ライフサイエンスコーパス: interlobular

1 perfused juxtamedullary nephron preparation, interlobular and afferent arteriolar diameter responses

2 12-DIHETE, had no effect on diameters of the interlobular and afferent arterioles at concentrations u

3 In contrast, 5,6-EET constricted the interlobular and afferent arterioles by 16 +/- 3% (N = 6

4 nM, 11,12-EET increased the diameters of the interlobular and afferent arterioles by 18 +/- 2% (N = 1

5 Diameters of interlobular and afferent arterioles preconstricted with

6 1,12(S,R)-EET increased the diameters of the interlobular and afferent arterioles.

7 caused graded increases in diameters of the interlobular and afferent arterioles.

8 e control diameter, whereas the diameters of interlobular and arcuate arteries declined to 50%+/-12%

9 here a significant difference between small (interlobular and bile ductules) and large (intrahepatic

10 gradients of development (i.e. early to late interlobular and posterior to anterior, respectively).

11 reshly isolated from afferent arterioles and interlobular arteries averaging between 10 to 40 microns

12 usside (SNP) increased the diameter of renal interlobular arteries preconstricted with phenylephrine

13 ETHODS AND Sprague-Dawley rat interlobar and interlobular arteries were examined in terms of superoxi

14 ithelial cells and endothelial cells of some interlobular arteries, in parallel with SODD, during acu

15 Interlobular arteriolar diameter and flow, afferent and

16 Interlobular arteriolar injury was not increased in the

17 forming arborizing networks connected to the interlobular bile duct by single tributaries.

18 e three-dimensional (3D) architecture of the interlobular bile duct during cholestasis, we used 3D co

19 e each of a portal vein, hepatic artery, and interlobular bile duct numbered 11 +/- 6 per biopsy (ran

20 The diameter of the interlobular bile duct remains constant after BDL, a res

21 In contrast, interlobular bile ducts and septal bile ducts had detect

22 were primarily localized around the damaged interlobular bile ducts in PBC.

23 ly by chronic non-suppurative destruction of interlobular bile ducts leading to advanced fibrosis, ci

24 Notably, on average there were 2.3 +/- 2.2 interlobular bile ducts per portal tract, compared to 2.

25 PDC-E2 monoclonal antibody (mAb) C355.1, the interlobular bile ducts showed typical aberrant apical s

26 The average minimum external diameter of interlobular bile ducts was 13 +/- 4 microm, of hepatic

27 Interlobular bile ducts, septal bile ducts, and prolifer

28 On average, there are two interlobular bile ducts, two hepatic arteries, and one p

29 disease characterized by the destruction of interlobular biliary ductules, which progressively leads

30 or protein was detected in interalveolar and interlobular connective tissue cells adjacent to glandul

31 We show a highly reproducible sequence of interlobular duct remodeling, where cholangiocyte prolif

32 Short segments of interlobular duct were microdissected from guinea-pig pa

33 was expressed in the glandular alveolar and interlobular ductal cells in the lacrimal gland and all

34 toring the luminal pH of isolated guinea-pig interlobular ducts after microinjection of an extracellu

35 yte growth factor likely collects within the interlobular ducts and becomes a component in normal tea

36 e cells and in the ductal cells of small and interlobular ducts in CP.

37 Interlobular ducts were isolated from the rat pancreas a

38 erminal ductal lobular units, rather than in interlobular ducts.

39 sists of acini connected by intercalated and interlobular ducts.

40 lls and associated with the cells lining the interlobular ducts.

41 1a-negative palisading granulomas in widened interlobular fibrous septa were detected.

42 acteristics: the intercalated, intralobular, interlobular, intralobar, interlobar, and main excretory

43 significantly more intralobular (P < 0.001), interlobular (P < 0.05), and total pancreatic fat (P < 0

44 s of peripheral, dilated airways (n = 3) and interlobular septa (n = 3).

45 A network of interlobular septa acted as a barrier or pathway to the

46 , reticular or granular opacities (thickened interlobular septa and ground-glass opacities at CT), cy

47 increased attenuation located in or near the interlobular septa can be seen on micro-CT studies.

48 es enlarge and approach each other along the interlobular septa, causing a fine reticular pattern on

49 d ASIR-HD images for small anatomic details (interlobular septa, centrilobular region, and small bron

50 revealed lymphadenopathy, nodules, thickened interlobular septa, focal consolidation, reticular opaci

51 facilitated along the renal vessels and the interlobular septa.

52 The predominant HRCT presentation of PAP was interlobular septal thickening (ILST;100%) and ground gl

53 omy visible on thin-section CT scans include interlobular septal thickening and diseases with periphe

54 nant HRCT presentation of idiopathic PAP was interlobular septal thickening and ground glass opacitie

55 Interlobular septal thickening and ground-glass attenuat

56 ; interquartile range, 23-38 years) and more interlobular septal thickening and mediastinal lymphaden

57 nd-glass opacity or airspace consolidation), interlobular septal thickening, and gastric distention a

58 n associated with autism, and the cerebellar interlobular variation in Spry3 expression coincident wi