ライフサイエンスコーパス: interstitial fibrosis

1 ctive oxygen species, and manifested cardiac interstitial fibrosis.

2 rstitial changes, and histologic findings of interstitial fibrosis.

3 ular hypertrophy, disarray of myofibers, and interstitial fibrosis.

4 p53/miR-199a-3p/SOCS7/STAT3 pathway in renal interstitial fibrosis.

5 re protected against cardiac dysfunction and interstitial fibrosis.

6 allografts showed less glomerular injury and interstitial fibrosis.

7 ant decreases in myocardial apoptosis and LV interstitial fibrosis.

8 g novel disease-modifying therapy, targeting interstitial fibrosis.

9 dysfunction by promoting tubular atrophy and interstitial fibrosis.

10 ilateral ureteral obstruction model of renal interstitial fibrosis.

11 tially improved cardiac function and reduced interstitial fibrosis.

12 in recovered more rapidly and exhibited less interstitial fibrosis.

13 essened bladder mucosa damage, and decreased interstitial fibrosis.

14 n content of the heart increases, leading to interstitial fibrosis.

15 developed concentration- and time-dependent interstitial fibrosis.

16 tracellular matrix, and development of renal interstitial fibrosis.

17 myocyte hypertrophy and disarray, as well as interstitial fibrosis.

18 n quantify the burden of global and regional interstitial fibrosis.

19 in association with myocyte hypertrophy and interstitial fibrosis.

20 There was no increase in interstitial fibrosis.

21 t RGC-32 is essential for the onset of renal interstitial fibrosis.

22 ch TIN, with most showing diffuse, expansile interstitial fibrosis.

23 ition important for the development of renal interstitial fibrosis.

24 cted kidney, with marked tubular atrophy and interstitial fibrosis.

25 thers to be responsible for nephron loss and interstitial fibrosis.

26 s glomerular sclerosis, tubular atrophy, and interstitial fibrosis.

27 t and correlated directly with the degree of interstitial fibrosis.

28 d seven showed > or =40% tubular atrophy and interstitial fibrosis.

29 ar hypertrophy, intrarenal inflammation, and interstitial fibrosis.

30 to acute rejection or to tubular atrophy and interstitial fibrosis.

31 ypertrophy and iron deposits with or without interstitial fibrosis.

32 atrix components in a large portion of renal interstitial fibrosis.

33 positive cells per glomerular tuft area, and interstitial fibrosis.

34 with a trend toward higher estimated percent interstitial fibrosis.

35 brocytes made no significant contribution to interstitial fibrosis.

36 tation, show a two- to four-fold increase in interstitial fibrosis.

37 s in the initiation and progression of renal interstitial fibrosis.

38 n NADPH oxidase activity and the increase in interstitial fibrosis.

39 pressure or scores of glomerulosclerosis or interstitial fibrosis.

40 ructure, and arrested glomerulosclerosis and interstitial fibrosis.

41 tubule cell (RPTC) injury, inflammation, and interstitial fibrosis.

42 d promoted macrophage persistence and severe interstitial fibrosis.

43 lar hypertrophy, left atrial dilatation, and interstitial fibrosis.

44 cued angiotensin II-induced perivascular and interstitial fibrosis.

45 tion slowing in an in vitro model of cardiac interstitial fibrosis.

46 ressive chronic kidney disease and pulmonary interstitial fibrosis.

47 ular cells is involved in the development of interstitial fibrosis.

48 chronic Banff components tubular atrophy and interstitial fibrosis.

49 ion of the kidney parenchyma and attenuating interstitial fibrosis.

50 of different rhubarb extract against tubulo-interstitial fibrosis.

51 glomerulosclerosis, and more tubular atrophy/interstitial fibrosis.

52 bal glomerulosclerosis, tubular atrophy, and interstitial fibrosis.

53 characterized by tubular cell apoptosis and interstitial fibrosis.

54 tractile dysfunction, and heart failure with interstitial fibrosis 1 week after pressure overload.

55 ve stress (1 day post-I/R), hypertrophy, and interstitial fibrosis (1 week).

56 ia length 4.4+/-0.4 versus 5.2+/-0.6 mg/mm), interstitial fibrosis (11.2+/-3.1% versus 18.5+/-3.5%),

57 nificantly increased NADPH oxidase activity, interstitial fibrosis (11.5+/-1.0% vs. 7.2+/-0.7%; P<0.0

58 phritis [23 of 25 (92%)], and glomerular and interstitial fibrosis [12 of 25 (48%)] were predominant

59 th minimal tubular atrophy (20%) and/or <20% interstitial fibrosis (24%).

60 Other features included interstitial fibrosis (5), poorly-formed granulomas (4),

61 es (0.17 versus 0.28%, P<0.05) and amount of interstitial fibrosis (5.0 versus 7.1%, P<0.05) in the r

62 ar biopsies, lower incidence and severity of interstitial fibrosis (67% vs. 45%, P=0.003) and tubular

63 Interstitial fibrosis, a common pathological feature of

64 llele are hypersensitive to developing renal interstitial fibrosis, a disease stimulated by TGF-beta

65 is associated with a dramatic inhibition of interstitial fibrosis, a fivefold reduction in alpha-smo

66 Interstitial fibrosis after ischemia/reperfusion was red

67 ce were protected against the development of interstitial fibrosis after obstructive injury.

68 recovery and late graft function and reduced interstitial fibrosis after transplant.

69 autophagy in the CryABR120G hearts decreased interstitial fibrosis, ameliorated ventricular dysfuncti

70 f cardiac disease, leading to an increase in interstitial fibrosis and a decrease in ventricular func

71 ignificantly attenuated cardiac hypertrophy, interstitial fibrosis and apoptosis, and better cardiac

72 nterstitial inflammation and subsequently to interstitial fibrosis and atrophy of the renal tubules i

73 directly associated with the development of interstitial fibrosis and bronchiolitis obliterans.

74 ) has demonstrated utility to detect diffuse interstitial fibrosis and changes in regional myocardial

75 cipients, but sensitization caused increased interstitial fibrosis and chronic vasculopathy.

76 severe AKI, characterized by development of interstitial fibrosis and CKD.

77 uroursodeoxycholic acid or BGP-15, decreased interstitial fibrosis and collagen deposition in ovaries

78 s at week 6 could predict the development of interstitial fibrosis and de novo donor specific anti-HL

79 in/GRP can induce features of BPD, including interstitial fibrosis and diminished alveolarization.

80 infiltration of inflammatory cells, enhanced interstitial fibrosis and elevated serum creatine kinase

81 activation of sodium transporters, and cause interstitial fibrosis and glomerular injury.

82 rine model of aristolochic acid (AA)-induced interstitial fibrosis and human AA nephropathy, we obser

83 pneumonia fibroblasts caused a more diffuse interstitial fibrosis and increased hydroxyproline level

84 itial pneumonia fibroblast injection, patchy interstitial fibrosis and increased hydroxyproline were

85 the thickness of interventricular septum and interstitial fibrosis and increases anterior wall thickn

86 they develop a cardiomyopathy that includes interstitial fibrosis and infiltration of the myocardium

87 study assessed the development of allograft interstitial fibrosis and inflammation (GIF+"i"), a hist

88 eas patients with graft loss frequently have interstitial fibrosis and inflammation (IF+i=ci>0+i>0).

89 he periostin gene showed less injury-induced interstitial fibrosis and inflammation and were protecte

90 Only a small increase in interstitial fibrosis and inflammation was found in tamo

91 nd histopathological abnormalities including interstitial fibrosis and inflammation, and either fully

92 Renal interstitial fibrosis and interstitial active inflammati

93 All biopsies showed less than 30% interstitial fibrosis and less than 30% to 40% glomerulo

94 ysyl oxidase-like 2 (Loxl2)-is essential for interstitial fibrosis and mechanical dysfunction of path

95 Kidney histology demonstrated interstitial fibrosis and nephrocalcinosis in addition t

96 ypertrophic remodeling, pulmonary edema, and interstitial fibrosis and prevents cardiac dysfunction a

97 ells before the onset of proteinuria delayed interstitial fibrosis and progression of glomerular scle

98 Chronic kidney disease is characterized by interstitial fibrosis and proliferation of scar-secretin

99 Biomarkers and new methods to assess interstitial fibrosis and regional myocardial function h

100 Kidney transplants with both interstitial fibrosis and subclinical inflammation but n

101 obstruction model, ICG-001 ameliorated renal interstitial fibrosis and suppressed renal expression of

102 egardless of age, pulmonary hypertension, or interstitial fibrosis and that this relation may be chan

103 ype mice, NOX4-deficient mice exhibited more interstitial fibrosis and tubular apoptosis after obstru

104 Subclinical rejection and interstitial fibrosis and tubular atrophy (IF/TA) in pro

105 ome of doubling of interstitium or ESRD from interstitial fibrosis and tubular atrophy (IF/TA) in the

106 Chronic allograft damage, defined by interstitial fibrosis and tubular atrophy (IF/TA), is a

107 Interstitial fibrosis and tubular atrophy (IFTA) associa

108 MicroRNA (miRNA) alterations accompanying interstitial fibrosis and tubular atrophy (IFTA) in kidn

109 t 6-month urinary CCL2: Cr is a predictor of interstitial fibrosis and tubular atrophy (IFTA) on 24-m

110 The biopsy diagnosis of interstitial fibrosis and tubular atrophy (IFTA) was bas

111 abnormal glomerulosclerosis, 23.9% abnormal interstitial fibrosis and tubular atrophy (IFTA), 4.8% a

112 ntation and if production is associated with interstitial fibrosis and tubular atrophy (IFTA).

113 py is a contributing factor to the origin of interstitial fibrosis and tubular atrophy (IFTA).

114 poorly characterized histopathologic entity interstitial fibrosis and tubular atrophy (IFTA).

115 lant patients with normal histology (n = 5), interstitial fibrosis and tubular atrophy (n = 6), subcl

116 emptive therapy group had moderate to severe interstitial fibrosis and tubular atrophy (odds ratio, 2

117 R at 12 months (P=0.001) and higher risk for interstitial fibrosis and tubular atrophy (P=0.01).

118 INK4a(-/-) mice developed significantly less interstitial fibrosis and tubular atrophy after ischemia

119 -dependent senescence, significantly reduced interstitial fibrosis and tubular atrophy and associated

120 P = 0.009), viral nephropathies (P = 0.002), interstitial fibrosis and tubular atrophy and inflammati

121 al cellular senescence in the development of interstitial fibrosis and tubular atrophy and kidney gra

122 lganciclovir therapy may lead to less severe interstitial fibrosis and tubular atrophy and to signifi

123 sult in chronic histological damage of which interstitial fibrosis and tubular atrophy are dominant f

124 Interstitial fibrosis and tubular atrophy are major cont

125 question in the human kidney allografts with interstitial fibrosis and tubular atrophy not otherwise

126 hose with normal histology, while those with interstitial fibrosis and tubular atrophy plus inflammat

127 sease process can be identified, what drives interstitial fibrosis and tubular atrophy progression in

128 After 1 year, interstitial fibrosis and tubular atrophy score was sign

129 Interstitial fibrosis and tubular atrophy score, interst

130 gher mean scores in the tacrolimus group for interstitial fibrosis and tubular atrophy with a trend t

131 Extensive interstitial fibrosis and tubular atrophy without a clea

132 cimens for the degree of glomerulosclerosis, interstitial fibrosis and tubular atrophy, and vascular

133 ic signs of AKI to CKD transition, including interstitial fibrosis and tubular atrophy, and with an a

134 nic allograft injury (CAI), characterized by interstitial fibrosis and tubular atrophy, leads to a pr

135 flamed histology (1.4 [0.4, 4.2], normal and interstitial fibrosis and tubular atrophy, n = 52), and

136 that together were associated with increased interstitial fibrosis and tubular atrophy.

137 een observed in renal transplants developing interstitial fibrosis and tubular atrophy; however, whet

138 le cardiac remodelling, such as hypertrophy, interstitial fibrosis, and abnormal activity of the card

139 renal injury, including glomerulosclerosis, interstitial fibrosis, and albuminuria.

140 global glomerulosclerosis, tubular atrophy, interstitial fibrosis, and arteriosclerosis.

141 te foot process effacement, tubular atrophy, interstitial fibrosis, and casts, were observed.

142 y associated with cardiomyocyte hypertrophy, interstitial fibrosis, and contractile dysfunction.

143 diffuse glomerulosclerosis, tubular atrophy, interstitial fibrosis, and decreased renal excretory fun

144 development of left ventricular hypertrophy, interstitial fibrosis, and diastolic dysfunction.

145 gy, CKD is characterized by tubular atrophy, interstitial fibrosis, and glomerulosclerosis.

146 kidney function, more glomerulosclerosis and interstitial fibrosis, and greater propensity to progres

147 ent, mitochondrial damage, apoptosis, and LV interstitial fibrosis, and hence contributes to both sys

148 iting pathological myofibroblast activation, interstitial fibrosis, and HF progression.

149 expression, attenuated tubular apoptosis and interstitial fibrosis, and improved renal function and t

150 tion, hypoperfusion, increased deposition of interstitial fibrosis, and increased apoptosis of endoth

151 very, reduced postinjury tubular atrophy and interstitial fibrosis, and increased the regenerative ca

152 ich had severe vascular intimal hyperplasia, interstitial fibrosis, and inflammation.

153 omyocyte necrosis with replacement fibrosis, interstitial fibrosis, and intercalated disc dissociatio

154 well as reduced levels of oxidative stress, interstitial fibrosis, and myocyte apoptosis.

155 in the thickness of interventricular septum, interstitial fibrosis, and phosphorylated p38 mitogen-ac

156 iron deposition, blocked the development of interstitial fibrosis, and prevented immune cell infiltr

157 tivity, cell proliferation, cyst growth, and interstitial fibrosis, and significantly improved renal

158 gnificantly less tubular senescence, reduced interstitial fibrosis, and superior renal function 30 da

159 in promotes cell proliferation, cyst growth, interstitial fibrosis, and the decline in renal function

160 hrough macrophage infiltration, resulting in interstitial fibrosis, and through altered paracrine sig

161 function through mitochondrial apoptosis, LV interstitial fibrosis, and to some extent to myocardial

162 teriolosclerosis, global glomerulosclerosis, interstitial fibrosis, and tubular atrophy) all increase

163 with increased fractional interstitial area, interstitial fibrosis, and tubular atrophy.

164 egulating the cardiac hypertrophic response, interstitial fibrosis, and ventricular remodeling follow

165 ormal mitochondria; extensive subendocardial interstitial fibrosis; and marked hypertrophy of arterio

166 and genetic blockade of p53 attenuated renal interstitial fibrosis, apoptosis, and inflammation in mi

167 In addition, these hearts develop interstitial fibrosis associated with diffuse N-cadherin

168 ificant reduction of cardiac hypertrophy and interstitial fibrosis at 14 days after ischemia.

169 atinocyte-derived chemokine and resulting in interstitial fibrosis at 28 days.

170 they showed signs of cardiac hypertrophy and interstitial fibrosis at 8 weeks of age.

171 inated the difference in obstruction-induced interstitial fibrosis between wild-type and VDR-null mic

172 role, not only in the pathogenesis of renal interstitial fibrosis but also in the onset of podocyte

173 ntly preserves cardiac function, and reduces interstitial fibrosis but does not alter the numbers of

174 centage of glomerulosclerosis, and degree of interstitial fibrosis but not immunomodulatory treatment

175 GRP receptor (GRPR)-null mice had increased interstitial fibrosis but reduced defects in alveolariza

176 Hypothyroidism increased atrial interstitial fibrosis, but connexin 43 was not affected.

177 us casts, with marked glomerulosclerosis and interstitial fibrosis by 6 weeks of age.

178 cardiac allografts significantly attenuated interstitial fibrosis, cardiac hypertrophy, and improved

179 aracterized by deteriorating graft function, interstitial fibrosis, cardiac hypertrophy, and occlusiv

180 Furthermore, fingolimod decreased interstitial fibrosis, cardiomyocyte hypertrophy, and ch

181 ntrarenal polyomavirus load levels and Banff interstitial fibrosis ci scores.

182 Moderate/severe interstitial fibrosis (ci>/=2) at 1 year was observed in

183 The total Banff score equaled the sum of interstitial fibrosis (CI), tubular atrophy, arteriolar

184 remodeling, smaller LV cavity size, and more interstitial fibrosis compared with high gradient (HG) n

185 Collagen deposition and interstitial fibrosis contribute to cardiac remodeling a

186 titin; and 5) both stiff cardiomyocytes and interstitial fibrosis contribute to high diastolic left

187 MCP-1 neutralization also inhibited interstitial fibrosis, decreasing left ventricular dysfu

188 ths were associated with moderate and severe interstitial fibrosis developed at 24 months after trans

189 ey pathology, including hyperplasia of PTCs, interstitial fibrosis, development of glomerular cysts,

190 Subclinical development of interstitial fibrosis during the first posttransplant ye

191 uding cardiac magnetic resonance measures of interstitial fibrosis (extracellular volume fraction), a

192 e breaks conditioned by strands of increased interstitial fibrosis (fibrous tissue content in heart f

193 eases in cardiomyocyte cell surface area and interstitial fibrosis following pressure overload.

194 ed this model to examine renal apoptosis and interstitial fibrosis following unilateral urethral obst

195 The presence of glomerulitis or chronic interstitial fibrosis (g and ci scores by Banff, respect

196 dences of acute mild arteriolopathy, striped interstitial fibrosis, glomerular congestion, and tubula

197 han 40% global glomerular sclerosis, or with interstitial fibrosis greater than 30% or for patients o

198 ailure progression with a 3-fold increase in interstitial fibrosis, greater accumulation of polyubiqu

199 Loss of kidney graft function due to interstitial fibrosis (IF) and tubular atrophy (TA) is t

200 Interstitial fibrosis (IF) and tubular atrophy (TA) was

201 The incidence of (interstitial fibrosis) IF/(tubular atrophy) TA at month

202 Ang-(1-7) concomitantly reduced interstitial fibrosis in association with a significant

203 crease in both myofibroblast recruitment and interstitial fibrosis in bleomycin-treated syndecan-4-nu

204 ities, suggesting that imaging biomarkers of interstitial fibrosis in cancer survivors are related to

205 liorate hypertension, renal hypertrophy, and interstitial fibrosis in diabetes.

206 ificantly to the tubular apoptosis and renal interstitial fibrosis in kidney diseases.

207 model can be used to monitor treatments for interstitial fibrosis in LN with drugs currently being d

208 e was significantly less lung congestion and interstitial fibrosis in MAFbx KO than in WT mice.

209 ific knockdown of Shroom3 markedly abrogated interstitial fibrosis in mice with unilateral ureteric o

210 oad-induced heart failure despite inhibiting interstitial fibrosis in mice.

211 paricalcitol significantly attenuated renal interstitial fibrosis in mouse kidney after ureteral obs

212 hat paricalcitol is able to ameliorate renal interstitial fibrosis in obstructive nephropathy, possib

213 r expansion (ECE), which has been related to interstitial fibrosis in previous clinical and subclinic

214 independently associated with the degree of interstitial fibrosis in protocol biopsies, scored accor

215 several inflammatory molecules and increased interstitial fibrosis in regenerating muscle.

216 ge infiltration after 3 to 5 days and marked interstitial fibrosis in the ischemic area after 7 days,

217 tant to aldosterone-dependent progression of interstitial fibrosis in the kidney.

218 with extensive fibrosis in the LAI group and interstitial fibrosis in the LCx group.

219 mmation in healing infarcts, but it prevents interstitial fibrosis in the noninfarcted myocardium and

220 tubular cell apoptosis and suppressed renal interstitial fibrosis in UUO.

221 rowth factor beta1 (TGF-beta) promotes renal interstitial fibrosis in vivo and the expression of mese

222 and we have shown that HA is a correlate of interstitial fibrosis in vivo.

223 ty of intrarenal factors lead to progressive interstitial fibrosis, including the newly described pro

224 erity of tubular atrophy, tubular casts, and interstitial fibrosis increased significantly in Nr4a1(-

225 However, KO hearts after TAC had increased interstitial fibrosis, increased apoptosis, and failed i

226 deletion of miR-214 significantly attenuated interstitial fibrosis induced by unilateral ureteral obs

227 ressive acinar cell vacuolization and death, interstitial fibrosis, inflammation, and circulatory rel

228 Interstitial fibrosis is an outcome measure of increasin

229 ate that the severity and structure of local interstitial fibrosis is associated with atrial conducti

230 Six weeks post-reperfusion, interstitial fibrosis is greater in aged kidneys based o

231 Diffuse interstitial fibrosis is present in diverse cardiomyopat

232 tion and sclerotic glomeruli; iii) decreased interstitial fibrosis; iv) preservation of kidney functi

233 ularization (at 28 days, post-I-R) and lower interstitial fibrosis, leading to improved left ventricu

234 myocardial apoptosis and the development of interstitial fibrosis, left atrial hypertrophy, and pulm

235 MCP-1-null mice had markedly diminished interstitial fibrosis, lower macrophage infiltration, an

236 Existing data suggest that increased interstitial fibrosis may occur abnormally in renal tran

237 sinus node dysfunction, cardiac hypertrophy, interstitial fibrosis, multi-focal myocardial fiber deat

238 =7) and those that had developed subclinical interstitial fibrosis (n=8) at 1 year.

239 P significantly decreased volume fraction of interstitial fibrosis, normalized sarcoplasmic reticulum

240 We studied the effect of interstitial fibrosis on conduction velocity (CV) in the

241 s as well as the percent tubular atrophy and interstitial fibrosis on renal biopsy were assessed.

242 so more likely to demonstrate the finding of interstitial fibrosis (OR 3.0 and 3.7, P<0.0001, respect

243 phocytic bronchiolitis (OR 5.1, P<0.0001) or interstitial fibrosis (OR 3.2, P<0.0001).

244 kidneys into C57BL6 mice was associated with interstitial fibrosis (P<0.0001), tubular atrophy (P<0.0

245 reservation of tubules (P=0.008) and reduced interstitial fibrosis (P=0.01), with decreased myofibrob

246 vealed a significant decrease in replacement/interstitial fibrosis, p-SMAD2/3 and p-ERK1/2.

247 Interstitial fibrosis plays a key role in the developmen

248 Cardiac interstitial fibrosis plays an important role in the pat

249 losclerosis, and greater tubular atrophy and interstitial fibrosis predicted ESRD.

250 plantation is an independent risk factor for interstitial fibrosis progression and is associated with

251 Pepstatin A treatment slowed interstitial fibrosis progression following IRI induced

252 al-derived Wnt ligand is sufficient to drive interstitial fibrosis provides strong support for the ma

253 on of EMT-related gene expression with early interstitial fibrosis (r<0.45) for size-mismatched allog

254 reversed cardiac and myocyte hypertrophy and interstitial fibrosis, reduced the propensity for ventri

255 P proteins in enhancing or suppressing renal interstitial fibrosis, respectively, we examined whether

256 l dysfunction, nitric oxide disarrays, renal interstitial fibrosis, sarcopenia, and worsening protein

257 t post was used to assess volume fraction of interstitial fibrosis, sarcoplasmic reticulum calcium AT

258 re observed for g, C4d, tubular atrophy, and interstitial fibrosis scores in early AMR patients and t

259 igh proximal tubular proliferation rates and interstitial fibrosis, similar to known polycystic kidne

260 ongitudinal conduction blocks coincided with interstitial fibrosis strands that were exaggerated in t

261 These mice developed progressive interstitial fibrosis surrounding collecting ducts with

262 nd CSCs similarly inhibited infarct size and interstitial fibrosis, SVPs were superior in inducing an

263 histologic scores for glomerulosclerosis and interstitial fibrosis than untreated controls.

264 tor (tPA) have fewer myofibroblasts and less interstitial fibrosis than wild-type controls.

265 bly, donor age correlated significantly with interstitial fibrosis, tubular atrophy, and glomeruloscl

266 Interstitial fibrosis, tubular atrophy, and glomeruloscl

267 disease, among other factors, the result is interstitial fibrosis, tubular atrophy, and graft failur

268 al end points, including glomerulosclerosis, interstitial fibrosis, tubular injury, and inflammation.

269 Interstitial fibrosis/tubular atrophy (IF/TA) contribute

270 KCC2, E-cadherin, and 18S rRNA diagnostic of interstitial fibrosis/tubular atrophy (IF/TA).

271 model included GFR (P<0.001) and presence of interstitial fibrosis/tubular atrophy (P=0.003) at diagn

272 VAN = 10, normal functioning graft = 73, and interstitial fibrosis/tubular atrophy = 59) from 168 uni

273 with increased allograft glomerulopathy and interstitial fibrosis/tubular atrophy lesions (P<0.001 f

274 tinine level, tacrolimus level, and grade of interstitial fibrosis/tubular atrophy or graft loss.

275 intended primary end point was the change in interstitial fibrosis/tubular atrophy score between impl

276 ejection and better renal function with less interstitial fibrosis/tubular atrophy versus calcineurin

277 l blood pressure, change in body mass index, interstitial fibrosis/tubular atrophy, and change in ren

278 Banff-2 antibody-mediated rejection, Banff-5 interstitial fibrosis/tubular atrophy, in samples from s

279 ibodies), in three different models of renal interstitial fibrosis, unilateral ureteral obstruction,

280 CR is a progressive disease defined by interstitial fibrosis, vascular neointimal development,

281 (type p, q, or r) opacities, whereas grade 3 interstitial fibrosis was associated (P = 0.02) with the

282 Finally, interstitial fibrosis was evident 20 weeks after radiati

283 Interstitial fibrosis was significantly increased within

284 ated apoptotic pathway and its role in renal interstitial fibrosis, we established a mouse model in w

285 Glomerulosclerosis and interstitial fibrosis were also significantly ameliorate

286 pillary and arteriole densities, and cardiac interstitial fibrosis were analyzed in the border zone o

287 icited increases in hypertrophic markers and interstitial fibrosis were blunted in the knockout anima

288 Cardiomyocyte size and interstitial fibrosis were increased equally in both gen

289 ar hyalinosis, mesangiolysis, and focal mild interstitial fibrosis were present.

290 Agt gene expression, renal hypertrophy, and interstitial fibrosis were studied.

291 d extracellular volume fraction, a marker of interstitial fibrosis, were determined with a model for

292 Ketamine treatment also enhanced bladder interstitial fibrosis, whereas ketamine + Cox-2 inhibito

293 atory cell infiltration, tubular damage, and interstitial fibrosis, whereas similar changes were abse

294 ar cells significantly reduced the extent of interstitial fibrosis, which correlated with reduced fib

295 ar cells may be involved in the induction of interstitial fibrosis, which in turn, leads to loss of r

296 cludes induction of renal cyst formation and interstitial fibrosis while the latter entails urine-con

297 tion within the allograft and development of interstitial fibrosis, whilst type III collagen depositi

298 Four had diffuse interstitial fibrosis with chronic inflammation, and two

299 ing, and fatal lung disease characterized by interstitial fibrosis with decreasing lung volumes and h

300 characterized by myocardial remodeling with interstitial fibrosis, with reduced diastolic function a