 gene, the i
36 with acute pulmonary embolism had the lowest insulin-like growth factor binding protein-1 level (5 2-
41 ver-operating-characteristic curve for serum insulin-like growth factor binding protein-1 was 0.98 0.
43 imately 70% decrease in the plasma levels of insulin-like growth factor binding protein-1, a marker o
44 on protein to activate transcription via the insulin-like growth factor binding protein-1-insulin res
48 cts with an insulin response sequence in the insulin-like growth factor-binding protein-1 (IGFBP-1) g
49 hosphoenolpyruvate carboxykinase (PEPCK) and insulin-like growth factor-binding protein-1 (IGFBP-1) g
50 binds to the insulin response element of the insulin-like growth factor-binding protein-1 (IGFBP-1) p
53 pG2 hepatoma cells show that FKHR stimulates insulin-like growth factor-binding protein-1 promoter ac
55 roteinase inhibitor and His(140)-Val(141) in insulin-like growth factor-binding protein-1, probably r
58 HLA-DR-degenerate epitope pool derived from insulin-like growth factor binding protein 2 (IGFBP-2).
59 tein 1 (TKA-1); colony stimulating factor-1; insulin-like growth factor binding protein 2 (IGFBP-2);
60 t grades revealed frequent overexpression of insulin-like growth factor binding protein 2 (IGFBP2) in
64 oblastomas multiforme) tumors and found that insulin-like growth factor binding protein 2 (IGFBP2) wa
66 sma macrophage inhibitory protein-1alpha and insulin-like growth factor binding protein 2 showed a si
70 e report here, we tested the hypothesis that insulin-like growth factor-binding protein 2 (IGFBP2) pr
71 R), which promotes HIF synthesis, as well as insulin-like growth factor-binding protein 2 (IGFBP2), a
72 everal genes, e.g., Sonic hedgehog (Shh) and Insulin-like growth factor-binding protein 2 (Igfbp2), t
73 xidase 3 (GPX3), apolipoprotein J/clusterin, insulin-like growth factor-binding protein 2, epithelial
74 enin to repress expression of the endogenous insulin-like growth factor binding protein-2 (IGFBP-2) g
75 ide, interleukin-6, soluble CD40 ligand, and insulin-like growth factor binding protein-2) and 5 clin
77 s the addition of a neutralizing antibody to insulin-like growth factor-binding protein-2 (IGFBP-2) r
79 on between prediagnostic levels of IGF-I and insulin-like growth factor binding protein 3 (IGFBP-3) a
83 that insulin-like growth factor 1 (IGF-1) or insulin-like growth factor binding protein 3 (IGFBP-3) w
84 sulin, insulin-like growth factor 1 (IGF-1), insulin-like growth factor binding protein 3 (IGFBP-3),
85 ons of insulin-like growth factor 1 (IGF-1), insulin-like growth factor binding protein 3 (IGFBP-3),
86 ular genes, including increased mRNA for the insulin-like growth factor binding protein 3 (IGFBP-3).
87 th factor I (IGF-I) and inversely related to insulin-like growth factor binding protein 3 (IGFBP-3).
88 owth factor binding protein 1 (IGFBP-1), and insulin-like growth factor binding protein 3 (IGFBP-3).
89 growth factor type 1 and its carrier protein insulin-like growth factor binding protein 3 (IGFBP-3).
90 secretome proteomics approach, we identified insulin-like growth factor binding protein 3 (IGFBP3) as
91 or induction of the proapoptotic target gene insulin-like growth factor binding protein 3 (IGFBP3) by
92 K regulates Runx1-dependent transcription of insulin-like growth factor binding protein 3 (IGFBP3), a
93 ugh a previously unappreciated activation of insulin-like growth factor binding protein 3 (IGFBP3), w
94 n binding of insulin-like growth factor-I to insulin-like growth factor binding protein 3 and the pre
95 factor-I, insulin-like growth factor-II, and insulin-like growth factor binding protein 3 and these l
96 tion of a specific protease directed against insulin-like growth factor binding protein 3 and to rela
97 Whites had higher 25-hydroxyvitamin D and insulin-like growth factor binding protein 3 but lower 1
98 vity, serum insulin-like growth factor I and insulin-like growth factor binding protein 3 concentrati
100 increasing insulin-like growth factor-I and insulin-like growth factor binding protein 3 concentrati
102 of insulin-like growth factor 1 (IGF-1) and insulin-like growth factor binding protein 3 in 6,520 wo
103 pression of p53 target genes, p21(waf-1) and insulin-like growth factor binding protein 3 in glioma c
104 onsurvivor, insulin-like growth factor-I and insulin-like growth factor binding protein 3 remained lo
105 trations of insulin-like growth factor-I and insulin-like growth factor binding protein 3 were higher
106 esence of protease activity directed against insulin-like growth factor binding protein 3 were invest
107 a survivor, insulin-like growth factor-I and insulin-like growth factor binding protein 3 were low in
108 S/FLI1 decreased the transcript half-life of insulin-like growth factor binding protein 3, a down-reg
109 tein 7, fibroblast growth factor receptor 2, insulin-like growth factor binding protein 3, and platel
113 nificant differences by supplement in plasma insulin-like growth factor-binding protein 3 (44 and 40
114 of insulin-like growth factor 1 (IGF-1) and insulin-like growth factor-binding protein 3 (IGFBP-3) a
117 d plasminogen activator inhibitor-1 (PAI-1), insulin-like growth factor-binding protein 3 (IGFBP-3),
119 for the suppression of HIF-responsive genes insulin-like growth factor-binding protein 3 (IGFBP3), D
120 characterized by down-regulation of Fas and insulin-like growth factor-binding protein 3 and by incr
121 the potential apoptotic targets Bax and the insulin-like growth factor-binding protein 3 gene (IGF-B
122 uences derived from the bax promoter and the insulin-like growth factor-binding protein 3 gene (IGF-B
123 m the p53-responsive portions of the Bax and insulin-like growth factor-binding protein 3 gene promot
125 messenger RNAs for the IGF-I transgene, and insulin-like growth factor-binding protein 3 were assaye
126 ree thyroxine, insulin-like growth factor 1, insulin-like growth factor-binding protein 3, bone alkal
129 cts of insulin-like growth factor-I (IGF-I), insulin-like growth factor binding protein-3 (IGFBP-3) a
130 n identified neutral endopeptidase (NEP) and insulin-like growth factor binding protein-3 (IGFBP-3) a
132 to determine whether Compound 49b stimulates insulin-like growth factor binding protein-3 (IGFBP-3) a
134 d with IGF-II expression and positively with insulin-like growth factor binding protein-3 (IGFBP-3) e
138 from this laboratory revealed that vitreous insulin-like growth factor binding protein-3 (IGFBP-3) i
139 to increased endothelial cell expression of insulin-like growth factor binding protein-3 (IGFBP-3) i
140 NKX3.1 expression in PC-3 cells increased insulin-like growth factor binding protein-3 (IGFBP-3) m
141 nd RXR-specific ligands on the regulation of insulin-like growth factor binding protein-3 (IGFBP-3) p
144 gulates the well-documented growth inhibitor insulin-like growth factor binding protein-3 (IGFBP-3).
146 ltered in the Id cKO hearts, but addition of Insulin-Like Growth Factor binding protein-3 (IGFbp3) re
148 asked whether the hypoxia-regulated factor, insulin-like growth factor binding protein-3 (IGFBP3), c
149 insulin-like growth factor-1 (P < 0.03) and insulin-like growth factor binding protein-3 (P < 0.02)
150 ulin-like growth factor-I and restoration of insulin-like growth factor binding protein-3 levels, and
151 ase in plasminogen activator inhibitor-I and insulin-like growth factor binding protein-3 mRNA levels
152 ssion of connective tissue growth factor and insulin-like growth factor binding protein-3 was observe
155 ng growth regulators (v-sis gene product and insulin-like growth factor binding protein-3, IGFBP-3) a
156 included serum insulin-like growth factor-1, insulin-like growth factor binding protein-3, prolactin,
157 The hepatic expression and serum levels of insulin-like growth factor-binding protein-3 (IGFBP-3) a
158 Insulin-like growth factor-I (IGF-I) and insulin-like growth factor-binding protein-3 (IGFBP-3) a
161 er retinoic acid receptor-beta (RARbeta) nor insulin-like growth factor-binding protein-3 (IGFBP-3) i
163 e same cells exhibited dramatic increases in insulin-like growth factor-binding protein-3 (IGFBP-3) m
166 identified in patients with ARDS, including insulin-like growth factor-binding protein-3 (IGFBP-3).
167 evels and the induction of wild-type p53 and insulin-like growth factor-binding protein-3 (IGFBP3) 24
169 tivates the insulin-response elements of the insulin-like growth factor-binding protein-3 and other i
170 g levels of insulin-like growth factor-1 and insulin-like growth factor-binding protein-3 early in cr
171 ced the anti-proliferative and pro-apoptotic insulin-like growth factor-binding protein-3 expression.
172 mulation (up to 5.8 fold; P < 0.05) of human insulin-like growth factor-binding protein-3 in mouse pl
173 854746 is also significantly associated with insulin-like growth factor-binding protein-3 levels in t
174 ciated with insulin-like growth factor-1 and insulin-like growth factor-binding protein-3 levels; Acu
181 nocerebellar ataxia [SCA1]), growth factors (insulin-like growth factor binding protein 4 and transfo
185 To identify the molecular mechanism by which insulin-like growth factor binding protein-4 (IGFBP-4) e
186 lagen type IV may suppress the expression of insulin-like growth factor binding protein-4 (IGFBP-4),
187 Furthermore, the differential expression of insulin-like growth factor binding protein 5 (IGFBP-5) w
189 scriptional changes, with down-regulation of insulin-like growth factor binding protein 5 (Igfbp5) re
191 tor, laminin receptor homolog, beta-tubulin, insulin-like growth factor binding protein 5, KIAA0179 p
194 d a strong upregulation in the expression of insulin-like growth factor-binding protein 5 (IGFBP5), a
195 eracts with the promoter of IGFBP5 (encoding insulin-like growth factor-binding protein 5) and displa
196 sformed BT549 cells include C-type lectin 2, insulin-like growth factor-binding protein 5, cathepsins
197 ins had an alteration in their expression of insulin-like growth factor binding protein-5 (IGFBP-5) a
198 The authors found transcript expression for insulin-like growth factor binding protein-5 (IGFBP-5) w
199 retinal glia and appeared to be mediated by insulin-like growth factor binding protein-5 (IGFBP-5),
200 crete an 88-kDa serine protease that cleaves insulin-like growth factor binding protein-5 (IGFBP-5).
207 Glucocorticoids inhibit the synthesis of insulin-like growth factor-binding protein-5 (IGFBP-5) i
208 gly Akt induced significant up-regulation of insulin-like growth factor-binding protein-5 (IGFBP-5),
209 cancer model and decreased the expression of insulin-like growth factor-binding protein-5 (IGFBP5).
210 nd up-regulation of one of its target genes, insulin-like growth factor-binding protein-5, which is t
211 pression of CITED1, claudin-10 (CLDN10), and insulin-like growth factor binding protein 6 (IGFBP6) bu
212 owth-regulatory biomarkers, such as IGFBP-6 (insulin-like growth factor-binding protein 6), RARbeta,
213 to the inflammation-associated transcripts, insulin-like growth factor-binding protein 6, macrophage
214 PAOh1 and ELF3, while prominent induction of insulin-like growth-factor-binding protein 6 (IGFBP6) st
215 -1, cartilage oligomeric matrix protein, and insulin-like growth factor binding protein 7 concentrati
218 tissue inhibitor of metalloproteinase-2 and insulin-like growth factor-binding protein 7 accurately
219 tissue inhibitor of metalloproteinase-2 and insulin-like growth factor-binding protein 7 in patients
220 tissue inhibitor of metalloproteinase-2 and insulin-like growth factor-binding protein 7 in this pop
221 tissue inhibitor of metalloproteinase-2 and insulin-like growth factor-binding protein 7 results in
222 tissue inhibitor of metalloproteinase-2 and insulin-like growth factor-binding protein 7 significant
223 tissue inhibitor of metalloproteinase-2 and insulin-like growth factor-binding protein 7 test was no
224 tissue inhibitor of metalloproteinase-2 and insulin-like growth factor-binding protein 7 was 0.84 (0
225 owed by a marker of cell cycle arrest (urine insulin-like growth factor-binding protein 7) and, final
226 onal induction of the Drosophila ortholog of insulin-like growth factor-binding protein 7, which syst
228 or-suppressor gene (period homolog 3 [PER3], insulin-like growth-factor-binding protein, acid labile
229 alog of IGF-I with very low affinity for the insulin-like growth factor-binding proteins (IGF-BPs), d
231 insulin and depressed levels of circulating insulin-like growth factor binding protein (IGFBP) -1, w
232 oth RA and TGF-beta2 increased the levels of insulin-like growth factor binding protein (IGFBP) 3 (2-
233 valuated the relationship of insulin, IGF-I, insulin-like growth factor binding protein (IGFBP) 3, an
234 , as expected if Notch1 loss altered the IGF/insulin-like growth factor binding protein (IGFBP) balan
235 nhibits prostate cancer growth and increases insulin-like growth factor binding protein (IGFBP) expre
236 ing indicated substantial down-regulation of insulin-like growth factor binding protein (Igfbp) genes
237 f the fetal insulin-like growth factor (IGF)-insulin-like growth factor binding protein (IGFBP) syste
238 To determine the cellular source of this insulin-like growth factor binding protein (IGFBP), 11 m
239 traction-promoting activity, the presence of insulin-like growth factor binding protein (IGFBP), and
242 es and insulin-like growth factor-I (IGF-I), insulin-like growth factor binding protein (IGFBP)-1, an
243 tor (PDGF) isoforms (PDGF-AA, -BB, and -AB), insulin-like growth factor binding protein (IGFBP)-2, an
247 , we examined the expression and function of insulin-like growth factor binding proteins (IGFBP)-3 an
248 d hepatocyte nuclear factor (HNF) 4alpha and insulin-like growth factor-binding protein (IGFBP) 1 mRN
249 CCN1 comprising completely or partially the insulin-like growth factor-binding protein (IGFBP) and v
250 quadricarinatus, we have identified a novel insulin-like growth factor-binding protein (IGFBP) terme
253 experiments demonstrated that PAPP-A cleaves insulin-like growth factor-binding protein (IGFBP)-2, bu
254 med gene expression profiling and identified insulin-like growth factor-binding protein (IGFBP)-3 as
259 Binding of STC2 prevents PAPP-A cleavage of insulin-like growth factor-binding protein (IGFBP)-4 and
263 ortic smooth muscle cells (SMCs) secrete two insulin-like growth factor-binding proteins (IGFBP), IGF
264 ts of IGFs on cells are modulated by various insulin-like growth factor-binding proteins (IGFBP).
267 early response (TIEG), SMAD5, SMAD7, SMAD2, insulin-like growth factor binding protein (IGFBP7), IGF
268 was associated with a marked upregulation of insulin-like growth factor binding proteins (IGFBPs) 3 a
269 igment epithelial (RPE) cells as a source of insulin-like growth factor binding proteins (IGFBPs) and
273 ase have been associated with changes in the insulin-like growth factor binding proteins (IGFBPs), a
274 gulated by six soluble binding proteins, the insulin-like growth factor binding proteins (IGFBPs), wh
275 ity are, in part, attributable to changes in insulin-like growth factor binding proteins (IGFBPs).
277 ine if ERT in vivo affects the production of insulin-like growth factor binding proteins (IGFBPs).
281 generation and the contributions of vitreous insulin-like growth factor-binding proteins (IGFBPs) tow
282 as to evaluate the contributions of vitreous insulin-like growth factor-binding proteins (IGFBPs) tow
283 acellular matrix (ECM) contains two forms of insulin-like growth factor-binding proteins (IGFBPs), IG
284 propeptide shares a 20-25% identity to human insulin-like growth factor-binding proteins (IGFBPs), su
285 atocyte growth factor, endocrine gland VEGF, insulin-like growth factor binding proteins, or endostat
288 25.1, a novel protein interacting with mac25/insulin-like growth factor-binding protein-related prote
289 on their apical surface, and the profile of insulin-like growth factor binding proteins resembled th
290 mains that bear sequence similarities to the insulin-like growth factor-binding proteins, von Willebr
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