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1 ostaining to identify the cells that produce interleukin 9.
4 riant natural killer T cells, basophils, and interleukin-9 are important in eosinophilic esophagitis
5 ed roles for interleukin-10, interleukin-18, interleukin-9, chemokines, activation of nuclear factor-
7 Immunostained cell smears showed that most interleukin 9 expression in bronchoalveolar lavage was b
8 regulatory mechanisms responsible for human interleukin 9 (IL-9) gene expression in a human T cell l
9 expressing the transcription factor PU.1 and interleukin 9 (IL-9) in patients with ulcerative colitis
16 PU.1 was required for the development of an interleukin 9 (IL-9)-secreting subset of helper T cells.
17 markers, with retention of flanking markers interleukin-9 (IL-9) and D5S414, and thus contained a sm
18 source of adipocytokines, 'alarmins' such as interleukin-9 (IL-9) and interleukin-33 (IL-33), and str
19 we identified a positive association between interleukin-9 (IL-9) and TSLP concentration in the serum
20 T cells lacking Atg3 or Atg5 have increased interleukin-9 (IL-9) expression upon differentiation int
22 e tyrosine phosphorylation of Stat3, such as interleukin-9 (IL-9) in MO7e cells or IL-6 in TF-1 cells
27 e differentiation of the recently identified interleukin-9 (IL-9) secreting T helper cell subset are
28 ow that, in mice and humans, a population of interleukin-9 (IL-9)-producing T cells activated via the
32 pression of canonical Notch target genes IL (interleukin)-9, IL-17 and interferon (IFN)gamma in the b
33 es on mouse models of asthma have identified interleukin-9 (IL9) as a determining factor in controlli
35 interleukin-4, interleukin-5, interleukin-6, interleukin-9, interleukin-10, interleukin-12, interleuk
36 een groups for interleukin-4, interleukin-6, interleukin-9, interleukin-15, interferon-gamma, granulo
48 l as pseudogenes for other genes such as the interleukin-9 receptor, within many subtelomeric regions
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