ライフサイエンスコーパス: intestinal bacteria

1 Mucolytic potential is widespread among intestinal bacteria.

2 the host immune system in the gut and in the intestinal bacteria.

3 atory agent cyclophosphamide (CTX) relies on intestinal bacteria.

4 enteropathy and the prevalence of pathogenic intestinal bacteria.

5 al stimulation that can functionally replace intestinal bacteria.

6 utualistic relationship between the host and intestinal bacteria.

7 uld be attenuated in the complete absence of intestinal bacteria.

8 s an exaggerated immune response directed at intestinal bacteria.

9 fense that protects against dissemination of intestinal bacteria.

10 lin or to different commensal and pathogenic intestinal bacteria.

11 on of IgM and complement-mediated control of intestinal bacteria.

12 e suggests a connection between diet and the intestinal bacteria.

13 tory IgA (SIgA) is found in association with intestinal bacteria.

14 IgT coated most intestinal bacteria.

15 gulation of the immune response to commensal intestinal bacteria.

16 tor-kappaB, are involved in the detection of intestinal bacteria.

17 s of oligosaccharides fermented by different intestinal bacteria.

18 Intestinal bacteria aid host health and limit bacterial

19 The reduction of intestinal bacteria also significantly improved the grow

20 y be due to excessive hydrogen production by intestinal bacteria altering the partial pressure of nit

21 Vancomycin altered the abundance of intestinal bacteria and fungi, measured by 16S and 18S r

22 n part, by the interaction between commensal intestinal bacteria and gut-associated lymphoid tissue (

23 cells, and the interrelationship between the intestinal bacteria and Tfh cell responses in the gut.

24 ng the complex interactions between resident intestinal bacteria and the immune system could improve

25 at host innate immune signaling can modulate intestinal bacteria and ultimately the host's susceptibi

26 ulting in tissue infiltration of 'symbiotic' intestinal bacteria and viruses that essentially become

27 of principles governing the localization of intestinal bacteria, and spatial relationships between b

28 strates that a substantial proportion of the intestinal bacteria are culturable.

29 inant daidzein metabolites produced by human intestinal bacteria are equol and O-desmethylangolensin.

30 Because intestinal bacteria are potent modulators of systemic im

31 Intestinal bacteria are required for development of gut-

32 Protein toxins released by certain intestinal bacteria are the cause of many diarrhoeal dis

33 Intestinal bacteria are thought to play a role in the pa

34 ealed that neomycin altered the abundance of intestinal bacteria bearing gamma-d-glutamyl-meso-diamin

35 resent a major nutrient source, not just for intestinal bacteria but also for microorganisms that occ

36 RegIIIgamma binds intestinal bacteria but lacks the complement recruitment

37 matic activity leads to growth inhibition of intestinal bacteria by causing local tryptophan depletio

38 hat commensal fungi can functionally replace intestinal bacteria by conferring protection against inj

39 how a marked proportion of oxygen-sensitive intestinal bacteria can be transmitted between individua

40 Our study highlights how protective intestinal bacteria can modify microbial-associated mole

41 Given that intestinal bacteria can produce ethanol, the aim of this

42 Interestingly, intestinal bacteria can promote infection of several mam

43 A recent study in mice suggests that intestinal bacteria can trigger mammary carcinoma.

44 An improved understanding of how intestinal bacteria cause disease has become increasingl

45 Finally, new understanding of how intestinal bacteria cause disease is revealing that ente

46 Reviewed here are the varied ways that intestinal bacteria cause disease, which provide fundame

47 Importantly, we found the intestinal bacteria contain glycolipids which can be pre

48 Our findings indicate that intestinal bacteria contribute to the development of NSA

49 t of progenitor cell responses to pathogenic intestinal bacteria could provide a measure of predispos

50 The absence of intestinal bacteria did not affect the incidence of alph

51 In animal models, intestinal bacteria drive colitis and in humans certain

52 Intestinal bacteria drive the formation of lymphoid tiss

53 Abnormal composition of intestinal bacteria--"dysbiosis"-is characteristic of Cr

54 Intestinal bacteria employ microbial metabolites from th

55 tively, these data demonstrate that resident intestinal bacteria enhance the stability of beta-cateni

56 Small intestinal bacteria exhibit distinct gluten metabolic pa

57 rmfree or colonized with a limited number of intestinal bacteria failed to develop inflammatory bowel

58 d to a considerable interest in manipulating intestinal bacteria for therapeutic benefit, and trials

59 Intestinal bacteria form a resident community that has c

60 Analysis of intestinal bacteria from the terminal ilea of Nod2-defic

61 , and to microbial products derived from the intestinal bacteria, has resulted in a distinctive local

62 Intestinal bacteria have been implicated in the pathogen

63 Human intestinal bacteria have many roles in human health, mos

64 We hypothesized that intestinal bacteria impact the pathogenesis of multiple

65 IgA coating uniquely identifies colitogenic intestinal bacteria in a mouse model of microbiota-drive

66 to equol and O-desmethylangolensin (ODMA) by intestinal bacteria in approximately 30-50% and 80-90% o

67 Bifidobacteria are the dominant intestinal bacteria in breastfed infants.

68 bacillus plantarum and its interactions with intestinal bacteria in mice undergoing switches between

69 Animal models suggest a role for intestinal bacteria in supporting the systemic immune re

70 microbiota, focusing on the role of resident intestinal bacteria in the development of immune respons

71 scuss the roles of known and novel commensal intestinal bacteria in the pathogenesis of inflammatory

72 Dysregulated immune responses to commensal intestinal bacteria, including Escherichia coli, contrib

73 gated whether oral inoculation with specific intestinal bacteria increased colon inflammation in the

74 sion, these results support the concept that intestinal bacteria induce endogenous signals that play

75 ring the fermentation of fiber by endogenous intestinal bacteria, induces mitochondrial function-depe

76 Intestinal bacteria influence mammalian physiology, but

77 Our results suggest that translocation of intestinal bacteria into liver may be involved as a one

78 Escape of intestinal bacteria into the ascites is involved in the

79 Increased translocation of intestinal bacteria is a hallmark of chronic liver disea

80 ses vs controls, we found that reactivity to intestinal bacteria is a normal property of the human CD

81 In this review the intestinal bacteria is discussed in the context of devel

82 innate immune inflammatory response against intestinal bacteria is sufficient to induce colon cancer

83 Although the inability to control intestinal bacteria is thought to underlie IBD, the role

84 Resident intestinal bacteria likely play an important role in the

85 Although intestinal bacteria live deep within the body, they are

86 accumulating to support the hypothesis that intestinal bacteria not only exchange resistance genes a

87 nce elicitor of systemic immune responses to intestinal bacteria observed in a loss-of-tolerance mode

88 al reservoir of particular importance is the intestinal bacteria of food-producing animals.

89 ing of the impact of translocating commensal intestinal bacteria on mucosal-associated T cell respons

90 eview examines the influence of signals from intestinal bacteria on the homeostasis of the mammalian

91 ed that stress promotes the translocation of intestinal bacteria or their toxins into the systemic co

92 rapidly respond to commensal and pathogenic intestinal bacteria, parasites and food components by po

93 a selective force in the evolution of their intestinal bacteria, particularly by increasing the prev

94 Here, we analyzed the intestinal bacteria presented in normal and infected wor

95 ation after antibiotic-mediated depletion of intestinal bacteria prevents colitis and influenza, thus

96 Host intestinal bacteria produce its precursor trimethylamine

97 Which intestinal bacteria provide resistance to C. difficile i

98 ead to exaggerated inflammatory responses to intestinal bacteria, raising the possibility that defect

99 Furthermore, the reduction in intestinal bacteria resulted in the elimination of the e

100 chain fatty acids (SCFA) are metabolites of intestinal bacteria resulting from fermentation of dieta

101 Using these collections, we found that intestinal bacteria selected on the basis of high coatin

102 tem targets non-self rather than self, these intestinal bacteria should be considered foreign and the

103 n probiotics, nutritional components and the intestinal bacteria should be considered when examining

104 entage of them become colonized by resistant intestinal bacteria such as extended-spectrum beta-lacta

105 ns that are highly specific for target Ag on intestinal bacteria, suggesting that an aberrant T cell

106 ry bowel disease, is attributed, in part, to intestinal bacteria that may initiate and perpetuate muc

107 MIA phenotypes in offspring require maternal intestinal bacteria that promote TH17 cell differentiati

108 e contact at birth with maternal vaginal and intestinal bacteria, the authors proposed that this coul

109 s review, we explore a more sinister side of intestinal bacteria; their role as traffickers in antibi

110 New evidence has implicated the lack of intestinal bacteria to be responsible for the degradatio

111 a, which may contribute to the adaptation of intestinal bacteria to the mucosal environment in health

112 Furthermore, supplement of killed intestinal bacteria was able to restore ConA-mediated NK

113 at colonization of GF mice with a variety of intestinal bacteria was capable of reducing T1D in MyD88

114 oxidative derivative of choline produced by intestinal bacteria, was positively associated with rect

115 erplay between the mucosal immune system and intestinal bacteria, we investigated the role of MIF in

116 The expanded intestinal bacteria were required for the parasite-induc

117 n of SAAs from meat to sulfide production by intestinal bacteria with use of both a model culture sys