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1 cations redistribute onto the newly created interstices.
2 Na(+)-ND(3) pairs residing in the octahedral interstices.
3 marker, acid lipase, to the stratum corneum interstices.
4 hich it delivers to the stratum corneum (SC) interstices.
5 and suppresses nanoparticle diffusion in gel interstices.
6 mellar bodies (LB), and secreted into the SC interstices, along with other LB-derived lipid hydrolase
7 eplete with mature lamellar membranes in the interstices and a functionally competent barrier appear.
8 ed lamellar body-derived membranes in the SC interstices, and depletion of histochemically detectable
9 ter/solute movement was restricted to the SC interstices, and further localized to non-lamellar domai
10 erved glycine residues rarely face the lipid interstices, and many of them participate in close helix
12 e the unfavorable free energy of hydrophobic interstices associated with the intermediates of the fus
13 d to protein aggregates jamming in the fluid interstices between bubbles and/or to the higher viscosi
14 ates that each of the K atoms in tetrahedral interstices between C60 spheres accepts three electrons
15 d that tip-sample adhesion was larger in the interstices between NPL complexes in the (110) crystal p
17 cs simulations suggest that lipids enter the interstices between the outer ends of the TM domains.
18 ranes containing hexadecane, known to reduce interstice energy in nonlamellar structures, provided ad
19 interbilayer space) and step 3 (reduction of interstice energy leading to expansion to a critical sta
21 rene-containing fluorescent probes to report interstices formed during the L(alpha)-H(II) transition
22 electride, with electron pairs localized in interstices, forming eight-centre two-electron bonds wit
23 also show here that increased Ca++ in the SC interstices in RXLI could contribute to corneocyte reten
24 give the RC QB site transient access to the interstices in the lattice, which might be of functional
27 ases in dermatology--from the gene to the SC interstices, its etiology and pathogenesis are becoming
29 thus providing the acidification of deep SC interstices necessary for lipid processing and barrier h
31 ogy of the aggregates that accumulate in the interstices of the brains of patients with autosomal dom
32 llen from these lines lacked tryphine in the interstices of the exine layer, displayed shrunken and c
34 ist in the relatively uniform low dielectric interstices of the lipid bilayer, largely devoid of wate
36 as additional collagen deposition within the interstices of the stent-graft that did not narrow the l
37 dues with short and nonpolar residues in the interstices of the TtCM x S tighten the intersubunit hyd
38 then form a non-lamellar phase within the SC interstices, shown by combined ruthenium tetroxide post-
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