ライフサイエンスコーパス: interleukin-22

1 ncluding interleukin 10, interleukin 13, and interleukin 22.

2 -type lectins Reg3gamma and Reg3beta, and of interleukin 22.

3 ing higher levels of the reparative cytokine interleukin-22.

4 vation of TLR5 on DCs leads to production of interleukin-22.

5 ing the interleukin-23/T helper 17 cytokine, interleukin-22.

6 lete loss of flagellin-induced production of interleukin-22.

7 r necrosis factor-alpha, interleukin-17, and interleukin-22.

8 was an inverse association between levels of interleukin 22 and serum levels of tryptophan.

9 This induction required the cytokines interleukin-22 and lymphotoxin in a commensal bacteria-d

10 Human CD4+ T cells that produce interleukin 22 are an essential component of skin defens

11 Serum concentration of interleukin 22 associated with disease activity in patie

12 rs exhibited low levels of tumor-suppressive interleukin-22 binding protein (IL-22BP) compared to nor

13 rom patients with IBD produce high levels of interleukin-22 binding protein (IL-22BP), the endogenous

14 h increased expression of interleukin-17 and interleukin-22 by day 5 after injury.

15 Immunity, Kryczek et al. (2014) reveal that interleukin-22 can also promote "stemness" in human colo

16 tective measures against such events include interleukin-22-driven systemic elimination of pathobiont

17 n in hematopoietic cells and is dependent on interleukin 22 expression.

18 e T cells homed to skin, where they produced interleukin 22 (IL-22) in response to CD1a on Langerhans

19 Previously, we found that interleukin 22 (IL-22) inhibits intracellular growth of

20 Interleukin 22 (IL-22) is a member of the IL-10 cytokine

21 Interleukin 22 (IL-22) is an IL-10-related cytokine prod

22 Interleukin 22 (IL-22) is emerging as a key cytokine for

23 Studies have shown that interleukin 22 (IL-22) is expressed at barrier surfaces

24 ntestinal mucosa, through interleukin 17 and interleukin 22 (IL-22) production.

25 ulosis for 24 hours yielded higher IL-10 and interleukin 22 (IL-22) transcript levels for tuberculosi

26 Interleukin 22 (IL-22), a cytokine of the IL-10 superfam

27 Interleukin 22 (IL-22), which is produced by cells of th

28 ntestinal mucosa from infection by secreting interleukin 22 (IL-22).

29 The mechanism of action of interleukin- 22 (IL-22) in inflammatory arthritis remain

30 Interleukin-22 (IL-22) and IL-17A are both effector cyto

31 We detail such a mechanism, centered on interleukin-22 (IL-22) and triggered by the depletion of

32 ILC3s resulted in deficiency of intrathymic interleukin-22 (IL-22) compared with transplant recipien

33 , CD4(+) LTi cells were a dominant source of interleukin-22 (IL-22) early during infection.

34 athogen, Citrobacter rodentium, we show that interleukin-22 (IL-22) has a crucial role in the early p

35 Interleukin-22 (IL-22) has important functions in host d

36 Reduced innate interleukin-22 (IL-22) in Ahr-deficient mice allowed exp

37 Here we detected interleukin-22 (IL-22) in patient colorectal cancer tiss

38 thway in which innate lymphoid cells produce interleukin-22 (IL-22) in response to loss of double pos

39 Here, we report a critical role for interleukin-22 (IL-22) in systemic protection against ba

40 Interleukin-22 (IL-22) is a recently described IL-10 fam

41 Interleukin-22 (IL-22) is a T helper 17 (Th17) T cell-as

42 Interleukin-22 (IL-22) is central to host protection aga

43 Interleukin-22 (IL-22) is highly induced in response to

44 The cytokine interleukin-22 (IL-22) is primarily expressed by T helpe

45 Interleukin-22 (IL-22) is released by immune cells and m

46 fection, and mice with and without exogenous interleukin-22 (IL-22) or anti-IL-22 antibodies.

47 Interleukin-22 (IL-22) plays a critical role in mucosal

48 Interleukin-22 (IL-22) plays an important role in host i

49 g HFD with inulin restored microbiota loads, interleukin-22 (IL-22) production, enterocyte proliferat

50 arly colonization resistance was mediated by interleukin-22 (IL-22) regulation of the microbiota.

51 ously identified human ILC3 distinguished by interleukin-22 (IL-22) secretion.

52 We set out to test the hypothesis that interleukin-22 (IL-22), a cytokine crucial for epithelia

53 Interleukin-22 (IL-22), a recently identified member of

54 rotective role in liver disease by releasing interleukin-22 (IL-22), a recently identified T cell-der

55 t induction of gamma interferon (IFN-gamma), interleukin-22 (IL-22), and IL-17 expression (genes Ifn-

56 forming growth factor beta, forkhead box P3, interleukin-22 (IL-22), and IL-17 mRNA but caused minima

57 genes encoding gamma interferon (IFN-gamma), interleukin-22 (IL-22), and IL-17 were detected by quant

58 express aryl hydrocarbon receptor (AHR) and interleukin-22 (IL-22), supporting a role in mucosal imm

59 Interleukin-22 (IL-22), which acts as either a proinflam

60 ic cells elicited production of the cytokine interleukin-22 (IL-22), which induced a protective gene

61 how here that attenuated ileitis observed in interleukin-22 (IL-22)-deficient mice was associated wit

62 an secondary lymphoid tissues (SLTs) contain interleukin-22 (IL-22)-producing cells with an immature

63 Here, we show that interleukin-22 (IL-22)-producing innate lymphoid cells (

64 heir homeostasis and driving them to produce interleukin-22 (IL-22).

65 ut immunity presumably through production of interleukin-22 (IL-22).

66 Similar differences were observed with interleukin 22 (IL22) mRNA showing early downregulation

67 We measured levels of interleukin 22 in serum from 28 patients by enzyme-linke

68 ; and interleukin 6, interleukin 10, IL-17A, interleukin 22, interleukin 23, interferon gamma, kynure

69 Interleukin-22 is produced by certain T helper cells sub

70 edly downregulated in DM, but not sIBM, were interleukin 22, Kallmann syndrome 1 (KAL-1), an adhesion

71 Interestingly, higher levels of interleukin 22, monocyte chemoattractant protein 1, TNF-

72 silosis induced much less interleukin 17 and interleukin 22 production as compared to C. albicans.

73 We show that the epithelial interleukin-22 receptor IL-22RA1 protects against lethal

74 show that a network including the epithelial interleukin-22 receptor protects against infection with

75 mately 200-fold expansion of interleukin 17+/interleukin 22+ T effectors with profound Th1 suppressio

76 nra arm was decreased, and the production of interleukin 22 was increased.

77 nt and survival, and produced high levels of interleukin-22, which prevented liver damage.

78 hnsen et al. (2014) report that the cytokine interleukin-22, which usually plays a protective role, p

79 he cytokines measured (with the exception of interleukin 22) with hemoglobin A1c levels.