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1 fection/pyelonephritis, viral nephritis, and interstitial nephritis.
2 aving renal dysfunction that suggested acute interstitial nephritis.
3 ocyte proliferation, lymphoid depletion, and interstitial nephritis.
4 had little effect on T cell infiltration and interstitial nephritis.
5 a rise in creatinine revealed polyoma virus interstitial nephritis.
6 basic protein, and renal tubular Ag-induced interstitial nephritis.
7 ients with the idiopathic variety of chronic interstitial nephritis.
8 the induction and persistence of leptospiral interstitial nephritis.
9 r epithelium, and varying degrees of chronic interstitial nephritis.
10 ng urinary tract infection (UTI) and chronic interstitial nephritis.
11 hocyte injury during allograft rejection and interstitial nephritis.
12 creas grafts and no evidence of recurrent PV interstitial nephritis 22 and 37 months after retranspla
13 odies, glomerular IgG and C3 deposition, and interstitial nephritis all developed by 3-5 wk of age.
14 idney transplant recipient who developed BKV interstitial nephritis and carcinoma of the bladder with
16 convoluted tubules of the kidney by causing interstitial nephritis and inducing the same cytokines i
23 ous adverse event during deferred treatment (interstitial nephritis) and one during the placebo phase
28 the parent strain caused severe cystitis and interstitial nephritis as determined by histopathologica
35 hemodynamics, proteinuria, tubular biology, interstitial nephritis, fibroblast formation, and fibros
36 c ESRD (ESRD caused by glomerulonephritis or interstitial nephritis) for which the SIR was 4.22 (95%
38 gnesium), rebound acid hypersecretion, acute interstitial nephritis, gastric carcinoid tumor, cardiov
41 ly-onset nephropathy with features of tubulo-interstitial nephritis, hypertension and tendency for hy
43 nsfer of CSF-1 or GM-CSF incited more severe interstitial nephritis in IFN-gammaR-deficient than in I
44 retroviral gene transfer approach to incite interstitial nephritis in MRL-Fas(lpr) mice that is rapi
45 primate polyomavirus SV40 is known to cause interstitial nephritis in primary infections and progres
47 ent neuropathy, granulomatous glomerular and interstitial nephritis, interstitial lung disease, arter
48 uld indicate that graft loss secondary to PV interstitial nephritis is not an absolute contraindicati
51 r, FAN1 mutations predispose to karyomegalic interstitial nephritis (KIN) and cancer rather than to F
53 mutations in FAN1 as a cause of karyomegalic interstitial nephritis (KIN), a disorder that serves as
54 ase 1 (FAN1) in humans leads to karyomegalic interstitial nephritis (KIN), a rare hereditary kidney d
55 Mutations in human Fan1 cause karyomegalic interstitial nephritis (KIN), but it is unclear whether
56 -Schiff D staining of infected kidney showed interstitial nephritis, mononuclear cell infiltrates, an
58 neumonia is inconsistent, and although acute interstitial nephritis, nutritional deficiencies (includ
61 male sex, age >/=50 years, diabetes, chronic interstitial nephritis, polycystic kidney disease, and 1
62 iation between SIGEI and risk for iatrogenic interstitial nephritis: SIGEI was seen in only 4 of 15 p
63 Using the MRL/MpJ fas<lpr> model of lupus interstitial nephritis, we found that KTC did not expres
64 ystemic complications of SS (ie, vasculitis, interstitial nephritis) were also more common in men (64
65 Far less common is the entity of primary interstitial nephritis wherein the glomerular and vascul
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