ライフサイエンスコーパス: internal organ

1 ding fibrotic disease affecting the skin and internal organs.

2 mutation D816V in the bone marrow and other internal organs.

3 autonomic and endocrine systems that control internal organs.

4 aging of tumors and gene expression in mouse internal organs.

5 nd excessive connective tissue deposition in internal organs.

6 vealed no apparent abnormality(ies) in vital internal organs.

7 establishes the L/R asymmetric placement of internal organs.

8 festing itself by fibrosis of skin and other internal organs.

9 ve dermal fibrosis with later progression to internal organs.

10 rooting selfhood in the neural monitoring of internal organs.

11 es the quantification of tumor growth within internal organs.

12 liver, and positive bacterial cultures from internal organs.

13 aberrima, has the capacity to regenerate its internal organs.

14 nd short persistence of the yaws pathogen in internal organs.

15 genetic disorders that affect both skin and internal organs.

16 ed by fibrosis of the skin, vasculature, and internal organs.

17 tumors without any apparent toxic effects on internal organs.

18 rmis, fascia, and the fibrous layers of many internal organs.

19 le, heart and blood with a decrease to other internal organs.

20 isease characterized by fibrosis of skin and internal organs.

21 hood is grounded in the neural monitoring of internal organs.

22 mussel gut, with little penetration in their internal organs.

23 It affects the skin and various internal organs.

24 Nominal expression was detected in internal organs.

25 er characterized by fibrosis of the skin and internal organs.

26 of subjacent muscles and usually sparing the internal organs.

27 respiratory tract and spreads by viremia to internal organs.

28 se dominated by the mechanics of pressurized internal organs.

29 er and line the surface of body cavities and internal organs.

30 forces contour the body and provide shape to internal organs.

31 of squamous cell carcinoma (SCC) in skin and internal organs.

32 n autoimmune disorder that involves skin and internal organs.

33 microscopes to minimally invasive imaging of internal organs.

34 SNA structures are virtually undetectable in internal organs.

35 metries in the structure and position of the internal organs.

36 f vibrissae, mineral deposits in a number of internal organs.

37 the site of inoculation and dissemination to internal organs.

38 histochemical analysis of excised tumors and internal organs.

39 es a consistent left-right (LR) asymmetry of internal organs.

40 are comparable to the dimensions of luminal internal organs.

41 and are required for the proper function of internal organs.

42 y lesions and satellite lesions, and infects internal organs.

43 se characterized by fibrosis of the skin and internal organs.

44 ity to clearly resolve and identify targeted internal organs.

45 plication, but pathology was observed in the internal organs.

46 eractions during epithelial morphogenesis of internal organs.

47 itis (OR = 59.1; CI, 14.1-247.8; P < .0001), internal organ abscess (OR = 2.9; CI, 1.2-6.4; P = .02),

48 ial meningitis, lymphoma/leukemia, HIV/AIDS, internal organ abscess, diabetes with ophthalmic manifes

49 dhere to epithelial surfaces, disseminate to internal organs, affect the body weight of adult mice an

50 er characterized by fibrosis of the skin and internal organs and autoimmunity.

51 position of ossifications in relation to the internal organs and blood vessels, allowing to schedule

52 he body cavity, where it directly bathes the internal organs and epidermis.

53 Combined information about its eyes, internal organs and gut contents obtained by X-ray micro

54 brosis is caused by scar tissue formation in internal organs and is associated with 45% of deaths in

55 is the study of the microbes colonizing the internal organs and orifices after death.

56 essed on the basis of the bacterial loads in internal organs and overall lethality.

57 lethality and reduces viral dissemination in internal organs and poxvirus lesions.

58 of the extracellular matrix in a variety of internal organs and skin.

59 Skin is the interface between the internal organs and the environment, and as such plays a

60 omach, ileum, colon) or in a broad series of internal organs and tissues (e.g. liver, brain, heart, p

61 ce were dissected to better visualize deeper internal organs and tissues.

62 These ganglia innervate internal organs and transmit information on heart rate,

63 s patches and mesenteric lymph nodes, to the internal organs and trigger the formation of abscesses;

64 is characterised by fibrosis of the skin and internal organs and vasculopathy.

65 immune activation, inflammation in skin and internal organs, and autoantibody generation.

66 ) mice develop severe pathology in colon and internal organs, and deteriorate rapidly during acute in

67 Internal organs are asymmetrically positioned inside the

68 ny species, changes in key energy-associated internal organs are common yet poorly characterised func

69 The asymmetries of internal organs are consistently oriented along the left

70 Besides skeletal dwarfism, internal organs are correspondingly smaller, indicating

71 The liver is engorged with lipid, and the internal organs are enlarged.

72 Neural inputs from internal organs are essential for normal autonomic funct

73 ine the serous cavities and outer surface of internal organs are involved in inflammatory responses i

74 m laterality relates to the L/R asymmetry of internal organs are poorly understood.

75 Most internal organs are situated in a coelomic cavity and ar

76 h through skin infection and then spreads to internal organs as reported previously for the wild-type

77 During development, the placement of internal organs asymmetrically along the left-right axis

78 ion in the colony counts of H. capsulatum in internal organs at 14 days after infection.

79 t a marker of parasympathetic denervation of internal organs, but further validation studies are need

80 oxin from the gastrointestinal tracts to the internal organs by in vivo imaging of whole animals over

81 ft/right (L/R) anatomical asymmetries of the internal organs can be traced to molecular events initia

82 dent changes in the thanatomicrobiome within internal organs can estimate the time of death as a huma

83 leishmaniasis (VL) is a fatal disease of the internal organs caused by the eukaryotic parasite Leishm

84 ion, bacteria proliferate extensively in the internal organs despite the massive infiltration of neut

85 Our results suggest a role for CPE-R in internal organ development and function during pre- and

86 ype 2 to replication in cultured cells or in internal organs during neonatal disseminated disease res

87 isrupted by the extensive damage observed in internal organs during the course of certain diseases.

88 xpression of vascularity markers in skin and internal organs, early immune activation, inflammation i

89 sease is the gradual accumulation of iron in internal organs, especially the liver, heart, and pancre

90 atfish by the immersion route and persist in internal organs for at least 48 h.

91 metabolous insects, the adult appendages and internal organs form anew from larval progenitor cells d

92 cordance and (2) that neuronal and visceral (internal organs) forms of bilateral asymmetry are coded

93 nsity focused ultrasound has been applied to internal organs from outside the body to ablate tissue.

94 Internal organ function improved (lung) or remained stab

95 cing physiological performance and buffering internal organ function while foraging in the cold, nutr

96 nd demonstrate the possibility of regulating internal organ function, leading to new bioelectronic th

97 ty and quality of life, and stabilization of internal organ function.

98 Overall, internal organ functions were stable to slightly worse a

99 This model of vascular development in an internal organ has a direct impact on the current dogma

100 uding thrombocytopenia, leukopenia, skin and internal organ hemorrhages, high viral replication, sple

101 Betaine has been shown to protect internal organs, improve vascular risk factors, and enha

102 mode endoscopy and show its ability to image internal organs in vivo, thus illustrating its potential

103 M formation in postnatal retinal vessels and internal organs including the gastrointestinal (GI) trac

104 looped hearts and randomized localization of internal organs including the pancreas, features typical

105 iciencies that cause defects in formation of internal organs, including circularization of the intest

106 that encapsulate the epithelial primordia of internal organs, including the kidney and lung, as well

107 IR-1 genetic pathways in murine versus human internal organs, including the lung and brain.

108 late mesoderm and randomize the placement of internal organs, indicating that the activity of Na,K-AT

109 ary motility, KV fluid flow and placement of internal organs induced by their knockdown could be supp

110 Non-lethal ischemia of internal organs induces local (ischemic preconditioning)

111 ole of vasculitic, as opposed to synovial or internal organ inflammation, may be release of secondary

112 erious brain injuries, spinal cord injuries, internal organ injuries, extremity fractures, and facial

113 The small bowel is the most common internal organ involved, resulting in considerable morbi

114 SSc-associated antibodies for diagnosis and internal organ involvement are becoming increasingly acc

115 the relationship between the skin score and internal organ involvement in dcSSc is more complex than

116 Mortality in severe diffuse disease with internal organ involvement is elevated.

117 dnan skin scores (mRSS) of more than 14, and internal organ involvement or restricted skin involvemen

118 ostic features (such as high skin scores and internal organ involvement).

119 r major form of recessive ichthyosis without internal organ involvement, lamellar ichthyosis, which i

120 der characterized by loose skin and variable internal organ involvement, resulting from paucity of el

121 of memory T cell subsets, which may reflect internal organ involvement.

122 had no significant differences in individual internal organ involvements, laboratory features, serum

123 She had neither erythema nor internal organ involvements.

124 Formation of numerous internal organs involves reciprocal epithelial-mesenchym

125 g physiological tolerance against self-Ag of internal organs is not yet defined.

126 in 25% to 68% of patients, dissemination to internal organs is rare.

127 ogy is that the final size of appendages and internal organs is regulated autonomously, within the st

128 examining microbial communities in different internal organs is to address the paucity of empirical d

129 s administration of [1-(11)C]-glucose for 24 internal organs, lens, blood and total body were calcula

130 ce of C. neoformans from the lungs and other internal organs, less is known about the protective mech

131 he skin, extremities, oral cavity and in the internal organs (lung, liver, intestine, spleen and brai

132 ellectual disability, increased frequency of internal organ malformations (including those of the hea

133 sults suggest that acupoints associated with internal organs may be identical to neurogenic inflammat

134 few vegetative forms could be recovered from internal organs of animals infected with the bslA mutant

135 A novel X-ray technique shows that the internal organs of crawling caterpillars slide past the

136 The assumption that amongst internal organs of early arthropods only the digestive s

137 Fibroblasts and myofibroblasts in skin and internal organs of patients with systemic sclerosis and

138 accessibility to various tissue sites in the internal organs of small animals.

139 nt role for TLR11 in preventing infection of internal organs of the urogenital system.

140 Laterality of the internal organs of vertebrates is determined by asymmetr

141 ing embryonic development and innervates the internal organs of vertebrates to modulate their stress

142 ient positioning and involuntary movement of internal organs, often necessitating labor-intensive non

143 osing processes affecting the skin, selected internal organs, or both in a characteristic pattern are

144 infection is characterized by the growth in internal organ parenchymae of fluid-filled structures (h

145 We propose that peripheral tolerance for internal organs relies on the control of autoreactive ef

146 bicans to translocate and disseminate to the internal organs, resulting in an uncontrolled immune res

147 talis, the complete mirror image reversal of internal organ situs (positioning).

148 R) asymmetry in the anatomy and placement of internal organs such as the heart.

149 blood may result from CPE pore formation in internal organs such as the liver.

150 to affect the colon but are not effective in internal organs such as the lungs.

151 processing associated with signals from the internal organs (such as the heart and the lung) plays a

152 mediator between nutrition and the growth of internal organs, such as imaginal disks, and is required

153 hood is grounded in the neural monitoring of internal organs, such as the heart.

154 These pathways enhance protection in internal organs, such as the nervous system, and in the

155 e elucidated, the presence of fetal cells in internal organs suggests that they could play a role in

156 disease features, frequency and severity of internal organ system involvement, and survival in Afric

157 tocytosis in the skin: MIS) and/or involving internal organs (systemic mastocytosis: SM).

158 lesions, and higher viral loads in skin and internal organs than mice inoculated in saline-exposed s

159 om, anterior differentiation, appendages, or internal organs that would suggest a bilateral body plan

160 nt (the epidermis and cornea) and in certain internal organs (the epithelium of the tongue, esophagus

161 bsorbed into the circulation to target other internal organs), the ability of isogenic agrB or luxS m

162 ing reliably estimates muscle quantity in an internal organ, the heart, and can longitudinally follow

163 ovirus (Ad) vector-mediated gene transfer to internal organs, this study evaluated the consequences o

164 challenge and reduces viral dissemination to internal organs, thus providing a shield for the develop

165 ion, followed by the binding of the toxin to internal organs to induce potassium leakage, which can c

166 se concomitant fractures usually also damage internal organs; together, these injuries are hypothesiz

167 in the extremities, progressive fibrosis of internal organs, vasoconstriction and altered expression

168 The organization of the epidermis and internal organs was also analyzed using a monoclonal ant

169 -deficient cells to the formation of various internal organs was analyzed.

170 enhanced, and recruitment of macrophages to internal organs was increased in septic PHD3-deficient m

171 However, while spread to other internal organs was rapid and efficient in CAST/EiJ mice

172 ite its name (visceral means associated with internal organs) was only thought to give rise to extrae

173 rotection against chemical carcinogenesis in internal organs, we treated XPC mutant (XPC-/-) mice wit

174 be seen through the skin of shaved mice, and internal organs were easily discernible.

175 Droppings, cecal tonsils and 5 internal organs were sampled and cultured at 6, 13 and 2

176 e co-registered anatomical maps of a mouse's internal organs, while also acquiring in vivo molecular

177 the mechanism that coordinates the growth of internal organs with overall somatic growth.

178 brates display a characteristic asymmetry of internal organs with the cardiac apex, stomach and splee

179 o a lesser extent, the intestinal tracts and internal organs; with limited histopathological changes

180 deling and fibrosis of the skin and multiple internal organs, yet the fundamental pathological defect