ライフサイエンスコーパス: intestinal epithelial cell

1 mited TNF-dependent apoptosis in transformed intestinal epithelial cells.

2 tion, and transcriptional activity of YAP in intestinal epithelial cells.

3 n polymerization and caspase-3 activation in intestinal epithelial cells.

4 pendent binding to sulfated proteoglycans on intestinal epithelial cells.

5 stress and the unfolded protein response in intestinal epithelial cells.

6 ell death by regulating ERK1/2 MAP kinase in intestinal epithelial cells.

7 turnover and caspase-dependent apoptosis of intestinal epithelial cells.

8 pression at the posttranscriptional level in intestinal epithelial cells.

9 ut activation of the Akt survival pathway in intestinal epithelial cells.

10 re susceptible to C. rodentium attachment to intestinal epithelial cells.

11 tor was explored using CB1-knockdown (CB1Kd) intestinal epithelial cells.

12 phorylation, and transcriptional activity in intestinal epithelial cells.

13 ssion of pro- and anti-inflammatory genes by intestinal epithelial cells.

14 ce of PrP(c) in the nucleus of proliferating intestinal epithelial cells.

15 sufficient to stimulate the proliferation of intestinal epithelial cells.

16 antly led to an increase in CFTR activity in intestinal epithelial cells.

17 ctivated the Tfr1 gene selectively in murine intestinal epithelial cells.

18 phagy enhances TJ barrier function in Caco-2 intestinal epithelial cells.

19 ffector" proteins into the cytoplasm of host intestinal epithelial cells.

20 ontrolled at the level of IL-18 signaling in intestinal epithelial cells.

21 f the Wnt signaling pathway in proliferating intestinal epithelial cells.

22 riptional responses in specific fractions of intestinal epithelial cells.

23 ive oxygen species generator known to damage intestinal epithelial cells.

24 mation in neurons, and bacterial adhesion to intestinal epithelial cells.

25 bonate, the intestinal pH buffer secreted by intestinal epithelial cells.

26 sa of patients with CD, adhere to and invade intestinal epithelial cells.

27 trol mice or mice with deletion of EGFR from intestinal epithelial cells.

28 somal cadherin and intercellular adhesion in intestinal epithelial cells.

29 promoter activity upon transfection in human intestinal epithelial cells.

30 ired for autophagy and autophagy response in intestinal epithelial cells.

31 uced a protective gene expression program in intestinal epithelial cells.

32 to Dsg2, decreased intercellular adhesion in intestinal epithelial cells.

33 ing EGFR signaling and cell proliferation in intestinal epithelial cells.

34 ling drives catenin-related transcription in intestinal epithelial cells.

35 and regulates lipid absorption and export in intestinal epithelial cells.

36 nsing of intraepithelial lymphocytes to kill intestinal epithelial cells.

37 ragweed did not affect the integrity of the intestinal epithelial cells.

38 cum gliadin-derived peptides in human Caco-2 intestinal epithelial cells.

39 was performed in monocytes, lymphocytes, and intestinal epithelial cells.

40 and restitution were reduced in the isolated intestinal epithelial cells.

41 vel and reduced glutathione concentration in intestinal epithelial cells.

42 Intestinal epithelial cells absorb nutrients, respond to

43 arget of TNF-alpha and was phosphorylated in intestinal epithelial cells after alcohol administration

44 The current study examines survivin in intestinal epithelial cells after ileocecal resection.

45 in human colonic epithelial cells and small intestinal epithelial cells after knockdown of MIR29 wit

46 and inactive precursor protein (proIL-18) in intestinal epithelial cells after T. gondii or Citrobact

47 terization of previously unknown subtypes of intestinal epithelial cell and their gene signatures.

48 Specifically, rat intestinal epithelial cells and a human intestinal epith

49 M2 mediates the caspase-1-dependent death of intestinal epithelial cells and bone marrow cells in res

50 strate that dietary adjustments affect small intestinal epithelial cells and can be used to modulate

51 ule relative to other signalling pathways in intestinal epithelial cells and colorectal cancer (CRC)

52 e in regulating proliferation of both normal intestinal epithelial cells and colorectal cancer cells.

53 coded adhesin, EaeH, engages the surfaces of intestinal epithelial cells and contributes to bacterial

54 es revealed exclusive expression of Clr-a by intestinal epithelial cells and crypt cells throughout t

55 ation of STAT3 and expression of survivin in intestinal epithelial cells and expression of IL6 in col

56 by in vitro studies involving mouse primary intestinal epithelial cells and human Caco-2 cells; both

57 Inhibition of MIR30C and MIR130A in cultured intestinal epithelial cells and in mouse enterocytes blo

58 fective in preventing amebic cytotoxicity in intestinal epithelial cells and macrophages.

59 immune signalling pathway that functions in intestinal epithelial cells and may present useful targe

60 ing HIF-1alpha [HIF-1alpha-knockout (KO)] in intestinal epithelial cells and mice lacking NTR1 [NTR1-

61 In analyses of intestinal epithelial cells and mice, we identified uc.1

62 xpression of a transgene increased growth of intestinal epithelial cells and organoids.

63 P2RX7 blockade enhanced proliferation of intestinal epithelial cells and protected them from apop

64 NLR Nlrp9b that is specifically expressed in intestinal epithelial cells and restricts rotavirus infe

65 Guanylyl cyclase C (GC-C) is expressed in intestinal epithelial cells and serves as the receptor f

66 sin-like serine proteinases, is expressed on intestinal epithelial cells and stimulates mitogenic pat

67 in driving ulcerative colitis by regulating intestinal epithelial cells and that TH9 cells represent

68 to many environmental factors that influence intestinal epithelial cells and the underlying mucosal i

69 NA) regulate the development and function of intestinal epithelial cells, and many viruses disrupt no

70 Nfil3 transcription oscillates diurnally in intestinal epithelial cells, and the amplitude of the ci

71 of extracellular vesicles (EVs) derived from intestinal epithelial cells, and these ANXA1-containing

72 pts polarity, growth, and differentiation of intestinal epithelial cells, and which impairs immune ce

73 est, Sgpp2 deficiency suppressed DSS-induced intestinal epithelial cell apoptosis and improved mucosa

74 ve colonic mucosal inflammation by promoting intestinal epithelial cell apoptosis and mucosal TH17 re

75 tes a direct contact-dependent activation of intestinal epithelial cell apoptosis signaling and progr

76 ethylation and associated gene activation in intestinal epithelial cells are significantly altered by

77 suppress larval diapause, is changed in the intestinal epithelial cells at larval diapause.

78 of cytoprotective IL-18 from IKKalpha-mutant intestinal epithelial cells because of elevated caspase

79 membrane-bound guanylyl cyclase expressed in intestinal epithelial cells, binds the paracrine hormone

80 y reduces adhesion of C. difficile to Caco-2 intestinal epithelial cells but does not affect activati

81 Echovirus 7 enters polarized Caco-2 intestinal epithelial cells by a clathrin-mediated endoc

82 In vitro, infection of polarized human intestinal epithelial cells by coxsackievirus B3 (CVB3)

83 nisms underlying interactions of CpClec with intestinal epithelial cells by using an Fc-tagged recomb

84 Intestinal epithelial cells (Caco-2 and IEC-6 lines) wer

85 It is now well accepted that intestinal epithelial cells can be induced to express an

86 exneri, which replicates in the cytoplasm of intestinal epithelial cells, can use the Embden-Meyerhof

87 Fucosylation of intestinal epithelial cells, catalyzed by fucosyltransfe

88 Targeted deletion of HuR in intestinal epithelial cells caused significant mucosal a

89 Overexpression of ectopic eIF3i in intestinal epithelial cells causes oncogenesis by direct

90 In rat intestinal epithelial cells, CCTalpha silencing increase

91 hat the expression levels of RALDH1 in small intestinal epithelial cells correlated with the activity

92 e early 1990s, my laboratory discovered that intestinal epithelial cells could alter their phenotype

93 to be required for regulation of TNF-induced intestinal epithelial cell death and survival.

94 d externally desynchronized WT mice to study intestinal epithelial cell death.

95 Deletion of Il18 or its receptor Il18r1 in intestinal epithelial cells (Delta/EC) conferred protect

96 essed HIF1alpha or HIF2alpha specifically in intestinal epithelial cells demonstrated an increase in

97 CVB3 infection of human intestinal epithelial cells depends on DAF at the apical

98 and to infect polarized monolayers of small-intestinal epithelial cells derived from DAF transgenic

99 and prevents the unlimited proliferation of intestinal epithelial cells despite constitutive beta-ca

100 Deletion of EGFR from intestinal epithelial cells did not affect tumor growth.

101 expression, and revealed that the process of intestinal epithelial cell differentiation upregulates p

102 t, analysis of genomic architecture in mouse intestinal epithelial cells disclosed that microbiota co

103 orders, and loss of Myo7b in differentiating intestinal epithelial cells disrupts intermicrovillar ad

104 equired for in vivo caspase-11 production in intestinal epithelial cells during DSS colitis.

105 Here, we show that adhesion of microbes to intestinal epithelial cells (ECs) is a critical cue for

106 In contrast, the targeted deletion of HuR in intestinal epithelial cells enhanced miR-675 production

107 s innate immune evasion enables infection of intestinal epithelial cells, escape from adaptive immuni

108 er Ikkepsilon phosphorylation in transformed intestinal epithelial cells, establishing a positive fee

109 alpha and TP53, which increases survivin and intestinal epithelial cell expansion during therapeutic

110 Human intestinal epithelial cells express the desmoglein-2 iso

111 Notch signaling largely determines intestinal epithelial cell fate.

112 lk1 expressing tuft cells regulate the whole intestinal epithelial cells following injury through par

113 tabolic and immunological responses in human intestinal epithelial cells following their co-culture w

114 mensal microbes in contact with living human intestinal epithelial cells for more than a week in vitr

115 te in different cell types in the intestine, intestinal epithelial cells for reovirus and intestinal

116 Intestinal epithelial cells form a barrier that is criti

117 The apical brush border membrane (BBM) of intestinal epithelial cells forms a highly structured an

118 Microarray analysis of intestinal epithelial cells from gnotobiotic mice reveal

119 commensal-specific CD4(+) T cells on primary intestinal epithelial cells from these samples.

120 SIgA in breast milk resulted in a pattern of intestinal epithelial cell gene expression in adult mice

121 At baseline, p85alpha-deficient intestinal epithelial cells had less Trp53 and more surv

122 roenvironmental O2 sufficiently to stabilize intestinal epithelial cell hypoxia-inducible factor (HIF

123 eomic screen for Dsg2-associated proteins in intestinal epithelial cells identified a lectin referred

124 (also called IL28A or interferon lambda2) in intestinal epithelial cell (IEC) activation, studying it

125 to decreased crypt proliferation, increased intestinal epithelial cell (IEC) apoptosis and increased

126 activation, beclin 1 and ATG5 cleavage, and intestinal epithelial cell (IEC) death compared with con

127 Intestinal epithelial cell (IEC) death is typical of inf

128 Claudins are expressed differentially during intestinal epithelial cell (IEC) differentiation.

129 ingle amino acid, glutamate (GLM), modulates intestinal epithelial cell (IEC) growth and EBF.

130 inal tract, IL-22 activates STAT3 to promote intestinal epithelial cell (IEC) homeostasis and tissue

131 kine interferon gamma (IFNgamma ) influences intestinal epithelial cell (IEC) homeostasis in a biphas

132 that DUSP10 knockout (KO) mice had increased intestinal epithelial cell (IEC) proliferation and migra

133 (EGF) and TLR signaling in the modulation of intestinal epithelial cell (IEC) proliferation; however,

134 esized that TNF exerts beneficial effects on intestinal epithelial cell (IEC) responses to injury.

135 The relative roles of intestinal epithelial cell (IEC) vs dendritic cell (DC)

136 Critical to these processes is the intestinal epithelial cell (IEC), which regulates immune

137 Using mice with intestinal epithelial cell (IEC)-specific deletions in e

138 Intestinal epithelial cell (IEC)-specific RIPK1 knockout

139 ine of defence via cytolysis of dysregulated intestinal epithelial cells (IEC) and cytokine-mediated

140 e miRNA-processing enzyme, Dicer, identified intestinal epithelial cells (IEC) and Hopx-positive cell

141 Using HT-29 intestinal epithelial cells (IEC) as a model we have dem

142 rylated beta-catenin (pbeta-Cat(Ser-552)) in intestinal epithelial cells (IEC) during colitis and col

143 ation patterns and transcriptomes of primary intestinal epithelial cells (IEC) of children newly diag

144 unctions as an intrinsic tumor suppressor in intestinal epithelial cells (IEC), by regulating their r

145 ORP4 silencing in non-transformed intestinal epithelial cells (IEC)-18 caused apoptosis ch

146 nteraction of S. suis with human and porcine intestinal epithelial cells (IEC).

147 The reduced butyrate in CD326(+) intestinal epithelial cells (IECs) after allo-BMT result

148 flammation-associated pathways are active in intestinal epithelial cells (IECs) and contribute to the

149 We deleted Cosmc in mouse intestinal epithelial cells (IECs) and found marked redu

150 xpressed abundantly on the apical surface of intestinal epithelial cells (IECs) and functions as the

151 directly affects expression of DRA in human intestinal epithelial cells (IECs) and in the intestines

152 de methylome- and transcriptome-profiling of intestinal epithelial cells (IECs) and sperm cells of ma

153 s in the intestine, including the surface of intestinal epithelial cells (IECs) and the interior of g

154 Intestinal epithelial cells (IECs) are exposed to profou

155 ways driving disease-specific alterations of intestinal epithelial cells (IECs) are largely unknown.

156 mbrane-derived microparticles (PMN-MPs) onto intestinal epithelial cells (IECs) during TEM leads to l

157 Furthermore, the increase in Smurf2 in intestinal epithelial cells (IECs) expressing lower leve

158 A similar phenotype occurs in mice whose intestinal epithelial cells (IECs) fail to express the t

159 Intestinal epithelial cells (IECs) form a critical barri

160 ptome and accessible chromatin landscapes in intestinal epithelial cells (IECs) from mice reared in t

161 Intestinal epithelial cells (IECs) have critical roles i

162 Ifnlr1 We found that expression of IFNLR1 on intestinal epithelial cells (IECs) in the small intestin

163 Rotavirus (RV) replicates efficiently in intestinal epithelial cells (IECs) in vivo despite the a

164 RNA and accessible chromatin data from adult intestinal epithelial cells (IECs) in zebrafish, stickle

165 dent protein kinase II gamma (CAMK2gamma) in intestinal epithelial cells (IECs) modulates inflammator

166 Proliferation and differentiation of intestinal epithelial cells (IECs) occur in part through

167 induced phosphorylation of STAT1-Y701 within intestinal epithelial cells (IECs) of suckling mice.

168 of Crohn's disease patients that can invade intestinal epithelial cells (IECs) or macrophages and su

169 Intestinal epithelial cells (IECs) play an indispensable

170 How intestinal epithelial cells (IECs) recognize pathogens a

171 Intestinal epithelial cells (IECs) regulate gut immune h

172 ntalization of Toll-like receptors (TLRs) in intestinal epithelial cells (IECs) regulates distinct im

173 (MNoV), we determine that a small number of intestinal epithelial cells (IECs) serve as the reservoi

174 ut mice, and show that mice lacking RIPK1 in intestinal epithelial cells (IECs) spontaneously develop

175 on of lymphoid cells that reside between the intestinal epithelial cells (IECs) that form the intesti

176 oteins villin 1 (VIL1) and gelsolin (GSN) in intestinal epithelial cells (IECs) to determine whether

177 mouse ISCs, progenitors, and differentiated intestinal epithelial cells (IECs) using Villin-Cre.

178 hemistry, and ultrastructural alterations in intestinal epithelial cells (IECs) were assessed by elec

179 Intestinal epithelial cells (IECs) were isolated and pur

180 Here we report that, unlike non-metastatic intestinal epithelial cells (IECs), metastatic IECs expr

181 Using human intestinal epithelial cells (IECs), we discovered that S

182 Here we show that mouse apoptotic intestinal epithelial cells (IECs), which undergo contin

183 FB3 expression and inflammatory responses in intestinal epithelial cells (IECs).

184 from various cell types in vitro, including intestinal epithelial cells (IECs).

185 ntium infection induced IL-7 production from intestinal epithelial cells (IECs).

186 tion and the induction of tnfa expression in intestinal epithelial cells (IECs).

187 or of coxsackievirus B3 (CVB) replication in intestinal epithelial cells (IECs).

188 n channel, subfamily V, member 1 (TRPV1), in intestinal epithelial cells (IECs).

189 e model deficient for LEPR-B specifically in intestinal epithelial cells (IECs).

190 h PMN-MPs upon PMN activation and binding to intestinal epithelial cells (IECs).

191 (T3SS)-mediated injection of effectors into intestinal epithelial cells (IECs); these effectors alte

192 he Wnt pathway in both cultured biopsies and intestinal epithelial cells implicated Wnt ligands drivi

193 t this is due to activation of C3 within the intestinal epithelial cells in a cathepsin-dependent man

194 y, targeted deletion of insulin receptors in intestinal epithelial cells in Apc(Min/+) mice did not c

195 RAGE was abundant in the intestinal epithelial cells in both suckling pups and ad

196 nd down-regulation of 196 lincRNAs in murine intestinal epithelial cells in culture.

197 levels of inflammatory cytokines produced by intestinal epithelial cells in response to LT are signif

198 also decreased secretion of ApoB-48 from rat intestinal epithelial cells in response to oleic acid st

199 rogramming of the gene expression profile in intestinal epithelial cells in response to TNF-alpha sti

200 te, repressed the accumulation of tetraploid intestinal epithelial cells in the Apc(Min/+) mouse mode

201 Rotavirus specifically infects the intestinal epithelial cells in the host small intestine

202 virulence genes and increases attachment to intestinal epithelial cells in vitro in a QseC-dependent

203 of the vitamin A-converting enzyme RALDH1 in intestinal epithelial cells in vivo and in vitro, respec

204 s, and modules of transcriptional targets in intestinal epithelial cells in vivo.

205 n the underlying intestinal mucosa, and that intestinal epithelial cells, in turn, serve as targets o

206 production of d-amino acid oxidase (DAO) by intestinal epithelial cells, including goblet cells, whi

207 As expected, cultured intestinal epithelial cells increased their expression o

208 eals that TFEB is critical for resistance to intestinal epithelial cell injury, potentially mediated

209 stimulates conversion of both rat and human intestinal epithelial cells into insulin-secreting cells

210 enterohemorrhagic Escherichia coli (EHEC) to intestinal epithelial cells is critical for colonization

211 The coordinated release of molecules by intestinal epithelial cells is crucial for activating in

212 ota-mediated signal transduction via TLR4 in intestinal epithelial cells is far more complex than wha

213 arget activation of an IKKbeta/NCoR1 loop in intestinal epithelial cells lead to derepression of gene

214 In vivo, VDR deletion in intestinal epithelial cells led to significant decreased

215 Here, using a human intestinal epithelial cell line (HCT116), we show that l

216 rat intestinal epithelial cells and a human intestinal epithelial cell line were infected with C. sa

217 there is deregulation in differentiation of intestinal epithelial cell lineages that may influence t

218 pullorum strains were investigated on human intestinal epithelial cell lines.

219 In the neobladder, the intestinal epithelial cells lose their tissue-specific e

220 Intestinal epithelial cells, macrophages, and dendritic

221 testinal inflammation and TNFRI signaling in intestinal epithelial cells mediate a disruption of the

222 To investigate whether TNFRI on intestinal epithelial cells mediates intestinal barrier

223 ice and attenuated LPS inhibitory effects on intestinal epithelial cell migration along the crypt-vil

224 Here, we showed that impairment of intestinal epithelial cell migration occurred in lipopol

225 sis of a complex miRNA regulatory program in intestinal epithelial cell models provides novel evidenc

226 s by targeting various cell types, including intestinal epithelial cells, mononuclear phagocytes, inn

227 ase using cell-selective (body wall muscles, intestinal epithelial cells, neurons, and pharyngeal mus

228 d circulating glucose was trapped within the intestinal epithelial cells of rats and humans that unde

229 rface, and expression of human DAF on murine intestinal epithelial cells permits their infection in v

230 Intestinal epithelial cell populations were purified; we

231 and compare global DNA methylation levels of intestinal epithelial cells pre- and post-neobladder con

232 There was impaired proliferation of intestinal epithelial cell progenitors, aberrant lipid h

233 human RIP140, we found that RIP140 inhibited intestinal epithelial cell proliferation and apoptosis.

234 vels observed are shown to result in reduced intestinal epithelial cell proliferation and increased c

235 However, intestinal epithelial cell proliferation is not impeded,

236 st-restricted deletion of Ikkbeta stimulates intestinal epithelial cell proliferation, suppresses tum

237 tgr1(-/-) and Mtg16(-/-) mice have increased intestinal epithelial cell proliferation.

238 ficiency of either UPR or autophagy in small intestinal epithelial cells promotes each other's compen

239 Deletion of EGFR from myeloid cells, but not intestinal epithelial cells, protects mice from colitis-

240 Intestinal epithelial cells provide an important coloniz

241 a suggest that the NOD1 signaling pathway in intestinal epithelial cells provides an important sentin

242 Boosting BAI1-mediated uptake by intestinal epithelial cells (rather than myeloid cells)

243 s, the role of these cytokines in regulating intestinal epithelial cell renewal is largely unknown.

244 The emerging picture indicates that intestinal epithelial cells represent an integral compon

245 enforcing specific expression of guanylin in intestinal epithelial cells restored GUCY2C signaling, e

246 Overexpression of Reg3g in intestinal epithelial cells restricts bacterial coloniza

247 (also known as Eif2ka4) in CD11c(+) APCs or intestinal epithelial cells resulted in enhanced intesti

248 Reactivation of TNFRI on intestinal epithelial cells resulted in increased intest

249 Knockdown of DEN-1 in human intestinal epithelial cells resulted in increased kineti

250 ghout the body or restricted specifically to intestinal epithelial cells resulted in loss of AHR-depe

251 Overexpression of Cdg7_FLc_0990 in intestinal epithelial cells resulted in significant chan

252 Here we show that loss of Erk1/2 in intestinal epithelial cells results in defects in nutrie

253 Furthermore, knockdown of IL-10R1 in intestinal epithelial cells results in impaired barrier

254 , pancreatic cells, smooth muscle cells, and intestinal epithelial cells results in phenotypically no

255 Initial in vitro studies using cultured intestinal epithelial cells revealed that the neddylatio

256 P promoter region are differentially used in intestinal epithelial cell(s) (IEC).

257 Thus, our data indicate that intestinal epithelial cells serve as gatekeepers for the

258 n-based recycling and degradation studies in intestinal epithelial cells show that SNX27 is required

259 In vitro studies using Caco2 intestinal epithelial cells showed that in the presence

260 Using mice with an intestinal epithelial cell-specific deletion of PPARdelt

261 From an investigation of mice with intestinal epithelial cell-specific deletion of the p38a

262 Colitis in mice with an intestinal epithelial cell-specific Hmgb1 deletion and p

263 ctivates p38gamma in mouse colon tissues and intestinal epithelial cell-specific p38gamma knockout (K

264 tutively active TLR4 (CD4-TLR4) linked to an intestinal epithelial cell-specific promoter.

265 t up-regulate GLUT5 in GLUT5-KO, KHK-KO, and intestinal epithelial cell-specific Rab11a-KO mice.

266 lonic wounds in a process involving FPR1 and intestinal epithelial-cell-specific NOX1-dependent redox

267 microbiota compared with miRNAs in any other intestinal epithelial cell subtype.

268 regulate miRNA expression in IESCs and other intestinal epithelial cell subtypes will elucidate a cri

269 (LPA) receptor 1 regulates proliferation of intestinal epithelial cells, such that the absence of LP

270 pression of C3/C3 fragments primarily in the intestinal epithelial cells, suggesting local involvemen

271 e promotes proliferation of cancer-initiated intestinal epithelial cells, suggesting that it can act

272 Microfold (M) cells are specialized intestinal epithelial cells that internalize particulate

273 ated a gut-restricted expression of Clr-f on intestinal epithelial cells that is spatially matched by

274 ogenic effect of feline T. foetus on porcine intestinal epithelial cells, the dependence of T. foetus

275 rom patients with CD were applied to healthy intestinal epithelial cells, the epithelial cells increa

276 shed light on how bacterial adhesion can cue intestinal epithelial cells to direct differentiation of

277 stimulation of the proliferative response of intestinal epithelial cells to GPCR agonists that act vi

278 y manipulating the host cell cytoskeleton of intestinal epithelial cells to promote bacterial attachm

279 nal enteroid (HIE) cultures contain multiple intestinal epithelial cell types that comprise the intes

280 ADAM10 is expressed in all intestinal epithelial cell types, but the requirement fo

281 ls for the study and application of multiple intestinal epithelial cell types.

282 Unlike other cells and tissues, intestinal epithelial cells undergo rapid cell death upo

283 enerated transgenic mice expressing IL-33 in intestinal epithelial cells (V33 mice).

284 e OMVs bind to and are internalised by human intestinal epithelial cells via dynamin-dependent and St

285 erformed experiments in mice lacking EGFR in intestinal epithelial cells (Villin-Cre; Egfr(f/f) and V

286 In infected mice, proliferation of small intestinal epithelial cells was compromised in an SseF/S

287 uanylin, and uroguanylin mRNA and protein by intestinal epithelial cells was preserved following leth

288 Loss of Bcl-3 in intestinal epithelial cells was sufficient to increase t

289 out (VDR(DeltaIEC)) mice, and cultured human intestinal epithelial cells, we demonstrate here that th

290 hepatocytes, pancreatic endocrine cells, and intestinal epithelial cells when treated with defined so

291 (TLRs) in murine intestinal mucosa and human intestinal epithelial cells where Jak3 interacted with a

292 knockdown of alphaSNAP induced detachment of intestinal epithelial cells, whereas overexpression of a

293 d K(+) channel activation and K(+) efflux by intestinal epithelial cells, which preceded cell killing

294 inflammasomes function for innate defense in intestinal epithelial cells, which represent the first l

295 that ERK5 provides a common bypass route in intestinal epithelial cells, which rescues cell prolifer

296 Treatment of mouse primary intestinal epithelial cells with [Thr(28),Nle(31)]CCK in

297 adhesion and uptake, we developed polarized intestinal epithelial cells with reduced microvilli ("mi

298 demonstrate that treatment of nontransformed intestinal epithelial cells with TGF-beta inhibited ARE-

299 s continuously proliferate and replenish all intestinal epithelial cells within days.

300 p in the translocation of cholera toxin into intestinal epithelial cells without exerting measurable