ライフサイエンスコーパス: internal capsule

1 asal ganglia, occipital WM, thickness of the internal capsule).

2 of the striatum and the anterior limb of the internal capsule.

3 homolog of the primate anterior limb of the internal capsule.

4 01 to <.03) except the posterior limb of the internal capsule.

5 lly medially across the inferior edge of the internal capsule.

6 ventromedial putamen, on either side of the internal capsule.

7 e zone, and, by P5, they reach the primitive internal capsule.

8 ed white matter volumes in pyramids and left internal capsule.

9 and inferior fronto-occipital fasciculi, and internal capsule.

10 wing the fornix and the anterior limb of the internal capsule.

11 e ventral border of the anterior limb of the internal capsule.

12 m, and to subcortical structures through the internal capsule.

13 te cells with an axon projecting through the internal capsule.

14 orpus callosum (CC), optic chiasm (Och), and internal capsule.

15 alamic radiations, and posterior limb of the internal capsule.

16 ially the frontal parts of the brain and the internal capsule.

17 for thalamic axons that failed to enter the internal capsule.

18 or commissure, subcortical fiber tracts, and internal capsule.

19 ductions in the frontal lobe, cerebellum and internal capsule.

20 neurons during electrical stimulation of the internal capsule.

21 the corticospinal tracts at the level of the internal capsule.

22 p=0.01), and abnormal posterior limb of the internal capsule (0.38, 0.17-0.85; p=0.02).

23 the corpus callosum and anterior limb of the internal capsule (-0.02; p =.01) at hospital discharge.

24 e, 130 x 10(-5) mm(2)/sec; posterior limb of internal capsule, 109 x 10(-5) mm(2)/sec) and three gray

25 10(-5) mm(2)/sec at birth; anterior limb of internal capsule, 130 x 10(-5) mm(2)/sec; posterior limb

26 ly lower axial diffusion in the thalamus and internal capsule across groups (P = .02).

27 sed atrophy in the anterior limb of the left internal capsule adjacent to the head of the left caudat

28 ccipital cortices, and posterior limb of the internal capsules after TBI.

29 cle, the anterior and posterior limbs of the internal capsule (ALIC and PLIC), the external capsule,

30 fibers that are part of the anterior limb of internal capsule (ALIC) in MDD and diabetic subjects usi

31 the diencephalon and growing out through the internal capsule among groups of back-labelled cells tha

32 kB-immunostained fibers were observed in the internal capsule and as two distinct fascicles within th

33 of fractional anisotropy in the genu of the internal capsule and bilateral increase of overall water

34 and myelin content in the right genu of the internal capsule and clusters of mineral depositions, co

35 w images for differentiation of GPi from the internal capsule and external globus pallidus, respectiv

36 e normally situated in the ventral thalamus, internal capsule and hypothalamus, were more dispersed i

37 tropy (FA) in the right anterior limb of the internal capsule and right uncinate fasciculus.

38 t on thalamic axons as they grow through the internal capsule and subplate but is not present in the

39 hich attracts initial axon growth toward the internal capsule and that this activity may be due to Ne

40 ns of white matter, particularly in the left internal capsule and the left frontal lobe (P = .02-.05)

41 imulated thalamic axons coursing through the internal capsule and, as a control, the basolateral (BL)

42 ding white matter density differences in the internal capsules and left centrum semiovale.

43 tter (anterior and posterior subcortical and internal capsule) and four of gray matter (cortex, thala

44 y low within areas of white matter (fimbria, internal capsule) and select neuronal fields (hippocampa

45 l external capsule and posterior limb of the internal capsule, and corona radiata.

46 ly in white matter tracts of the cerebellum, internal capsule, and corpus callosum.

47 tria terminalis, medial amygdala, claustrum, internal capsule, and globus pallidus.

48 trum semiovale, in the posterior limb of the internal capsule, and in the cerebral peduncle; the thal

49 lly located in the cerebral white matter and internal capsule, and infrequently in the brain stem.

50 le, bilateral retrolenticular region part of internal capsule, and left posterior thalamic radiation.

51 ped myelination of the posterior limb of the internal capsule, and more immature gyral folding than t

52 c radiation, the retrolenticular part of the internal capsule, and the sagittal stratum (p<.05, corre

53 l globus pallidus, the ventral pallidum, the internal capsule, and the substantia innominata.

54 , caudate nuclei, lentiform nuclei, thalami, internal capsules, and cerebellar hemispheres.

55 Fibers in the internal capsule are organized according to destination.

56 motor system through the corona radiata and internal capsule are well described in non-human primate

57 ein (MBP) at the fimbria hippocampus and the internal capsule areas in the 7-day-old BACO rat brain w

58 matter in the corpus callosum, subcortex and internal capsule areas while largely spared cortical neu

59 rmation in the corpus callosum, external and internal capsule areas.

60 knock-out mice pioneer the projection to the internal capsule as they do in wild-type mice.

61 xons show increased fasciculation within the internal capsule, as well as abnormal turning and branch

62 ractional anisotropy in the anterior limb of internal capsule at discharge and in genu of corpus call

63 -0.03 to 0.02) in the posterior limb of the internal capsule between the two groups.

64 m, bilateral anterior and posterior limbs of internal capsule, bilateral retrolenticular region part

65 e we demonstrate that a set of neurons in an internal-capsule bordering regions of the primate dorsal

66 fected 293T cells, as it had been toward the internal capsule, but not toward control-transfected 293

67 tical axon outgrowth was directed toward the internal capsule, but outgrowth was nondirected and supp

68 tions, superior fronto-occipital fasciculus, internal capsule, callosal isthmus, and the corona radia

69 igma)) were measured in the corpus callosum, internal capsule, caudate nucleus, lentiform nucleus, an

70 ontrol subjects in the posterior limb of the internal capsule, corona radiata, posterior frontal whit

71 st in the centrum semiovale, corona radiata, internal capsule, corpus callosum, and subcortical white

72 Major central WM tracts, including internal capsule, corpus callosum, anterior commissure,

73 addition, reduced FA in anterior limb of the internal capsule correlated significantly with an increa

74 ctional anisotropy with age was found in the internal capsule, corticospinal tract, left arcuate fasc

75 Parietal operculum, corona radiata, and internal capsule differences between cases and controls

76 ed FA values were located bilaterally in the internal capsule extending into the globus pallidus and

77 nferior longitudinal fasciculi, limbs of the internal capsule, external capsule and cerebellum (p < 0

78 ngs in the putamen into the rostral putamen, internal capsule, external capsule, caudate nucleus, and

79 beta = -0.01 (P = .04)] and retrolenticular internal capsule [FA, beta = -0.01 (P = .002); RD, beta

80 ralateral side (FAcontra) and to that in the internal capsule (FAint) was calculated.

81 Development of anterior cortical and internal capsule fibers may be particularly vulnerable t

82 for Isl1 in diencephalic cells bordering the internal capsule for the normal development of the stria

83 VPN neurons by electrical stimulation of the internal capsule from P3.

84 out the striatal neuropil, as well as in the internal capsule, globus pallidus, and substantia nigra.

85 of myelination in the posterior limb of the internal capsule, gyral maturation, signal intensity abn

86 fiber tracts within the anterior limb of the internal capsule have a significantly higher (P < 0.01)

87 n began at postnatal day 5 (P5) (E36) in the internal capsule (IC) and at P11 in the medial corpus ca

88 tor regions through the corona radiata (CR), internal capsule (IC) and crus cerebri of the cerebral p

89 udinal fasciculus (LF), corona radiata (CR), internal capsule (IC) and external capsule (EC).

90 al projection in the corona radiata (CR) and internal capsule (IC) can assist in evaluating a patient

91 upon DA application or by stimulation of the internal capsule (IC), which also supports self-stimulat

92 the pallial-subpallial boundary to form the internal capsule (IC).

93 ly and orthodromically by stimulation of the internal capsule (IC; conduction velocity, 2.4 +/- 0.2 m

94 ite matter volume around the optic tract and internal capsule in anophthalmic subjects showed a large

95 including the corpus callosum, fimbria, and internal capsule in the brain, and pyramidal tracts and

96 major CNS axon tracts including those of the internal capsule, in part via cell-nonautonomous mechani

97 In rats, the internal capsule is pioneered on E14, but the corpus cal

98 ns pioneer the pathway from neocortex to the internal capsule, leading to the proposal that they are

99 02) and a lower FA of the cranial CST at the internal capsule level (p = 0.001).

100 onal anisotropy in the posterior limb of the internal capsule, measured with magnetic resonance spect

101 tently lower diffusivity in the thalamus and internal capsule of all tobacco-exposed infants suggests

102 ion restriction in the posterior limb of the internal capsule, often also optic radiation, brainstem

103 In contrast, bilateral involvement of the internal capsule on brain MRI was associated with poorer

104 ury was prominent in the corpus callosum and internal capsule on day 3 and then partially recovered o

105 gets of cortical efferent axons, the nascent internal capsule, or the medial wall of the dorsal telen

106 bocyanine dye tracing from the callosum, the internal capsule, or the primary somatosensory cortex.

107 ventral portion of the anterior limb of the internal capsule over a 20-year period using the Leksell

108 ccumbens (P = .03), and anterior limb of the internal capsule (P = .02).

109 and the contralateral posterior limb of the internal capsule (PLIC) after age correction and were co

110 l tracts (CSTs) in the posterior limb of the internal capsule (PLIC).

111 corpus callosum (SPCC) and posterior limb of internal capsule (PLIC).

112 orpus callosum (SCC), posterior limbs of the internal capsules (PLIC), superior longitudinal fascicul

113 ttern beginning in the cerebellum, pons, and internal capsule; proceeding caudocranially from the spl

114 ared with hypothermia alone in the thalamus, internal capsule, putamen and caudate, and there was red

115 mid, and anterior cingulate, as well as the internal capsule, putamen, and globus pallidus and basal

116 nd vessel walls of the posterior limb of the internal capsule, putamen, globus pallidus and cerebellu

117 hen cocultured with MDT, suggesting that the internal capsule releases a chemoattractant for cortical

118 icothalamic and thalamocortical axons in the internal capsule requires Fz3 expression in the ventral

119 ateral amydalofugal pathway and ventromedial internal capsule, respectively).

120 the cortical afferents arriving through the internal capsule, respectively.

121 Early in the study, heating of the internal capsule resulted in 2 cases (8%) of mild hemipa

122 ion of thalamic/brainstem fibers through the internal capsule, results in a complex mingling of thala

123 ial corticospinal tracts at the level of the internal capsule (right Z score 5.21, p=0.0081; left Z s

124 us callosum, the right posterior limb of the internal capsule, right external capsule, and the right

125 nal capsule, the retrolenticular part of the internal capsule (RLIC), the body and splenium of the co

126 to Broca's area (RR, 2.5; 95% CI, 1.3-5.0), internal capsule (RR, 2.2; 95% CI, 1.1-4.4), Wernicke's

127 Labeled fibers descend in the internal capsule (SMA in anterior limb and genu; M1 in p

128 oscillations became the typical response to internal capsule stimulation at P12.

129 sterior limb and the retrolenticular part of internal capsule, superior and posterior corona radiata,

130 WM loss was concentrated in the brainstem, internal capsule, temporal and frontal regions and the m

131 rticospinal tract), corpus callosum, fornix, internal capsule (thalamocortical and corticothalamic tr

132 f the cerebellum, cingulum, corpus callosum, internal capsule, thalamus, basal forebrain, occipital,

133 tures (corpus callosum, anterior commissure, internal capsule, thalamus, caudoputamen, and cortex).

134 sociated with increased FA of regions of the internal capsule that contain auditory thalamocortical a

135 -compared with controls-in the pyramids, the internal capsule, the cerebral peduncle and the hand are

136 ntricular regions, the posterior limb of the internal capsule, the external capsule, and the pyramida

137 defined and topographic anterior limb of the internal capsule, the specific locations and organizatio

138 th abnormalities in the anterior limb of the internal capsule, the white matter (WM) bundle carrying

139 ither through the ventrolaterally positioned internal capsule to subcortical targets or through the d

140 showed cerebral cortex, corpus callosum, or internal capsule volume differences from control.

141 ng the ventral striatum/anterior limb of the internal capsule (VS/ALIC) in 10 patients with poststrok

142 Myelination of the posterior limb of the internal capsule was less developed, and gyral maturatio

143 FA of the posterior limb of the left internal capsule was positively correlated with reaction

144 nisotropy (FA) in the posterior limbs of the internal capsules was determined with diffusion-weighted

145 chemoattractant Netrin-1 is expressed in the internal capsule, we cocultured cortical explants with E

146 us pallidus and the adjacent optic tract and internal capsule were identified with microelectrode rec

147 Dysmorphic basal ganglia with an abnormal internal capsule were the most consistent feature.

148 the corpus callosum, the frontal WM, and the internal capsule; were compared with those of five age-m

149 substantia innominata, globus pallidus, and internal capsule, where PHA-L-labeled terminals abutted

150 l axon growth is directed toward the nascent internal capsule, which could account for the timing dif

151 orticospinal tract at the level of the right internal capsule (Z score 4.01, p<0.0001).

152 l capsule (Z score 4.30, p=0.0021), the left internal capsule (Z score 4.27, p=0.0278), and left cere

153 rticospinal tracts at the level of the right internal capsule (Z score 4.30, p=0.0021), the left inte

154 d growth of dorsal thalamic axons toward the internal capsule zone of ventral telencephalic explants