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1 membrane pemphigoid and mAb GoH3 and BQ16 to integrin alpha6.
2 tes with the down-regulation of cell surface integrin alpha6.
3 we developed a platelet-specific knockout of integrin alpha6.
4             This process entirely depends on integrin alpha6, a receptor for laminin.
5 in luminal cells that express keratin 14 and integrin-alpha6, a phenotype that is usually expressed e
6 ring the cells on membranes coated with anti-integrin alpha6 and beta1 antibodies.
7 maintained on feeders or off feeders express integrin alpha6 and beta1, which may form a laminin-spec
8          This promotion is inhibited by anti-integrin alpha6 and beta4 antibodies and by phosphatidyl
9 e proband revealed negative staining for the integrin alpha6 and markedly reduced staining for the be
10 ch as plectin, collagen type XVII/BP180, and integrin alpha6 and ss4 chains, seen in conventional Lam
11 l TACE constructs demonstrated not only that integrins alpha6 and beta1 bind to TACE via the disinteg
12                          The contribution of integrins alpha6 and beta1 to differentiation of fetal e
13 pe IV collagen, type VII collagen, perlecan, integrin alpha6, and epithelial cell differentiation mar
14                       In organ culture, anti-integrin alpha6 antibody and wortmannin reduce tooth ger
15 uced apoptosis, whereas those plated on anti-integrin alpha6 antibody were not.
16                                              Integrin alpha6 antisense-containing cell clones exhibit
17 gment E8, and dependence of this response on integrin alpha6 beta1 and at least one other long arm-bi
18 ls were a subset of the previously described integrin alpha6(+)CD34(+) bulge cell population, and 28.
19  enhanced expression of CD90, c-Kit (CD117), integrin alpha6 (CD49f), and CXCR4 (CD184).
20        The p67 LBP+ T cells also express the integrin alpha6 chain (CD49f), which is known to associa
21 s, suggesting that the formation of the CD82-integrin alpha6 complex reduces alpha6 integrin cell sur
22 h as CD9, CD81, and CD151 did not block this integrin alpha6-dependent morphogenesis.
23                            All sera bound to integrin alpha6 in DU145 cell lysate by immunoprecipitat
24 xpected, CD82 physically associated with the integrin alpha6 in Du145-CD82 transfectant cells, sugges
25 ment significantly reduced the expression of integrin alpha6, integrin beta4, Fak, paxillin, Rac1/2/3
26 st therapeutic interventions to inhibit CCN1-integrin alpha6 interactions to sensitize gliomas to vir
27 Finally, the internalization of cell surface integrin alpha6 is significantly enhanced upon CD82 expr
28                                  An SNP near integrin alpha6 (ITGA6) reached genome-wide significance
29 sing cytokeratin15 (KRT15), CD200, CD34, and integrin, alpha6 (ITGA6) were quantitated via flow cytom
30 reshly isolated club cells express Sca-1 and integrin alpha6, markers commonly used to characterize l
31 )-dependent hepatocyte proliferation through integrin alpha6-mediated accumulation of reactive oxygen
32 a peptide within the extracellular domain of integrin alpha6 molecule, to which Abs in the sera from
33                                 Blocking AR, integrin alpha6, NF-kappaB, or Bcl-xL concurrent with in
34                                       Unlike Integrin-alpha6(+) or c-Kit(+) cells, E-Cad/Lgr6(+) sing
35                                 We find that integrin alpha6 participates in this pathway, but either
36 he fluorescently labeled proteins identified integrin alpha6 precursor as a protein associated in a c
37                        The identification of integrin alpha6 precursor was confirmed by Western blot
38 our results indicate that 1) CD82 attenuates integrin alpha6 signaling during a cellular morphogenic
39                                          The integrin alpha6 subunit mediates an attachment of the ce
40 Control sera did not bind to the full-length integrin alpha6 subunit nor any of the cloned fragments.
41  the binding of OP autoantibodies within the integrin alpha6 subunit, by cloning four overlapping fra
42 tein complex is required in conjunction with integrin alpha6 to reduce muscle degeneration.
43 s; and 3) the accelerated internalization of integrin alpha6 upon CD82 expression correlates with the
44                                  hEpiSC-rich integrin-alpha6(+ve) hERM cells derived by fluorometry c
45 t the lateral membrane, and beta-catenin and integrin alpha6 were located at the basolateral membrane

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