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   1  encoded by herpesvirus Saimiri and to human interleukin-8 receptors.                                
  
     3 oid cells transfected with formyl peptide or interleukin-8 receptors, agonist stimulation activated n
     4 ine transfected with human formyl peptide or interleukin-8 receptors and normal human neutrophils as 
     5 ncise syntheses of (+/-)-frondosin B (1), an interleukin-8 receptor antagonist, have been achieved fr
     6 ously that chemokine receptors including the interleukin-8 receptor B (CXCR2) and the Duffy blood gro
  
     8  2, angiotensin receptors types 1 and 2, and interleukin-8 receptor could not activate SRF in the pre
     9 ng the top of the lamellipodium, whereas the interleukin 8 receptors CXCR1 and CXCR2 and the integrin
  
  
    12 functional domains of the two CXC chemokine (interleukin-8) receptors, CXCR1 (formally IL-8RA) and CX
    13 ocrine tumor cells expressed another type of interleukin-8 receptor, CXCR2, suggesting an autocrine m
  
    15  Gialpha subfamily members and it attenuates interleukin-8 receptor-mediated mitogen-activated protei
    16 ctase, bcl-2, interferon regulatory factors, interleukin 8 receptor, neural cell adhesion molecule-li
  
    18 of intracellular loops between the MCP-1 and interleukin-8 receptors to create chimeric receptors rev
    19 leukin-8 (IL-8) that allow high affinity and interleukin-8 receptor type 1 (IL8R1)-specific binding o
    20 ins from IL-8, which binds to both IL8R1 and interleukin-8 receptor type 2 (IL8R2) with high affinity
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