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1 fficking that contributes to invasion is the integrin alpha6beta4.
2 wn to form a complex with the hemidesmosomal integrin alpha6beta4.
7 ponse of the actin cytoskeleton and specific integrins (alpha6beta4, alpha6beta1, and alpha3beta1) to
9 diated by transmembrane proteins such as the integrin alpha6beta4 and bullous pemphigoid antigen 2 wi
10 t that the interaction between laminin-10/11-integrin alpha6beta4 and the phosphatidylinositol 3-kina
12 age and motility of epithelial cells through integrins alpha6beta4 and alpha3beta1, respectively.
13 n expression patterns, in which the exosomal integrins alpha6beta4 and alpha6beta1 were associated wi
15 e effects by forming a complex with HER2 and integrin alpha6beta4 at the cell surface that disrupts d
16 en established, this study demonstrates that integrin alpha6beta4 can dramatically impact the epigeno
18 Likewise, we provide the novel finding that integrin alpha6beta4 confers an enhanced ability on cell
19 utant BM could not induce stable adhesion by integrin alpha6beta4, consistent with the presence of ju
22 rate that increased autotaxin secretion from integrin alpha6beta4 expressing cells acts to enhance ch
24 tment of lung disease by identifying a novel integrin alpha6beta4-expressing alveolar epithelial cell
26 ethylation were up-regulated dramatically by integrin alpha6beta4 expression, including S100A4, FST,
27 n abrogates strictly polarized expression of integrin alpha6beta4 in basal keratinocytes and negative
28 tor (EGF-R) combines with the hemidesmosomal integrin alpha6beta4 in both normal and neoplastic kerat
31 r of the epithelium, whereas in mutant mice, integrin alpha6beta4 is expressed around the cell surfac
35 iles of MDA-MB-435 cells that stably express integrin alpha6beta4 (MDA/beta4) and vector-only-transfe
36 cytes and negatively impacts the laminin-332/integrin alpha6beta4 signaling axis guiding keratinocyte
37 y bisulfate sequencing, thus suggesting that integrin alpha6beta4 signaling can lead to the demethyla
39 me bisulfite sequencing (WGBS) revealed that integrin alpha6beta4 signaling promotes an overall hypom
40 eover, matrix metalloproteinase activity and integrin alpha6beta4 signaling were required for AREG se
42 Taken together, these data illustrate that integrin alpha6beta4 stimulates invasion by promoting au
43 laminin 5, collagen, or an antibody against integrin alpha6beta4, suggesting that signaling through
44 e many known signaling functions mediated by integrin alpha6beta4 that promote invasive properties ha
47 ich mediates rapid tumor cell migration, and integrin alpha6beta4, which often mediates stable cell a
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