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1 l in milieus that mimicked gastric and small intestinal content.
2 ence analysis of bacterial DNA prepared from intestinal content.
3 were also evaluated to simulate postprandial intestinal contents.
4 n drinking water) led to alkalization of the intestinal contents.
5 ISA, and PEC was detected by IEM in feces or intestinal contents.
6 and proteolytic activities were measured in intestinal contents.
7 physical barrier between the host tissue and intestinal contents, a separation that is vital for heal
8 raises the possibility that dysbiosis of the intestinal content also influences the outcome of cancer
9 hese transitions include dramatic changes in intestinal contents and require appropriate adaptations
11 in net fluid hyperabsorption, dehydration of intestinal contents, and a propensity to inspissated int
12 a, and lower viral loads in liver, bile, and intestinal content collected at three different necropsy
15 LLC-PK cells, but only in the presence of an intestinal content fluid filtrate from gnotobiotic pigs.
16 tected by immunoelectron microscopy (IEM) in intestinal contents from all the WT-PEC-inoculated pigs,
18 Pigs inoculated intravenously with serum or intestinal contents from the viremic virulent HRV-inocul
19 n cell culture but only with the addition of intestinal contents from uninfected gnotobiotic pigs.
21 om physiologic polymicrobial cultures (e.g., intestinal contents, human feces) and reduce TMAO levels
22 I/4/HS66/2001/US strain or with pig-passaged intestinal contents (IC) and euthanized acutely (n = 43)
23 rofiles were determined in serum, feces, and intestinal contents (IC) of the HuNoV-HS66-inoculated ca
24 (reg)] IL-10) cytokine profiles in serum and intestinal contents (IC) of the HuNoV-HS66-inoculated pi
25 tion in hilA or invG were recovered from the intestinal contents, intestinal tissues, and systemic ti
26 r V. cholerae The induction of TCP by murine intestinal contents is counteracted when Ca(2+) is deple
27 levels in the liver, intestine, plasma, and intestinal content, led to drastic decreases in the mRNA
28 g or infectious virus titers in the feces or intestinal contents measured by enzyme-linked immunosorb
29 n this study, U.S. PDCoV strain OH-FD22 from intestinal contents of a diarrheic pig from Ohio was iso
30 ain, PC177, by blind serial passaging of the intestinal contents of a diarrheic piglet in Vero cell c
31 zyme-linked immunosorbent assay in serum and intestinal contents of neonatal gnotobiotic pigs and IL-
32 [OH-FD22(DC44) strain] in LLC-PK cells from intestinal contents of PDCoV OH-FD22 strain-inoculated g
33 nt IFN-gamma responses occurred in serum and intestinal contents of the AttHRV-infected pigs, compare
37 mune response predisposes to accumulation of intestinal contents that breach the mucosal barrier of t
38 uring vortex mixing and downward flow of the intestinal contents, thereby persisting long enough for
39 Levels of breath hydrogen were measured and intestinal content was assessed by MRI before and at var
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