ライフサイエンスコーパス: insult to

1 increased from a minimum at the end of brain insult to a maximum of 43 +/- 17 torr (5.7 +/- 2.3 kPa)

2 uries, yet nRT cells generally survive these insults to a greater degree than TCR cells.

3 roles in coupling hypoxic-ischemic neuronal insults to activation of p53 and hypoxic cell death.

4 mod accelerated barrier recovery after acute insults to aged and young skin.

5 he data may also help to explain why genetic insults to alpha4 subunits are associated with working m

6 The results also explain why genetic insults to alpha7-nAChR would profoundly disrupt cogniti

7 eriments provide strong evidence for a local insult to axons in the optic nerve.

8 ophy of RGCs, whereas the second involves an insult to axons, which causes the degeneration of axon p

9 s) allele, which is known to protect against insults to axons, strongly protects against DBA/2J glauc

10 Given that transient insults to brain may lead to long-term brain damage, the

11 arated mechanisms of action: (i) an indirect insult to cell bodies, requiring the presence of Schwann

12 UV irradiation is one of the major external insults to cells and can cause skin aging and cancer.

13 s protection against a wide variety of toxic insults to cells, including cytotoxic cancer chemotherap

14 biologic basis for the syndrome is likely an insult to cerebral vascular autoregulation.

15 re key steps in the continuum from allograft insult to chronic allograft dysfunction.

16 ntribute to the transition from acute tissue insult to chronic pain and to pain conditions for which

17 rate descriptions of the underlying physical insults to complex neuron structures at the nano-meter s

18 The nature of the insult to cultures was quantified by the resazurin-reduc

19 The prolonged period from insult to death suggests that the window for successful

20 The disorder results from various insults to different areas within the developing nervous

21 cretion and may be perturbed by diabetogenic insults to disrupt glucose homeostasis in humans.

22 age-related cognitive decline, while genetic insults to DNC proteins are commonly linked to schizophr

23 athway activated by a wide range of cellular insults to elicit innate immune defenses through the act

24 These data suggest a link between fetal insults to epigenetic modification of genes and the resu

25 Insults to ER homeostasis activate the unfolded protein

26 this deadly transition from early myocardial insult to heart failure and sudden death is a key goal f

27 rehabilitation following other neurological insults to higher cognitive systems.

28 well as the ability of a number of oxidative insults to induce synuclein oligomerization.

29 Reperfusion adds further insult to injury.

30 port that excitotoxic (N-methyl-D-aspartate) insults to mature cerebrocortical neurons activate caspa

31 plausible pathway linking external seasonal insults to mothers with thymic development in their infa

32 ld produce recurrent and cumulative ischemic insults to multiple organs, such as the brain.

33 lead to secondary hyperalgesia triggered by insult to muscle afferents.

34 ance of Delay cells on NMDAR may explain why insults to NMDARs in schizophrenia or Alzheimer's diseas

35 Insults to nuclear DNA induce multiple response pathways

36 induced by an interaction between metabolic insult to oligodendrocytes and inflammatory mediators.

37 this feature can be used to localize soluble insults to one compartment for up to 20 h after each med

38 on is safe and can be performed with minimal insult to overall renal function.

39 g to a wide variety of potentially oncogenic insults to prevent tumour development.

40 adation of the nuclear lamina upon oncogenic insults to reinforce cellular senescence.

41 d-go technique results in a massive ischemic insult to several major organ systems.

42 process, which is accelerated by an ischemic insult to the allograft.

43 A mechanical insult to the brain drastically alters the microenvironm

44 Acquired epilepsy (i.e., after an insult to the brain) is often considered to be a progres

45 Insult to the central nervous system (CNS) induces many

46 Inflammatory insult to the colon on post-natal day 10 caused an aberr

47 We investigated whether inflammatory insult to the colons of neonatal rats induced GHS in adu

48 Inflammatory insult to the colons of rat pups leads to GHS in adult l

49 of premature birth caused by nitro-oxidative insult to the developing retinal vasculature during ther

50 xtravasated and oxidized LDL is an important insult to the diabetic retina.

51 absence of Xblm, even without any exogenous insult to the DNA, may help to explain the genesis of ch

52 ult of severe periodontal disease acts as an insult to the endothelium.

53 mpal recordings of epileptogenesis (from the insult to the first spontaneous seizure) from two labora

54 Endotoxin causes an ischemic insult to the gallbladder similar to that seen in acalcu

55 h abdominal pain resulting from an ischaemic insult to the gastrointestinal tract.

56 demonstrate that a viral infection, a toxic insult to the gut, commensal bacteria, and a Crohn's dis

57 T/HS cause ischemic insult to the gut, resulting in the release of biologica

58 Pressure overload is a common pathological insult to the heart and the resulting hypertrophy is an

59 nd and persistent memory loss resulting from insult to the hippocampus in infancy becomes evident in

60 hological observations suggest that a direct insult to the host vasculature may also be important.

61 ental disruption associated with the initial insult to the immature brain, injury processes can persi

62 de pannus invasion following an inflammatory insult to the joint.

63 that drive fibrogenesis after an initiating insult to the kidney are incompletely understood.

64 yte FGFR4 protects against acute and chronic insult to the liver and prevents accompanying fibrosis.

65 Alcohol insult to the liver sets off a complex sequence of infla

66 eroxidation were similar irrespective of the insult to the livers that resulted in fatty liver.

67 structive lung disease that follows a severe insult to the lower respiratory tract, resulting in fibr

68 ute lung injury (ALI, not caused by a direct insult to the lung) represents the first organ dysfuncti

69 t acute lung injury (ALI not due to a direct insult to the lung), the contribution of various regulat

70 Heat-shock is an acute insult to the mammalian proteome.

71 enon and a manifestation of greater ischemic insult to the myocardium.

72 be caused by traumatic, toxic, or metabolic insult to the nerve.

73 ollow deafening can be prevented by a second insult to the nervous system--the lesion of a basal gang

74 ve cell loss, suggesting that a time-limited insult to the nigrostriatal system can set in motion a s

75 in SS is determined by the direct autoimmune insult to the ocular surface epithelia, whereas in MGD p

76 A primary insult to the pancreatic islets of Langerhans, leading t

77 on of this analog up to 1h after an ischemic insult to the rat brain had a potent neuroprotective act

78 cell (RGC) loss/survival following a defined insult to the retina is a prerequisite in order to allow

79 lso protective against a subsequent ischemic insult to the same kidney, revealing that systemic uremi

80 drome may develop following a focal vascular insult to the sixth nerve trunk with axonal degeneration

81 It is particularly controversial whether an insult to the type II AEC initiates an inflammatory resp

82 us system development and after pathological insults to the adult nervous system.

83 Insults to the airway epithelium play a key role in cons

84 levels of responsiveness produced by various insults to the airway.

85 th PAMPs and DAMPs can be liberated by early insults to the allograft, including ischemia/reperfusion

86 er cognition and help to explain why genetic insults to the alpha4 subunit weaken cognitive and atten

87 cent theories which link early developmental insults to the amygdala with the ToM impairments which a

88 th genetic barrier defects and environmental insults to the barrier with AD suggests that epidermal b

89 part of the innate immune response following insults to the body.

90 leukin-1 (IL-1) is induced immediately after insults to the brain, and elevated levels of IL-1 have b

91 dence for a compensatory mechanism following insults to the brain, and insight into the dynamic natur

92 bolism commonly occur following a variety of insults to the brain.

93 in inhibition on the BBB following traumatic insults to the brain.

94 ctive agent against neural damage by various insults to the brain.

95 of CK2 as a neuroprotectant after oxidative insults to the brain.

96 d by neuro-degenerative disease or traumatic insults to the brain.

97 Chronic pain caused by insults to the CNS (central neuropathic pain) is widely

98 thesized to occur in epilepsy after acquired insults to the CNS as well.

99 nefits may be achieved for a wide variety of insults to the CNS by regulating tPA activity to preserv

100 ility of children to trauma or to infectious insults to the CNS.

101 Our data suggest that severe nutritional insults to the developing brain in utero may be capable

102 logical studies showing associations between insults to the developing lung during prenatal and early

103 n that fungal pathogens and yeast respond to insults to the endoplasmic reticulum (ER) by initiating

104 bles proper inflammatory response to harmful insults to the host, and increases resilience of the ent

105 e immune responses to sterile and infectious insults to the host.

106 s demonstrate that SIV produces long-lasting insults to the humoral immune system beginning very earl

107 MP fluxes are secondary to pathophysiologic insults to the liver or might reflect compensatory respo

108 d clinical disorders caused by many clinical insults to the lung or because of predispositions to lun

109 neutralize potentially harmful and frequent insults to the lung, and appropriate regulation of leuko

110 isease occurring after years of inhalational insults to the lungs, pinpointing abnormalities or disru

111 low distinction of acute vs. remote ischemic insults to the myocardium.

112 tective effects have not been assessed after insults to the neonatal brain.

113 t of neuropathic pain, triggered by multiple insults to the nervous system, is a clinical challenge b

114 ce caused by a wide variety of exposures and insults to the oral cavity.

115 ulfatase are sufficient to accommodate acute insults to the permeability barrier.

116 of the sequelae of environmental and genetic insults to the postnatal cerebrum.

117 We determined SPN responses to two common insults to the preterm brain: hypoxia-ischemia and hypox

118 duced by mild-to-moderate severity traumatic insults to the rat spinal cord.

119 Despite this insult to their genetic integrity, these cells survive w

120 toxicity in vitro, and mild hypoxic/ischemic insult to these mice in vivo results in significantly in

121 Insult to this pathway may underlie BBB dysfunction and

122 ation with use of yttrium 90, inflict lethal insult to tumors while preserving normal hepatic parench