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1                           In each heart, the interatrial and internodal pathways were similarly invol
2 d left atria, right and left ventricles, and interatrial and interventricular septa.
3 pling interval, from the right atrium to the interatrial area and finally to the left atrium.
4 bility of fetal catheterization to create an interatrial communication and describes technical consid
5 with significant increase in the size of the interatrial communication and fossa ovalis, and decrease
6 gent catheterization to create or enlarge an interatrial communication before surgical palliation.
7   The patent foramen ovale (PFO) is a normal interatrial communication during fetal life that persist
8 (HLHS) have been correlated with restrictive interatrial communication or intact atrial septum (RAS)
9 tients underwent transcatheter closure of an interatrial communication using the CardioSEAL (n = 22),
10 c defect resulting in a small but persistent interatrial communication.
11 d with a matched control group with adequate interatrial communication.
12 ormal other than for the demonstration of an interatrial communication.
13                                              Interatrial communications may play a role in the etiolo
14 e been reported in hundreds of patients with interatrial communications, patent ductus arteriosus, an
15 n 12% of adults and has been associated with interatrial communications.
16 e (BB) is considered to be the main route of interatrial conduction and to play a role in development
17 malities, 68 had atrial fibrillation (AF) or interatrial conduction block (IAB) (P wave duration, >or
18   Pathologic analysis revealed that complete interatrial conduction block was associated with conflue
19                     Recent data suggest that interatrial conduction can be important in triggering an
20 arization (Ta-TcD) are believed to represent interatrial conduction defect.
21 he slower atria after ablation close to main interatrial conduction fascicles (P=0.035).
22          Therefore, a method for attenuating interatrial conduction may have therapeutic value.
23                                              Interatrial conduction occurs in specific zones.
24 ught to characterize a method of attenuating interatrial conduction using radiofrequency ablated lesi
25 Cx40 plays an essential role in establishing interatrial conduction velocity heterogeneity in the mur
26                          In 13 healthy pigs, interatrial conduction was evaluated before and after se
27                    After ablation of zone 1, interatrial conduction was slowed, but there was no bloc
28 lation of the right atrial septum attenuated interatrial conduction without disturbing atrioventricul
29 lation of the right atrial septum, targeting interatrial conduction zones.
30  frequency domain analysis of a wide bipolar interatrial electrogram describes the global organizatio
31                                           An interatrial fenestration is designed to maintain cardiac
32               In patients with potential for interatrial flow, this may lead to a right-to-left shunt
33 onfidence interval, 6.1-6.23; P=0.021), with interatrial frequency gradients established by the spati
34 scent at E7.5, and in the outflow tract, the interatrial groove, the atrioventricular canal and right
35 TAPVR, PDA, cardiac apex and heart chambers, interatrial or interventricular septal defects, pericard
36 syndrome patients subdivided into those with interatrial (pretricuspid) versus interventricular or gr
37                               One year after interatrial septal shunt device implantation, there were
38 rved ejection fraction patients 1 year after interatrial septal shunt device implantation.
39 nary analyses have demonstrated that a novel interatrial septal shunt device that allows shunting to
40  participated in the open-label study of the interatrial septal shunt device.
41 ; wall thickness (r=-0.54 to -0.63, P<0.04); interatrial septal thickness (r=-0.52, P=0.001); and dia
42 sisting of a bridge (suture) element between interatrial septal wall and great cardiac vein anchors.
43 o 91% for all scallops of both MV leaflets), interatrial septum (84%), left atrial appendage (86%), a
44  regions of the adult heart: clusters in the interatrial septum and around the pulmonary veins, scatt
45             Two thrombi became wedged in the interatrial septum and incompletely withdrawn into the R
46 thickening of the mitral valve leaflets, and interatrial septum and mild pericardial effusion.
47 measurements of enhancement thickness at the interatrial septum and the number of days after ablation
48 ivation of the right and left aspects of the interatrial septum is discordant.
49              Targeting specific sites of the interatrial septum is followed by an increase in heart r
50 ber architecture of right and left atria and interatrial septum provide a means of investigating the
51 ntegrated backscatter image sequences of the interatrial septum to internally calibrate the left vent
52  of the patients) or right side (36%) of the interatrial septum was observed to be responsible for >/
53 ated Langendorff-perfused rabbit hearts, the interatrial septum was perforated to equalize biatrial p
54 l valve (MV), aortic valve, tricuspid valve, interatrial septum, and left atrial appendage were obtai
55 ght atrium, pacing from novel sites like the interatrial septum, Bachman bundle, and dual-site or bia
56 arly along the right and left aspects of the interatrial septum, is not clear.
57  both atria, but were mainly confined to the interatrial septum, pulmonary veins, roof of left atrium
58 corresponded to the expected position of the interatrial septum.
59 n experience with an implanted left-to-right interatrial shunt demonstrates initial safety and early
60 domized, open-label studies, a transcatheter interatrial shunt device (IASD, Corvia Medical) was asso
61 he performance and safety of a transcatheter interatrial shunt device (IASD, Corvia Medical, Tewkesbu
62                        A novel transcatheter interatrial shunt device has been developed and evaluate
63 he rationale for a therapeutic transcatheter interatrial shunt device in HFpEF, and we describe the d
64                           Implantation of an interatrial shunt device is feasible, seems to be safe,
65 e of MHA and assessed whether closure of the interatrial shunt in patients with MHA would result in i
66 ed to indicate the presence of a significant interatrial shunt that was eventually detected following
67 hesized that the creation of a left-to-right interatrial shunt to decompress the left atrium (without
68 n associated atrial septal aneurysm or large interatrial shunt, the rate of stroke recurrence was low
69 n associated atrial septal aneurysm or large interatrial shunt, to transcatheter PFO closure plus lon
70 ential efficacy of therapeutic left-to-right interatrial shunting in patients with heart failure with
71 y considering the physiologic effects during interatrial shunting.
72 basis of clinical experience with congenital interatrial shunts in mitral stenosis, it has been hypot
73 o determine whether transcatheter closure of interatrial shunts is an effective treatment for MHA.
74 .2 years), of whom 81% had moderate or large interatrial shunts.

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