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1 and Bloom's complexes at the site of the DNA interstrand crosslink.
2 be informative about the metabolism of other interstrand crosslinks.
3 and facilitates replication traverse of DNA interstrand crosslinks.
4 the stimulation of DNA synthesis by psoralen interstrand crosslinks.
5 fficient in vitro DNA resynthesis induced by interstrand crosslinks.
6 NA polymerase-helicase involved in repair of interstrand crosslinks.
7 s, including monoadducts and intrastrand and interstrand crosslinks.
8 links were preferred targets for the ER over interstrand crosslinks.
9 f damage, such as double-stranded breaks and interstrand crosslinks.
10 ncreased activity of DNA2 and WRN at the DNA interstrand crosslinks.
11 core complex that mediates the repair of DNA interstrand crosslinks.
12 that senses and repairs damage caused by DNA interstrand crosslinks.
13 interfere with normal cellular processing of interstrand crosslinks.
14 A core complex to chromatin in repairing DNA interstrand crosslinks.
15 -distorting DNA damage and the repair of DNA interstrand crosslinks.
16 hor complex that recognizes damage caused by interstrand crosslinks, a multisubunit ubiquitin ligase
17 nts cause defective cellular response to DNA interstrand crosslinking agent and telomere maintenance,
18 diazepine (PBD) dimer, is a highly efficient interstrand crosslinking agent that reacts with guanine
19 n, knockdown of NONO sensitizes cells to the interstrand crosslinking agent, cisplatin, whereas knock
20 Upon exposure to ionizing radiation or an interstrand crosslinking agent, the number of cells exhi
21 characterized by cellular sensitivity to DNA interstrand crosslinking agents and a high risk of cance
22 en shown to be specifically sensitive to DNA interstrand crosslinking agents but not sensitive to mon
23 y can result in a marked hypersensitivity to interstrand crosslinking agents, such as mitomycin C.
29 fective in promoting replication traverse of interstrand crosslinks and is also inefficient in promot
30 involved in the recognition or repair of DNA interstrand crosslinks and perhaps other forms of DNA da
31 re rapidly recruited to forks stalled by DNA interstrand crosslinks, and both are required for cellul
34 stards selectively formed two base-staggered interstrand crosslinks between the 5'G and the G opposit
36 as well as trioxsalen (psoralen)-induced DNA interstrand crosslinks, but not to angelicin monoadducts
37 ke MUS81-EME1, is required for repair of DNA interstrand crosslinks, but this role appears to be inde
40 ified NEIL1 protein bound stably to psoralen interstrand crosslink-containing synthetic oligonucleoti
41 onoaziridinyl-1,4-benzoquinones were able to interstrand crosslink DNA after reduction and were cytot
43 otoxic agents that generate alkylated bases, interstrand crosslinks, DNA-protein crosslinks, and doub
44 acted doses of 14 alkylating and similar DNA interstrand crosslinking drugs from medical records.
45 this groove for efficient digestion past DNA interstrand crosslinks, facilitating the key DNA repair
46 ifference in the extent of N-alkyl purine or interstrand crosslink formation in the N-ras, c-myc or C
49 resolution of non-B DNA structures including interstrand crosslinks, G quadruplexes and DNA triplexes
50 roduces a number of DNA adducts with the DNA interstrand crosslink (ICL) considered to be the critica
51 pression, abrogation of BRIP1 foci after DNA interstrand crosslink (ICL) damage and hypersensitivity
53 other key cellular processes, including DNA interstrand crosslink (ICL) repair and DNA double-strand
54 s roles in nucleotide excision repair (NER), interstrand crosslink (ICL) repair, homologous recombina
60 date factors affecting TFO-directed psoralen interstrand crosslink (ICL)-induced recombination, we co
62 oligonucleotides (TFOs) conjugated to a DNA interstrand crosslinking (ICL) agent, psoralen (pTFO-ICL
63 (FA) patients are extremely sensitive to DNA interstrand crosslinking (ICL) agents, but the molecular
68 tients and mouse models are sensitive to DNA interstrand crosslinks (ICLs) and FA mice are moderately
69 ay plays a central role in the repair of DNA interstrand crosslinks (ICLs) and regulates cellular res
70 CA pathway is critical for the repair of DNA interstrand crosslinks (ICLs) and the maintenance of chr
81 esolving double-strand DNA breaks (DSBs) and interstrand crosslinks (ICLs) by homologous recombinatio
82 ins thought to function in the repair of DNA interstrand crosslinks (ICLs) comprise what is known as
86 ed that fludarabine enhanced accumulation of interstrand crosslinks (ICLs) in specific regions of the
89 not essential for cells to survive toxic DNA interstrand crosslinks (ICLs), although MMR proteins bin
91 otein network is necessary for repair of DNA interstrand crosslinks (ICLs), but its control mechanism
92 Several important anti-tumor agents form DNA interstrand crosslinks (ICLs), but their clinical effici
94 le-deficient cells are hypersensitive to DNA interstrand crosslinks (ICLs), indicating that BRCA1 has
96 r hypersensitivity to agents that induce DNA interstrand crosslinks (ICLs), such as mitomycin C (MMC)
97 agents used in cancer chemotherapy cause DNA interstrand crosslinks (ICLs), which covalently link bot
99 itin are required for localizing SLX4 to DNA interstrand crosslinks (ICLs), yet how SLX4 is targeted
108 NEIL1 recognizes specifically and distinctly interstrand crosslinks in DNA, and can obstruct the effi
109 es or a novel tetrafunctional mustard caused interstrand crosslinks in the target DNA and were more m
111 that EcoR124I can translocate past covalent interstrand crosslinks, inconsistent with an obligatory
112 veloped a mammalian cell free assay in which interstrand crosslinks induce DNA synthesis in both dama
115 the cellular level, hypersensitivity to DNA interstrand crosslinks is the defining feature in Fancon
116 es profound cellular hypersensitivity to DNA interstrand crosslink lesions in vivo, highlighting the
117 itutional genomic disorders, suggesting that interstrand crosslinks may play a pathogenic role in suc
119 to aid replication machines to traverse DNA interstrand crosslinks prior to post-replication repair.
120 emonstrated that neither mismatch repair nor interstrand crosslink repair affects the production of t
121 la MUS308, which is essential for normal DNA interstrand crosslink repair and is unique in that it co
122 ing replication and its participation in DNA interstrand crosslink repair and/or heteroduplex rejecti
123 CM participates in recombination-independent interstrand crosslink repair by facilitating recruitment
124 esults suggest that FAN1 has a minor role in interstrand crosslink repair compared with true FA genes
126 roper RAD51 function is important during DNA interstrand crosslink repair outside of homologous recom
127 the deficiency in replication-dependent DNA interstrand crosslink repair pathway commonly referred t
128 protein families, namely the PSO2 (SNM1) DNA interstrand crosslink repair proteins and the 73-kD subu
129 ce that BRCA1 plays an important role in DNA interstrand crosslink repair that is distinct from its e
130 including nucleotide excision repair (NER), interstrand crosslink repair, and meiotic recombination.
131 connected to a pathway that is deficient in interstrand crosslink repair, and that at least one othe
132 genetic complementation groups implicated in interstrand crosslink repair, FANCJ encodes a DNA helica
133 ty-DNA double-strand break (DSB) repair, DNA interstrand crosslink repair, repair of stalled replicat
134 monoubiquitinated FANCD2 and is required for interstrand crosslink repair, suggesting that mutation o
135 lication-dependent and transcription-coupled interstrand crosslink repair, while SNM1B/Apollo is requ
142 were exposed to chemicals that generate DNA interstrand crosslinks (repaired by FA proteins), but no
145 n in vitro assay in which the presence of an interstrand crosslink stimulates the incorporation of ra
146 alter the accumulation of NEIL1 at sites of interstrand crosslinks, suggesting distinct recognition
147 leotides (TFOs) that target a psoralen (pso) interstrand crosslink to a specific chromosomal site in
148 n C, which is consistent with the ability of interstrand crosslinks to induce homologous recombinatio
149 to oxidative DNA damage and psoralen-induced interstrand crosslinks was differentially affected by th
150 repair (BER) proteins in the response to DNA interstrand crosslinks, which block replication and tran
151 Their main role is in the repair of DNA interstrand crosslinks, which, by covalently binding the
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