ライフサイエンスコーパス: interventricular septum

1 ference toward the lateral wall, but not the interventricular septum.

2 most severe hypertrophy involving the basal interventricular septum.

3 lation, sharing a common pericardial sac and interventricular septum.

4 tions on the right ventricular aspect of the interventricular septum.

5 hom the critical part of the VT involved the interventricular septum.

6 1.1 mL, lesions extended anteriorly into the interventricular septum.

7 , a critical component was identified in the interventricular septum.

8 those obtained in the left ventricle and the interventricular septum.

9 ythrocytes in the subepicardium and muscular interventricular septum.

10 pressed in the left ventricular apex and the interventricular septum.

11 profiling of the four heart chambers and the interventricular septum.

12 attern of Ito expression was observed in the interventricular septum.

13 nts using a transvenous approach through the interventricular septum.

14 the heart, the left ventricle free wall and interventricular septum.

15 heart and is essential for formation of the interventricular septum.

16 An additional transducer was placed in the interventricular septum.

17 wall 973 +/- 42 vs 923 +/- 12 ms; P < 0.005; interventricular septum 1003 +/- 31 vs 974 +/- 21 ms, P

18 Cardiac hypertrophy (interventricular septum, 12+/-4 [7-23] mm; left ventricu

19 es were incomplete formation of the muscular interventricular septum and an abnormal and novel positi

20 asia of the right ventricular myocardium and interventricular septum and display profound ventricular

21 creased from 10 to 14 mm (p < 0.001) for the interventricular septum and from 10 to 13 mm for the pos

22 1 in cardiomyocytes reduces the thickness of interventricular septum and interstitial fibrosis and in

23 The magnitude of CV-IB was analyzed at the interventricular septum and left ventricular (LV) poster

24 hic finding of an increased thickness of the interventricular septum and posterior wall.

25 7), P=0.005), due to a reduction in both the interventricular septum and the left ventricular posteri

26 patients had scarring at the junction of the interventricular septum and the right ventricular (RV) f

27 transgene expression in the right ventricle, interventricular septum, and atrial ventricular canal; U

28 pment of the trabeculae, compact myocardium, interventricular septum, and endocardial cushion.

29 on right ventricular size, curvature of the interventricular septum, and maximal tricuspid regurgita

30 rise to the majority of the right ventricle, interventricular septum, and outflow tract in mammals an

31 ng of ventricular myocardium, especially the interventricular septum, and reduction of both ventricul

32 lular volume at the RV insertion points, the interventricular septum, and the left ventricular latera

33 ntricular and semilunar valves, the muscular interventricular septum, and the ventricular myocardium.

34 cardiac neural crest in the formation of the interventricular septum, and therefore could play a role

35 activities in the muscular component of the interventricular septum at embryonic day 12.5, when fusi

36 t became quite strong along the crest of the interventricular septum by E16.5.

37 Post-infarction VT involving the interventricular septum can involve the endocardial musc

38 bryos and that defects of the outflow tract, interventricular septum, cardiac vasculature, and hyposp

39 .50; 95% confidence interval, 1.22 to 5.11), interventricular septum diameter (odds ratio, 1.18; 95%

40 f the larger CoreValve prosthesis, increased interventricular septum diameter and prolonged QRS durat

41 ising from the mid and apical portion of the interventricular septum dissecting into the basal part.

42 ions, resulting in the failure of membranous interventricular septum formation.

43 ased interventricular septal wall thickness (interventricular septum in diastole Z value, +0.45 +/- 0

44 nd abnormal vasculature in the myocardium of interventricular septum in E15.5 Cited2(-/-) hearts were

45 revalence of the re-entry circuit within the interventricular septum in post-infarction patients refe

46 ncomitant with increases in the thickness of interventricular septum, interstitial fibrosis, and phos

47 Aneurysm of the muscular interventricular septum is a rare entity as compared to

48 ing lead by transvenous approach through the interventricular septum is feasible in patients.

49 g from within a perforator branch within the interventricular septum is helpful in identifying the si

50 Little is known about the formation of the interventricular septum (IVS), a central event during ca

51 the lateral wall occurred compared with the interventricular septum (IVS; P = .001); at 21 to 28 day

52 0.2 mmol/kg gadopentetate dimeglumine in the interventricular septum, left ventricular (LV) free wall

53 rdiac phenotype, including thickening of the interventricular septum, left ventricular volume reducti

54 The hypoxia of the outflow tract and interventricular septum peaked at E13.5 and dissipated b

55 ance between the anterior mitral leaflet and interventricular septum, septal base function and the an

56 S developed more regularly in vessels of the interventricular septum than in the right or left ventri

57 analysis a significant direct association of interventricular septum thickness (odds ratio for 1 SD i

58 h time point), RD significantly reduced mean interventricular septum thickness from 14.1 +/- 1.9 mm t

59 ficantly positive correlation with diastolic interventricular septum thickness in those athletes.

60 he correlation between J-point elevation and interventricular septum thickness suggests a possible me

61 between linoleic acid (18:2n6) and diastolic interventricular septum thickness.

62 hways from the great cardiac vein across the interventricular septum to create cerclage.

63 eshaped guiding catheter, driven through the interventricular septum to the LVS.

64 etected in the myocardium, especially in the interventricular septum, ventricular wall, and outflow t

65 93 patients (8%), an intramural focus in the interventricular septum was identified.

66 , the myocardium was remarkably thinner, and interventricular septum was incompletely formed.

67 mapping and local EGM assessment of the left interventricular septum was performed during RV basal se

68 All VTs mapped to the interventricular septum were acutely successfully ablate

69 TDE color M-mode images of the interventricular septum were recorded from the apical 4-

70 1 expression was also high in the developing interventricular septum, where expression of the BMP-1 g