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1  placenta are localized predominantly in the intervillous space.
2 lated with monocyte density in the placental intervillous space.
3 te to the accumulation of macrophages in the intervillous space.
4 leukin (IL)-10, in plasma collected from the intervillous space.
5 al endometrium to initiate blood flow to the intervillous space.
6  microm over the villous tips throughout the intervillous space.
7 of transport rates via ease of access to the intervillous space.
8 recruit B cells to further produce Ig in the intervillous spaces.
9 depositions of IgG3 and IgM were observed in intervillous spaces.
10                         Epithelial leaks and intervillous spaces also increased significantly in CLE+
11 it was found that IRBCs adhere mainly in the intervillous space and also at significant levels to the
12 to an accumulation of macrophages within the intervillous space and increased production of tumor nec
13         Although CSPG is present both in the intervillous space and on the syncytiotrophoblast surfac
14  antigens caused immediate breaks, increased intervillous spaces, and increased IELs in the intestina
15                           Epithelial breaks, intervillous spaces, and the number of intraepithelial l
16 ous CSPGs, we conclude that the CSPGs of the intervillous spaces are the receptors for placental IRBC
17 ction showed parasitized erythrocytes in the intervillous space but no hemozoin in macrophages nor in
18               However, the structures of the intervillous-space CSPGs changed considerably during the
19 ly, monocyte infiltration into the placental intervillous space has been identified as a key risk fac
20  this model, the depth of diffusion into the intervillous space is inversely proportional to the effi
21 infected erythrocytes (IEs) sequester in the intervillous space (IVS) of the placenta causing placent
22 sulfate proteoglycans (CSPGs) present in the intervillous spaces mediate the adherence of IRBCs in th
23 lfated, extracellular CSPGs localized in the intervillous spaces, minor amounts of two cell-associate
24  the placenta, the low sulfated CSPGs of the intervillous spaces most efficiently bind IRBCs.
25 eoglycan (CSPG) present predominantly in the intervillous space of the placenta.
26  cells (IRBCs) selectively accumulate in the intervillous spaces of placenta, leading to poor fetal o
27 droitin sulfate proteoglycans (CSPGs) in the intervillous spaces of the placenta are the receptors fo
28 an mediate the sequestration of IRBCs in the intervillous spaces of the placenta during the entire se
29 droitin sulfate proteoglycans (CSPGs) in the intervillous spaces of the placenta mediate the IRBC adh
30 arum-infected red blood cells (IRBCs) in the intervillous spaces of the placenta.
31 emonstrate that the CSPGs are present in the intervillous spaces throughout the second and third trim
32  a significant correlate of most components (intervillous space, villous, trophoblast, and capillary
33 placental weight (r = 0.41, P = 0.0009), and intervillous space volume (r = 0.38, P = 0.0024).
34                                              Intervillous space volume was increased in OT1DM (OT1DM
35  uterus are suspended as cell columns in the intervillous space, where they experience significant am
36 increased, epithelial leaks/gaps formed, and intervillous spaces widened.

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