ライフサイエンスコーパス: interleukin-8

1 neutrophil spreading onto glass coated with interleukin 8.

2 expression or secretion of interleukin 6 and interleukin 8.

3 rkers, intercellular adhesion molecule 1 and interleukin 8.

4 ing to significant increases in secretion of interleukin 8.

5 the expression of tumor necrosis factor and interleukin 8.

6 proinflammatory cytokines interleukin-6 and interleukin-8.

7 ivin (BIRC5) and the CSC-associated cytokine interleukin-8.

8 a modulation of the proinflammatory cytokine interleukin-8.

9 n 3 (two transcription factors); and induced interleukin-8.

10 nd protein production of NF-kappaB-regulated interleukin-8.

11 ophils via vascular smooth muscle release of interleukin-8.

12 ignaling to transcription and translation of interleukin-8.

13 n-activated protein kinase and production of interleukin-8.

14 motif chemokine ligand 2, interleukin-6, and interleukin-8.

15 ligodeoxynucleotides, ammonium chloride, and interleukin-8.

16 elease of pro-inflammatory mediators such as interleukin-8.

17 is factor (44.73 vs 21.68 pg/mL; p < 0.001), interleukin-8 (124.76 vs 47.48 pg/mL; p = 0.028), and in

18 +/- 20.2 vs 111.5 +/- 29.6 pg/mL, P = .02), interleukin-8 (2196.1 +/- 737.9 vs 3753.2 +/- 1106.0 pg/

19 osis factor: 26.95 vs 18.38 pg/mL, p = 0.02; interleukin-8: 70.75 vs 27.86 pg/mL, p = 0.002; interleu

20 (grades 3 and 4), the greatest of which was interleukin-8 (92,940 pg/mL, grade 4).

21 ne exposure, we observed a 98.9% increase in interleukin-8, a 21.4% decrease in plasminogen activator

22 ine MIP-2 is the functional homolog of human interleukin-8, a known risk factor for human TRALI.

23 BAL parameters, including neutrophil count, interleukin-8, alpha defensins and MMP-9, demonstrate hi

24 eron gamma, tumor necrosis factor alpha, and interleukin 8 and 10) were similar in both groups.

25 The association between interleukin 8 and GBA mutation status was replicated (P

26 receptor 9 was induced by costimulation with interleukin-8 and ammonia.

27 direct and indirect mechanisms by targeting interleukin-8 and CXCL1 secreted by the tumour endotheli

28 dysfunction, as demonstrated through reduced interleukin-8 and elevated leptin in VEDS.

29 ar to each other for 24 of 27 analytes, with interleukin-8 and interleukin-13 higher in AML and vascu

30 th of proinflammatory response, particularly interleukin-8 and interleukin-6 concentrations, inversel

31 nsfusion-related acute lung injury, systemic interleukin-8 and interleukin-6 levels were higher compa

32 udy identifies an association between plasma interleukin-8 and microvesicle tissue factor activity me

33 o analyze the effects of Klotho signaling on interleukin-8 and monocyte chemoattractant protein-1 pro

34 In contrast, the chemokines interleukin-8 and monocyte chemotactic protein-1 stayed

35 Lavage interleukin-8 and neutrophil levels were elevated.

36 BALF was assessed for pepsin, bile salts, interleukin-8 and neutrophils.

37 receptor 9 expression correlated with plasma interleukin-8 and peak ammonia concentration (r = 0.6; p

38 was associated with serum concentrations of interleukin-8 and RANTES.

39 The gene expression of interleukin-6, interleukin-8 and tumor necrosis factor alpha was signif

40 Levels of interleukin-8 and tumor necrosis factor-alpha produced b

41 Macrophages produced cytokines, such as interleukin-8 and tumor necrosis factor-alpha, to stimul

42 patients, men had higher admission levels of interleukin-8 and tumor necrosis factor-alpha.

43 ontained relatively higher concentrations of interleukin-8 and VEGF due to GRM1-mediated activation o

44 tein kinases, resulting in superinduction of interleukin-8 and, ultimately, absorption of Stx into th

45 luding interleukin 1beta, interleukin 6, and interleukin 8, and marked inflammatory cervical infiltra

46 nducible protein 10/CXC chemokine ligand 10, interleukin 8, and monocyte chemotactic protein-1/CC che

47 onic acid and higher d-dimer, interleukin 6, interleukin 8, and soluble CD14 levels were associated w

48 C-reactive protein, temperature, interleukin 8, and soluble tumor necrosis factor recepto

49 as defensin HNP-3, the lambda-Cro repressor, interleukin 8, and the ribonuclease H domain of HIV-1 re

50 20, hBD-2, interleukin 1beta, interleukin 6, interleukin 8, and tumor necrosis factor alpha) that can

51 (vascular endothelial growth factor [VEGF], interleukin-8, and basic fibroblast growth factor [bFGF]

52 matrix-degrading enzymes (MMP-2 and MMP-9), interleukin-8, and chemokine receptors (CXCR1, CXCR4, an

53 increase in plasma levels of interleukin-6, interleukin-8, and elastase-alpha1-antitrypsin complexes

54 ition, we measured changes in interleukin-6, interleukin-8, and interleukin-10 blood levels.

55 opoietin-2) and inflammation (interleukin-6, interleukin-8, and interleukin-33 and soluble suppressio

56 the production of the neutrophil chemotaxin interleukin-8, and recruitment of neutrophils at 24 hour

57 rations of tumor necrosis factor receptor-1, interleukin-8, and regenerating islet-derived protein 3-

58 including interleukin-6, C-reactive peptide, interleukin-8, and soluble tumor necrosis factor recepto

59 r respiratory rate condition, interleukin-6, interleukin-8, and tumor necrosis factor-alpha concentra

60 s, protein, interferon-gamma, interleukin-6, interleukin-8, and tumor necrosis factor-alpha, compared

61 ssion model incorporating oxygenation index, interleukin-8, and tumor necrosis factor-R2 was superior

62 of sepsis-related biomarkers have identified interleukin-8 as a robust outcome biomarker in pediatric

63 , arterial ammonia concentration, and plasma interleukin-8 associated with neutrophil exhaustion.

64 F6 by RNA interference and neutralization of interleukin-8, basic fibroblast growth factor, and vascu

65 1 (growth-related oncogene alpha) and CXCL8 (interleukin-8), both classes of nanobodies displayed inv

66 rease in concentrations of procalcitonin and interleukin-8, but not of interleukin-6.

67 ing pathway responsible for the induction of interleukin 8 by wood smoke extract in lung epithelial c

68 at of monocyte chemoattractant protein 1 and interleukin-8 by cultured adipocytes.

69 cytokine secretion (interleukin-6 P<0.0001, interleukin-8/C-X-C motif chemokine ligand 8 P=0.04, and

70 ysteine reduced the levels of interleukin-6, interleukin-8/C-X-C motif chemokine ligand 8, and monocy

71 ophage inflammatory protein-1alpha/CCL3, and interleukin-8/CCL8) and cytokines (interleukin-6, interl

72 noassay was evaluated by detecting the human interleukin 8 chemokine.

73 Serum levels of interleukin 8, chemokine (C-C motif) ligand 3, and tumor

74 ney injury marker 1 (Kim-1) and the cytokine interleukin-8 compared to the 2D cell cultures.

75 n in airway epithelial cells correlated with interleukin-8 concentrations and absolute neutrophil num

76 er interleukin-15 concentrations, and higher interleukin-8 concentrations, than were no acute exacerb

77 l airway abnormality, and interleukin-15 and interleukin-8 concentrations.

78 spholipase A2; at CYP2F1, with higher plasma interleukin-8 concentrations; at TREH, with lower concen

79 findings in pediatric septic shock, a plasma interleukin-8 cutoff <220 pg/mL had a negative predictiv

80 In adults aged <40 yrs, a plasma interleukin-8 cutoff <220 pg/mL had a negative predictiv

81 ant 1-month increases in CRP, D-dimer, IL-6, interleukin 8, CXCL10, TNF-alpha, and interferon-gamma l

82 cell effector function in human newborns is interleukin-8 (CXCL8) production, which has the potentia

83 Interleukin-8 (CXCL8) release by neutrophils after IgLC

84 roduction around day +14 was associated with interleukin-8-driven neutrophil recovery.

85 ated genes, and secreted large quantities of interleukin-8 during exposure to HCV core and NS3 protei

86 eolar lavage fluid, levels of interleukin-6, interleukin-8, elastase-alpha1-antitrypsin complexes, th

87 Interleukin 8 expression was evaluated by using wild-typ

88 TFF2 and Muc2 was inversely correlated with interleukin-8 expression in CD (p = 0.027).

89 ection were assessed by measuring release of interleukin 8 from AGS cells (to detect cag pathogenicit

90 n the change in bronchoalveolar lavage fluid interleukin-8 from baseline to day 4 (p = .37) or day 8

91 city-induced release of the pro-inflammatory interleukin-8 from human corneal epithelial cells and re

92 The interleukin 8 gene single-nucleotide polymorphism rs4073

93 onstructs, we showed that flagellin-mediated interleukin-8 gene transcription is heavily dependent on

94 nhibition of p38 has no detectable effect on interleukin-8 gene transcription, even though flagellin-

95 eterious non-synonymous SNPs (nsSNPs) in the interleukin-8 gene using three steps.

96 s of RANKL, OPG, and inflammatory cytokines (interleukin-8, granulocyte colony-stimulating factor) we

97 Interleukin-4, interleukin-8, granulocyte macrophage colony-stimulating

98 protein BMP4 and several cytokines including interleukin-8, growth-related protein (GRO), and CCL2.

99 tor-alpha, interleukin-2, interleukin-6, and interleukin-8) have been associated with AF.

100 merged from initial screening-interleukin 6, interleukin 8, hepatocyte growth factor (HGF), tissue in

101 ion of macrophage inflammatory protein 2 (an interleukin-8 homolog) in lung epithelial cells and lung

102 ring inflammatory stimulation showed reduced interleukin 8 (IL-8) and CXCL1 secretion (P < .001).

103 miR-126 KD cells showed lower expression of interleukin 8 (IL-8) and CXCL1, both on the mRNA and pro

104 We determined fecal interleukin 8 (IL-8) and lactoferrin protein concentrati

105 itochondrial depolarization and secretion of interleukin 8 (IL-8) and leukotriene B4 (LTB4) and was p

106 ation of NF-kappaB p65 and the expression of interleukin 8 (IL-8) following ectopic expression and du

107 SP6 also elicited higher levels of interleukin 8 (IL-8) in the lower genital tract and incr

108 Specifically, CCL3 levels, but not CXCL8/interleukin 8 (IL-8) levels, were higher within discrete

109 Specific inhibition of neutrophil influx by interleukin 8 (IL-8) neutralization resulted in the coor

110 lly, Xps, and predominantly StmPr1, degraded interleukin 8 (IL-8) secreted by A549 cells during cocul

111 ants were tested for their ability to induce interleukin 8 (IL-8) secretion and translocation of Stx2

112 ng N. lactamica PorB induces lower levels of interleukin 8 (IL-8) secretion than wild-type N. meningi

113 by the gastric cell "hummingbird" phenotype, interleukin 8 (IL-8) secretion, and CagA translocation a

114 results also indicate the essential role of interleukin 8 (IL-8) signaling for the acquisition and/o

115 tumor necrosis factor alpha (TNF-alpha), and interleukin 8 (IL-8) were compared between dichotomous g

116 mediated by FOXA1 binding to the promoter of interleukin 8 (IL-8), a chemokine previously shown eleva

117 tor alpha (TNF-alpha), interleukin 6 (IL-6), interleukin 8 (IL-8), and interleukin 10 (IL-10) concent

118 n 1, growth-related oncogene, interleukin 6, interleukin 8 (IL-8), and tumor necrosis factor were det

119 were associated with increased expression of interleukin 8 (IL-8), CXCL2, IL-1beta, tumor necrosis fa

120 R was blocked by TAK1M expression, including interleukin 8 (IL-8), Gro1, IkappaBalpha, COX-2, cIAP2,

121 induces apical and basolateral secretion of interleukin 8 (IL-8), interferon-inducible protein 10 (I

122 ell adhesion molecule 1 (PECAM-1), CD99, and interleukin 8 (IL-8).

123 ty and with certain cytokines, in particular interleukin 8 (IL-8)/CXCL8.

124 Interleukin-8 (IL-8 or CXCL8) plays a critical role in o

125 for their anti-inflammatory activity against interleukin-8 (IL-8) and for the inhibition of matrix me

126 Both interleukin-8 (IL-8) and human beta-defensin 2 (HbetaD2)

127 associations between dupA cluster status and interleukin-8 (IL-8) and IL-12 production were also exam

128 We observed that epithelial production of interleukin-8 (IL-8) and IL-6 in response to bacterial s

129 es), we show the induction of TLR2-dependent interleukin-8 (IL-8) and IL-6 secretion in vitro, the in

130 atory activity response relationship against interleukin-8 (IL-8) and inhibition of matrix metallopro

131 kinase (MAPK) and proinflammatory cytokines interleukin-8 (IL-8) and KC (from human and mouse cells,

132 other cell types secreted various levels of interleukin-8 (IL-8) and monocyte chemoattractant protei

133 expression of the proinflammatory cytokines interleukin-8 (IL-8) and monocyte chemoattractant protei

134 induces greater expression of the chemokine interleukin-8 (IL-8) and the cytokine tumor necrosis fac

135 ant release of the immunomodulatory cytokine interleukin-8 (IL-8) as well as the antimicrobial peptid

136 tolerance, with continued high production of interleukin-8 (IL-8) but diminished secretion of tumor n

137 n2 (Ent+Lcn2) leads to enhanced secretion of interleukin-8 (IL-8) compared to that induced by either

138 Two mutants stimulated interleukin-8 (IL-8) expression at both the mRNA level a

139 The 15(S)-HETE induced interleukin-8 (IL-8) expression in Jak2-STAT-5B-dependen

140 ropean strains promoted significantly higher interleukin-8 (IL-8) expression than did African strains

141 and the contribution of ERK1/2 activation to interleukin-8 (IL-8) expression.

142 escape coincided with increased secretion of interleukin-8 (IL-8) from tumors into the plasma, and co

143 Interleukin-8 (IL-8) gene polymorphisms have been consid

144 MIT 97-6194-5 induced significant levels of interleukin-8 (IL-8) in HT-29 cell culture supernatants

145 Here we show that interleukin-8 (IL-8) increases mRNA expression levels fo

146 Interleukin-8 (IL-8) is a potent PMN chemoattractant.

147 cancer cells, and that adipokines including interleukin-8 (IL-8) mediate these activities.

148 n of TREM-1 on the GEC led to an increase in interleukin-8 (IL-8) mRNA levels.

149 their inhibitory activity against chemokine interleukin-8 (IL-8) production before and after cigaret

150 lation of nuclear factor-kappaB, and reduced interleukin-8 (IL-8) production in human neutrophils.

151 ing sites abolished or decreased IKK-induced interleukin-8 (IL-8) promoter activity.

152 y active, highly angiogenic homologue of the interleukin-8 (IL-8) receptors that signals in part via

153 SF from these patients did not stimulate interleukin-8 (IL-8) release from TLR transfectants.

154 culture cells and mouse ilium, resulting in interleukin-8 (IL-8) release.

155 -induced NF-kappaB and inflammatory cytokine interleukin-8 (IL-8) reporter activation depended on Rel

156 ) cells and neutrophils as well as a reduced interleukin-8 (IL-8) response.

157 ad cells, lactate dehydrogenase release, and interleukin-8 (IL-8) secretion and decreased apical cili

158 nocyte chemoattractant protein-1 (MCP-1) and interleukin-8 (IL-8) secretion from 3T3-L1 adipocytes.

159 rB is also responsible for the inhibition of interleukin-8 (IL-8) secretion from gingival epithelial

160 occal supernatants and tended to induce less interleukin-8 (IL-8) secretion in primary human fallopia

161 T serine-threonine kinase activity and serum interleukin-8 (IL-8) secretion.

162 A significantly dysregulated interleukin-8 (IL-8) signaling network was identified in

163 from a C. jejuni 11168H cdtA mutant induced interleukin-8 (IL-8) to the same extent as did wild-type

164 y detecting the approximately 8 kDa cytokine interleukin-8 (IL-8) with a limit of detection and dynam

165 genously produced chemokines (in particular, interleukin-8 (IL-8)).

166 ctant protein-1 (MCP-1), a CC chemokine, and interleukin-8 (IL-8), a CXC chemokine, along with their

167 Interleukin-8 (IL-8), a key modulator of neutrophil func

168 igh-affinity receptors for the CXC chemokine interleukin-8 (IL-8), a major mediator of immune and inf

169 CXCR1 is a receptor for the chemokine interleukin-8 (IL-8), a mediator of immune and inflammat

170 get for anti-angiogenic-based therapies, and interleukin-8 (IL-8), a potent proinflammatory mediator.

171 d sensor for the detection in serum of human interleukin-8 (IL-8), a pro-angiogenic chemokine implica

172 for tumor necrosis factor alpha (TNF-alpha), interleukin-8 (IL-8), and gamma interferon (IFN-gamma).

173 of tumor necrosis factor alpha (TNF-alpha), interleukin-8 (IL-8), and IL-6, respectively, in THP-1 c

174 nd secreted high levels of a cytokine called interleukin-8 (IL-8), attracting neutrophils and increas

175 Fibronectin, interleukin-8 (IL-8), biotin, and neutrophil gelatinase-

176 Expression of interleukin-8 (IL-8), cyclooxygenase-2 (COX-2), and heme

177 interferon gamma-induced protein 10 (IP-10), interleukin-8 (IL-8), epidermal growth factor (EGF), mat

178 f vascular endothelial growth factor (VEGF), interleukin-8 (IL-8), human beta-defensin 1 (hBD-1), and

179 del, CLA activation of MAPK and induction of interleukin-8 (IL-8), IL-6, IL-1beta, and cyclo-oxygenas

180 activating factors for phagocytes, including interleukin-8 (IL-8), monocyte chemotactic protein 1 (MC

181 WF levels of angiopoietin-2, interleukin-8 (IL-8), tumor necrosis factor-alpha (TNF-a

182 secretion, and suppresses hypersecretion of interleukin-8 (IL-8).

183 roduction of the pro-inflammatory chemokine, interleukin-8 (IL-8).

184 of fibronectin, fibrinogen, and the cytokine interleukin-8 (IL-8).

185 l secretion of the proinflammatory chemokine interleukin-8 (IL-8).

186 shedding and the production of the cytokine interleukin-8 (IL-8).

187 expression of inflammatory genes, including interleukin-8 (IL-8).

188 mutated DNA damage pathway and the chemokine interleukin-8 (IL-8).

189 is factor-alpha (TNF-alpha) or the chemokine interleukin-8 (IL-8, CXCL8) compared to controls.

190 Proinflammatory and pro-angiogenic chemokine interleukin-8 (IL-8, CXCL8) contributes to ovarian cance

191 The human chemokine interleukin-8 (IL-8; CXCL8) is a key mediator of innate

192 by strong CXCL2 (mouse functional homolog of interleukin-8 [IL-8]) and tumor necrosis factor alpha (T

193 rum leptin, adiponectin, KC (mouse analog of interleukin-8 [IL-8]), monokine induced by interferon-ga

194 s in MDS, AML, and control samples and found interleukin 8 (IL8) to be consistently overexpressed in

195 chemokine (C-C motif) ligand 20 (CCL20) and interleukin 8 (IL8).

196 Interleukin-8 (IL8) receptors IL8RA and IL8RB on neutrop

197 ered genes include humanin-like-8 (MTRNRL8), interleukin-8 (IL8), and serpin peptidase inhibitor, cla

198 pressing gingipain-associated degradation of interleukin-8 (IL8).

199 production of the neutrophil-chemoattractant interleukin 8 in the lungs.

200 lating endogenous DNA as well as ammonia and interleukin-8 in a synergistic manner inducing systemic

201 crosis factor -alpha, interleukin-1beta, and interleukin-8 in a temporal manner may indicate early ba

202 ession of monocyte chemotactic protein-1 and interleukin-8 in porcine jugular vein ECs.

203 lpha were reduced in the injured lung and of Interleukin-8 in the contralateral lung, respectively.

204 or necrosis factor alpha, interleukin-6, and interleukin-8 in the respiratory tract and central nervo

205 rulent strains to translocate CagA or induce interleukin 8, indicating that hypervirulent phenotypes

206 d an area under the curve of 0.59 for plasma interleukin-8, indicating that plasma interleukin-8 is a

207 agellin and lipopolysaccharide are the major interleukin-8 inducers.

208 Interleukin-8 induction by wood smoke extract in lung ep

209 and activator of transcription protein 3 and interleukin-8 induction.

210 in 3 activation by inhibitors attenuated the interleukin-8 induction.

211 and activator of transcription protein 3 and interleukin-8 induction.

212 AMP-activated protein kinase activation, and interleukin-8 induction.

213 ulated kinase and Jun N-terminal kinase, and interleukin-8 induction.

214 ge colony-stimulating factor, interleukin-6, interleukin-8, interferon-inducible protein-10, monocyte

215 decreased interleukin 1beta, interleukin 6, interleukin 8, interleukin 10, and tumor necrosis factor

216 cytokines (interleukin 1beta, interleukin 6, interleukin 8, interleukin 17, interferon gamma, and tum

217 A group consisting of interleukin-6, interleukin-8, interleukin-10, and fractalkine was ident

218 mponent 1, mainly loaded with interleukin-6, interleukin-8, interleukin-10, and fractalkine, was sign

219 statistically significant increase in plasma interleukin-8, interleukin-10, and interleukin-1 recepto

220 interleukin-4, interleukin-5, interleukin-6, interleukin-8, interleukin-10, interleukin-12/interleuki

221 rations of interleukin-1beta, interleukin-6, interleukin-8, interleukin-10, interleukin-18, and tumor

222 Interleukin-6, interleukin-8, interleukin-10, interleukin-18, and tumor

223 eukin-1ra, interleukin-1beta, interleukin-6, interleukin-8, interleukin-10, interleukin-18, monocyte

224 ytokine covariates, including interleukin-6, interleukin-8, interleukin-10, interleukin-1beta, interl

225 With ubiquitin treatment, tissue levels of Interleukin-8, Interleukin-10, Tumor Necrosis Factor alp

226 rleukin-1beta, interleukin-4, interleukin-6, interleukin-8, interleukin-10, tumor necrosis factor-alp

227 senescence-associated secretory phenotypes (interleukin-8, interleukin-12, GRO, and MDC).

228 ls of tissue necrosis factor, interleukin-6, interleukin-8, interleukin-17A, and interleukin-10 were

229 nt protein D, angiopoietin-2, interleukin-6, interleukin-8, interleukin-33, and soluble suppression o

230 ases, bronchoalveolar lavage fluid levels of interleukin-8, interleukin-6, and elastase-alpha1-antitr

231 plasma interleukin-8, indicating that plasma interleukin-8 is a poor predictor of mortality in this g

232 Interleukin-8 level (bronchoalveolar lavage) did not dif

233 ut human beta-defensin 2 (HBD-2), HBD-3, and interleukin 8 levels decreased after unprotected sex.

234 Clara cell secretory protein (CCSP) and interleukin 8 levels were assessed in the bronchoalveola

235 Bronchoalveolar lavage CCSP and CCSP/interleukin 8 levels were low and decreasing early after

236 Plasma interleukin-8 levels > or =220 pg/mL were significantly

237 Secondary outcomes included interleukin-8 levels and neutrophil counts from nasal wa

238 Median change in interleukin-8 levels and neutrophil counts were also not

239 ntrast to similar pediatric patients, plasma interleukin-8 levels are not an effective risk stratific

240 Plasma interleukin-8 levels of <220 pg/mL have an excellent neg

241 alpha, interleukin-1beta, interleukin-6, and interleukin-8 levels were detected as early as 4 days pr

242 alpha, interleukin-1beta, interleukin-6, and interleukin-8 levels were observed within patients with

243 ion, microvesicle tissue factor activity and interleukin-8 levels were significantly and positively c

244 y end point was bronchoalveolar lavage fluid interleukin-8 levels.

245 conditions regarding the capture of an anti-interleukin 8 mAb, using a multimodal ligand (2-benzamid

246 those measured after placebo treatment) and interleukin-8 (mean peak levels of 57.0% [p<0.05 vs plac

247 of tumor necrosis factor alpha (TNFalpha) or interleukin-8 messenger RNA.

248 a-B (NF-kappaB) activation and production of interleukin-8, monocyte chemoattactrant protein-1, and i

249 ivariable analysis included higher levels of interleukin-8, monocyte chemoattractant protein-1, resis

250 lial cell (EC) expression of chemokines (eg, interleukin-8, monocyte chemotactic protein-1) and other

251 Betalains dampened cyclooxygenase-2 and interleukin-8 mRNA expression after lipopolysaccharide i

252 CFA treatment induced CXCL1 and interleukin-8 mRNA in BEAS-2B and primary human bronchia

253 Ks), p38 activation, and a superinduction of interleukin-8 mRNA.

254 PBP induced interleukin 8 or CXCL2, tumor necrosis factor alpha, int

255 gnificantly associated with plasma levels of interleukin 8 (P = .001), monocyte chemotactic protein 1

256 concentrations of interleukin 6 (p<0.0001), interleukin 8 (p=0.002), and osteopontin (p<0.0001) were

257 high concentrations (relative to median) of interleukin 8 (p=0.006), osteopontin (p=0.0004), HGF (p=

258 differentiate PGD3 cases from controls were interleukin-8 (P < 0.001, AUC = 0.93) and growth-regulat

259 ses were reduced, and leukocyte recruitment (interleukin-8 pathway, P = 1.41E-5) and extravasation pr

260 ction efficiency) and short interfering RNA (interleukin-8, PCNA, and Bax), as a validation of effect

261 s of interest-angiopoietin 2, interleukin 6, interleukin 8, placental growth factor, soluble TIE-1, s

262 ed LPS-induced inflammation, as monitored by interleukin 8 production.

263 E. faecalis MN1 inhibited interleukin-8 production from human vaginal epithelial c

264 ed nuclear factor-kappaB activation, reduced interleukin-8 production, and decreased epithelial injur

265 s which appear responsible for inhibition of interleukin-8 production, as well as inhibition of T cel

266 mmatory actions, which include reductions in interleukin-8 production, neutrophil migration and/or fu

267 d are capable of inducing AEC activation and interleukin-8 production.

268 evident at the protein level, with increased interleukin-8 protein detectable following cotreatment w

269 ffects the posttranscriptional regulation of interleukin-8 protein expression induced by flagellin.

270 tivation of host p38 is critical for maximal interleukin-8 protein expression.

271 ng the top of the lamellipodium, whereas the interleukin 8 receptors CXCR1 and CXCR2 and the integrin

272 on was also shown to manifest an increase in interleukin 8 release from A549 lung epithelial cells bu

273 n of human neutrophils with 85 and 65% lower interleukin-8 released at 12 and 24 h, respectively.

274 apoptosis and inhibition of cell growth and interleukin-8, RHAMM, HAS2, CD44, and MMP-9 expression.

275 n of TLR10 completely abrogated TLR2-induced interleukin 8 secretion by HEK-293 cells in response to

276 region and CagA expression levels influence interleukin 8 secretion by the host gastric epithelial c

277 RIP1 kinase triggered JNK3/MAPK10-dependent interleukin-8 secretion and FADD-mediated proapoptotic c

278 gA EPIYA and CRPIA domains, V225d stimulates interleukin-8 secretion and the hummingbird phenotype in

279 6, only SAA induced a 10-50-fold increase of interleukin-8 secretion in CD36-overexpressing HEK293 ce

280 tion of a JAK2-dependent pathway, leading to interleukin-8 secretion.

281 and induce caspase-9-mediated apoptosis and interleukin-8 secretion.

282 A reduced set of three biomarkers (interleukin-8, soluble tumor necrosis factor receptor-1,

283 1/CCL-2, MIP-1alpha/CCL-3, RANTES/CCL-5, and interleukin 8) than SARS-CoV.

284 -inflammatory factors including the cytokine interleukin-8, the chemokines (C-X-C motif) ligand 1 and

285 hase: granulocyte colony-stimulating factor, interleukin-8, tissue necrosis factor-alpha, interleukin

286 The ratio of proinflammatory (interleukin-8) to anti-inflammatory (interleukin-10) pla

287 attractant protein-1, interleukin-1beta, and interleukin-8 transcripts levels compared with control.

288 tor-alpha, interleukin-1beta, interleukin-6, interleukin-8, transforming growth factor-beta, and proc

289 T-cells (NFAT) pathway and the production of interleukin 8 triggered by histamine in HUVECs.

290 (IL-1alpha), IL-1beta, interleukin 6 (IL-6), interleukin 8, tumor necrosis factor alpha, and interleu

291 report that the procancerous cytokine IL-8 (interleukin-8) upregulates CXCR7 expression along with l

292 es identified associations of interleukin 6, interleukin 8, VEGF, osteopontin, E-selectin, and HGF wi

293 dhesion molecule-1, and leptin and decreased interleukin-8 versus controls.

294 The protein level of interleukin-8 was also increased.

295 Interleukin-8 was quantified in serum and bronchoalveola

296 n addition, neutrophil chemotaxis induced by interleukin-8 was significantly enhanced when cells were

297 Interleukin-8 was significantly higher in nonsurvivors c

298 In interleukin-8, we substituted Pro for each of the three

299 ) and p50, and KC, the mouse analog of human interleukin-8, were markedly reduced by silencing BCL10

300 Whether plasma interleukin-8 would have similar utility in adults with