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1 d a mononuclear cell infiltration within the interstitial tissue.
2 blast-like cells that surrounded a pulp-like interstitial tissue.
3 type I and type VI expression was within the interstitial tissue.
4 nd showed that expression was limited to the interstitial tissue.
5 always within blood vessel lumens and not in interstitial tissue.
6 three treated groups without an increase in interstitial tissue.
7 hich cohesive groups of cells invade through interstitial tissue.
8 lls to pass from the vascular space into the interstitial tissues.
9 le preventing microbial contamination of the interstitial tissues.
10 droitin-6-sulphate were expressed within the interstitial tissues.
12 ain permanent access to type I collagen-rich interstitial tissues, an experimental platform was estab
13 parenchymal basement membranes, move through interstitial tissue, and enter and exit the vasculature.
15 grating cells penetrate type I collagen-rich interstitial tissues by mobilizing undefined proteolytic
16 reduced immunoglobulin concentrations in the interstitial tissue, confirmed in patients, may have imp
17 in extracellular matrix or abnormal myocyte-interstitial tissue coupling may be important for the ge
18 ch progressed to dilated cardiomyopathy with interstitial tissue fibrosis and death by 6 months in ma
22 y located at the interface between blood and interstitial tissues, placing thus endothelial cell as a
23 tivated by its paracrine ligand HGF from the interstitial tissues rather than by an autocrine loop or
24 ively to the main body of nephrons and renal interstitial tissues, respectively, indicating a lineage
26 re evidenced in vivo in hemocyte-infiltrated interstitial tissues surrounding wounds, whereas they we
27 disorganized with apparent disintegration of interstitial tissue that normally surrounds the seminife
30 peripheral airways, alveoli, and surrounding interstitial tissue which develops as the result of a no
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